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Neuroscience Letters, ISSN 0304-3940, 2006, Volume 396, Issue 1, pp. 67 - 72
The progressive degenerative process associated with sporadic Parkinson's disease (sPD) is characterized by formation of α-synuclein-containing inclusion... 
Neurotropic pathogen | Enteric nervous system | Retrograde axonal transport | Lewy neurites/bodies | Sporadic Parkinson's disease | retrograde axonal transport | HELICOBACTER-PYLORI INFECTION | ENTERIC NERVOUS-SYSTEM | GASTROINTESTINAL DYSFUNCTION | ALZHEIMERS-DISEASE | NEURONS | enteric nervous system | sporadic Parkinson's disease | NEUROSCIENCES | neurotropic pathogen | LEWY BODIES | Myenteric Plexus - pathology | Neurons - pathology | Humans | Middle Aged | Male | Myenteric Plexus - metabolism | Neural Pathways - pathology | Brain - metabolism | Aged, 80 and over | Female | Gastric Mucosa - physiopathology | Neurons - metabolism | Parkinson Disease - metabolism | Disease Transmission, Infectious | Neural Pathways - physiopathology | Submucous Plexus - metabolism | Inclusion Bodies - metabolism | Nerve Net - physiopathology | Prions - metabolism | Parkinson Disease - pathology | Submucous Plexus - physiopathology | Vagus Nerve - metabolism | Brain - physiopathology | Submucous Plexus - pathology | Axonal Transport - physiology | Gastric Mucosa - innervation | Parkinson Disease - physiopathology | Nerve Net - pathology | Protein Folding | Vagus Nerve - physiopathology | Myenteric Plexus - physiopathology | Nerve Net - metabolism | Brain - pathology | Inclusion Bodies - pathology | Neural Pathways - metabolism | Aged | Models, Neurological | alpha-Synuclein - metabolism | Index Medicus
Journal Article
Brain Research, ISSN 0006-8993, 2013, Volume 1584, pp. 59 - 72
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 09/2009, Volume 33, Issue 9, pp. 1401 - 1408
Gastrointestinal stromal tumors (GISTs), KIT or platelet derived growth factor receptor alpha (PDGFRA) signaling driven mesenchymal tumors of the... 
PDGFRA | Chloride channel protein | Sarcoma | DOG1 | KIT | Mutation | Gastrointestinal stromal tumor | PDGFRA MUTATIONS | GASTRIC TUMORS | SURGERY | sarcoma | MARKER | KINASE-C-THETA | MONOCLONAL-ANTIBODY | PATHOLOGY | mutation | chloride channel protein | gastrointestinal stromal tumor | TERM-FOLLOW-UP | EXPRESSION | PKC-THETA | MORPHOLOGY | Immunohistochemistry | Myenteric Plexus - pathology | Predictive Value of Tests | Anoctamin-1 | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Stomach Neoplasms - metabolism | Leiomyoma - metabolism | Myenteric Plexus - metabolism | Neoplasm Proteins - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Esophageal Neoplasms - pathology | Leiomyoma - pathology | Chloride Channels | Gastrointestinal Stromal Tumors - diagnosis | Esophageal Neoplasms - metabolism | Mesenchymoma - diagnosis | Biomarkers, Tumor - metabolism | Membrane Proteins - metabolism | Intestinal Neoplasms - diagnosis | Colorectal Neoplasms - metabolism | Peritoneal Neoplasms - pathology | Mesenchymoma - metabolism | Diagnosis, Differential | Stomach Neoplasms - diagnosis | Intestinal Neoplasms - metabolism | Sarcoma, Synovial - metabolism | Peritoneal Neoplasms - metabolism | Gastrointestinal Stromal Tumors - metabolism | Mesenchymoma - secondary | Colorectal Neoplasms - pathology | Sarcoma, Synovial - pathology | Index Medicus
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2015, Volume 82, pp. 99 - 113
Abstract Lewy pathology affects the gastrointestinal tract in Parkinson's disease (PD) and recent reports suggest a link between the disorder and gut... 
Neurology | Neuroprotection | Immunophenotype | Intestine | GPER1 | 17β-estradiol | Inflammation | Myenteric plexus | MPTP | Ileum | Innate immune response | CYCLASE-ACTIVATING POLYPEPTIDE | WILD-TYPE | 17 beta-estradiol | ENTERIC NERVOUS-SYSTEM | NEURODEGENERATION | COLONIC BIOPSIES | ALPHA-SYNUCLEIN | DOPAMINERGIC-NEURONS | NEUROSCIENCES | ESTROGEN | MICE | Myenteric Plexus - pathology | Neuroprotective Agents - therapeutic use | Immunomodulation - drug effects | Myenteric Plexus - immunology | Receptors, Estrogen - metabolism | Receptors, G-Protein-Coupled - metabolism | Dopaminergic Neurons - pathology | Immunomodulation - genetics | NF-kappa B - metabolism | Myenteric Plexus - metabolism | Neuroprotection - drug effects | Quinolines - pharmacology | Parkinsonian Disorders - metabolism | Neuroprotective Agents - pharmacology | Parkinsonian Disorders - drug therapy | Dopaminergic Neurons - metabolism | Dopaminergic Neurons - drug effects | Estradiol - pharmacology | Dopaminergic Neurons - immunology | Receptors, Estrogen - genetics | Estradiol - therapeutic use | Myenteric Plexus - drug effects | Neuroprotection - genetics | Animals | Parkinsonian Disorders - pathology | Mice | Receptors, G-Protein-Coupled - genetics | Parkinsonian Disorders - immunology | Benzodioxoles - pharmacology | Nitric Oxide Synthase Type II - metabolism | Analysis | Gastrointestinal system | Estrogen | Neurosciences | Immune response | G proteins | Macrophages | Estradiol | Index Medicus
Journal Article
Journal Article
Aging Cell, ISSN 1474-9718, 12/2012, Volume 11, Issue 6, pp. 996 - 1004
In senescent cells, a DNA damage response drives not only irreversible loss of replicative capacity but also production and secretion of reactive oxygen... 
