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BMC genomics, ISSN 1471-2164, 2019, Volume 20, Issue 1, pp. 878 - 18
Background The function of Toll-like receptor 2 (TLR2) in host defense against pathogens, especially Mycobacterium tuberculosis (Mtb) is poorly understood. To... 
ACYLATION STATE | Mycobacterium | IMMUNE-RESPONSE | ACTIVATED PROTEIN-KINASE | INNATE | Zebrafish | PROINFLAMMATORY CYTOKINES | RNAseq | C-MAF | CUTTING EDGE | Tuberculosis | Transcriptomics | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | HOST-DEFENSE | RNA deep sequencing | TLR2 | Chemokine CXCL11 - genetics | Toll-Like Receptor 2 - genetics | Zebrafish - immunology | Interleukin-1beta - genetics | Host-Pathogen Interactions - immunology | Lymphotoxin-alpha - genetics | Toll-Like Receptor 2 - deficiency | Larva - immunology | Zebrafish Proteins - immunology | Chemokine CXCL11 - immunology | Mycobacterium Infections, Nontuberculous - immunology | Disease Resistance - genetics | Macrophages - microbiology | CCAAT-Enhancer-Binding Protein-beta - genetics | Fish Diseases - genetics | Zebrafish - genetics | Metabolic Networks and Pathways - genetics | Host-Pathogen Interactions - genetics | Mycobacterium marinum - immunology | CCAAT-Enhancer-Binding Protein-beta - immunology | Maf Transcription Factors - immunology | Receptors, CXCR3 - genetics | Embryo, Nonmammalian | Receptors, CXCR3 - immunology | Toll-Like Receptor 2 - immunology | Proto-Oncogene Proteins c-fos - genetics | Larva - microbiology | Zebrafish Proteins - genetics | Receptors, Immunologic - genetics | Maf Transcription Factors - genetics | Transcriptome - immunology | Zebrafish - microbiology | Mycobacterium marinum - pathogenicity | Lymphotoxin-alpha - immunology | Metabolic Networks and Pathways - immunology | Gene Expression Regulation, Developmental | Mycobacterium Infections, Nontuberculous - microbiology | Larva - growth & development | Fish Diseases - immunology | Larva - genetics | Neutrophils - microbiology | Receptors, Immunologic - immunology | Macrophages - immunology | Fish Diseases - microbiology | Neutrophils - immunology | Interleukin-1beta - immunology | Proto-Oncogene Proteins c-fos - immunology | Immunity, Innate | Zebrafish - growth & development | Animals | Mycobacterium Infections, Nontuberculous - veterinary | Mycobacterium Infections, Nontuberculous - genetics | RNA sequencing | Bacterial infections | RNA | Calcifediol | DNA binding proteins | Genetic transcription | Infection | Vitamin D | Physiological aspects | Genetic research | Genetic aspects | Alfacalcidol | Comparative analysis | Health aspects | Larvae | Granuloma | Transcription factors | Genes | Microbiomes | Infections | Macrophages | Protein turnover | Gene sequencing | Proteins | Receptors | Pathways | Interleukin 1 | Toll-like receptors | Pathogens | Antigens | Phenotypes | Immune response | Cytokines | Neutrophils | Leukocytes (neutrophilic) | CXCR3 protein | Ribonucleic acid--RNA | Mammals | Gene expression | Metabolism | Embryos | Defense | Mutants | Infectious diseases | TLR2 protein | Metabolic pathways | CXCL11 protein | Mutation | Vitamin D receptors | Chemokines
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 51, pp. E7128 - E7137
This paper reviews the developments that have occurred in the field of human genetics of infectious diseases from the second half of the 20th century onward.... 
