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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2012, Volume 303, Issue 5, pp. 605 - 618
.... Little is known about the role of apelin in the homing of vascular progenitor cells (PCs) and cardiac functional recovery postmyocardial infarction... 
Stromal cell-derived factor-1α/C-X-C chemokine receptor-4 | Vascular progenitor cell | Myocardial repair | c-kit | Jagged1/notch3 | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | c-Kit | ANGIOGENESIS | HEART-FAILURE | myocardial repair | vascular progenitor cell | APJ RECEPTOR | PROTEIN-COUPLED RECEPTOR | EMBRYONIC STEM-CELLS | PERIPHERAL VASCULAR DISEASE | LIGAND | stromal cell-derived factor-1 alpha/C-X-C chemokine receptor-4 | jagged1/notch3 | TOPCARE-AMI | ISCHEMIC-STROKE | PARACRINE | Apoptosis - drug effects | Receptors, Notch - metabolism | Cardiomegaly - pathology | Vascular Endothelial Growth Factor A - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Time Factors | Serrate-Jagged Proteins | Antigens, Ly - metabolism | Myocardial Infarction - physiopathology | Proto-Oncogene Proteins c-akt - metabolism | Apelin | Disease Models, Animal | Jagged-1 Protein | Biomarkers - metabolism | Ventricular Function, Left - drug effects | Cardiomegaly - physiopathology | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Cell Movement - drug effects | Myocytes, Cardiac - pathology | Regeneration - drug effects | Cardiomegaly - prevention & control | Chemokine CXCL12 - metabolism | Myocytes, Cardiac - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Fibrosis | Myocytes, Cardiac - metabolism | Stem Cells - pathology | Mice | Myocardial Infarction - genetics | Neovascularization, Physiologic - drug effects | Glycoproteins - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Stem Cells - metabolism | Recovery of Function | Intercellular Signaling Peptides and Proteins - metabolism | Myocardial Infarction - pathology | Membrane Proteins - metabolism | Nitric Oxide Synthase Type III - metabolism | Calcium-Binding Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Adipokines | AC133 Antigen | Animals | Myocardial Infarction - drug therapy | Stem Cells - drug effects | Receptor, Notch3 | Physiological aspects | Care and treatment | Health aspects | Ligands (Biochemistry) | Heart attack | stromal cell-derived factor-1α | notch3 | Integrative Cardiovascular Physiology and Pathophysiology | jagged1 | C-X-C chemokine receptor-4
Journal Article
Circulation, ISSN 0009-7322, 01/2013, Volume 127, Issue 2, pp. 213 - 223
...(+) cardiac stem cells (CSCs) in vivo and in vitro, we hypothesized that combining human (h) MSCs with c-kit(+) hCSCs produces greater infarct size reduction compared with either cell administered alone after myocardial infarction... 
heart failure | stem cells | mesenchymal stem cells | myocardial infarction | CHRONIC ISCHEMIC CARDIOMYOPATHY | CARDIAC & CARDIOVASCULAR SYSTEMS | MULTIPOTENT | RANDOMIZED PHASE-1 TRIAL | REGENERATION | RELAXATION | INTRAMYOCARDIAL INJECTION | HEART | THERAPY | CONSEQUENCES | PERIPHERAL VASCULAR DISEASE | Humans | Heart Failure - physiopathology | Transplantation, Heterologous | Proto-Oncogene Proteins c-kit - metabolism | Heart Failure - therapy | Myocardial Infarction - therapy | Mesenchymal Stromal Cells - cytology | Myocardial Infarction - pathology | Systole - physiology | Recovery of Function - physiology | Myocardial Infarction - physiopathology | Ventricular Function, Left - physiology | Disease Models, Animal | Myocytes, Cardiac - cytology | Bone Marrow Cells - cytology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Treatment Outcome | Hematopoietic Stem Cells - metabolism | Heart Failure - pathology | Diastole - physiology | Mesenchymal Stem Cell Transplantation - methods | Animals | Hematopoietic Stem Cells - cytology | Hematopoietic Stem Cell Transplantation - methods | Sus scrofa | Bone Marrow Cells - metabolism | Usage | Care and treatment | Patient outcomes | Heart cells | Cellular therapy | Stem cells | Transplantation | Research | Heart attack | Methods
Journal Article
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2014, Volume 70, pp. 74 - 82
.... The current review manuscript discusses the dynamic phenotypic and functional alterations of cardiac fibroblasts following myocardial infarction... 
Cardiovascular | Myocardial infarction | Extracellular matrix | Inflammation | Cardiac remodeling | Transforming growth factor (TGF)-β | Fibroblast | SMOOTH MUSCLE ACTIN | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | MATRIX-METALLOPROTEINASE | MATRICELLULAR PROTEINS | CELL BIOLOGY | CARDIAC FIBROBLASTS | MARROW-DERIVED CELLS | MOUSE MODEL | COLLAGEN DEGRADATION | GROWTH FACTOR-2 | MYOFIBROBLAST DIFFERENTIATION | Transforming growth factor (TGF)-beta | Inflammation - pathology | Cell Proliferation | Cytokines - metabolism | Signal Transduction | Extracellular Matrix Proteins - genetics | Humans | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Extracellular Matrix - metabolism | Myocardium - pathology | Myocardial Infarction - metabolism | Extracellular Matrix - chemistry | Intercellular Signaling Peptides and Proteins - metabolism | Inflammation - metabolism | Animals | Myocardial Infarction - pathology | Myocardium - metabolism | Fibroblasts - cytology | Wound Healing - physiology | Cytokines - genetics | Extracellular Matrix Proteins - metabolism | Cell Movement | Fibroblasts - metabolism | Proteins | Proteases | Heart attack | Medical colleges | Interleukins | Protein biosynthesis | Transforming growth factors | Cardiology | cytokine | fibroblast | inflammation | myocardial infarction | cardiac remodeling | extracellular matrix
Journal Article
Circulation research, ISSN 1524-4571, 2009, Volume 104, Issue 2, pp. e9 - e18
Persistent inflammatory response has adverse effects on left ventricular (LV) function and remodeling following acute myocardial infarction... 
Myocardial infarction | Inflammation | Cardiac remodeling | Cytokines | IL-10 | CARDIAC-FUNCTION | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | myocardial infarction | INTERLEUKIN-10 | FACTOR-ALPHA | MATRIX METALLOPROTEINASES | TUMOR-NECROSIS-FACTOR | inflammation | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | cytokines | DYSFUNCTION | HEMATOLOGY | EXPRESSION | ELAV-Like Protein 1 | NIH 3T3 Cells | RNA-Binding Proteins - genetics | Phosphorylation | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Anti-Inflammatory Agents - metabolism | ELAV Proteins | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | RNA Interference | Time Factors | Matrix Metalloproteinase 9 - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Inflammation Mediators - metabolism | Interleukin-10 - administration & dosage | Antigens, Surface - metabolism | Interleukin-10 - metabolism | Anti-Inflammatory Agents - administration & dosage | Myocardial Infarction - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Capillaries - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Recombinant Proteins - metabolism | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Antigens, Surface - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Animals | Arterioles - metabolism | Fibrosis | Inflammation - prevention & control | Mice | Ventricular Remodeling - drug effects | Neovascularization, Physiologic | RNA-Binding Proteins - metabolism | Apoptosis | Inflammation - physiopathology | RNA, Small Interfering - metabolism
Journal Article
JACC: Cardiovascular Imaging, ISSN 1936-878X, 09/2018, Volume 11, Issue 9, pp. 1248 - 1256
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 91, pp. 114 - 122
Journal Article