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Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7527, pp. 431 - 435
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2005, Volume 11, Issue 10, pp. 1096 - 1103
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 04/2011, Volume 79, Issue 8, pp. 853 - 860
Journal Article
Nature, ISSN 0028-0836, 01/2008, Volume 451, Issue 7178, pp. 578 - 582
Understanding cellular response to environmental stress has broad implications for human disease. AMP-activated protein kinase (AMPK) orchestrates the... 
TRANSLOCATION | MULTIDISCIPLINARY SCIENCES | DISEASE SEVERITY | FACTOR MIF GENE | INJURY | CARDIAC DYSFUNCTION | CYTOKINE | REGULATORY ROLE | POLYMORPHISM | GLUCOSE-UPTAKE | BINDING | Myocardial Ischemia - genetics | Myocardial Ischemia - metabolism | Humans | Multienzyme Complexes - metabolism | Macrophage Migration-Inhibitory Factors - metabolism | Macrophage Migration-Inhibitory Factors - secretion | Promoter Regions, Genetic - genetics | AMP-Activated Protein Kinases | Hypoxia - metabolism | Macrophage Migration-Inhibitory Factors - deficiency | Macrophage Migration-Inhibitory Factors - genetics | Myocardium - metabolism | Histocompatibility Antigens Class II - metabolism | Protein-Serine-Threonine Kinases - metabolism | Antigens, Differentiation, B-Lymphocyte - genetics | Genetic Predisposition to Disease | Hypoxia - enzymology | Signal Transduction | Rats | Genotype | Myocardial Ischemia - enzymology | Myocardial Reperfusion Injury - physiopathology | Hypoxia - genetics | Myocardium - enzymology | Animals | Coronary Artery Disease - genetics | Glucose - metabolism | Polymorphism, Genetic - genetics | Mice | Antigens, Differentiation, B-Lymphocyte - metabolism | Enzyme Activation | Histocompatibility Antigens Class II - genetics | Myocardial Reperfusion Injury - prevention & control | Control | Cytokines | Myocardial ischemia | Cyclic adenylic acid | Research | Macrophages | Health aspects | Protein kinases | Risk factors | Cell migration | Cellular biology | Cardiovascular disease | Hormones | Kinases | Stress | Cell adhesion & migration | Apoptosis | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 2013, Volume 3, Issue 1, pp. 2767 - 2767
Myocardial ischemia, while causing cardiomyocyte injury, can activate innate protective processes, enhancing myocardial tolerance to ischemia. Such processes... 
BETA-KLOTHO | SURVIVAL | ENDOTHELIAL PROGENITOR CELLS | ARTERIAL ELASTIC LAMINAE | PPAR-ALPHA | ACTIVATION | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | BONE-MARROW | C-KIT | AKT | Up-Regulation | Liver - pathology | Phosphorylation | Cardiotonic Agents - metabolism | Adipose Tissue - physiopathology | Caspase 3 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Glucuronidase | Liver - physiopathology | Phosphatidylinositol 3-Kinases | Myocardial Reperfusion Injury - enzymology | Myocytes, Cardiac - enzymology | Adipose Tissue - metabolism | Fibroblast Growth Factors - metabolism | Myocardial Reperfusion Injury - pathology | Endocrine System - metabolism | Myocardial Ischemia - physiopathology | bcl-Associated Death Protein - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Function Tests | Heart Ventricles - pathology | Signal Transduction | Liver - metabolism | Adipose Tissue - pathology | Gene Silencing | Myocardial Ischemia - prevention & control | Myocardial Ischemia - enzymology | Myocardial Reperfusion Injury - physiopathology | Endocrine System - pathology | Myocardial Ischemia - pathology | Myocytes, Cardiac - pathology | Animals | Heart Ventricles - physiopathology | Protein Binding | Heart Ventricles - metabolism | Mice | Hemodynamics | RNA, Small Interfering - metabolism | Myocardial infarction | AKT1 protein | Adipose tissue | Liver | Caspase | Myocardial ischemia | Cardiomyocytes | Adipocytes | Caspase-3 | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Reperfusion | Ischemia | Cell death | Klotho protein | Rodents | Myocardium | Fibroblast growth factor receptor 1 | Index Medicus
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 12/2017, Volume 1863, Issue 12, pp. 3265 - 3276
Journal Article