Pediatrics | Immunology | Infectious diseases | Human genetics | Primary immunodeficiency | human genetics | LINKED LYMPHOPROLIFERATIVE SYNDROME | CALMETTE-GUERIN INFECTION | RECEPTOR BETA-1 DEFICIENCY | MULTIDISCIPLINARY SCIENCES | infectious diseases | ANHIDROTIC ECTODERMAL DYSPLASIA | T-CELL IMMUNODEFICIENCY | CHRONIC MUCOCUTANEOUS CANDIDIASIS | PRIMARY IMMUNODEFICIENCY DISEASES | PYOGENIC BACTERIAL-INFECTIONS | pediatrics | primary immunodeficiency | immunology | HERPES-SIMPLEX ENCEPHALITIS | INHERITED CARD9 DEFICIENCY | Encephalitis, Herpes Simplex - immunology | Humans | Genetic Diseases, Inborn - genetics | Lymphoproliferative Disorders - immunology | Mycobacterium Infections - immunology | Young Adult | Pneumococcal Infections - immunology | Mycobacterium Infections - genetics | Pneumococcal Infections - genetics | Complement System Proteins - genetics | Encephalitis, Herpes Simplex - genetics | Epidermodysplasia Verruciformis - immunology | Interferon-gamma - genetics | Immunologic Deficiency Syndromes - immunology | Child | Genetic Predisposition to Disease | Neisseria - immunology | Tinea - immunology | Infection - immunology | Candidiasis, Chronic Mucocutaneous - genetics | Malaria - immunology | Candidiasis, Chronic Mucocutaneous - immunology | Genetic Diseases, Inborn - immunology | Tinea - genetics | Epidermodysplasia Verruciformis - genetics | Lymphoproliferative Disorders - genetics | Infection - genetics | Models, Immunological | Neisseria - pathogenicity | Influenza, Human - genetics | Interferon-gamma - immunology | Adolescent | Immunologic Deficiency Syndromes - genetics | Models, Genetic | Malaria - genetics | Influenza, Human - immunology | Development and progression | Genetic aspects | Disease susceptibility | Immune response | Communicable diseases | Biological Sciences | PNAS Plus
Journal Article
Journal Article
Journal Article
Journal of innate immunity, ISSN 1662-8128, 2014, Volume 6, Issue 2, pp. 152 - 158
We have recently shown that BCG (Bacillus Calmette-Guérin) vaccination in healthy volunteers induces epigenetic reprogramming of monocytes, leading to... 
Research Article | BCG vaccination | Innate immunity | Trained immunity | MORTALITY | CHILD SURVIVAL | WEST-AFRICA | PROTECTION | RANDOMIZED-TRIAL | CALMETTE-GUERIN VACCINATION | IMMUNOLOGY | CANDIDA-ALBICANS | MEMORY | GUINEA-BISSAU | RESISTANCE | Tumor Necrosis Factor-alpha - metabolism | Receptors, Pattern Recognition - immunology | Humans | Vaccination | Interleukin-17 - immunology | Monocytes - metabolism | Tuberculosis - microbiology | Host-Pathogen Interactions - drug effects | Monocytes - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Lipopolysaccharides - immunology | Host-Pathogen Interactions - immunology | Th1 Cells - metabolism | Young Adult | Receptors, Pattern Recognition - metabolism | Flow Cytometry | Th17 Cells - metabolism | Time Factors | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Adult | Adaptive Immunity - immunology | Enzyme-Linked Immunosorbent Assay | Mycobacterium - immunology | Mycobacterium - physiology | Interleukin-1beta - immunology | Monocytes - drug effects | Tuberculosis - immunology | Immunity, Innate - immunology | Interleukin-17 - metabolism | Interferon-gamma - immunology | Tuberculosis - prevention & control | BCG Vaccine - immunology | Lipopolysaccharides - pharmacology | Th17 Cells - immunology | BCG Vaccine - administration & dosage | Adaptive Immunity | Mycobacterium | Interferon-gamma | Interleukin-1beta | Immunity, Innate | Tumor Necrosis Factor-alpha | Lipopolysaccharides | Host-Pathogen Interactions | Life Sciences | Receptors, Pattern Recognition | Monocytes | Tuberculosis | Immunology | Th1 Cells | BCG Vaccine | Th17 Cells | Interleukin-17 | trained immunity
Journal Article
Trends in Immunology, ISSN 1471-4906, 2011, Volume 32, Issue 10, pp. 470 - 477
Environmental signals at the site of inflammation mediate rapid monocyte mobilization and dictate differentiation programs whereby these cells give rise to... 
