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Circulation research, ISSN 1524-4571, 2013, Volume 112, Issue 4, pp. 675 - 688
RATIONALE:Matrix metalloproteinase (MMP)-28 regulates the inflammatory and extracellular matrix responses in cardiac aging, but the roles of MMP-28 after myocardial infarction (MI... 
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | LYSYL OXIDASE | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | VENTRICULAR-FUNCTION | HEART-FAILURE | CORONARY MICROEMBOLIZATION | REPAIR | MATRIX-METALLOPROTEINASE-9 | PERIPHERAL VASCULAR DISEASE | EPILYSIN MMP-28 | TNF-ALPHA | TARGETED DELETION | HEMATOLOGY | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis
Journal Article
JAMA : the journal of the American Medical Association, ISSN 0098-7484, 04/2016, Volume 315, Issue 15, pp. 1591 - 1599
IMPORTANCE: p38 Mitogen-activated protein kinase (MAPK)–stimulated inflammation is implicated in atherogenesis, plaque destabilization, and maladaptive processes in myocardial infarction (MI... 
APOPTOSIS | MEDICINE, GENERAL & INTERNAL | ACTIVATED PROTEIN-KINASE | INHIBITION | EVENTS | PATHWAY | HEART-FAILURE | MAPK | ACUTE CORONARY SYNDROME | Recurrence | Myocardial Infarction - mortality | Humans | Middle Aged | Male | Secondary Prevention | Cyclopropanes - adverse effects | Protein Kinase Inhibitors - adverse effects | Cyclopropanes - therapeutic use | Pyridines - adverse effects | Treatment Failure | Female | C-Reactive Protein - analysis | Myocardial Infarction - enzymology | Pyridines - therapeutic use | Double-Blind Method | Drug Administration Schedule | Myocardial Ischemia - prevention & control | Hospitalization | Myocardial Revascularization | Algorithms | Myocardial Infarction - drug therapy | Protein Kinase Inhibitors - therapeutic use | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Aged | Myocardial Ischemia - surgery | Clinical trials | Care and treatment | Usage | Dosage and administration | Extracellular signal-regulated kinases | Heart attack | Patient safety | Inhibitor drugs | Heart attacks | Clinical outcomes | Pyridines/adverse effects/therapeutic use | Cyclopropanes/adverse effects/therapeutic use | C-Reactive Protein/analysis | Klinisk medicin | Clinical Medicine | p38 Mitogen-Activated Protein Kinases/antagonists & inhibitors | Myocardial Ischemia/prevention & control/surgery | Protein Kinase Inhibitors/adverse effects/therapeutic use | Myocardial Infarction/drug therapy/enzymology/mortality
Journal Article
Circulation research, ISSN 1524-4571, 2013, Volume 113, Issue 8, pp. 1004 - 1012
.... After myocardial infarction, phagocytes are recruited to the heart and promote clearance of dying cardiomyocytes... 
Myocardial infarction | Efferocytosis | Inflammation | Macrophages | Phagocytosis | phagocytosis | CLEARANCE | CELL ACCUMULATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHATIDYLSERINE RECEPTOR | efferocytosis | myocardial infarction | HEART-FAILURE | PLAQUE NECROSIS | ACCELERATES ATHEROSCLEROSIS | MEDIATED PHAGOCYTOSIS | MICE LACKING | macrophages | inflammation | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | MER | HEMATOLOGY | Inflammation - pathology | Myocardial Infarction - genetics | CD36 Antigens - deficiency | CD36 Antigens - genetics | Ventricular Function, Left | Myocytes, Cardiac - immunology | Coculture Techniques | Male | Wound Healing | Myocardial Infarction - immunology | Recovery of Function | Myocytes, Cardiac - enzymology | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | c-Mer Tyrosine Kinase | Bone Marrow Transplantation | Antigens, Ly - metabolism | Female | Myocardial Infarction - physiopathology | Macrophages - immunology | Disease Models, Animal | Myocardial Infarction - enzymology | Myocardial Contraction | Proto-Oncogene Proteins - metabolism | Receptor Protein-Tyrosine Kinases - deficiency | Macrophages - pathology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Proto-Oncogene Proteins - genetics | Inflammation - immunology | Proto-Oncogene Proteins - deficiency | Receptor Protein-Tyrosine Kinases - metabolism | Transplantation Chimera | Macrophages - enzymology | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Receptor Protein-Tyrosine Kinases - genetics | Inflammation - genetics | Mice | Inflammation - enzymology | Apoptosis
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2012, Volume 59, Issue 2, pp. 153 - 163
Journal Article
Current pharmaceutical design, ISSN 1381-6128, 01/2007, Volume 13, Issue 3, pp. 333 - 346
The matrix metalloproteinase family of enzymes has been a pharmaceutical target for over 20 years. In that time, many drugs have been developed but none have... 
