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Journal Article
Cancer cell, ISSN 1535-6108, 2008, Volume 13, Issue 6, pp. 483 - 495
Faithful modeling of mixed-lineage leukemia in murine cells has been difficult to achieve... 
STEMCELL | CELLCYCLE | HEMATOPOIETIC-CELLS | B-CELLS | STEM-CELLS | ONCOLOGY | MLL REARRANGEMENTS | ACUTE LYMPHOBLASTIC-LEUKEMIA | ACUTE MYELOGENOUS LEUKEMIA | THERAPEUTIC TARGET | GENE-EXPRESSION PROFILES | ACUTE MYELOID-LEUKEMIA | FLT3 MUTATIONS | Translocation, Genetic | Antigens, CD34 - analysis | Myeloid-Lymphoid Leukemia Protein - metabolism | Cell Proliferation | Fetal Stem Cells - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Multipotent Stem Cells - metabolism | rac GTP-Binding Proteins - metabolism | Aneuploidy | Gene Expression Profiling | Stem Cell Transplantation | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | rac GTP-Binding Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Culture Techniques | Fetal Blood - immunology | Transduction, Genetic | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Genotype | Cell Lineage | Multipotent Stem Cells - pathology | Phenotype | Environment | Mice, Inbred NOD | Mice | Fetal Stem Cells - pathology | Oncogene Proteins, Fusion - metabolism | Species Specificity | Gene Expression Regulation, Neoplastic | Neoplastic Stem Cells - immunology | Multipotent Stem Cells - immunology | Fetal Stem Cells - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Leukemia, Experimental - pathology | Fetal Blood - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Myeloid-Lymphoid Leukemia Protein - genetics | Oncogene Proteins, Fusion - genetics | Chromosomes, Human, Pair 11 | Cell Transformation, Neoplastic - pathology | Cell Line, Transformed | Leukemia, Experimental - metabolism | Apoptosis | Leukemia, Myeloid, Acute - genetics | Cytogenetics | Models | Growth factors | Analysis | Leukemia | Stem cells
Journal Article
Journal Article
Cancer research (Chicago, Ill.), ISSN 1538-7445, 2013, Volume 73, Issue 8, pp. 2435 - 2444
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 5, pp. 1827 - 1832
CD1d-restricted natural killer T (NKT) cells include two major subgroups. The most widely studied are Vα14Jα18⁺ invariant NKT (iNKT... 
T lymphocytes | Antigens | Reactivity | Natural killer T cells | Hybridomas | Antigen presenting cells | Lipids | Infections | Phospholipids | T cell antigen receptors | CD1 antigen presentation | Innate immunity | Adaptive immunity | Type II natural killer T cell | SULFATIDE | REPERTOIRE | POPULATION | ACTIVATION | MULTIDISCIPLINARY SCIENCES | DISTINCT | type II natural killer T cell | RECEPTOR | adaptive immunity | CD1 | innate immunity | KILLER T-CELLS | INNATE-LIKE | GLYCOLIPIDS | Hybridomas - metabolism | Corynebacterium glutamicum - genetics | Antigens, CD1d - immunology | Transferases (Other Substituted Phosphate Groups) - genetics | Antigens, Bacterial - immunology | Mycobacterium tuberculosis - immunology | Transferases (Other Substituted Phosphate Groups) - immunology | Cardiolipins - immunology | Galactosylceramides - metabolism | Receptors, Antigen, T-Cell, alpha-beta - immunology | Corynebacterium glutamicum - metabolism | Transferases (Other Substituted Phosphate Groups) - metabolism | Natural Killer T-Cells - metabolism | Myeloid Differentiation Factor 88 - immunology | Antigens, CD1d - genetics | Bacterial Proteins - immunology | Phospholipids - metabolism | Toll-Like Receptors - metabolism | Mycobacterium tuberculosis - metabolism | Phosphatidylglycerols - metabolism | Macrophages - immunology | Macrophages - microbiology | Cell Line | Antigen-Presenting Cells - metabolism | Mice, Inbred C57BL | Phosphatidylglycerols - immunology | Phospholipids - immunology | Toll-Like Receptors - immunology | Bacterial Proteins - genetics | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Cardiolipins - metabolism | Antigen-Presenting Cells - immunology | Antigens, CD1d - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Galactosylceramides - immunology | Hybridomas - immunology | Bacterial Proteins - metabolism | Mice | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Corynebacterium glutamicum - immunology | Antigens, Bacterial - metabolism | Myeloid Differentiation Factor 88 - metabolism | Natural Killer T-Cells - immunology | Autoimmunity | Killer cells | Antigen receptors, T cell | Receptors | Research | Properties | T cells | Biological Sciences | glycolipids | KazuyoshiKawakami | distinct | Mikrobiologi inom det medicinska området | VictorNizet | population | MoriyaTsuji | cd1 | receptor | Gurdyal S.Besra | Microbiology in the medical area | killer t-cells | innate-like | sulfatide | repertoire | KNizet | VBesra | GSTsuji | activation | Kawakami
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 4, pp. 2080 - 2088
Cross-talk between NK cells and dendritic cells (DCs) is critical for the potent therapeutic response to dsRNA, but the receptors involved remained controversial... 