Allergy and Immunology | URINARY-TRACT-INFECTION | MYCOBACTERIUM-TUBERCULOSIS | CELLS IN-VIVO | ATHEROSCLEROTIC PLAQUES | DENDRITIC CELLS | IMMUNE-RESPONSES | BONE-MARROW | LYMPH-NODES | MYD88-DEPENDENT ACTIVATION | BACTERIAL-INFECTION | IMMUNOLOGY | Inflammation - pathology | Spleen - immunology | Monocytes - cytology | Dendritic Cells - immunology | Humans | Monocytes - immunology | Cell Movement - immunology | Signal Transduction - immunology | Inflammation - metabolism | Listeria - immunology | Listeriosis - immunology | Dendritic Cells - metabolism | Macrophages - immunology | Chemokines - immunology | Acute Disease | Atherosclerosis - pathology | Listeriosis - metabolism | Atherosclerosis - immunology | Receptors, Chemokine - metabolism | Rats | Inflammation - immunology | Spleen - cytology | Receptors, Chemokine - immunology | Immunity, Innate | Cell Differentiation - immunology | Macrophages - metabolism | Animals | Listeriosis - microbiology | Chemokines - metabolism | Mice | Chronic Disease | Development and progression | Inflammation | Macrophages | Analysis | Atherosclerosis | Studies | Heart attacks | Listeria | Ligands | Infections | Gene expression | Chemokines | Immune system | Spleen | Dendritic Cells | Signal Transduction | Listeriosis | Life Sciences | Monocytes | Immunology | Cell Differentiation | Receptors, Chemokine | Cell Movement
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2007, Volume 282, Issue 36, pp. 26014 - 26025
Mycobacterium tuberculosis lipomannans (LMs) modulate the host innate immune response. The total fraction of Mycobacterium bovis BCG LM was shown both to... 
BACILLUS-CALMETTE-GUERIN | C-TYPE LECTIN | DENDRITIC CELLS | MULTIACYLATED FORMS | SURFACTANT PROTEIN-A | NITRIC-OXIDE SYNTHASE | DC-SIGN | BIOCHEMISTRY & MOLECULAR BIOLOGY | SELECTIVE RECOGNITION | MYO-INOSITOL ANCHOR | 2-DEFICIENT MICE | Mannose-Binding Lectins - metabolism | Mycobacterium bovis - immunology | Toll-Like Receptor 2 - agonists | Membrane Glycoproteins - metabolism | Humans | Lectins, C-Type - immunology | Mycobacterium tuberculosis - immunology | Lipopolysaccharides - metabolism | Cell Adhesion Molecules - immunology | Lipopolysaccharides - immunology | Lectins, C-Type - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Macrophages - secretion | Toll-Like Receptor 1 - agonists | Myeloid Differentiation Factor 88 - immunology | Tumor Necrosis Factor-alpha - immunology | Toll-Like Receptor 4 - agonists | Interleukin-12 Subunit p40 - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Acylation | Cell Line | Nitric Oxide - immunology | Immunity, Innate - drug effects | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Tumor Necrosis Factor-alpha - secretion | Receptors, Cell Surface - metabolism | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Nitric Oxide - secretion | Animals | Mannose-Binding Lectins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Toll-Like Receptor 1 - immunology | Macrophage Activation - drug effects | Myeloid Differentiation Factor 88 - metabolism | Toll-Like Receptor 1 | Toll-Like Receptor 2 | Toll-Like Receptor 4 | Cell Adhesion Molecules | Receptors, Cell Surface | Macrophages | Lipopolysaccharides | Life Sciences | Immunology | Membrane Glycoproteins | Mannose-Binding Lectins | Mycobacterium bovis | Signal Transduction | Lectins, C-Type | Inflammation | Myeloid Differentiation Factor 88 | Nitric Oxide | Tumor Necrosis Factor-alpha | Immunity, Natural | Macrophage Activation | Mycobacterium tuberculosis | Receptors, Interleukin-1 | Interleukin-12 Subunit p40
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e32769
A safe and effective HIV vaccine is required to significantly reduce the number of people becoming infected with HIV each year. In this study wild type... 
