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Journal Article
Biochemical Journal, ISSN 0264-6021, 07/2011, Volume 437, Issue 2, pp. 199 - 213
FGFs (fibroblast growth factors) and their receptors (FGFRs) play essential roles in tightly regulating cell proliferation, survival, migration and... 
Receptor tyrosine kinase | Therapy | Fibroblast growth factor (FGF) | Signalling | Fibroblast growth factor receptor (FGFR) | Cancer | MULTIPLE-MYELOMA | BLADDER-CANCER | fibroblast growth factor receptor (FGFR) | therapy | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE MAMMARY-TUMORS | POTENT ANTITUMOR-ACTIVITY | PROSTATE-CANCER | SQUAMOUS-CELL CARCINOMA | fibroblast growth factor (FGF) | cancer | receptor tyrosine kinase | FGFR4 GLY388ARG POLYMORPHISM | signalling | ACTIVATING MUTATIONS | GENOME-WIDE ASSOCIATION | Receptors, Fibroblast Growth Factor - drug effects | Cell Proliferation | Humans | Receptor Protein-Tyrosine Kinases - physiology | Fibroblast Growth Factors - genetics | Antibodies, Monoclonal - therapeutic use | Male | Prostatic Neoplasms - physiopathology | Lung Neoplasms - physiopathology | Breast Neoplasms - physiopathology | Recombinant Fusion Proteins | Cell Differentiation - genetics | Urinary Bladder Neoplasms - physiopathology | Receptors, Fibroblast Growth Factor - genetics | Female | Neoplasms - physiopathology | Fibroblast Growth Factors - physiology | Multiple Myeloma - physiopathology | Signal Transduction - genetics | Animals | Rhabdomyosarcoma - physiopathology | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Polymorphism, Single Nucleotide | Receptors, Fibroblast Growth Factor - physiology
Journal Article
Science, ISSN 0036-8075, 6/2010, Volume 328, Issue 5986, pp. 1689 - 1693
Elevated leukocyte cell numbers (leukocytosis), and monocytes in particular, promote atherosclerosis; however, how they become increased is poorly understood.... 
Myeloid cells | Leukocytosis | Transgenic animals | HDL lipoproteins | REPORTS | Atherosclerosis | Transgenes | Macrophages | Lesions | Cholesterols | Progenitor cells | CHOLESTEROL | MULTIDISCIPLINARY SCIENCES | MACROPHAGE ABCA1 | LIPID RAFTS | GENE-EXPRESSION | ATHEROSCLEROSIS | MICE | ACCUMULATION | ABCG1 | HIGH-DENSITY-LIPOPROTEIN | APOLIPOPROTEIN-A-I | Cell Proliferation | Lipoproteins - genetics | Lipoproteins, HDL - metabolism | Apolipoprotein A-I - genetics | Leukocytosis - metabolism | ATP-Binding Cassette Transporters - genetics | Atherosclerosis - therapy | Leukocytosis - therapy | Myeloproliferative Disorders - metabolism | Bone Marrow Transplantation | Leukocytosis - physiopathology | Lipoproteins - metabolism | ATP-Binding Cassette Transporters - metabolism | Hematopoietic Stem Cells - physiology | Mice, Inbred DBA | ATP Binding Cassette Transporter 1 | Multipotent Stem Cells - physiology | ATP Binding Cassette Transporter, Subfamily G, Member 1 | Myeloproliferative Disorders - therapy | Signal Transduction | Atherosclerosis - physiopathology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Receptors, Interleukin-3 - metabolism | Apolipoprotein A-I - metabolism | Cholesterol - metabolism | Atherosclerosis - metabolism | Macrophages - metabolism | Phenotype | Animals | Myeloid Progenitor Cells - physiology | Myeloproliferative Disorders - physiopathology | Mice | Hypercholesterolemia - metabolism | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Cell proliferation | High density lipoproteins | Physiological aspects | Development and progression | Research | Health aspects | Hematopoietic stem cells | Adenosine triphosphate
Journal Article
Toxicology Letters, ISSN 0378-4274, 10/2016, Volume 260, pp. 36 - 45
Sorbitol is a polyol used by the food industry as a sweetener. Women are consuming diet and light products containing sorbitol during pregnancy and in the... 
Sorbitol | Myelotoxicity | Food safety | Lactation | Hepatotoxicity | Genotoxicity | HEPATOCYTES | COMET ASSAY | OBESE RATS | PARAMETERS | MUTAGENICITY | SUPPLEMENTATION | TRANSPORT | CALCIUM | ADIPOSITY | TOXICOLOGY | MILK | Hepatic Insufficiency - physiopathology | Milk - chemistry | Maternal Nutritional Physiological Phenomena | Rats, Wistar | Growth Disorders - pathology | Humans | Growth Disorders - etiology | Male | Myeloproliferative Disorders - pathology | Growth Disorders - blood | Liver - physiopathology | Non-Nutritive Sweeteners - adverse effects | Myeloproliferative Disorders - blood | Triglycerides - analysis | Mutagenicity Tests | Non-Nutritive Sweeteners - analysis | Prenatal Exposure Delayed Effects - blood | Sorbitol - administration & dosage | Female | Non-Nutritive Sweeteners - administration & dosage | Biomarkers - blood | Prenatal Exposure Delayed Effects - physiopathology | Prenatal Exposure Delayed Effects - etiology | Sorbitol - analysis | Hepatic Insufficiency - blood | Hep G2 Cells | Pregnancy | Animals | Mutagens - adverse effects | Fructose - analysis | Hepatic Insufficiency - pathology | Myeloproliferative Disorders - etiology | Myeloproliferative Disorders - physiopathology | Sorbitol - adverse effects | Hepatic Insufficiency - etiology | Prenatal Exposure Delayed Effects - pathology | Growth Disorders - physiopathology | Index Medicus | Consumption | Reduction | Alterations | Exposure | Intakes | Milk
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
B-CELL DEVELOPMENT | HUMAN EPITHELIAL-CELLS | STEM-CELLS | IN-VITRO | MOUSE KERATINOCYTES | BIOLOGY | MICE | T-CELL | GROWTH-FACTOR | EXPRESSION | THYMIC STROMAL LYMPHOPOIETIN | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Bone | Hematopoietic system | Mutation | Eosinophils
Journal Article
Annual Review of Pathology: Mechanisms of Disease, ISSN 1553-4006, 5/2016, Volume 11, Issue 1, pp. 101 - 126
Journal Article