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Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 9, pp. 7692 - 7704
Journal Article
Infection and Immunity, ISSN 0019-9567, 12/2013, Volume 81, Issue 12, pp. 4399 - 4407
Journal Article
Scientific Reports, ISSN 2045-2322, 07/2015, Volume 5, Issue 1, p. 11888
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 9, pp. 7070 - 7081
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 07/2015, Volume 67, Issue 7, pp. 1922 - 1932
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2015, Volume 52, Issue 5, pp. 611 - 621
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 01/2012, Volume 23, Issue 1, pp. 49 - 62
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2011, Volume 286, Issue 28, pp. 24638 - 24648
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 4, pp. 1272 - 1282
Journal Article
PLOS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0172105
Estrogen, a natural immunomodulator, regulates the development and function of diverse immune cell types. There is now renewed attention on neutrophils and... 
MENSTRUAL-CYCLE | B-CELLS | INTERFERON-GAMMA | AUTOIMMUNE-DISEASES | MULTIDISCIPLINARY SCIENCES | IN-VIVO | NITRIC-OXIDE | CATHEPSIN-G | CELL-DIFFERENTIATION | T-LYMPHOCYTES | 17-BETA-ESTRADIOL | Leukocyte Elastase - metabolism | Serine Proteases - genetics | Estrogens - pharmacology | Gene Expression - drug effects | Species Specificity | Male | Spleen - drug effects | Myeloblastin - genetics | Mice, Inbred MRL lpr | Flow Cytometry | Inflammation Mediators - metabolism | Female | Neutrophils - metabolism | Myeloblastin - metabolism | Serine Proteases - metabolism | Cytokines - metabolism | Neutrophils - enzymology | Cathepsin G - genetics | Mice, Inbred C57BL | Neutrophils - drug effects | Cells, Cultured | Leukocyte Elastase - genetics | Spleen - cytology | Mice, Inbred NZB | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Cathepsin G - metabolism | Animals | Spleen - metabolism | Peroxidase - genetics | Estrogens - administration & dosage | Peroxidase - metabolism | Spleen | Lupus | Neutrophils | Estrogen | Dosage and administration | Research | Drug therapy | Veterinary colleges | Proteinase | Estrogens | Serine | Veterinary medicine | Blood | Lipopolysaccharides | Interleukin 6 | Splenocytes | Lymphocytes | Rodents | Bone marrow | Peroxidase | Elastase | Cytokines | Dendritic cells | Cathepsin G | Gender differences | Leukocytes (neutrophilic) | Inflammation | Tumor necrosis factor-α | Commonality | Infectious diseases | Womens health | γ-Interferon | Mice | Autoimmune diseases | Chemokines | Monocyte chemoattractant protein 1 | Proteinase 3
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e38317
Background: The complement system is one of the important contributing factors in the development of antineutrophil cytoplasmic antibody (ANCA)-associated... 
REGULATED KINASE | GLOMERULONEPHRITIS | AUTOANTIBODIES | MYELOPEROXIDASE | INHIBITION | ANTIMYELOPEROXIDASE ANTIBODIES | MOLECULAR-STRUCTURE | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | ANTINEUTROPHIL CYTOPLASMIC ANTIBODIES | VASCULITIS | Neutrophil Activation - immunology | Phosphorylation | Neutrophils - enzymology | Signal Transduction | Neutrophils - drug effects | Enzyme Inhibitors - pharmacology | Neutrophil Activation - drug effects | Neutrophils - immunology | Complement C5a - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Protein Transport - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Respiratory Burst - immunology | Cell Degranulation - immunology | Antibodies, Antineutrophil Cytoplasmic - immunology | Respiratory Burst - drug effects | Antibodies, Antineutrophil Cytoplasmic - metabolism | Cell Membrane - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Complement C5a - immunology | Proto-Oncogene Proteins c-akt - metabolism | Myeloblastin - metabolism | Cell Degranulation - drug effects | Peroxidase - metabolism | Mitogens | Protein kinases | Immunoglobulin G | Lactoferrins | Flow cytometry | Transcription factors | Nephrology | Laboratories | Complement component C5a | Fluorescence | Activation | Kinases | Recruitment | Proteins | Signal transduction | Lactoferrin | Cell activation | Vasculitis | Pathways | Antineutrophil cytoplasmic antibodies | Tumor necrosis factor-TNF | Translocation | Antigens | Immunoglobulins | Incubation | Neutrophils | Extracellular signal-regulated kinase | c-Jun protein | MAP kinase | Priming | Leukocytes (neutrophilic) | 1-Phosphatidylinositol 3-kinase | Medicine | Signaling | Complement activation | Hospitals | Inhibitors | Protein kinase | Degranulation | Kidney diseases | Respiratory burst
Journal Article