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Journal Article
Journal Article
Journal Article
JNEN: Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 07/2002, Volume 61, Issue 7, pp. 623 - 633
Activated microglia and macrophages (CNS macrophages) have been implicated in the secondary or “bystander” pathology (e.g. axon injury, demyelination) that... 
Neuroinflammation | Funiculotomy | Functional recovery | Spinal cord injury | Axonal injury | Demyelination | spinal cord injury | MARROW-DERIVED ELEMENTS | KAPPA-B ACTIVATION | NECROSIS-FACTOR-ALPHA | DORSOLATERAL FUNICULI | MICROGLIAL CELLS | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | MYELIN PHAGOCYTOSIS | axonal injury | SPINAL-CORD INJURY | GROWTH-FACTOR-BETA | neuroinflammation | demyelination | CENTRAL-NERVOUS-SYSTEM | functional recovery | INFLAMMATORY RESPONSE | funiculotomy | Immunohistochemistry | Microglia - metabolism | Myelitis - chemically induced | Membrane Glycoproteins - metabolism | Zymosan - pharmacology | Chemotaxis, Leukocyte - physiology | Spinal Cord Injuries - pathology | Chemotaxis, Leukocyte - drug effects | Nerve Fibers, Myelinated - pathology | Gait Disorders, Neurologic - pathology | Disease Models, Animal | Microglia - cytology | Microinjections | Myelitis - physiopathology | Nerve Fibers, Myelinated - drug effects | Myelitis - pathology | Rats | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Macrophages - metabolism | Blood Proteins | Axons - pathology | Spinal Cord - physiopathology | Antigens, CD | Spinal Cord - drug effects | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Myelin Sheath - drug effects | Gliosis - physiopathology | Nerve Degeneration - chemically induced | Myelin Sheath - metabolism | Basigin | Gliosis - pathology | Spinal Cord - pathology | Gait Disorders, Neurologic - physiopathology | Female | Gait Disorders, Neurologic - chemically induced | Antigens, Neoplasm | Denervation - methods | Microglia - drug effects | Myelin Sheath - pathology | Axons - drug effects | Axons - metabolism | Macrophages - cytology | Animals | Avian Proteins | Macrophages - drug effects | Antigens, Surface | Spinal Cord Injuries - physiopathology
Journal Article
Neurology, ISSN 0028-3878, 2009, Volume 73, Issue 12, pp. 949 - 953
Journal Article
Journal of the American Veterinary Medical Association, ISSN 0003-1488, 02/2012, Volume 240, Issue 3, pp. 269 - 271
Journal Article
Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 10/2009, Volume 68, Issue 10, pp. 1061 - 1072
Neuroinvasive West Nile virus (WNV) infections may cause acute flaccid paralysis (AFP); in fatal cases, anterior horn cell loss is presumed to be caused by... 
Astrocyte | Glutamate transporters | Excitotoxic injury | Acute flaccid paralysis | West Nile virus | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RECEPTOR ANTAGONISTS | AMYOTROPHIC-LATERAL-SCLEROSIS | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | VIRAL ENCEPHALOMYELITIS | EXCITOTOXICITY | SINDBIS VIRUS | MULTIPLE-SCLEROSIS | ENCEPHALITIS | MICE | REDUCED EXPRESSION | Glutamate Plasma Membrane Transport Proteins - metabolism | Dendrites - pathology | Neurons - pathology | Spinal Cord - metabolism | Humans | Middle Aged | Astrocytes - pathology | Male | Paralysis - pathology | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | Paralysis - metabolism | Synapses - metabolism | Astrocytes - immunology | Dendrites - immunology | Spinal Cord - pathology | Female | Neurons - metabolism | West Nile Fever - complications | Paralysis - etiology | West Nile Fever - pathology | Dendrites - metabolism | Acute Disease | Cricetinae | West Nile Fever - immunology | Synapses - immunology | Myelitis - pathology | Neurons - immunology | Paralysis - immunology | West Nile Fever - metabolism | Nerve Fibers, Unmyelinated - immunology | Excitatory Amino Acid Transporter 2 - metabolism | Spinal Cord - immunology | Nerve Fibers, Unmyelinated - pathology | Animals | Nerve Fibers, Unmyelinated - metabolism | Myelitis - metabolism | Aged | Astrocytes - metabolism
Journal Article
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 10/2008, Volume 25, Issue 10, pp. 1217 - 1225
Inflammation in the CNS predominantly involves microglia and macrophages, and is believed to be a significant cause of secondary injury following trauma. This... 
