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Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 08/2015, Volume 86, Issue 8, pp. 873 - 878
Journal Article
Development, ISSN 0950-1991, 07/2011, Volume 138, Issue 13, pp. 2673 - 2680
In peripheral nerves, Schwann cells form the myelin sheath that insulates axons and allows rapid propagation of action potentials. Although a number of... 
Gpr126 | Mouse | Myelin | Schwann cell | SYSTEM | CLAW-PAW MUTATION | DEVELOPMENTAL BIOLOGY | GLIAL-CELLS | DESERT-HEDGEHOG | CYCLIC-AMP | PROTEIN-COUPLED RECEPTOR | TRANSCRIPTION FACTOR SOX10 | GENE-EXPRESSION | SCHWANN-CELL DIFFERENTIATION | Immunohistochemistry | Peripheral Nerves - ultrastructure | Cochlear Nerve - ultrastructure | Receptors, G-Protein-Coupled - metabolism | Early Growth Response Protein 2 - genetics | Octamer Transcription Factor-6 - genetics | Cochlear Nerve - metabolism | Myelin Basic Protein - genetics | Octamer Transcription Factor-6 - metabolism | Peripheral Nerves - metabolism | Peripheral Nervous System Diseases - genetics | Microscopy, Electron, Transmission | Myelin Basic Protein - metabolism | Cochlear Nerve - abnormalities | Schwann Cells - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Peripheral Nerves - pathology | Mice, Knockout | Peripheral Nervous System Diseases - metabolism | Animals | Myelin P0 Protein - genetics | Early Growth Response Protein 2 - metabolism | Mice | Receptors, G-Protein-Coupled - genetics | Myelin P0 Protein - metabolism | Peripheral Nerves - growth & development | Index Medicus | Peripheral nerves | Schwann cells | perineurium | Action potential | Peripheral neuropathy | Myelin basic protein | EGR-2 protein | Axons | Signal transduction | Myelination | Fibroblasts | Mutation | Myelin P0 protein | Sciatic nerve
Journal Article
Journal Article
Glia, ISSN 0894-1491, 04/2019, Volume 67, Issue 4, pp. 650 - 667
Demyelination occurs following many neurological insults, most notably in multiple sclerosis (MS). Therapeutics that promote remyelination could slow the... 
myelination | oligodendrocytes | in vivo | zebrafish | myelin protein zero | RED PHOTOCONVERSION | SPINAL-CORD | CNS | RESCUES BEHAVIORAL-CHANGES | NEUROSCIENCES | OLIGODENDROCYTE PRECURSORS | WHITE-MATTER DAMAGE | MULTIPLE-SCLEROSIS | REMYELINATION | CENTRAL-NERVOUS-SYSTEM | AXON FORMATION | Spinal Cord - metabolism | Demyelinating Diseases - genetics | Gene Expression Regulation, Developmental - genetics | Culture Media, Conditioned - pharmacology | Green Fluorescent Proteins - genetics | Demyelinating Diseases - metabolism | Myelin Basic Protein - genetics | Oligodendroglia - drug effects | Oligodendroglia - physiology | Demyelinating Diseases - pathology | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Green Fluorescent Proteins - metabolism | Myelin Sheath - physiology | Myelin Basic Protein - metabolism | Animals, Genetically Modified | SOXE Transcription Factors - metabolism | Zebrafish Proteins - metabolism | Gene Expression Regulation, Developmental - drug effects | Zebrafish | Sirolimus - pharmacology | Spinal Cord - embryology | Animals | Larva | Embryo, Nonmammalian | Myelin P0 Protein - genetics | Luminescent Proteins - genetics | Neuroglia - metabolism | Myelin Sheath - ultrastructure | Embryonic Stem Cells | Zebrafish Proteins - genetics | Myelin P0 Protein - metabolism | Luminescent Proteins - metabolism | SOXE Transcription Factors - genetics | Nervous system diseases | Multiple sclerosis | Imaging systems | Fluorescence | Cytogenetics | Animal genetic engineering | Myelin proteins | Drugs | Larvae | Regulators | Myelin | Transgenic | Drug development | Myelin basic protein | Proteins | Organic chemistry | Axons | Screening | Biomedical materials | Myelination | Demyelination | Cascades | Oligodendrocytes | Green fluorescent protein | Biocompatibility | In vivo methods and tests | Myelin P0 protein
Journal Article
Brain, ISSN 0006-8950, 12/2012, Volume 135, Issue 12, pp. 3551 - 3566
Charcot–Marie–Tooth disease type 1B is caused by mutations in myelin protein zero. R98C mice, an authentic model of early onset Charcot–Marie–Tooth disease... 
Charcot-Marie-Tooth disease 1B | unfolded protein response | peripheral neuropathy | myelin protein zero | curcumin | TRANSCRIPTION FACTORS | PROTEIN | PHOSPHORYLATION | ENDOPLASMIC-RETICULUM STRESS | KAPPA-B | NEUROSCIENCES | CLINICAL NEUROLOGY | MYELIN | GENE | MUTATIONS | PELIZAEUS-MERZBACHER DISEASE | C-JUN | Action Potentials - genetics | Muscle Strength - genetics | Age Factors | Neuromuscular Junction - drug effects | Humans | Cercopithecus aethiops | Motor Activity - drug effects | Green Fluorescent Proteins - genetics | COS Cells - drug effects | X-Box Binding Protein 1 | Charcot-Marie-Tooth Disease - genetics | Cysteine - genetics | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-6 - metabolism | Transfection | Protein Folding - drug effects | Arginine - genetics | Neuromuscular Junction - genetics | Electric Stimulation - methods | Action Potentials - drug effects | Disease Models, Animal | Schwann Cells - drug effects | Animals, Newborn | Curcumin - therapeutic use | Gene Expression Regulation - genetics | Cells, Cultured | Mice, Transgenic | Charcot-Marie-Tooth Disease - drug therapy | Transcription Factors - genetics | Charcot-Marie-Tooth Disease - pathology | DNA-Binding Proteins - genetics | Mutation - genetics | Regulatory Factor X Transcription Factors | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Motor Activity - genetics | Rotarod Performance Test | Animals | Analysis of Variance | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Cell Differentiation - drug effects | Proto-Oncogene Proteins c-jun - metabolism | Myelin P0 Protein - genetics | Early Growth Response Protein 2 - metabolism | Mice | Muscle Strength - drug effects | Myelin P0 Protein - metabolism | Index Medicus | Abridged Index Medicus | Original
Journal Article