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Circulation: Cardiovascular Imaging, ISSN 1941-9651, 05/2013, Volume 6, Issue 3, pp. 363 - 372
Background-Although the recent surgical treatment of ischemic heart failure substudy reported that revascularization of viable myocardium did not improve... 
Prognosis | Ischemia | Positron emission tomography | Viability | Revascularization | HEART-RATE RECOVERY | SURVIVAL | revascularization | CARDIAC & CARDIOVASCULAR SYSTEMS | METAANALYSIS | CARDIOLOGY | VALIDATION | prognosis | EMISSION COMPUTED-TOMOGRAPHY | EXERCISE | ischemia | viability | MEDICAL THERAPY | positron emission tomography | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | CORONARY-ARTERY-DISEASE | Predictive Value of Tests | Ventricular Function, Left | Humans | Middle Aged | Male | Positron-Emission Tomography | Ventricular Dysfunction, Left - mortality | Recovery of Function | Patient Selection | Ventricular Dysfunction, Left - therapy | Time Factors | Ventricular Dysfunction, Left - pathology | Myocardial Ischemia - diagnostic imaging | Myocardial Ischemia - physiopathology | Female | Myocardial Ischemia - mortality | Myocardial Stunning - diagnostic imaging | Radiopharmaceuticals | Rubidium Radioisotopes | Myocardial Revascularization - mortality | Myocardial Stunning - physiopathology | Risk Assessment | Risk Factors | Proportional Hazards Models | Myocardial Stunning - therapy | Myocardium - pathology | Treatment Outcome | Chi-Square Distribution | Myocardial Stunning - mortality | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Myocardial Ischemia - pathology | Myocardial Perfusion Imaging - methods | Myocardial Stunning - pathology | Stroke Volume | Cardiovascular Agents - therapeutic use | Propensity Score | Myocardial Ischemia - therapy | Fluorodeoxyglucose F18 | Tissue Survival | Aged | Hemodynamics | Myocardial Revascularization - adverse effects | Index Medicus
Journal Article
Science, ISSN 0036-8075, 6/2009, Volume 324, Issue 5935, pp. 1710 - 1713
MicroRNAs (miRs) are small noncoding RNAs that regulate gene expression by binding to target messenger RNAs (mRNAs), leading to translational repression or... 
Heart | Angiogenesis | Human umbilical vein endothelial cells | Messenger RNA | Ischemia | Sprouts | Small interfering RNA | Reports | Gene expression regulation | Integrins | Tumors | GENE | MULTIDISCIPLINARY SCIENCES | VASCULAR INTEGRITY | FIBRONECTIN | POLYCISTRON | ALPHA | ENDOTHELIAL-CELL MIGRATION | DICER | EXPRESSION | Up-Regulation | MicroRNAs - antagonists & inhibitors | Apoptosis - drug effects | Humans | MicroRNAs - metabolism | Muscle, Skeletal - metabolism | Gene Expression Profiling | RNA, Messenger - metabolism | Oligoribonucleotides - pharmacology | Myocardial Infarction - pathology | Myocardium - metabolism | Myocardial Infarction - physiopathology | Ischemia - pathology | Endothelial Cells - metabolism | Down-Regulation | Mice, Inbred C57BL | RNA, Messenger - genetics | Ventricular Function, Left - drug effects | Ischemia - metabolism | Zebrafish | Ischemia - physiopathology | Myocardial Infarction - metabolism | Regional Blood Flow | Oligoribonucleotides - therapeutic use | Animals | Ischemia - drug therapy | Hindlimb - blood supply | Integrin alpha5 - metabolism | Mice | Integrin alpha5 - genetics | Neovascularization, Physiologic | Development and progression | Genetic aspects | Neovascularization | Research | Gene expression | Cell growth | Cellular biology | Rodents | Blood vessels | Ribonucleic acid--RNA | Index Medicus
Journal Article
Journal Article
Journal Article
Science Translational Medicine, ISSN 1946-6234, 06/2017, Volume 9, Issue 395
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e75872 - e75872
Background: Ischemic postconditioning (IPost) protects the reperfused heart from infarction which has drawn much attention recently. However, studies to date... 
