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Nature Medicine, ISSN 1078-8956, 02/2016, Volume 22, Issue 2, pp. 175 - 182
Regulated necrosis (necroptosis) and apoptosis are crucially involved in severe cardiac pathological conditions, including myocardial infarction,... 
MEDICINE, RESEARCH & EXPERIMENTAL | ISCHEMIA/REPERFUSION INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | HEART-FAILURE | RECEPTOR INTERACTING PROTEIN-3 | MIXED LINEAGE KINASE | PROGRAMMED NECROSIS | DOMAIN-LIKE | CELL-DEATH | CELL BIOLOGY | MITOCHONDRIAL PERMEABILITY TRANSITION | INDEPENDENT ACTIVATION | Immunohistochemistry | Myocardial Ischemia - genetics | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Phosphorylation | Antibiotics, Antineoplastic - toxicity | Reactive Oxygen Species | Oxidative Stress | Apoptosis - genetics | Necrosis - pathology | Membrane Potential, Mitochondrial | Myocardial Reperfusion Injury - genetics | Real-Time Polymerase Chain Reaction | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | In Situ Nick-End Labeling | Mitochondrial Membrane Transport Proteins - metabolism | Cell Survival | Mice, Inbred C57BL | Rats | Heart Failure - genetics | Heart Failure - pathology | Rats, Sprague-Dawley | Blotting, Western | Doxorubicin - toxicity | Mice, Knockout | Myocardial Ischemia - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Microscopy, Confocal | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Myocardial Ischemia - complications | Necrosis - etiology | Myocytes, Cardiac - metabolism | Mice | Necrosis - genetics | Heart Failure - chemically induced | Oxidative stress | Medical research | Physiological aspects | Medicine, Experimental | Research | Heart diseases | Necrosis | Apoptosis | Signal transduction | Cardiovascular disease | Cardiomyocytes | Ischemia | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2005, Volume 11, Issue 10, pp. 1096 - 1103
Journal Article
Circulation, ISSN 0009-7322, 03/2012, Volume 125, Issue 9, pp. 1134 - 1146
Background-Rheb is a GTP-binding protein that promotes cell survival and mediates the cellular response to energy deprivation (ED). The role of Rheb in the... 
apoptosis | signal transduction | obesity | ischemia | MAMMALIAN TARGET | RAPAMYCIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | MTOR | SKELETAL-MUSCLE | INFARCTION | IMPACT | PATHWAY | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Myocardial Ischemia - metabolism | TOR Serine-Threonine Kinases - metabolism | Metabolic Syndrome - metabolism | Ras Homolog Enriched in Brain Protein | Autophagy - physiology | TOR Serine-Threonine Kinases - genetics | Dietary Fats - pharmacology | Metabolic Syndrome - physiopathology | Apoptosis Regulatory Proteins - genetics | Myocardial Ischemia - physiopathology | Neuropeptides - genetics | Energy Metabolism - physiology | Cell Survival - physiology | Beclin-1 | Myocytes, Cardiac - cytology | Cells, Cultured | Rats | Neuropeptides - metabolism | Obesity - physiopathology | Glucose - pharmacology | Monomeric GTP-Binding Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Myocardial Ischemia - pathology | Obesity - metabolism | Obesity - pathology | Animals | Myocytes, Cardiac - drug effects | Monomeric GTP-Binding Proteins - metabolism | Myocytes, Cardiac - physiology | Signal Transduction - physiology | Mice | Metabolic Syndrome - pathology | Endoplasmic Reticulum Stress - physiology | Physiological aspects | Autophagy (Cytology) | Obesity | Myocardial ischemia | Research | Metabolic syndrome X | Index Medicus | Abridged Index Medicus | myocardial ischemia | autophagy | Rheb | mTORC1
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 4, pp. H812 - H838
Myocardial infarction is a prevalent major cardiovascular event that arises from myocardial ischemia with or without reperfusion, and basic and translational... 
Heart failure | Myocardial infarction | Animal models | Rigor and reproducibility | Reperfusion | Cardiac remodeling | heart failure | CHRONIC HEART-FAILURE | animal models | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | POSTMYOCARDIAL INFARCTION | myocardial infarction | HIBERNATING MYOCARDIUM | REPERFUSION INJURY | NO-REFLOW PHENOMENON | CELL-DERIVED CARDIOMYOCYTES | BLOOD-FLOW | reperfuson | CORONARY COLLATERAL CIRCULATION | CARDIOPROTECTIVE THERAPIES CAESAR | LEFT-VENTRICULAR DYSFUNCTION | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | rigor and reproducibility | Myocardial Ischemia - metabolism | Data Accuracy | Cells, Cultured | Myocardium - pathology | Cardiology - standards | Myocardial Infarction - metabolism | Periodicals as Topic - standards | Consensus | Myocardial Ischemia - pathology | Isolated Heart Preparation - standards | Animals | Myocardial Infarction - pathology | Myocardium - metabolism | Physiology - standards | Myocardial Ischemia - physiopathology | Myocardial Infarction - physiopathology | Mice | Biomedical Research - standards | Disease Models, Animal | Quality Control | Myocardial ischemia | Biological models | Usage | Models | Heart attack | Occlusion | Heart attacks | Cardiovascular disease | Angina | Guidelines | Salvage | Ischemia | Reproducibility | Research methodology | Hibernation | Infarction | Best practice | Structure-function relationships | Index Medicus | Review | reperfusion
Journal Article
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 08/2011, Volume 109, Issue 4, pp. 428 - 436
RATIONALE:A growing number of patients with coronary disease have refractory angina. Preclinical and early-phase clinical data suggest that intramyocardial... 
stem cells | angiogenesis | endothelial progenitor cells (EPC) myocardial ischemia | myocardial regeneration | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | CELL TRANSPLANTATION | RANDOMIZED CONTROLLED-TRIAL | RESEARCH ISSUES | METASTATIC COLORECTAL-CANCER | PERCUTANEOUS CORONARY INTERVENTION | HEART-DISEASE | UNIVERSAL DEFINITION | DOUBLE-BLIND | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ARTERY-DISEASE | Antigens, CD34 - metabolism | Prospective Studies | Angina Pectoris - mortality | Angina Pectoris - physiopathology | United States | Humans | Middle Aged | Endothelial Cells - transplantation | Tomography, Emission-Computed, Single-Photon | Male | Transplantation, Autologous | Angina Pectoris - etiology | Blood Component Removal | Angina Pectoris - pathology | Microcirculation | Exercise Test | Time Factors | Hematopoietic Stem Cell Transplantation - adverse effects | Myocardial Ischemia - physiopathology | Female | Surveys and Questionnaires | Myocardial Ischemia - mortality | Hematopoietic Stem Cell Mobilization | Biomarkers - metabolism | Coronary Circulation | Double-Blind Method | Risk Assessment | Risk Factors | Myocardium - pathology | Treatment Outcome | Myocardial Ischemia - pathology | Endothelial Cells - immunology | Cardiovascular Agents - therapeutic use | Regeneration | Regression Analysis | Angina Pectoris - surgery | Myocardial Ischemia - complications | Exercise Tolerance | Least-Squares Analysis | Aged | Myocardial Ischemia - surgery | Neovascularization, Physiologic | Index Medicus
Journal Article