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Lancet Neurology, The, ISSN 1474-4422, 2017, Volume 16, Issue 4, pp. 264 - 264
  Several topics discussed at the stakeholder meeting were the same as those identified in the Duchenne muscular dystrophy discussion, including variability of... 
Neurology | CLINICAL NEUROLOGY | Muscular Atrophy, Spinal | Spinal Muscular Atrophies of Childhood | Humans | Medical colleges | Ophthalmology | Patents | Stakeholders | Collaboration | Muscular dystrophy | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 01/2017, Volume 93, Issue 1, pp. 3 - 5
Spinal muscular atrophy is the most common genetic killer of infants. A therapy shows promise in the clinic, but there is a potential limit to its efficacy. In... 
spinal muscular atrophy | lncRNA | SMN | SMA | antisense | RESCUE | RNA | ANTISENSE OLIGONUCLEOTIDES | NEUROSCIENCES | MOUSE MODEL | Genetic Therapy | Muscular Atrophy, Spinal - genetics | Humans | Spinal muscular atrophy | Medical colleges | Atrophy, Muscular | Neurosciences | Proteins | Disease | Neurons | Rodents | Clinical trials | Gene therapy | Gene expression | Drug dosages | Index Medicus
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 11/2017, Volume 377, Issue 18, pp. 1713 - 1722
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 02/2018, Volume 378, Issue 5, pp. 487 - 488
To the Editor: In two reports regarding patients with type 1 spinal muscular atrophy (Nov. 2 issue), 1,2 researchers describe therapies involving antisense... 
MEDICINE, GENERAL & INTERNAL | Muscular Atrophy, Spinal | Spinal Muscular Atrophies of Childhood | Humans | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, pp. e41701 - e41701
Background: Heart failure (HF) is known to lead to skeletal muscle atrophy and dysfunction. However, intracellular mechanisms underlying HF-induced myopathy... 
CARDIAC-FUNCTION | PATHWAY | HYPERACTIVITY | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | PROTEIN EXPRESSION | GENETIC MODEL | IMPROVES | SATELLITE CELLS | MICE | DYSFUNCTION | Oxidative Stress | Humans | Muscular Atrophy - therapy | Heart Failure - physiopathology | Ubiquitin - metabolism | Male | Exercise Therapy | Heart Failure - therapy | Muscular Atrophy - physiopathology | Muscle Proteins - metabolism | Aged, 80 and over | Female | Muscular Atrophy - etiology | Physical Conditioning, Animal | Heart Failure - complications | Muscular Atrophy - metabolism | Heart Failure - genetics | Muscular Atrophy - genetics | Heart Failure - metabolism | Ubiquitin - genetics | Mice, Knockout | Muscle Proteins - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Aged | Mice | Lipid Peroxidation - genetics | Proteasome Endopeptidase Complex - metabolism | Heart failure | Ubiquitin | Oxidative stress | Chymotrypsin | Exercise | Ligases | Analysis | Muscles | Delivery services | Model testing | Hyperactivity | Lipid peroxidation | Activation | mRNA | Antioxidants | Training | Atrophy | Ethics | Rodents | Physical education | Heart diseases | Fitness training programs | Age | Chronic illnesses | Carbonyls | Cardiac muscle | Patients | Skeletal muscle | Musculoskeletal system | Intolerance | Proteasomes | Aerobic capacity | Myopathy | Index Medicus | Stresses | Stress
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International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 10/2013, Volume 45, Issue 10, pp. 2119 - 2119
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 02/2018, Volume 378, Issue 5, p. 487
Antibodies to scAAV9 could present a barrier to repeated administration.[...]we are curious to know whether infants who were treated with scAAV9 were tested... 
Studies | Proteins | Medical research | Immunoglobulins | Gene therapy | Gene expression | Vectors (Biology) | Children & youth | Spinal muscular atrophy
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Journal Article
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