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Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, pp. 16105 - 16105
Journal Article
Science Signaling, ISSN 1945-0877, 08/2012, Volume 5, Issue 236, pp. ra56 - ra56
Journal Article
Science, ISSN 0036-8075, 12/2004, Volume 306, Issue 5702, pp. 1796 - 1799
Most mutations in the dystrophin gene create a frameshift or a stop in the mRNA and are associated with severe Duchenne muscular dystrophy. Exon skipping that... 
Proteins | Legs | Messenger RNA | Reverse transcriptase polymerase chain reaction | Exons | Duchenne muscular dystrophy | Muscles | Small nuclear RNA | Reports | Muscle fibers | Genetic mutation | SKELETAL-MUSCLE | HEMOPHILIA-B | MULTIDISCIPLINARY SCIENCES | MDX MOUSE MUSCLE | MUTATION | DUCHENNE MUSCULAR-DYSTROPHY | GENE-TRANSFER | EXPRESSION | PRE-MESSENGER-RNA | DELIVERY | VECTOR | Dependovirus - genetics | Genetic Therapy | Muscular Dystrophy, Animal - genetics | Muscular Dystrophy, Animal - physiopathology | Muscle, Skeletal - metabolism | RNA, Messenger - metabolism | Muscular Dystrophy, Animal - pathology | RNA Splicing | Transfection | Muscular Dystrophy, Duchenne - physiopathology | RNA, Small Nuclear - genetics | Mice, Inbred mdx | Muscular Dystrophy, Animal - therapy | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Oligonucleotides, Antisense - pharmacology | Introns | RNA, Messenger - genetics | Muscular Dystrophy, Duchenne - pathology | Muscle, Skeletal - physiology | Muscle Fibers, Skeletal - immunology | Animals | Muscle Contraction | Dystrophin - genetics | Muscle Fibers, Skeletal - pathology | Mice | Muscle, Skeletal - pathology | Muscular Dystrophy, Duchenne - genetics | Mutation | RNA, Small Nuclear - metabolism | Genetic Vectors | Usage | Care and treatment | Genetic aspects | Gene therapy | Exon (Molecular genetics) | Ribonucleic acid--RNA | Genes | Rodents | Muscular dystrophy | Index Medicus
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 2, pp. 583 - 592
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20614 - 20619
Duchenne muscular dystrophy (DMD) is a devastating genetic muscular disorder of childhood marked by progressive debilitating muscle weakness and wasting, and... 
Artificial satellites | Plasmids | Duchenne muscular dystrophy | Stem cells | Muscles | Electroporation | Muscle fibers | Skeletal muscle | Hypertrophy | Myoblasts | Noncanonical Wnt signaling | REGENERATION | MULTIDISCIPLINARY SCIENCES | MOUSE | SUSCEPTIBILITY | INJURY | SATELLITE STEM-CELLS | MDX MICE | DAMAGE | noncanonical Wnt signaling | SKELETAL-MUSCLE | CONTRACTION | PATHWAY | skeletal muscle | Genetic Therapy | Muscular Dystrophy, Animal - genetics | Muscular Dystrophy, Animal - drug therapy | Muscular Dystrophy, Animal - physiopathology | Humans | Male | RNA, Messenger - metabolism | Muscular Dystrophy, Animal - pathology | Gene Knockdown Techniques | Wnt Proteins - genetics | Mice, Inbred mdx | Plasmids - genetics | Electrochemotherapy | MEF2 Transcription Factors | Myogenic Regulatory Factors - genetics | Signal Transduction | Mice, Inbred C57BL | RNA, Messenger - genetics | Myogenic Regulatory Factors - metabolism | Wnt Proteins - therapeutic use | Regeneration - physiology | Animals | Muscle Contraction - physiology | Muscle, Skeletal - physiopathology | Mice | Wnt Proteins - physiology | Muscle, Skeletal - pathology | Plasmids - administration & dosage | Physiological aspects | Care and treatment | Rapamycin | Health aspects | Muscular dystrophy | Signal transduction | Genetic disorders | Rodents | Cells | Index Medicus | Biological Sciences
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2019, Volume 116, Issue 9, pp. 3508 - 3517
Duchenne muscular dystrophy (DMD) is a genetic disorder caused by loss of the protein dystrophin. In humans, DMD has early onset, causes developmental delays,... 
C. elegans | Degeneration | Exercise | Muscular dystrophy | Hypertrophy | hypertrophy | CALCIUM HOMEOSTASIS | MULTIDISCIPLINARY SCIENCES | SARCOLEMMA | exercise | MECHANISMS | PATHOLOGY | degeneration | muscular dystrophy | CAENORHABDITIS-ELEGANS | GENE | INTEGRITY | MUTATIONS | Muscular Dystrophy, Animal - genetics | Physical Exertion - genetics | Muscle, Skeletal - growth & development | Caenorhabditis elegans - genetics | Muscular Dystrophy, Animal - physiopathology | Physical Exertion - physiology | Humans | Caenorhabditis elegans - physiology | Animals | Muscular Dystrophy, Duchenne - physiopathology | Muscle Contraction - physiology | Muscle, Skeletal - physiopathology | Mice, Inbred mdx | Muscular Dystrophy, Animal - therapy | Mice | Muscular Dystrophy, Duchenne - genetics | Muscular Dystrophy, Duchenne - therapy | Caenorhabditis elegans Proteins - genetics | Physical Conditioning, Animal | Disease Models, Animal | Signs and symptoms | Phenotypes | Animal models | Physical activity | Calcium | Therapeutic applications | Pathogenesis | Contractility | Muscles | Lethality | Machinery | Muscle contraction | Necrosis | Proteins | Locomotion | Mitochondria | Calcium-binding protein | Duchenne's muscular dystrophy | Structural damage | Dystrophy | Dystrophin | Calmodulin | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 11/2013, Volume 22, Issue 21, pp. 4368 - 4382
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2012, Volume 109, Issue 27, pp. 10978 - 10983
Journal Article