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Journal of Neuroscience, ISSN 0270-6474, 06/2012, Volume 32, Issue 26, pp. 8855 - 8864
Journal Article
Glia, ISSN 0894-1491, 04/2013, Volume 61, Issue 4, pp. 567 - 586
Journal Article
Journal Article
Glia, ISSN 0894-1491, 11/2017, Volume 65, Issue 11, pp. 1762 - 1776
Proteolipid protein (PLP) is the most abundant integral membrane protein in compact central nervous system myelin, and null mutations of the PLP1 gene cause... 
glia‐axonal support | myelin | FA2H | axonopathy | proteolipid protein (PLP) | neuroinflammation | oligodendrocyte | hereditary spastic paraplegia type 2 (SPG2) | neurodegeneration | GJC2/CX47 | glia-axonal support | PROTEOLIPID PROTEIN GENE | SOMA-RESTRICTED PRODUCTS | CX47 | MYELIN-DEFICIENT RAT | NEUROSCIENCES | CELLS CONTRIBUTE | GJC2 | AXONAL SWELLINGS | CNS MYELIN | PELIZAEUS-MERZBACHER-DISEASE | CENTRAL-NERVOUS-SYSTEM | MONOCLONAL-ANTIBODIES | TRANSGENIC MICE | Spastic Paraplegia, Hereditary - genetics | Oligodendroglia - metabolism | Age Factors | Male | Antigens, CD - metabolism | Ribosomal Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - genetics | Amyloid beta-Protein Precursor - metabolism | Female | Myelin Proteolipid Protein - deficiency | Neurons - metabolism | Spastic Paraplegia, Hereditary - pathology | Disease Models, Animal | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation - genetics | Ribosomal Proteins - genetics | Mice, Inbred C57BL | Axons - metabolism | Mice, Transgenic | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - metabolism | Animals | Myelin Proteolipid Protein - genetics | Axons - pathology | Corpus Callosum - pathology | Mice | Neurons | Surgery | Paralysis, Spastic | Aquaporins | Recombination | Myelin | Abnormalities | Central nervous system | Inflammation | Cre recombinase | Membrane proteins | Axons | Paraplegia | Myelin proteolipid protein | Neurodegeneration | Rodents | Proteolipid protein | Degeneration | Paralysis | Mutation | Spastic paraplegia | Dissection
Journal Article
Glia, ISSN 0894-1491, 01/2016, Volume 64, Issue 1, pp. 155 - 174
Protein zero (P0) is the major structural component of peripheral myelin. Lack of this adhesion protein from Schwann cells causes a severe dysmyelinating... 
glia‐axonal support | myelin | Schwann cell | Dejerine‐Sottas syndrome | oligodendrocyte | neurodegeneration | neuropathy | Charcot‐Marie‐tooth disease | hereditary spastic paraplegia (SPG‐2) | Dejerine-Sottas syndrome | Oligodendrocyte | Myelin | Neurodegeneration | Charcot-Marie-tooth disease | Glia-axonal support | Neuropathy | Hereditary spastic paraplegia (SPG-2) | MARIE-TOOTH-DISEASE | COLONY-STIMULATING FACTOR | glia-axonal support | MAJOR STRUCTURAL PROTEIN | NEUROSCIENCES | MICE HETEROZYGOUSLY DEFICIENT | hereditary spastic paraplegia (SPG-2) | MOUSE MODEL | PELIZAEUS-MERZBACHER-DISEASE | CENTRAL-NERVOUS-SYSTEM | SCHWANN-CELL DEVELOPMENT | BASIC-PROTEIN | INHERITED DEMYELINATING NEUROPATHIES | Motor Activity - physiology | Optic Nerve - pathology | Phrenic Nerve - pathology | Myelin-Associated Glycoprotein - metabolism | Myelin Sheath - metabolism | Brain - metabolism | Sciatic Nerve - metabolism | Myelin Proteolipid Protein - metabolism | Female | Phrenic Nerve - metabolism | Myelin Sheath - pathology | Sciatic Nerve - pathology | Mice, Inbred C57BL | Kaplan-Meier Estimate | Axons - metabolism | Neural Conduction - physiology | Mice, Knockout | Mortality, Premature | Animals | Myelin Proteolipid Protein - genetics | Axons - pathology | Brain - pathology | Myelin P0 Protein - genetics | Myelin P0 Protein - metabolism | Optic Nerve - metabolism | Analysis | Myelin proteins | Proteins | Preservation | Rodents
Journal Article
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 1/2007, Volume 64, Issue 1, pp. 50 - 65
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2002, Volume 22, Issue 3, pp. 876 - 885
Transgenic mice expressing enhanced green fluorescent protein (EGFP) driven by the mouse myelin proteolipid protein (PLP) gene promoter have been developed to... 
Oligodendroglia | NG2 proteoglycan | Enhanced green fluorescent protein | Myelin | Progenitor cells | Proteolipid protein | myelin | PDGF ALPHA-RECEPTOR | proteolipid protein | OLIGODENDROCYTE PRECURSOR CELLS | progenitor cells | SPINAL-CORD | green fluorescent protein | NEUROSCIENCES | oligodendroglia | enhanced | PROTEIN GENE-PRODUCT | DM-20 MESSENGER-RNA | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | DIFFERENTIATION | RAT OPTIC-NERVE | EXPRESSION | Green Fluorescent Proteins | Oligodendroglia - metabolism | Central Nervous System - metabolism | Lateral Ventricles - metabolism | Stem Cells - cytology | Lateral Ventricles - cytology | Peripheral Nervous System - embryology | Stem Cells - metabolism | Central Nervous System - embryology | Cerebral Cortex - cytology | Peripheral Nervous System - cytology | Recombinant Fusion Proteins - metabolism | Cell Movement - physiology | Cerebral Cortex - metabolism | Stem Cells - classification | Cell Differentiation | Oligodendroglia - cytology | Transgenes | Organ Specificity - physiology | Animals, Newborn | Proteoglycans - biosynthesis | Mice, Transgenic | Cell Division - physiology | Cell Lineage | Animals | Promoter Regions, Genetic - physiology | Central Nervous System - cytology | Myelin Proteolipid Protein - genetics | Peripheral Nervous System - metabolism | Cerebral Cortex - growth & development | Recombinant Fusion Proteins - genetics | Antigens - biosynthesis | Luminescent Proteins - genetics | Mice
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2009, Volume 29, Issue 26, pp. 8363 - 8371
Journal Article