LISTERIA-MONOCYTOGENES | VACCINE DEVELOPMENT | RESPONSES | BACILLUS-CALMETTE-GUERIN | CD8-T-CELL MEMORY | CD4-T-CELL HELP | TUBERCULOSIS | BIOLOGY | IMMUNODEFICIENCY-VIRUS TYPE-1 | INFECTION | SURFACE PROTEIN-A | Tumor Necrosis Factor-alpha - metabolism | HIV Infections - prevention & control | Mycobacterium bovis - immunology | Spleen - immunology | AIDS Vaccines - immunology | Interferon-gamma - metabolism | Vaccinia virus - genetics | BCG Vaccine - genetics | HIV Infections - immunology | Vaccination - methods | Tumor Necrosis Factor-alpha - immunology | CD8-Positive T-Lymphocytes - metabolism | Interleukin-6 - metabolism | Pantothenic Acid - metabolism | Mycobacterium bovis - genetics | Gene Products, gag - immunology | Gene Products, gag - genetics | Enzyme-Linked Immunosorbent Assay | Immunization, Secondary - methods | Spleen - cytology | HIV-1 - genetics | Mice, SCID | HIV-1 - immunology | Animals | Mycobacterium bovis - metabolism | Spleen - metabolism | Interferon-gamma - immunology | BCG Vaccine - immunology | Interleukin-6 - immunology | AIDS Vaccines - genetics | Mice | Mice, Inbred BALB C | AIDS Vaccines - administration & dosage | CD8-Positive T-Lymphocytes - immunology | BCG Vaccine - administration & dosage | HIV Infections - metabolism | Hsp60 protein | Laboratories | CD8 antigen | Vaccination | Viruses | Lymphocytes T | Infections | Vaccines | Gene deletion | Tropical diseases | Proteins | Immunology | Listeria | Gag protein | Acquired immune deficiency syndrome--AIDS | Clonal deletion | Lymphocytes | Human immunodeficiency virus--HIV | Immune system | Antigens | Research & development--R&D | BCG | Priming | Gene expression | Virology | CD4 antigen | Medicine | Disease prevention | Tuberculosis | Infectious diseases | Research & development | Acquired immune deficiency syndrome | AIDS | R&D | HIV | Human immunodeficiency virus
Journal Article
Vaccine, ISSN 0264-410X, 2011, Volume 29, Issue 40, pp. 6959 - 6966
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 5, pp. 1827 - 1832
CD1d-restricted natural killer T (NKT) cells include two major subgroups. The most widely studied are Vα14Jα18⁺ invariant NKT (iNKT) cells that recognize the... 
T lymphocytes | Antigens | Reactivity | Natural killer T cells | Hybridomas | Antigen presenting cells | Lipids | Infections | Phospholipids | T cell antigen receptors | CD1 antigen presentation | Innate immunity | Adaptive immunity | Type II natural killer T cell | SULFATIDE | REPERTOIRE | POPULATION | ACTIVATION | MULTIDISCIPLINARY SCIENCES | DISTINCT | type II natural killer T cell | RECEPTOR | adaptive immunity | CD1 | innate immunity | KILLER T-CELLS | INNATE-LIKE | GLYCOLIPIDS | Hybridomas - metabolism | Corynebacterium glutamicum - genetics | Antigens, CD1d - immunology | Transferases (Other Substituted Phosphate Groups) - genetics | Antigens, Bacterial - immunology | Mycobacterium tuberculosis - immunology | Transferases (Other Substituted Phosphate Groups) - immunology | Cardiolipins - immunology | Galactosylceramides - metabolism | Receptors, Antigen, T-Cell, alpha-beta - immunology | Corynebacterium glutamicum - metabolism | Transferases (Other Substituted Phosphate Groups) - metabolism | Natural Killer T-Cells - metabolism | Myeloid Differentiation Factor 88 - immunology | Antigens, CD1d - genetics | Bacterial Proteins - immunology | Phospholipids - metabolism | Toll-Like Receptors - metabolism | Mycobacterium tuberculosis - metabolism | Phosphatidylglycerols - metabolism | Macrophages - immunology | Macrophages - microbiology | Cell Line | Antigen-Presenting Cells - metabolism | Mice, Inbred C57BL | Phosphatidylglycerols - immunology | Phospholipids - immunology | Toll-Like Receptors - immunology | Bacterial Proteins - genetics | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Cardiolipins - metabolism | Antigen-Presenting Cells - immunology | Antigens, CD1d - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Galactosylceramides - immunology | Hybridomas - immunology | Bacterial Proteins - metabolism | Mice | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Corynebacterium glutamicum - immunology | Antigens, Bacterial - metabolism | Myeloid Differentiation Factor 88 - metabolism | Natural Killer T-Cells - immunology | Autoimmunity | Killer cells | Antigen receptors, T cell | Receptors | Research | Properties | T cells | Biological Sciences | glycolipids | KazuyoshiKawakami | distinct | Mikrobiologi inom det medicinska området | VictorNizet | population | MoriyaTsuji | cd1 | receptor | Gurdyal S.Besra | Microbiology in the medical area | killer t-cells | innate-like | sulfatide | repertoire | KNizet | VBesra | GSTsuji | activation | Kawakami
Journal Article