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PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 4, p. e0215590
..., Elaine Minatel * Introduction Duchenne muscular dystrophy (DMD) is the most common muscular dystrophy, whose genetic defect is identified in the X chromosome gene... 
PREDNISONE | MULTIDISCIPLINARY SCIENCES | MOUSE | MUSCULAR-DYSTROPHY | PERMEABLE RADICAL SCAVENGER | MECHANISMS | DUCHENNE | DIAPHRAGM MUSCLE | DAMAGE | RODENT MODEL | Muscular Dystrophy, Animal - genetics | Muscular Dystrophy, Animal - physiopathology | Humans | Muscle, Skeletal - metabolism | Cyclic N-Oxides - administration & dosage | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Spin Labels | Muscular Dystrophies - genetics | Muscular Dystrophy, Animal - pathology | Cyclic N-Oxides - pharmacology | Muscular Dystrophy, Duchenne - physiopathology | Muscle, Skeletal - drug effects | Mice, Inbred mdx | Diaphragm - metabolism | Muscle Fibers, Skeletal - physiology | Diaphragm - physiopathology | Disease Models, Animal | Mice, Inbred C57BL | Injections, Intraperitoneal | Muscular Dystrophy, Duchenne - pathology | Antioxidants - pharmacology | Muscular Dystrophies - pathology | Phenotype | Animals | Muscle, Skeletal - physiopathology | Antioxidants - administration & dosage | Muscle Strength - drug effects | Muscular Dystrophy, Duchenne - genetics | Muscular Dystrophies - physiopathology | Genetic aspects | Mice | Corticosteroids | Prednisolone | Medical research | Care and treatment | Rats as laboratory animals | Medicine, Experimental | Dosage and administration | Research | Health aspects | Muscular dystrophy | Oxidative stress | Phenotypes | Muscles | Tempol | Inflammatory response | Biochemistry | Biology | Inflammation | Saline solutions | Genotype & phenotype | Angiogenesis | Ischemia | Rodents | Physiology | Degeneration | Myonecrosis | Muscular strength | Diaphragm
Journal Article
Mammalian genome, ISSN 0938-8990, 2012, Volume 23, Issue 1-2, pp. 85 - 108
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20614 - 20619
Duchenne muscular dystrophy (DMD) is a devastating genetic muscular disorder of childhood marked by progressive debilitating muscle weakness and wasting, and ultimately death in the second or third decade of life... 
Artificial satellites | Plasmids | Duchenne muscular dystrophy | Stem cells | Muscles | Electroporation | Muscle fibers | Skeletal muscle | Hypertrophy | Myoblasts | Noncanonical Wnt signaling | REGENERATION | MULTIDISCIPLINARY SCIENCES | MOUSE | SUSCEPTIBILITY | INJURY | SATELLITE STEM-CELLS | MDX MICE | DAMAGE | noncanonical Wnt signaling | SKELETAL-MUSCLE | CONTRACTION | PATHWAY | skeletal muscle | Genetic Therapy | Muscular Dystrophy, Animal - genetics | Muscular Dystrophy, Animal - drug therapy | Muscular Dystrophy, Animal - physiopathology | Humans | Male | RNA, Messenger - metabolism | Muscular Dystrophy, Animal - pathology | Gene Knockdown Techniques | Wnt Proteins - genetics | Mice, Inbred mdx | Plasmids - genetics | Electrochemotherapy | MEF2 Transcription Factors | Myogenic Regulatory Factors - genetics | Signal Transduction | Mice, Inbred C57BL | RNA, Messenger - genetics | Myogenic Regulatory Factors - metabolism | Wnt Proteins - therapeutic use | Regeneration - physiology | Animals | Muscle Contraction - physiology | Muscle, Skeletal - physiopathology | Mice | Wnt Proteins - physiology | Muscle, Skeletal - pathology | Plasmids - administration & dosage | Physiological aspects | Care and treatment | Rapamycin | Health aspects | Muscular dystrophy | Biological Sciences
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e49350
In patients with Duchenne muscular dystrophy (DMD) and the standard mdx mouse model of DMD, dystrophin deficiency causes loss of neuronal nitric oxide synthase (nNOS mu... 
