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Blood, ISSN 0006-4971, 04/2019, Volume 133, Issue 15, pp. 1677 - 1690
Key Points The absence of Cdk6 ameliorates MPN hallmarks, lessens splenomegaly, and enhances survival of JAK2V617F mice. CDK6 facilitates MPN by enhancing... 
Myeloid Neoplasia
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 5341 - 14
Journal Article
Blood, ISSN 0006-4971, 09/2014, Volume 124, Issue 23, pp. 3441 - 3449
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 06/2017, Volume 16, Issue 6, pp. 991 - 1001
The receptor tyrosine kinase fms-like tyrosine kinase 3 (FLT3), involved in regulating survival, proliferation, and differentiation of hematopoietic... 
AML | POTENT INHIBITOR | INTENSIVE CHEMOTHERAPY | PTK INHIBITORS | ONCOLOGY | PROMOTES RESISTANCE | RISK MYELODYSPLASTIC SYNDROME | KINASE INHIBITOR | MUTATIONS | SORAFENIB | PHASE-I | fms-Like Tyrosine Kinase 3 - antagonists & inhibitors | Leukemia, Myeloid, Acute - pathology | Humans | Protein Multimerization | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Treatment Outcome | Clinical Trials as Topic | Tandem Repeat Sequences | fms-Like Tyrosine Kinase 3 - chemistry | fms-Like Tyrosine Kinase 3 - metabolism | Leukemia, Myeloid, Acute - mortality | fms-Like Tyrosine Kinase 3 - genetics | Drug Resistance, Neoplasm - genetics | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Leukemia, Myeloid, Acute - drug therapy | Protein Binding | Protein Interaction Domains and Motifs | Protein Kinase Inhibitors - pharmacology | Mutation | Drug Evaluation, Preclinical | Leukemia, Myeloid, Acute - genetics | Cell proliferation | Tyrosine | Drugs | Medical research | Cell survival | Myeloid leukemia | Leukemia | Clinical trials | Kinases | Survival | Patients | Progenitor cells | Proteasome inhibitors | Hemopoiesis | Signaling | Chemotherapy | Inhibitors | Cells (biology) | Protein-tyrosine kinase receptors | Flt3 protein | Acute myeloid leukemia | Protein-tyrosine kinase | Cancer | FLT3 internal tandem duplication | FLT3 point mutation | FLT3 inhibitors | acute myeloid leukemia | resistance
Journal Article
Blood, ISSN 0006-4971, 02/2012, Volume 119, Issue 5, pp. 1123 - 1129
Journal Article
Science, ISSN 0036-8075, 5/2013, Volume 340, Issue 6132, pp. 622 - 626
A number of human cancers harbor somatic point mutations in the genes encoding isocitrate dehydrogenases 1 and 2 (IDH1 and IDH2). These mutations alter... 
Molecules | Enzymes | Dehydrogenases | Blasts | Myeloid leukemia | REPORTS | Stem cells | Bone marrow cells | Cellular differentiation | Hematopoietic stem cells | Mesenchymal stem cells | ISOCITRATE DEHYDROGENASE | ONCOMETABOLITE 2-HYDROXYGLUTARATE | SUFFICIENT | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC PROGENITORS | MUTATION | Cell Proliferation | Humans | Protein Multimerization | Crystallography, X-Ray | Erythropoiesis - drug effects | Isocitrate Dehydrogenase - antagonists & inhibitors | Molecular Targeted Therapy | Hematopoiesis - drug effects | Antineoplastic Agents - metabolism | Leukemia, Myeloid, Acute - enzymology | Phenylurea Compounds - chemistry | Enzyme Inhibitors - chemistry | Leukemia, Myeloid, Acute - drug therapy | Antineoplastic Agents - pharmacology | Phenylurea Compounds - metabolism | Mutant Proteins - antagonists & inhibitors | Catalytic Domain | Enzyme Inhibitors - metabolism | Protein Structure, Secondary | Sulfonamides - chemistry | Leukemia, Myeloid, Acute - pathology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Isocitrate Dehydrogenase - genetics | Mutant Proteins - metabolism | Gene Expression Regulation, Leukemic | Antineoplastic Agents - chemistry | Sulfonamides - pharmacology | Point Mutation | Isocitrate Dehydrogenase - chemistry | Small Molecule Libraries | Mutant Proteins - chemistry | Allosteric Site | Sulfonamides - metabolism | Cell Line, Tumor | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Phenylurea Compounds - pharmacology | Leukemia, Erythroblastic, Acute | Leukemia, Myeloid, Acute - genetics | Leukemia | Cancer cells | Physiological aspects | Research | Oxidoreductases | Cell differentiation | Health aspects | Mutation | Cellular biology | Drug therapy | Cells
Journal Article
Blood, ISSN 0006-4971, 2015, Volume 125, Issue 20, pp. 3144 - 3152
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 05/2013, Volume 368, Issue 19, pp. 1781 - 1790
Journal Article
Blood, ISSN 0006-4971, 04/2012, Volume 119, Issue 14, pp. 3203 - 3210
Journal Article