HEART | PERCUTANEOUS CORONARY INTERVENTION | ACTIVATION | CARDIOMYOCYTE APOPTOSIS | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | ATTENUATION | ACUTE MYOCARDIAL-INFARCTION | CARDIOPROTECTION | EXPRESSION | MICRORNA | Myocardial Ischemia - genetics | Oligonucleotides - genetics | Apoptosis - drug effects | Gene Expression Regulation - genetics | Enzyme Inhibitors - pharmacology | PTEN Phosphohydrolase - metabolism | Apoptosis - genetics | Male | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Myocardial Ischemia - pathology | Gene Expression Regulation - drug effects | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Reperfusion Injury - prevention & control | Signal Transduction - drug effects | Ventricular Dysfunction, Left - genetics | Myocardial Ischemia - therapy | Ventricular Dysfunction, Left - pathology | Ischemic Postconditioning | Myocardial Ischemia - physiopathology | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-akt - metabolism | Heart | Ischemia | MicroRNA | Myocardial infarction | Heart attacks | Myocardial ischemia | AKT protein | Kinases | Signal transduction | Reperfusion | Rodents | Cardiology | Heart diseases | Cytokines | MiRNA | Cardiomyocytes | Gene expression | Molecular chains | 1-Phosphatidylinositol 3-kinase | Signaling | Ostomy | Injury prevention | Hospitals | Molecular modelling | MicroRNAs | Infarction | Ventricle | Laboratory animals | PTEN protein | Apoptosis | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 2013, Volume 3, Issue 1, pp. 2767 - 2767
Myocardial ischemia, while causing cardiomyocyte injury, can activate innate protective processes, enhancing myocardial tolerance to ischemia. Such processes... 
BETA-KLOTHO | SURVIVAL | ENDOTHELIAL PROGENITOR CELLS | ARTERIAL ELASTIC LAMINAE | PPAR-ALPHA | ACTIVATION | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | BONE-MARROW | C-KIT | AKT | Up-Regulation | Liver - pathology | Phosphorylation | Cardiotonic Agents - metabolism | Adipose Tissue - physiopathology | Caspase 3 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Glucuronidase | Liver - physiopathology | Phosphatidylinositol 3-Kinases | Myocardial Reperfusion Injury - enzymology | Myocytes, Cardiac - enzymology | Adipose Tissue - metabolism | Fibroblast Growth Factors - metabolism | Myocardial Reperfusion Injury - pathology | Endocrine System - metabolism | Myocardial Ischemia - physiopathology | bcl-Associated Death Protein - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Function Tests | Heart Ventricles - pathology | Signal Transduction | Liver - metabolism | Adipose Tissue - pathology | Gene Silencing | Myocardial Ischemia - prevention & control | Myocardial Ischemia - enzymology | Myocardial Reperfusion Injury - physiopathology | Endocrine System - pathology | Myocardial Ischemia - pathology | Myocytes, Cardiac - pathology | Animals | Heart Ventricles - physiopathology | Protein Binding | Heart Ventricles - metabolism | Mice | Hemodynamics | RNA, Small Interfering - metabolism | Myocardial infarction | AKT1 protein | Adipose tissue | Liver | Caspase | Myocardial ischemia | Cardiomyocytes | Adipocytes | Caspase-3 | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Reperfusion | Ischemia | Cell death | Klotho protein | Rodents | Myocardium | Fibroblast growth factor receptor 1 | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 37, pp. 13511 - 13516
Journal Article
Annals of Neurology, ISSN 0364-5134, 05/2014, Volume 75, Issue 5, pp. 670 - 683
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 08/2013, Volume 2, Issue 4, pp. e000249 - n/a
Background-Coronary artery disease leading to myocardial ischemia is the most common cause of heart failure. Apelin (APLN), the endogenous peptide ligand of... 
ischemia‐reperfusion injury | heart failure | cardiomyopathy | angiogenesis | myocardial infarction | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIAC DYSFUNCTION | LIGAND APELIN | AMERICAN-HEART-ASSOCIATION | FAILING HEARTS | ischemia-reperfusion injury | APJ RECEPTOR | IN-VIVO | BLOOD-VESSELS | ENDOGENOUS INOTROPE APELIN | Myocardial Ischemia - genetics | Myocardial Ischemia - metabolism | Ventricular Function, Left | Myocardial Reperfusion Injury - mortality | Humans | Heart Failure - physiopathology | Male | Stem Cells - metabolism | Recovery of Function | Heart Failure - prevention & control | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardium - metabolism | Myocardial Ischemia - mortality | Intercellular Signaling Peptides and Proteins - deficiency | Myocardial Reperfusion Injury - genetics | Apelin | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Myocardial Ischemia - prevention & control | Myocardium - pathology | Heart Failure - metabolism | Adipokines | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Cardiovascular Agents - pharmacology | Mice, Knockout | Myocardial Ischemia - pathology | Peptides - pharmacology | Ventricular Dysfunction, Left - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Intercellular Signaling Peptides and Proteins - pharmacology | Stem Cells - pathology | Mice | Endothelial Cells - pathology | Ventricular Remodeling - drug effects | Myocardial Reperfusion Injury - prevention & control | Neovascularization, Physiologic | Index Medicus
Journal Article