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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2017, Volume 114, Issue 16, pp. 4213 - 4218
Duchenne muscular dystrophy (DMD) is a lethal genetic disorder caused by an absence of the dystrophin protein in bodywide muscles, including the heart.... 
Cardiac Purkinje fibers | Dystrophic dog model | Peptide-conjugated morpholinos | Duchenne muscular dystrophy | Exon skipping | MUSCLE PATHOLOGY | MULTIDISCIPLINARY SCIENCES | DMD GENE | CARDIOMYOPATHY | BODYWIDE | dystrophic dog model | peptide-conjugated morpholinos | MDX52 MICE | OLIGONUCLEOTIDES | RESCUE | exon skipping | LABORATORY-ANIMALS | cardiac Purkinje fibers | EXPRESSION | EXONS 45-55 | Genetic Therapy | Muscular Dystrophy, Animal - genetics | Exons | Male | Muscle, Skeletal - metabolism | Muscular Dystrophy, Animal - complications | Muscular Dystrophy, Duchenne - complications | Cardiomyopathies - etiology | Cardiomyopathies - therapy | Cell-Penetrating Peptides - pharmacology | Animals | Dogs | Morpholinos - pharmacology | Female | Muscular Dystrophy, Animal - therapy | Muscle, Skeletal - pathology | Muscular Dystrophy, Duchenne - genetics | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Disease Models, Animal | Heart | Physiological aspects | Health aspects | Conduction | Intravenous administration | Peptides | Toxicity | Cardiomyopathy | Effects | Muscular dystrophy | Fibers | Oligomers | Nerve conduction | Arginine | Duchenne's muscular dystrophy | Degeneration | Polymers | Heart diseases | Dystrophin | EKG | Cardiac muscle | Abnormalities | Muscles | Skeletal muscle | Purkinje fibers | Myocardium | Dystrophy | Index Medicus | Biological Sciences
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 2014, Volume 23, Issue 15, pp. 3990 - 4000
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, pp. e48478 - e48478
The GRMD (Golden retriever muscular dystrophy) dog has been widely used in pre-clinical trials targeting DMD (Duchenne muscular dystrophy), using in many cases... 
SKELETAL-MUSCLE | STEM-CELLS | EFFICACY | GAIT | ATROPHY | MULTIDISCIPLINARY SCIENCES | MYOTUBES | GENE-EXPRESSION | CALCINEURIN | PREDNISOLONE | TRANSPLANTATION | Tetany - physiopathology | Follow-Up Studies | Tetany - complications | Cyclosporine - pharmacology | Muscular Dystrophy, Animal - drug therapy | Muscular Dystrophy, Animal - physiopathology | Humans | Immunosuppressive Agents - therapeutic use | Motor Activity - drug effects | Muscular Dystrophy, Animal - complications | Muscular Dystrophy, Duchenne - physiopathology | Biomechanical Phenomena - drug effects | Muscular Dystrophy, Animal - blood | Immunosuppressive Agents - pharmacology | Principal Component Analysis | Disease Models, Animal | Creatine Kinase - blood | Muscular Dystrophy, Duchenne - drug therapy | Tetany - blood | Muscular Dystrophy, Duchenne - complications | Cyclosporine - therapeutic use | Gait - drug effects | Animals | Dogs | Muscular Dystrophy, Duchenne - blood | Accelerometry | Medical research | Corticosteroids | Duchenne muscular dystrophy | Utrophin | Medicine, Experimental | Cyclosporine | Dystrophin | Transplants & implants | Gait | Cyclosporins | Medical services | Clinical trials | Kinases | Muscular dystrophy | Isometric | Rodents | Duchenne's muscular dystrophy | Evolution | Drug dosages | Prednisolone | Accelerometers | Evaluation | Force measurement | Medical treatment | Gene expression | Studies | Musculoskeletal system | Immunosuppression | Biopsy | Dystrophy | Quality assessment | Laboratory animals | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 5, pp. e10763 - e10763
Background: Duchenne muscular dystrophy (DMD) is the most common, lethal disease of childhood. One of 3500 new-born males suffers from this universally-lethal... 
