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Immunology, ISSN 0019-2805, 05/2018, Volume 154, Issue 1, pp. 144 - 155
Summary A recent study indicated that Lectin‐type oxidized LDL receptor‐1 (LOX‐1) was a distinct surface marker for human polymorphisms myeloid‐derived... 
polymorphonuclear myeloid‐derived suppressor cell | lectin‐type oxidized LDL receptor‐1 | prognosis | endoplasmic reticulum stress | hepatocelluar carcinoma patients | polymorphonuclear myeloid-derived suppressor cell | lectin-type oxidized LDL receptor-1 | LYMPHOCYTE RATIO ACTS | PROGNOSTIC-FACTOR | DISEASE | IMMUNOLOGY | Cell Proliferation | Reactive Oxygen Species - metabolism | Coculture Techniques | Humans | Arginase - metabolism | Lewis X Antigen - metabolism | Case-Control Studies | T-Lymphocytes - metabolism | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - immunology | Myeloid-Derived Suppressor Cells - metabolism | Myeloid-Derived Suppressor Cells - pathology | Myeloid-Derived Suppressor Cells - immunology | Signal Transduction | Lymphocyte Activation | Cells, Cultured | Liver Neoplasms - immunology | Fucosyltransferases - metabolism | Scavenger Receptors, Class E - metabolism | Endoplasmic Reticulum Stress | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Interferons - metabolism | T-Lymphocytes - immunology | Carcinoma, Hepatocellular - metabolism | Oxidases | Hepatitis | Liver | Low density lipoproteins | Lectins | Stress (Physiology) | Hepatoma | T cells | Liver cirrhosis | Protein binding | Cell proliferation | Lipoproteins (low density) | Arginase | Hepatocellular carcinoma | Lymphocytes T | Suppressor cells | NAD(P)H oxidase | Liver cancer | Cell growth | Lymphocytes | CYBB protein | Hepatitis B virus | Stresses | Cell survival | Nucleotide sequence | Gene expression | Ribonucleic acid--RNA | Patients | Stress | Cirrhosis | Inhibitors | Liquid oxygen | Medical prognosis | Interferon | Endoplasmic reticulum | Surface markers | Hepatitis B | Original
Journal Article
CANCER RESEARCH, ISSN 0008-5472, 05/2017, Volume 77, Issue 10, pp. 2607 - 2619
Checkpoint inhibitors are relatively inefficacious in head and neck cancers, despite an abundance of genetic alterations and a T-cell-inflamed phenotype. One... 
HEAD | ACTIVATION | ONCOLOGY | IMMUNE-RESPONSES | REGULATORY T-CELLS | RESISTANCE | MECHANISMS | PI3K-GAMMA | EXPRESSION | CARCINOMA | NECK-CANCER | Neoplasms - metabolism | Immunomodulation - drug effects | Myeloid-Derived Suppressor Cells - drug effects | Lymphocyte Activation - immunology | Isoquinolines - pharmacology | Lymphocytes, Tumor-Infiltrating - metabolism | Antineoplastic Agents - pharmacology | Epitopes, T-Lymphocyte - immunology | Myeloid-Derived Suppressor Cells - metabolism | Disease Models, Animal | Myeloid-Derived Suppressor Cells - immunology | Purines - pharmacology | Antibodies, Monoclonal - pharmacology | Lymphocytes, Tumor-Infiltrating - drug effects | Class I Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Neoplasms - drug therapy | Tumor Microenvironment - immunology | Animals | B7-H1 Antigen - antagonists & inhibitors | Lymphocyte Activation - drug effects | Neoplasms - immunology | Survival Analysis | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Neoplasms - pathology | Lymphocytes, Tumor-Infiltrating - immunology | Cell culture | Myeloid cells | Effectiveness | Cell survival | CD8 antigen | Effector cells | Lymphocytes T | Inflammation | Suppressor cells | Recruitment | Immunosuppression | Inhibitors | Immune checkpoint | Lymphocytes | PD-L1 protein | Isoforms | Head and neck | Head & neck cancer | Inhibition | Cancer | Immune system | immunosuppression | MDSC | IPI-145 | PI3K | head and neck cancer
Journal Article
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 06/2016, Volume 76, Issue 11, pp. 3156 - 3165
Journal Article
STEM CELLS, ISSN 1066-5099, 08/2016, Volume 34, Issue 8, pp. 2026 - 2039
Shifting the balance away from tumor‐mediated immune suppression toward tumor immune rejection is the conceptual foundation for a variety of immunotherapy... 
Macrophage migration inhibitory factor | Tumor microenvironment | Immunotherapy | Glioblastoma | MIF | Cancer stem cells | Myeloid‐derived suppressor cells | Tumor immune suppression | Myeloid-derived suppressor cells | GLIOMA | SELF-RENEWAL | RECEPTOR | TUMOR-INITIATING CELLS | BRAIN-TUMORS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | IN-VITRO | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH | HEMATOLOGY | PROMOTES | PROGRESSION | Tumor Microenvironment - drug effects | Cell Survival - drug effects | Neoplastic Stem Cells - secretion | Neoplastic Stem Cells - drug effects | Humans | Mice, Inbred C57BL | Arginase - metabolism | Brain Neoplasms - pathology | Culture Media, Conditioned - pharmacology | Myeloid-Derived Suppressor Cells - drug effects | Macrophage Migration-Inhibitory Factors - secretion | Carcinogenesis - metabolism | Carcinogenesis - pathology | Animals | Brain Neoplasms - immunology | Glioblastoma - immunology | Mice, Nude | Glioblastoma - pathology | Immune Evasion - drug effects | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Female | Myeloid-Derived Suppressor Cells - metabolism | Analysis | Stem cells | T cells | Macrophages | Glioblastoma multiforme | Cancer | Cell proliferation | Populations | Leukocyte migration | Syngeneic grafts | Brain tumors | Stem cell transplantation | Cytotoxicity | Arginase | Lymphocytes T | Suppressor cells | Bearing | Rodents | Mathematical models | Immune system | Proximity | Cell survival | Immune response | Tumor cells | CXCR2 protein | Patients | Survival | Rejection | Depletion | Glioma | Migration inhibitory factor | Antitumor activity | Cell migration
Journal Article