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Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 4, pp. 2080 - 2088
Cross-talk between NK cells and dendritic cells (DCs) is critical for the potent therapeutic response to dsRNA, but the receptors involved remained controversial... 
TOLL-LIKE RECEPTOR-3 | CROSS-TALK | ADAPTIVE IMMUNITY | NATURAL-KILLER-CELLS | ACTIVATION | INNATE RESISTANCE | RECOGNITION | PATHWAY | IMMUNOLOGY | BACTERIAL PEPTIDOGLYCAN | CYTOTOXICITY | Myeloid Cells - cytology | Humans | Interferon-gamma - metabolism | Interferon-Induced Helicase, IFIH1 | Dose-Response Relationship, Drug | Transfection | Dendritic Cells - drug effects | Myeloid Cells - drug effects | Toll-Like Receptor 3 - genetics | DEAD-box RNA Helicases - metabolism | Dendritic Cells - metabolism | Cell Line | DEAD Box Protein 58 | Poly I-C - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Poly A-U - pharmacology | Toll-Like Receptor 3 - metabolism | Mice, Knockout | DEAD-box RNA Helicases - genetics | Killer Cells, Natural - cytology | Animals | Lymphocyte Activation - drug effects | Adaptor Proteins, Signal Transducing - genetics | RNA, Double-Stranded - pharmacology | Myeloid Cells - metabolism | Dendritic Cells - cytology | Killer Cells, Natural - drug effects | Mice | Killer Cells, Natural - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Poly A-U | Interferon-gamma | Toll-Like Receptor 3 | DEAD-box RNA Helicases | Dendritic Cells | Killer Cells, Natural | Lymphocyte Activation | Myeloid Cells | Life Sciences | Adaptor Proteins, Signal Transducing | Immunology | RNA, Double-Stranded | Poly I-C
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e14733
Background: Differentiation of pluripotent stem cells in vitro provides a powerful means to investigate early developmental fates, including hematopoiesis... 
IMMUNOGENICITY | MULTIDISCIPLINARY SCIENCES | EXPANSION | HUMAN FIBROBLASTS | GROWTH-FACTOR | FLT3 LIGAND | APLASTIC-ANEMIA | CULTURES | LINES | Myeloid Cells - cytology | Embryonic Stem Cells - metabolism | Antigens, CD34 - metabolism | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Humans | Myeloid Cells - physiology | Pluripotent Stem Cells - physiology | Cells, Cultured | Cell Size | Hematopoietic Stem Cells - metabolism | Embryonic Stem Cells - physiology | Cell Lineage - physiology | Animals | Cell Differentiation - drug effects | Hematopoietic Stem Cells - cytology | Pluripotent Stem Cells - drug effects | Culture Media - pharmacology | Hematopoietic Stem Cells - physiology | Myeloid Cells - metabolism | Cell Culture Techniques | Culture Media - chemistry | Culture Media, Serum-Free - pharmacology | Induced Pluripotent Stem Cells - cytology | Cell culture | Flow cytometry | Senescence | Laboratories | Oct-4 protein | Differentiation (biology) | Erythrocytes | Embryo cells | Stem cell transplantation | Leukocytes | Kinases | Assaying | Experiments | Blood | Thrombopoietin | Stem cell factor | Red blood cells | Allografts | Immunology | Cord blood | Hematopoiesis | Rodents | Fibroblasts | Bone marrow | Vascular endothelial growth factor | CD34 antigen | Medical research | Food sources | Colonies | Anemia | Fetuses | Gynecology | Runx1 protein | Gene expression | Erythroid cells | Cell differentiation | Embryos | Obstetrics | Hemopoiesis | Endothelium | White blood cells | Pathology | Chemotherapy | Stem cells | Animal protein | Ligands | Pluripotency
Journal Article
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2009, Volume 119, Issue 5, pp. 1109 - 1123
...), but the frequency of resistance increases in advancing stages of disease. Elimination of BCR/ABL-dependent intracellular signals triggers apoptosis, but it is unclear whether this activates additional cell survival and/or death pathways... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 511, Issue 7509, pp. 312 - 318
Journal Article
Cancer cell, ISSN 1535-6108, 2008, Volume 13, Issue 6, pp. 483 - 495
Faithful modeling of mixed-lineage leukemia in murine cells has been difficult to achieve... 
STEMCELL | CELLCYCLE | HEMATOPOIETIC-CELLS | B-CELLS | STEM-CELLS | ONCOLOGY | MLL REARRANGEMENTS | ACUTE LYMPHOBLASTIC-LEUKEMIA | ACUTE MYELOGENOUS LEUKEMIA | THERAPEUTIC TARGET | GENE-EXPRESSION PROFILES | ACUTE MYELOID-LEUKEMIA | FLT3 MUTATIONS | Translocation, Genetic | Antigens, CD34 - analysis | Myeloid-Lymphoid Leukemia Protein - metabolism | Cell Proliferation | Fetal Stem Cells - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Multipotent Stem Cells - metabolism | rac GTP-Binding Proteins - metabolism | Aneuploidy | Gene Expression Profiling | Stem Cell Transplantation | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | rac GTP-Binding Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Culture Techniques | Fetal Blood - immunology | Transduction, Genetic | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Genotype | Cell Lineage | Multipotent Stem Cells - pathology | Phenotype | Environment | Mice, Inbred NOD | Mice | Fetal Stem Cells - pathology | Oncogene Proteins, Fusion - metabolism | Species Specificity | Gene Expression Regulation, Neoplastic | Neoplastic Stem Cells - immunology | Multipotent Stem Cells - immunology | Fetal Stem Cells - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Leukemia, Experimental - pathology | Fetal Blood - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Myeloid-Lymphoid Leukemia Protein - genetics | Oncogene Proteins, Fusion - genetics | Chromosomes, Human, Pair 11 | Cell Transformation, Neoplastic - pathology | Cell Line, Transformed | Leukemia, Experimental - metabolism | Apoptosis | Leukemia, Myeloid, Acute - genetics | Cytogenetics | Models | Growth factors | Analysis | Leukemia | Stem cells
Journal Article