MESSENGER-RNAS | FACTOR-BETA | TGF-BETA | NITRIC-OXIDE SYNTHASE | MOUSE | BIOLOGY | DEFICIENT SKELETAL-MUSCLE | PULMONARY-FUNCTION | TRANSFORMING-GROWTH-FACTOR | TRANSCRIPTIONAL REGULATION | EXPRESSION | Inflammation - pathology | Cardiomyopathy, Dilated - pathology | Cardiomyopathy, Dilated - enzymology | Arginase - metabolism | Male | Muscle, Skeletal - metabolism | Muscular Dystrophy, Animal - complications | Protein Transport - drug effects | Kyphosis - complications | Muscular Dystrophy, Animal - pathology | Th2 Cells - drug effects | Arginine - administration & dosage | Inflammation - complications | Dystrophin - deficiency | Gene Deletion | Myocardium - metabolism | Mice, Inbred mdx | Dystrophin - metabolism | Muscle, Skeletal - enzymology | Cytokines - metabolism | Macrophages - pathology | Myocardium - pathology | Macrophages - enzymology | Kyphosis - enzymology | Macrophages - metabolism | Animals | Muscular Dystrophy, Animal - enzymology | Arginine - pharmacology | Nitric Oxide Synthase Type I - metabolism | Fibrosis | Macrophages - drug effects | Mice | Muscle, Skeletal - pathology | Inflammation - enzymology | Kyphosis - pathology | Arginine - metabolism | Heart | Arginine | Analysis | Utrophin | Dietary supplements | Physiological aspects | Muscles | Children | Macrophages | Muscular dystrophy | Diseases | Corticoids | Kyphosis | Laboratories | Pathogenesis | Arginase | Matrix metalloproteinase | Males | Polyamines | Proteins | Metabolites | Rodents | Duchenne's muscular dystrophy | Physiology | Oxidation | Metalloproteinase | Supplementation | Lesions | Heart diseases | Phenotypes | Echocardiography | Corticosteroids | Cytokines | Cardiac muscle | Metabolism | Skeletal muscle | Medicine | Pathology | Musculoskeletal system | Diet | Nitric oxide | Mutation | Dystrophy | Long-term effects | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e30063 - e30063
Increased utrophin expression is known to reduce pathology in dystrophin-deficient skeletal muscles. Transgenic overexpression of PGC-1 alpha has been shown to... 
RESVERATROL | MITOCHONDRIAL BIOGENESIS | CALCIUM | BIOLOGY | GENE-EXPRESSION | DUCHENNE MUSCULAR-DYSTROPHY | MICE | NEUROMUSCULAR-JUNCTION | SIRT1 | UTROPHIN GENE | CELL-DEATH | Stilbenes - administration & dosage | Muscle Fibers, Fast-Twitch - drug effects | Recovery of Function - drug effects | Muscular Dystrophy, Animal - physiopathology | Body Weight - drug effects | Muscular Dystrophy, Animal - complications | Muscle Fibers, Slow-Twitch - drug effects | Stilbenes - pharmacology | Dependovirus - drug effects | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Mice, Inbred mdx | Recovery of Function - physiology | Myosins - metabolism | Muscle Fatigue | Gene Transfer Techniques | Dependovirus - metabolism | Organ Size | Biomechanical Phenomena | Animals | Muscle Contraction | Muscle Fibers, Slow-Twitch - pathology | Muscle Fibers, Fast-Twitch - pathology | Trans-Activators - metabolism | Mice | Transcription Factors | Dietary Supplements | Neonates | Oxidative stress | Phosphorylation | Transgenic | Viruses | mRNA | Insulin-like growth factors | Contraction | Muscular dystrophy | Crack propagation | Fibers | Proteins | Signal transduction | Mitochondria | Oxidation resistance | Fatigue strength | Rodents | Myosin | Resveratrol | Physiology | Localization | Dystrophin | Recombinant | Muscles | Fatigue | Glycoproteins | Histology | Pharmacology | Fatigue failure | Gene expression | Muscle contraction | Skeletal muscle | Medicine | Musculoskeletal system | Signaling | Overexpression | Utrophin | Muscle function | Gene therapy | Index Medicus
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2012, Volume 303, Issue 5, pp. H559 - H568
Cheng YJ, Lang D, Caruthers SD, Efimov IR, Chen J, Wickline SA. Focal but reversible diastolic sheet dysfunction reflects regional calcium mishandling in... 
Calcium transients | Diffusion tensor magnetic resonance imaging | Duchenne muscular dystrophy | Myocardial sheet | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | myocardial sheet | diffusion tensor magnetic resonance imaging | CONTRACTION-INDUCED INJURY | LEFT-VENTRICLE | SARCOPLASMIC-RETICULUM | HISTOLOGICAL VALIDATION | calcium transients | ATRIAL-FIBRILLATION | DIFFUSION TENSOR MRI | VASCULAR VOLUME | PERIPHERAL VASCULAR DISEASE | DUCHENNE MUSCULAR-DYSTROPHY | RABBIT HEART | Muscular Dystrophy, Animal - genetics | Excitation Contraction Coupling | Heart Failure - physiopathology | Ventricular Function | Male | Ventricular Dysfunction - etiology | Muscular Dystrophy, Animal - complications | Recovery of Function | Action Potentials | Time Factors | Myocardium - metabolism | Mice, Inbred mdx | Muscular Dystrophy, Animal - metabolism | Heart Failure - etiology | Calcium Signaling | Ventricular Dysfunction - physiopathology | Disease Models, Animal | Ventricular Dysfunction - metabolism | Myocardial Contraction | Ventricular Dysfunction - pathology | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Voltage-Sensitive Dye Imaging | Muscular Dystrophy, Duchenne - complications | Disease Progression | Diffusion Tensor Imaging | Biomechanical Phenomena | Animals | Perfusion | Fibrosis | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Complications and side effects | Heart beat | Cardiomyopathy | Physiological aspects | Calcium metabolism | Development and progression | Heart diseases | Health aspects | Risk factors | Heart | Nuclear magnetic resonance--NMR | Calcium | Rodents | Mortality | Muscular dystrophy | Index Medicus | Muscle Mechanics and Ventricular Function
Journal Article