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Seminars in Hematology, ISSN 0037-1963, 04/2017, Volume 54, Issue 2, pp. 94 - 97
While early presentation of familial leukemia syndromes is typical, long disease anticipation may mask cases of familial traits in seemingly spontaneous... 
AML | Mutations | DDX41 | MDS | Familial leukemia | GERMLINE DDX41 MUTATION | LEUKEMIA | HEMATOLOGY | TRANSPLANTATION | FAMILY | DEAD-box RNA Helicases - genetics | Leukemia, Myeloid, Acute - pathology | Humans | Germ-Line Mutation | Myelodysplastic Syndromes - genetics | Myelodysplastic Syndromes - pathology | Leukemia, Myeloid, Acute - genetics
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 06/2012, Volume 21, Issue 6, pp. 836 - 847
Stromal responses elicited by early stage neoplastic lesions can promote tumor growth. However, the molecular mechanisms that underlie the early recruitment of... 
COLONY-STIMULATING FACTORS | T-CELL RESPONSES | TUMOR PROGRESSION | P21 RAS | ONCOLOGY | DUCTAL ADENOCARCINOMA | INFLAMMATION | MOUSE | MICE | SUPPRESSOR-CELLS | CANCER | CELL BIOLOGY | Immunohistochemistry | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Pancreatic Neoplasms - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Immunoblotting | Green Fluorescent Proteins - genetics | Pancreatic Ducts - metabolism | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | RNA Interference | HEK293 Cells | CD8-Positive T-Lymphocytes - metabolism | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Green Fluorescent Proteins - metabolism | Pancreatic Ducts - cytology | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Receptors, Chemokine - metabolism | Cells, Cultured | Mice, Transgenic | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Animals | Cell Transformation, Neoplastic | Myeloid Cells - metabolism | Mice | CD11b Antigen - metabolism | Microscopy, Fluorescence | T cells | Tumors | granulocyte-macrophage colony stimulating factor | immunosuppression | Kras | cancer | inflammation | pancreas
Journal Article
WORLD JOURNAL OF GASTROENTEROLOGY, ISSN 1007-9327, 08/2019, Volume 25, Issue 30, pp. 4261 - 4277
Journal Article
Leukemia, ISSN 0887-6924, 01/2018, Volume 32, Issue 1, pp. 203 - 213
BCR-ABL1-targeting tyrosine kinase inhibitors (TKIs) have revolutionized treatment of Philadelphia chromosome-positive (Ph+) hematologic neoplasms.... 
CHRONIC MYELOGENOUS LEUKEMIA | HEMATOPOIETIC-CELLS | COILED-COIL DOMAIN | STEM-CELLS | SIGNALING PATHWAYS | ONCOLOGY | KINASE DOMAIN MUTATIONS | ACUTE LYMPHOBLASTIC-LEUKEMIA | BCR-ABL | HEMATOLOGY | PROTEIN-TYROSINE-PHOSPHATASE | CHRONIC MYELOID-LEUKEMIA | Apoptosis - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Apoptosis - genetics | Hematologic Neoplasms - pathology | Myeloproliferative Disorders - pathology | src Homology Domains - genetics | Myeloproliferative Disorders - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Cell Transformation, Neoplastic - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | HEK293 Cells | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Cell Line | Cell Proliferation - genetics | Mice, Inbred C57BL | Myeloproliferative Disorders - drug therapy | Signal Transduction - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Drug Resistance, Neoplasm - genetics | Fusion Proteins, bcr-abl - genetics | Animals | Signal Transduction - drug effects | Adaptor Proteins, Signal Transducing - genetics | Hematologic Neoplasms - drug therapy | Cell Proliferation - drug effects | Hematologic Neoplasms - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - genetics | Cell Transformation, Neoplastic - pathology | Drug Resistance, Neoplasm - drug effects | Molecular targeted therapy | Care and treatment | Leukemia | Development and progression | Esterases | Genetic aspects | Health aspects | Oncogenes | Cell proliferation | Transcription | Syngeneic grafts | Bone marrow transplantation | Transplantation | Myc protein | Kinases | Neoplasms | Cell activation | Clonal deletion | Bone marrow | Deletion | Protein-tyrosine kinase | Tyrosine | Acute lymphatic leukemia | Myeloid leukemia | Extracellular signal-regulated kinase | MAP kinase | Chronic myeloid leukemia | Lymphatic leukemia | Philadelphia chromosome | Lymphocytes B | Scaffolding | Leukemogenesis
Journal Article
Blood, ISSN 0006-4971, 10/2018, Volume 132, Issue 14, pp. 1535 - 1544
Publisher's Note: There is a Blood Commentary on this article in this issue. Crbn I391V mice degrade known thalidomide derivative targets and recapitulate... 
Myeloid Neoplasia
Journal Article
Leukemia, ISSN 0887-6924, 05/2019, Volume 33, Issue 5, pp. 1184 - 1194
Journal Article
Blood, ISSN 0006-4971, 04/2019, Volume 133, Issue 15, pp. 1677 - 1690
Key Points The absence of Cdk6 ameliorates MPN hallmarks, lessens splenomegaly, and enhances survival of JAK2V617F mice. CDK6 facilitates MPN by enhancing... 
Myeloid Neoplasia
Journal Article
Virchows Archiv, ISSN 0945-6317, 2019, Volume 474, Issue 3, pp. 353 - 363
Immunoglobulin light chain-derived (AL) amyloidosis may occur as a systemic disease usually with dismal prognosis and a localized variant with favorable... 
Amyloid | Congo red | B cell lymphoma | Light chain | PLASMA-CELL | IMMUNOGLOBULIN | FOLLICULAR LYMPHOMA | PATHOLOGY | L265P SOMATIC MUTATION | IMMUNOHISTOCHEMICAL CLASSIFICATION | MONOCLONAL GAMMOPATHY | CLONALITY | MARGINAL ZONE LYMPHOMA | MYD88 L265P | MALT-TYPE | Immunohistochemistry | Prospective Studies | Humans | Middle Aged | Male | Lymphoma, B-Cell - genetics | Plasma Cells - pathology | Waldenstrom Macroglobulinemia - pathology | Immunoglobulin kappa-Chains - immunology | DNA Mutational Analysis | Immunoglobulin lambda-Chains - genetics | Lymphoma, B-Cell - immunology | Myeloid Differentiation Factor 88 - immunology | Aged, 80 and over | Adult | Female | Gene Rearrangement, B-Lymphocyte, Light Chain | B-Lymphocytes - pathology | Immunoglobulin Heavy Chains - immunology | Immunoglobulin Light-chain Amyloidosis - immunology | Immunoglobulin lambda-Chains - immunology | Immunoglobulin Light-chain Amyloidosis - genetics | Myeloid Differentiation Factor 88 - genetics | Waldenstrom Macroglobulinemia - immunology | Immunoglobulin kappa-Chains - genetics | Phenotype | B-Lymphocytes - immunology | Gene Rearrangement, B-Lymphocyte, Heavy Chain | Lymphoma, B-Cell - pathology | Immunoglobulin Light-chain Amyloidosis - pathology | Aged | Biomarkers, Tumor - genetics | Mutation | Immunoglobulin Heavy Chains - genetics | Waldenstrom Macroglobulinemia - genetics | Plasma Cells - immunology | Lymphomas | Amyloidosis | B cells | Histochemistry | Plasma | Immunoglobulins | Goats | Chains | Hybridization | Lymphoma | Patients | Histopathology | Lymphocytes B | Light | Gene rearrangement | Plasma cells
Journal Article