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PloS one, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9258 - e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Hematopoietic system | Mutation | Eosinophils | Index Medicus
Journal Article
Blood, ISSN 1528-0020, 03/2016, Volume 127, Issue 10, pp. 1325 - 1335
Journal Article
The New England journal of medicine, ISSN 1533-4406, 12/2006, Volume 355, Issue 23, pp. 2452 - 2466
Journal Article
Cell cycle (Georgetown, Tex.), ISSN 1551-4005, 10/2014, Volume 6, Issue 5, pp. 550 - 566
Myeloproliferative disorders (MPDs) constitute a group of hematopoietic malignancies that feature enhanced proliferation and survival of one or more myeloid lineage cells... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Thrombocythemia | Myelofibrosis | JAK2 | Polycythemia | JAK2V617F | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Animals | Humans | Myeloproliferative Disorders - metabolism | Signal Transduction - genetics | Mutation | Myeloproliferative Disorders - genetics | Oncogenes | Index Medicus
Journal Article
Blood, ISSN 1528-0020, 01/2009, Volume 113, Issue 12, pp. 2619 - 2628
... in DS-leukemia research. With the increased knowledge of the way in which trisomy 21 affects hematopoiesis and the specific genetic mutations that are found in DS-associated leukemias, we are well on our way toward designing improved strategies for treating both myeloid and lymphoid malignancies in this high-risk population. (Blood. 2009; 113: 2619-2628) 
Life Sciences & Biomedicine | Hematology | Science & Technology | Liver - pathology | Precursor Cell Lymphoblastic Leukemia-Lymphoma - epidemiology | Humans | Hematopoiesis, Extramedullary - genetics | Liver - embryology | Myeloproliferative Disorders - genetics | Incidence | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Cell Transformation, Neoplastic - genetics | Preleukemia - genetics | Down Syndrome - blood | Down Syndrome - complications | Myeloproliferative Disorders - epidemiology | Preleukemia - epidemiology | Leukemia, Megakaryoblastic, Acute - genetics | Neoplasm Proteins - genetics | Myeloproliferative Disorders - congenital | Disease Models, Animal | Preleukemia - etiology | Genetic Predisposition to Disease | Janus Kinases - genetics | Leukemia, Megakaryoblastic, Acute - etiology | Leukemia, Megakaryoblastic, Acute - epidemiology | Myeloproliferative Disorders - drug therapy | Gene Expression Regulation, Leukemic | Preleukemia - drug therapy | Chromosomes, Human, Pair 21 - genetics | Disease Progression | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Preleukemia - congenital | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Down Syndrome - genetics | GATA1 Transcription Factor - genetics | RNA, Neoplasm - genetics | Myeloproliferative Disorders - etiology | Leukemia, Megakaryoblastic, Acute - drug therapy | Mice | MicroRNAs - genetics | Mutation | Precursor Cell Lymphoblastic Leukemia-Lymphoma - etiology | Index Medicus | Abridged Index Medicus | Review
Journal Article