TOLL-LIKE RECEPTOR-3 | CROSS-TALK | ADAPTIVE IMMUNITY | NATURAL-KILLER-CELLS | ACTIVATION | INNATE RESISTANCE | RECOGNITION | PATHWAY | IMMUNOLOGY | BACTERIAL PEPTIDOGLYCAN | CYTOTOXICITY | Myeloid Cells - cytology | Humans | Interferon-gamma - metabolism | Interferon-Induced Helicase, IFIH1 | Dose-Response Relationship, Drug | Transfection | Dendritic Cells - drug effects | Myeloid Cells - drug effects | Toll-Like Receptor 3 - genetics | DEAD-box RNA Helicases - metabolism | Dendritic Cells - metabolism | Cell Line | DEAD Box Protein 58 | Poly I-C - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Poly A-U - pharmacology | Toll-Like Receptor 3 - metabolism | Mice, Knockout | DEAD-box RNA Helicases - genetics | Killer Cells, Natural - cytology | Animals | Lymphocyte Activation - drug effects | Adaptor Proteins, Signal Transducing - genetics | RNA, Double-Stranded - pharmacology | Myeloid Cells - metabolism | Dendritic Cells - cytology | Killer Cells, Natural - drug effects | Mice | Killer Cells, Natural - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Poly A-U | Interferon-gamma | Toll-Like Receptor 3 | DEAD-box RNA Helicases | Dendritic Cells | Killer Cells, Natural | Lymphocyte Activation | Myeloid Cells | Life Sciences | Adaptor Proteins, Signal Transducing | Immunology | RNA, Double-Stranded | Poly I-C
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2014, Volume 129, Issue 15, pp. 1551 - 1559
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 506, Issue 7487, pp. 240 - 244
textabstractCells of the osteoblast lineage affect the homing and the number of long-term repopulating haematopoietic stem cells, haematopoietic stem cell mobilization and lineage determination and B cell lymphopoiesis... 
PROGENITOR CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | BONE-MARROW NICHE | NOTCH ACTIVATION | STEM-CELL NICHE | MYELOID-LEUKEMIA | MICE | DIFFERENTIATION | EXPRESSION | MYELODYSPLASTIC SYNDROMES | Osteoblasts - secretion | Receptors, Notch - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Hematopoietic Stem Cells - pathology | Anemia - pathology | Male | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - deficiency | Anemia - genetics | Cell Nucleus - metabolism | Tumor Microenvironment - genetics | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Base Sequence | Female | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - deficiency | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Myelodysplastic Syndromes - metabolism | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Calcium-Binding Proteins - deficiency | Hematopoietic Stem Cells - metabolism | Mutation - genetics | beta Catenin - metabolism | beta Catenin - genetics | Anemia - metabolism | Cell Lineage | Osteoblasts - pathology | Animals | Chromosome Aberrations | Myeloid Cells - metabolism | Ligands | Mice | Myelodysplastic Syndromes - genetics | Myeloid Cells - pathology | Cell Transformation, Neoplastic - pathology | Myelodysplastic Syndromes - pathology | Osteoblasts - metabolism | Calcium-Binding Proteins - genetics | Leukemia, Myeloid, Acute - genetics
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2015, Volume 194, Issue 7, pp. 3259 - 3266
.... No studies have investigated whether H. pylori affects HIF signaling in immune cells and a potential role for HIF in H... 
