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Journal Article
Circulation (New York, N.Y.), ISSN 0009-7322, 03/2012, Volume 125, Issue 9, pp. 1134 - 1146
.... Methods and Results-Rheb is inactivated during cardiomyocyte (CM) glucose deprivation (GD) in vitro, and during acute myocardial ischemia in vivo... 
apoptosis | signal transduction | obesity | ischemia | MAMMALIAN TARGET | RAPAMYCIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | MTOR | SKELETAL-MUSCLE | INFARCTION | IMPACT | PATHWAY | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Myocardial Ischemia - metabolism | TOR Serine-Threonine Kinases - metabolism | Metabolic Syndrome - metabolism | Ras Homolog Enriched in Brain Protein | Autophagy - physiology | TOR Serine-Threonine Kinases - genetics | Dietary Fats - pharmacology | Metabolic Syndrome - physiopathology | Apoptosis Regulatory Proteins - genetics | Myocardial Ischemia - physiopathology | Neuropeptides - genetics | Energy Metabolism - physiology | Cell Survival - physiology | Beclin-1 | Myocytes, Cardiac - cytology | Cells, Cultured | Rats | Neuropeptides - metabolism | Obesity - physiopathology | Glucose - pharmacology | Monomeric GTP-Binding Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Myocardial Ischemia - pathology | Obesity - metabolism | Obesity - pathology | Animals | Myocytes, Cardiac - drug effects | Monomeric GTP-Binding Proteins - metabolism | Myocytes, Cardiac - physiology | Signal Transduction - physiology | Mice | Metabolic Syndrome - pathology | Endoplasmic Reticulum Stress - physiology | Physiological aspects | Autophagy (Cytology) | Obesity | Myocardial ischemia | Research | Metabolic syndrome X | myocardial ischemia | autophagy | Rheb | mTORC1
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 3, p. e0193967
Objectives A novel paradigm of diastolic left ventricular (LV) dysfunction proposes involvement of the cardiac microvasculature. Vitamin K dependent matrix Gla... 
POWERFUL PREDICTOR | GENERAL-POPULATION | MENAQUINONE-7 SUPPLEMENTATION | ARTERIAL STIFFNESS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | K-DEPENDENT PROTECTION | VITAMIN-K | PRESERVED EJECTION FRACTION | BONE MORPHOGENETIC PROTEIN-4 | FILLING PRESSURE | Multivariate Analysis | Myocardial Ischemia - metabolism | Cardiomyopathy, Dilated - pathology | Cardiomyopathy, Dilated - surgery | Humans | Middle Aged | Male | Young Adult | Netherlands | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Aged, 80 and over | Electrocardiography | Myocardial Ischemia - diagnostic imaging | Adult | Female | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Ventricular Dysfunction, Left - epidemiology | Heart Transplantation | Myocardium - pathology | Cardiomyopathy, Dilated - metabolism | Switzerland | Ventricular Dysfunction, Left - diagnostic imaging | Heart - diagnostic imaging | Myocardial Ischemia - pathology | Ventricular Dysfunction, Left - metabolism | Cardiomyopathy, Dilated - diagnostic imaging | Aged | Myocardial Ischemia - surgery | Cohort Studies | Physiological aspects | Development and progression | Calcium-binding proteins | Heart enlargement | Health aspects | Heart muscle | Health care | Heart | Nephrology | Cardiomyopathy | Antibodies | Interstitial cells | Epidemiology | Proteins | Ischemia | Measurement methods | Population | Blood pressure | Capillaries | Cardiology | Heart diseases | Enzyme-linked immunosorbent assay | Vitamin K | Hypertension | Research & development--R&D | Hospitals | Dilated cardiomyopathy | Calcification | Myocardium | Microvasculature | Ventricle | Integrity | Research & development | R&D
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2012, Volume 125, Issue 25, pp. 3170 - 3181
Journal Article
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-682X, 08/2017, Volume 74, Issue 15, pp. 2795 - 2813
...), a non-selective channel in the inner mitochondrial membrane, is involved in the pathogenesis of cardiac ischemia-reperfusion and can be targeted to attenuate reperfusion-induced damage to the myocardium... 
Oxidative stress | Cyclophilin D | Ischemia–reperfusion injury | Cardioprotection | Mitochondrial permeability transition pores | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADP/ATP CARRIER | PHOSPHATE CARRIER | CELL-DEATH | CELL BIOLOGY | Ischemia-reperfusion injury | CYCLOSPORINE-A | MYOCARDIAL INFARCT SIZE | ATP SYNTHASE | NITRIC-OXIDE | CA2+ UPTAKE | ADENINE-NUCLEOTIDE TRANSLOCASE | Myocardial Ischemia - metabolism | Mitochondrial Membrane Transport Proteins - metabolism | Mitochondrial Membrane Transport Proteins - antagonists & inhibitors | Calcium - metabolism | Protein Interaction Maps - drug effects | Humans | Cyclophilins - metabolism | Cardiotonic Agents - pharmacology | Molecular Targeted Therapy | Drug Discovery | Myocardial Ischemia - drug therapy | Cyclophilins - antagonists & inhibitors | Myocardial Reperfusion Injury - metabolism | Animals | Protein Processing, Post-Translational - drug effects | Myocardium - metabolism | Heart - drug effects | Myocardial Reperfusion Injury - drug therapy | Oxidative Stress - drug effects | Energy Metabolism - drug effects | Medical colleges | Medical research | Ischemia | Analysis | Cardiac patients | Medicine, Experimental | Permeability | Heart | Myocardial infarction | Animal models | Mitochondrial permeability transition pore | Cyclosporins | Pathogenesis | Membrane permeability | Mitochondrial DNA | Information dissemination | Reperfusion | Bioenergetics | Attenuation | Physiology | Inhibition | Damage | Cardiology | Heart diseases | Pharmacology | Metabolism | Patients | Injury prevention | Inhibitors | Myocardium | Infarction
Journal Article