Cardiovascular disease | Inflammation | Topical | Acute therapy | Remodeling | SMOKE-INDUCED EMPHYSEMA | cardiovascular disease | remodeling | topical | ABDOMINAL AORTIC-ANEURYSMS | HEART-FAILURE | BLOOD-BRAIN-BARRIER | RESPIRATORY-DISTRESS-SYNDROME | MUSCLE-CELL-MIGRATION | OBSTRUCTIVE PULMONARY-DISEASE | TISSUE-PLASMINOGEN-ACTIVATOR | inflammation | acute therapy | MACROPHAGE ELASTASE MMP-12 | PHARMACOLOGY & PHARMACY | PREVENTS CARDIAC RUPTURE | Hypertrophy, Left Ventricular - enzymology | Humans | Eye Diseases - enzymology | Protease Inhibitors - pharmacology | Atherosclerosis - enzymology | Drug Design | Hypertrophy, Left Ventricular - drug therapy | Protease Inhibitors - therapeutic use | Vascular Diseases - drug therapy | Extracellular Matrix Proteins - metabolism | Myocardial Infarction - enzymology | Atherosclerosis - drug therapy | Heart Rupture, Post-Infarction - enzymology | Neoplasms - enzymology | Skin Diseases - drug therapy | Stroke - drug therapy | Respiratory Distress Syndrome, Adult - drug therapy | Vascular Diseases - enzymology | Arthritis - enzymology | Neoplasms - drug therapy | Pulmonary Disease, Chronic Obstructive - enzymology | Stroke - enzymology | Animals | Myocardial Infarction - drug therapy | Eye Diseases - drug therapy | Arthritis - drug therapy | Matrix Metalloproteinase Inhibitors | Matrix Metalloproteinases - metabolism | Heart Rupture, Post-Infarction - drug therapy | Respiratory Distress Syndrome, Adult - enzymology | Skin Diseases - enzymology | Pulmonary Disease, Chronic Obstructive - drug therapy
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 91, pp. 134 - 140
Journal Article
Cardiovascular pathology, ISSN 1054-8807, 2016, Volume 25, Issue 1, pp. 40 - 46
.... Materials and methods Thrombectomy materials ( N = 120) obtained from patients with acute myocardial infarction were histologically classified in three groups based on thrombus age: fresh (< 1 day), lytic (1–5 days), or organized (> 5 days) thrombi... 
Pathology | Immunohistochemistry | Granulocytes | Coronary artery | Atherosclerosis | Inflammation | Matrix metalloproteinase | Acute myocardial infarction | Thrombosis | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | INSTABILITY | COLLAGENASE | DEATH | PATHOLOGY | HUMAN-NEUTROPHILS | ATHEROSCLEROTIC PLAQUE RUPTURE | TISSUE INHIBITOR | INTERVENTION | PROGRESSION | Myocardial Infarction - genetics | Coronary Artery Disease - surgery | Matrix Metalloproteinases - genetics | Coronary Thrombosis - genetics | Humans | Coronary Artery Disease - enzymology | Time Factors | Myocardial Infarction - pathology | Coronary Artery Disease - pathology | Granulocytes - pathology | Myocardial Infarction - enzymology | Myocardial Infarction - surgery | Coronary Thrombosis - surgery | Coronary Vessels - pathology | Coronary Vessels - enzymology | Coronary Thrombosis - enzymology | Coronary Thrombosis - pathology | Plaque, Atherosclerotic | Reverse Transcriptase Polymerase Chain Reaction | Tissue Inhibitor of Metalloproteinases - metabolism | Granulocytes - enzymology | Gene Expression Regulation, Enzymologic | Thrombectomy | Biopsy | Coronary Artery Disease - genetics | Coronary Vessels - surgery | Matrix Metalloproteinases - metabolism | Heart attack | Blood clot
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2016, Volume 68, Issue 22, pp. 2454 - 2464
Journal Article
Endocrinology, ISSN 0013-7227, 01/2016, Volume 157, Issue 1, pp. 346 - 357
Journal Article