ACTIVATION | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | NITRIC-OXIDE SYNTHASE | MACROPHAGES | HOST | INFECTION | MICE | KAPPA-B | HIF-1-ALPHA | IMMUNOLOGY | EXPRESSION | Helicobacter pylori - immunology | Gastric Mucosa - microbiology | Gastritis - metabolism | Humans | Gastric Mucosa - pathology | Helicobacter Infections - complications | Gastric Mucosa - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Gastritis - etiology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Gastritis - pathology | Myeloid Cells - immunology | Inflammation Mediators - metabolism | Helicobacter Infections - metabolism | Neutrophils - microbiology | Cytokines - genetics | Neutrophils - metabolism | Macrophages - immunology | Macrophages - microbiology | Disease Models, Animal | Cytokines - metabolism | Neutrophils - immunology | Mice, Transgenic | Helicobacter Infections - immunology | Helicobacter Infections - genetics | Gastric Mucosa - immunology | Macrophages - metabolism | Animals | Biopsy | Myeloid Cells - metabolism | Mice | Inflammation Mediators/metabolism | Macrophages/microbiology | Helicobacter Infections/genetics | Macrophages/metabolism | Neutrophils/microbiology | Macrophages/immunology | Gastritis/metabolism | Cellular Biology | Myeloid Cells/immunology | Life Sciences | Basic Helix-Loop-Helix Transcription Factors/metabolism | Neutrophils/immunology | Cytokines/genetics | Cytokines/metabolism | Cell Behavior | Helicobacter pylori/immunology | Hypoxia-Inducible Factor 1/metabolism | Neutrophils/metabolism | Gastric Mucosa/immunology | Gastritis/etiology | Helicobacter Infections/metabolism | Bacteriology | Gastritis/pathology | Microbiology and Parasitology | Gastric Mucosa/microbiology | Helicobacter Infections/immunology | Helicobacter Infections/complications | Gastric Mucosa/metabolism | Myeloid Cells/metabolism | Gastric Mucosa/pathology
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 525, Issue 7570, pp. 538 - 542
.... Here we use primary mouse haematopoietic stem and progenitor cells immortalized with the fusion protein MLL-AF9 to generate several single-cell clones that demonstrate resistance, in vitro... 
SELECTIVE-INHIBITION | ACCURATE | CHROMATIN | MECHANISM | MULTIDISCIPLINARY SCIENCES | ACUTE MYELOID-LEUKEMIA | DRUG-RESISTANCE | MUTATIONS | SEQUENCING DATA | CANCER | DISCOVERY | Chromatin - metabolism | Transcription, Genetic - drug effects | Clone Cells - drug effects | Neoplastic Stem Cells - drug effects | Epigenesis, Genetic | Humans | Leukemia, Myeloid, Acute - metabolism | Molecular Targeted Therapy | Neoplastic Stem Cells - metabolism | Leukemia, Myeloid, Acute - drug therapy | Neoplastic Stem Cells - pathology | Gene Expression Regulation, Neoplastic - drug effects | Hematopoietic Stem Cells - drug effects | Benzodiazepines - pharmacology | Leukemia, Myeloid, Acute - pathology | Cells, Cultured | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Hematopoietic Stem Cells - metabolism | Clone Cells - metabolism | beta Catenin - metabolism | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Clone Cells - pathology | Wnt Signaling Pathway - drug effects | Genes, myc - genetics | Hematopoietic Stem Cells - cytology | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Mice | Cell Cycle Proteins | Drug Resistance, Neoplasm - drug effects | Leukemia, Myeloid, Acute - genetics | Proteins | Leukemia | Cloning | Cell cycle | Stem cells | Epigenetics | Apoptosis
Journal Article