SYNTHASE | NNOS | IN-VITRO | THERAPY | RAT | MULTIDISCIPLINARY SCIENCES | L-ARGININE | SARCOLEMMA | DEFICIENT SKELETAL-MUSCLE | SYMPATHETIC NEURAL-CONTROL | Flurbiprofen - therapeutic use | Muscular Dystrophy, Animal - drug therapy | Muscular Dystrophy, Animal - physiopathology | Muscles - physiopathology | Male | Muscular Dystrophy, Animal - pathology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Hindlimb - physiopathology | Time Factors | Muscular Dystrophy, Duchenne - physiopathology | Biomechanical Phenomena - drug effects | Weight Gain - drug effects | Nitric Oxide Donors - therapeutic use | Nitric Oxide Donors - pharmacology | Ischemia - pathology | Disease Models, Animal | Flurbiprofen - analogs & derivatives | Muscular Dystrophy, Duchenne - drug therapy | Mice, Inbred C57BL | Muscular Dystrophy, Duchenne - pathology | Ischemia - physiopathology | Vasoconstriction - drug effects | Feeding Behavior - drug effects | Animals | Muscle Contraction | Ischemia - drug therapy | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Muscles - drug effects | Hemodynamics - drug effects | Mice | Norepinephrine | Muscles - blood supply | Flurbiprofen - pharmacology | Ischemia | Duchenne muscular dystrophy | Utrophin | Nitric oxide | Soybean | Nonsteroidal anti-inflammatory drugs | Dystrophin | Femur | Experiments | Blood | Muscular dystrophy | Contraction | Proteins | Rodents | Duchenne's muscular dystrophy | Soybean oil | Drug dosages | Sarcolemma | Medical research | Vasoconstriction | Muscles | Inflammation | Muscle contraction | Nitric-oxide synthase | Blood flow | Resistance | Musculoskeletal system | Flow velocity | Oils & fats | Conductance | Dystrophy | Laboratory animals
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e48478
The GRMD (Golden retriever muscular dystrophy) dog has been widely used in pre-clinical trials targeting DMD... 
SKELETAL-MUSCLE | STEM-CELLS | EFFICACY | GAIT | MULTIDISCIPLINARY SCIENCES | CALCINEURIN | EXPRESSION | PREDNISOLONE | TRANSPLANTATION | Tetany - physiopathology | Follow-Up Studies | Tetany - complications | Cyclosporine - pharmacology | Muscular Dystrophy, Animal - drug therapy | Muscular Dystrophy, Animal - physiopathology | Humans | Immunosuppressive Agents - therapeutic use | Motor Activity - drug effects | Muscular Dystrophy, Animal - complications | Muscular Dystrophy, Duchenne - physiopathology | Biomechanical Phenomena - drug effects | Muscular Dystrophy, Animal - blood | Immunosuppressive Agents - pharmacology | Principal Component Analysis | Disease Models, Animal | Creatine Kinase - blood | Muscular Dystrophy, Duchenne - drug therapy | Tetany - blood | Muscular Dystrophy, Duchenne - complications | Cyclosporine - therapeutic use | Gait - drug effects | Animals | Dogs | Muscular Dystrophy, Duchenne - blood | Accelerometry | Medical research | Corticosteroids | Duchenne muscular dystrophy | Utrophin | Medicine, Experimental | Cyclosporine | Dystrophin | Transplants & implants | Gait | Cyclosporins | Clinical trials | Kinases | Muscular dystrophy | Isometric | Rodents | Duchenne's muscular dystrophy | Evolution | Drug dosages | Prednisolone | Accelerometers | Evaluation | Force measurement | Medical treatment | Gene expression | Studies | Musculoskeletal system | Immunosuppression | Biopsy | Quality assessment | Dystrophy | Laboratory animals
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 484, Issue 7394, pp. 394 - 398
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 7/2012, Volume 109, Issue 27, pp. 10978 - 10983
Journal Article