Brain | Spinal cord | Inflammation | Injury | Macrophage | OPTIC-NERVE | RAT | NEUROTROPHIC FACTOR | AXON REGENERATION | MOTOR FUNCTION | macrophage | CELLULAR REACTION | MICROGLIAL CELLS | brain | METHYLPREDNISOLONE | NEUROSCIENCES | CLINICAL NEUROLOGY | inflammation | CENTRAL-NERVOUS-SYSTEM | ACTIVATED MACROPHAGES | injury | spinal cord | CRITICAL CARE MEDICINE | Neuroprotective Agents - therapeutic use | Rats, Inbred F344 | Gliosis - physiopathology | Brain Injuries - physiopathology | Gliosis - pathology | Encephalitis - physiopathology | Microglia - immunology | Neuroprotective Agents - pharmacology | Brain Injuries - immunology | CD11b Antigen - analysis | Myelitis - immunology | Time Factors | Nerve Fibers, Myelinated - pathology | Spinal Cord - pathology | Anti-Inflammatory Agents - therapeutic use | Microglia - pathology | Female | Gliosis - immunology | Macrophages - immunology | Disease Models, Animal | Biomarkers - metabolism | Macrophages - pathology | Anti-Inflammatory Agents - pharmacology | Myelitis - physiopathology | Brain - physiopathology | Biomarkers - analysis | Myelitis - pathology | Encephalitis - pathology | Rats | Encephalitis - immunology | Spinal Cord - immunology | Animals | Nerve Fibers, Myelinated - immunology | Brain - pathology | Spinal Cord - physiopathology | CD11b Antigen - metabolism | Brain - immunology | Brain Injuries - pathology | Complications and side effects | Medical examination | Physiological aspects | Spinal cord injuries | Research | Diagnosis | Drug therapy | Macrophages | Health aspects | Risk factors | Comparative studies | Neurology | Brain damage | Inflammatory diseases | Injuries
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 04/2010, Volume 27, Issue 4, pp. 739 - 751
Spinal cord injury results in disruption of the cord microstructure, which is followed by inflammation leading to additional deterioration. Perivascular... 
Glia cell response to injury | Extracellular matrix | Inflammation | Spinal cord injury | spinal cord injury | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | COLLAGEN-IV | GLIAL SCAR | COMPRESSION INJURY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | REPAIR | inflammation | EXTRAVASATION | glia cell response to injury | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | EXPRESSION | extracellular matrix | CRITICAL CARE MEDICINE | Immunohistochemistry | Monocytes - cytology | Spinal Cord Injuries - complications | Blood Vessels - pathology | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Extracellular Matrix - metabolism | Myelitis - etiology | Gliosis - physiopathology | Basement Membrane - pathology | Chemotaxis, Leukocyte - physiology | Spinal Cord Injuries - pathology | Blood Vessels - physiopathology | Gliosis - pathology | Spinal Cord - pathology | Female | Monocytes - physiology | Gliosis - etiology | Macrophages - physiology | Endothelial Cells - metabolism | Myelitis - physiopathology | Mice, Inbred C57BL | Myelitis - pathology | Macrophages - cytology | Spinal Cord - blood supply | Basement Membrane - physiopathology | Blood-Brain Barrier - pathology | Animals | Mice | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Endothelial Cells - pathology | Extracellular Matrix - pathology | Astrocytes - metabolism | Physiological aspects | Care and treatment | Spinal cord injuries | Models | Patient outcomes | Neuroglia | Membranes | Cellular biology | Rodents | Inflammatory diseases
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0167473 - e0167473
Journal Article
Journal Article