senescence | aging | brain | inflammation | neurons | oxidative stress | Aging | Brain | Oxidative stress | Senescence | Inflammation | Neurons | SECRETORY PHENOTYPE | HUMAN-CELLS | TRIGGERS SENESCENCE | AGING MICE | DIETARY RESTRICTION | ONCOGENE-INDUCED SENESCENCE | CELLULAR SENESCENCE | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | CALORIC RESTRICTION | MITOCHONDRIAL DYSFUNCTION | Myenteric Plexus - pathology | Reactive Oxygen Species - metabolism | Caloric Restriction | Mitosis | Cell Count | Humans | Myenteric Plexus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - deficiency | RNA - genetics | Telomerase - deficiency | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Telomerase - genetics | Aging - genetics | Gene Expression Regulation, Developmental | beta-Galactosidase - metabolism | DNA Damage - genetics | Telomere - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | Telomere - genetics | Cellular Senescence - genetics | Purkinje Cells - metabolism | Interleukin-6 - genetics | Signal Transduction | Mice, Inbred C57BL | p38 Mitogen-Activated Protein Kinases - genetics | Aging - pathology | Phenotype | Telomere - pathology | Animals | Mice | beta-Galactosidase - genetics | Purkinje Cells - pathology | Aging - metabolism | Immunohistochemistry | Peptides | Developmental biology | DNA damage | DNA repair | Interleukins | DNA | Genetic aspects | Universities and colleges | Chromosomes | Telomerase | Genotype & phenotype | Index Medicus | Original
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY, ISSN 0193-1857, 04/2017, Volume 312, Issue 4, pp. G374 - G389
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 11/2013, Volume 354, Issue 2, pp. 381 - 394
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 10/2017, Volume 174, Issue 20, pp. 3623 - 3639
Background and Purpose Gut microbiota is essential for the development of the gastrointestinal system, including the enteric nervous system (ENS).... 
SMOOTH-MUSCLE | PROTEIN | CONCISE GUIDE | COLONIC DYSMOTILITY | ENTERIC NERVOUS-SYSTEM | PHARMACOLOGY | RECEPTOR | PHARMACOLOGY & PHARMACY | MYENTERIC PLEXUS | GLIAL-CELLS | BRAIN | Ileum - innervation | Myenteric Plexus - pathology | Ileum - pathology | Dysbiosis - chemically induced | Stomach - pathology | Mice, Inbred C57BL | Gastrointestinal Microbiome | Myenteric Plexus - drug effects | Gastrointestinal Motility - drug effects | Male | Dysbiosis - physiopathology | Cecum - drug effects | Cecum - pathology | Muscle, Smooth - drug effects | Anti-Bacterial Agents | Ileum - drug effects | Animals | Muscle Contraction - drug effects | Muscle, Smooth - physiology | Synaptic Transmission - drug effects | Ileum - physiology | Dysbiosis - pathology | Stomach - drug effects | Immunohistochemistry | Microbiota (Symbiotic organisms) | Antibiotics | Analysis | Gastrointestinal diseases | Gastrointestinal system | Mice | Liquid chromatography | Mass spectrometry | Deoxycholic acid | Dysbacteriosis | Digestive system | Homeostasis | Mass spectroscopy | Intestinal motility | Muscle contraction | Neuronal-glial interactions | Enteric nervous system | Developmental stages | Intestine | TLR2 protein | Rodents | Toll-like receptors | Myenteric plexus | Digestive tract | Neurotransmission | Bile | Structure-function relationships | Index Medicus | Research Papers | Research Paper
Journal Article
Nature Medicine, ISSN 1078-8956, 04/2012, Volume 18, Issue 4, pp. 600 - 604
Inflammatory bowel diseases (IBDs) are chronic relapsing and remitting conditions associated with long-term gut dysfunction resulting from alterations to the... 
MEDICINE, RESEARCH & EXPERIMENTAL | STIMULATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNERVATION | RELEASE | CELL BIOLOGY | RECOVERY | INHIBITION | P2X RECEPTORS | MYENTERIC NEURONS | GLIA | ATP | MOTILITY | Myenteric Plexus - pathology | Calcium - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Gastrointestinal Motility - drug effects | Colitis - pathology | Male | Glial Fibrillary Acidic Protein - metabolism | Cytoskeletal Proteins - deficiency | Adenosine Triphosphate - pharmacology | Dose-Response Relationship, Drug | Caspases - metabolism | Time Factors | Estrenes - pharmacology | Pyrrolidinones - pharmacology | Cytoskeletal Proteins - metabolism | Neurons - metabolism | Cell Death - drug effects | Dinitrofluorobenzene - analogs & derivatives | Neurons - drug effects | Disease Models, Animal | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Gastrointestinal Motility - genetics | Phosphodiesterase Inhibitors - pharmacology | Purinergic P2X Receptor Agonists - pharmacology | Adenosine Triphosphate - analogs & derivatives | Connexins - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Apoptosis Regulatory Proteins | Dinitrofluorobenzene - pharmacology | Analysis of Variance | Cell Death - physiology | Signal Transduction - drug effects | CARD Signaling Adaptor Proteins | Nitric Oxide Synthase Type I - metabolism | Receptors, Purinergic P2X7 - metabolism | Mice | Neural receptors | Neurons | Physiological aspects | Development and progression | Genetic aspects | Research | Colitis | Inflammatory bowel disease | Nervous system | Gene expression | Apoptosis | Index Medicus
Journal Article