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The New England journal of medicine, ISSN 1533-4406, 2011, Volume 365, Issue 25, pp. 2377 - 2388
The authors report that INF2 mutations are present in patients with focal segmental glomerulosclerosis (FSGS) associated with Charcot–Marie–Tooth neuropathy.... 
GLOMERULOSCLEROSIS | MEDICINE, GENERAL & INTERNAL | MYELIN | PROTEIN | NEUROPATHY | EPITHELIAL-CELLS | GENE | RHO | MEDIATED TRANSPORT | FORMIN | NEPHROPATHY | Humans | Middle Aged | Proteolipids - metabolism | Actins - metabolism | Male | Charcot-Marie-Tooth Disease - genetics | Young Adult | Kidney - metabolism | Glomerulosclerosis, Focal Segmental - etiology | Myelin and Lymphocyte-Associated Proteolipid Proteins | Adult | Female | Membrane Transport Proteins - metabolism | Microfilament Proteins - metabolism | Child | Microfilament Proteins - genetics | Charcot-Marie-Tooth Disease - complications | Schwann Cells - metabolism | Phenotype | Animals | Adolescent | Age of Onset | Heterozygote | Mice | Mutation | Myelin Proteins - metabolism | Glomerulonephritis | Gene mutations | Charcot-Marie-Tooth disease | Causes of | Genetic aspects | Research | Myelin proteins | Cdc42 protein | Disease | Exons | Genes | Amino acids | Nervous system | Neuropathy | Guanine nucleotide-binding protein | Proteins | Myelin P0 protein | Peripheral myelin protein 22 | Localization | Deoxyribonucleic acid--DNA | Kidneys | Schwann cells | Polymerization | Guanosine triphosphatases | Myelination | Genotyping | Biopsy | Glomerulus | Cytoskeleton | Genetic testing | Cytoplasm | Schwann Cells | Genomics | Kidney | Charcot-Marie-Tooth Disease | Life Sciences | Proteolipids | Biochemistry, Molecular Biology | Actins | Microfilament Proteins | Membrane Transport Proteins | Myelin Proteins | Glomerulosclerosis, Focal Segmental
Journal Article
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2010, Volume 66, Issue 5, pp. 663 - 670
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2009, Volume 183, Issue 6, pp. 4067 - 4076
Journal Article
Brain Research, ISSN 0006-8993, 2015, Volume 1641, Issue Pt A, pp. 43 - 63
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2005, Volume 25, Issue 6, pp. 1354 - 1365
Myelin-forming oligodendrocytes facilitate saltatory nerve conduction and support neuronal functions in the mammalian CNS. Although the processes of... 
Myelin assembly | Gene knock-out | Mouse models | Olig genes | Axonal integrity | Oligodendrocytes | mouse models | PROTEOLIPID PROTEIN | TRANSCRIPTION FACTORS | axonal integrity | myelin assembly | oligodendrocytes | OLIG2 | NEUROSCIENCES | gene knock-out | IN-VITRO | MYELIN | DISRUPTION | GENES | BASIC-PROTEIN | EXPRESSION | CELL | Glial Fibrillary Acidic Protein - genetics | Spinal Cord - metabolism | Transcription Factors - deficiency | Myelin-Associated Glycoprotein - genetics | Cercopithecus aethiops | Axons - physiology | Helix-Loop-Helix Motifs | Myelin-Associated Glycoprotein - biosynthesis | DNA-Binding Proteins - deficiency | Mice, Neurologic Mutants | Myelin Sheath - metabolism | Brain - metabolism | Genes, Lethal | Myelin Basic Protein - genetics | Myelin Basic Protein - biosynthesis | Oligodendroglia - physiology | Transfection | Spinal Cord - pathology | Nerve Tissue Proteins - biosynthesis | Cell Differentiation | Basic Helix-Loop-Helix Transcription Factors | DNA-Binding Proteins - physiology | Gliosis - genetics | Myelin Sheath - physiology | Transcription Factors - physiology | Mice, Inbred C57BL | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Mice, Knockout | Myelin Proteolipid Protein - biosynthesis | Glial Fibrillary Acidic Protein - biosynthesis | Phenotype | Transcription, Genetic - physiology | Animals | Myelin Proteolipid Protein - genetics | Brain - pathology | Cells, Cultured - metabolism | Mice | Nerve Degeneration | COS Cells | Plasticity | Development | Repair
Journal Article
Journal Article
Immunology Letters, ISSN 0165-2478, 2017, Volume 183, pp. 79 - 85
Highlights • Multiple sclerosis is an autoimmune disease with poorly understood aetiology. • Molecular mimicry between myelin proteins and endogenous... 
Allergy and Immunology | Autoimmunity | Human endogenous retroviruses | Multiple sclerosis | Molecular mimicry | HLA DR2 haplotype | Myelin proteins | CELLS | EPITOPE | DNA | EAE | IMMUNOLOGY | BINDING | Myelin-Oligodendrocyte Glycoprotein - immunology | Pregnancy Proteins - chemistry | Gene Products, env - immunology | Humans | Male | Pregnancy Proteins - metabolism | Endogenous Retroviruses - chemistry | Gene Products, env - metabolism | Molecular Mimicry | Endogenous Retroviruses - metabolism | T-Lymphocytes - metabolism | Myelin Basic Protein - chemistry | Peptide Fragments - immunology | Viral Envelope Proteins - metabolism | Myelin Proteins - immunology | Female | Multiple Sclerosis - metabolism | Amino Acid Sequence | Computational Biology - methods | Peptide Fragments - metabolism | Myelin Basic Protein - metabolism | HLA-DR2 Antigen - metabolism | Pregnancy Proteins - immunology | Myelin Basic Protein - immunology | HLA-DR2 Antigen - immunology | Sequence Homology, Amino Acid | Myelin Proteins - chemistry | Myelin-Oligodendrocyte Glycoprotein - chemistry | Peptide Fragments - chemistry | Myelin-Oligodendrocyte Glycoprotein - metabolism | Gene Products, env - chemistry | Viral Envelope Proteins - chemistry | Multiple Sclerosis - pathology | Protein Binding | T-Lymphocytes - immunology | Multiple Sclerosis - etiology | Myelin Proteins - metabolism | Antigenic determinants
Journal Article