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Journal Article
Advances in experimental medicine and biology, ISSN 0065-2598, 2019, Volume 1127, p. 117
Ischemic heart disease is the main cause of morbidity and mortality in the developed world. Although reperfusion therapies are currently the best treatment for... 
Dyslipidemias - complications | Oxidative Stress | Ischemic Preconditioning, Myocardial | Myocardial Reperfusion Injury - complications | Humans | Myocardial Ischemia - complications | Risk Factors | Ischemic Postconditioning
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 91, pp. 11 - 22
Journal Article
European Heart Journal, ISSN 0195-668X, 04/2017, Volume 38, Issue 13, pp. 935 - 941d
Targeting reperfusion injury in patients with ST-segment elevation myocardial infarction: trials and tribulations 
PERMEABILITY TRANSITION PORE | PROTEIN KINASE-C | MICROVASCULAR OBSTRUCTION | PERCUTANEOUS CORONARY INTERVENTION | CARDIAC & CARDIOVASCULAR SYSTEMS | CYCLOSPORINE-A | CELLULAR BIOLOGY | BYPASS GRAFT-SURGERY | ISCHEMIA-REPERFUSION | LEFT-VENTRICULAR FUNCTION | LONG-TERM BENEFIT | Myocardial Reperfusion Injury - etiology | Nucleotides, Cyclic - metabolism | Ischemic Postconditioning - methods | Vasodilator Agents - therapeutic use | Humans | Oximes - therapeutic use | Nitric Oxide - therapeutic use | Oligopeptides - therapeutic use | Patient Selection | ST Elevation Myocardial Infarction - therapy | Cardiotonic Agents - therapeutic use | Mitochondrial Membrane Transport Proteins - physiology | Adenosine - therapeutic use | Hypothermia, Induced - methods | ST Elevation Myocardial Infarction - complications | ST Elevation Myocardial Infarction - etiology | Coronary Vessels - physiology | Metoprolol - therapeutic use | Combined Modality Therapy | Protein Kinase C - antagonists & inhibitors | Enzyme Inhibitors - therapeutic use | Nitrites - therapeutic use | Cyclosporine - therapeutic use | Myocardial Reperfusion - adverse effects | Signal Transduction - physiology | Clinical Trials as Topic - methods | Nitric Oxide - metabolism | Secosteroids - therapeutic use | Myocardial Reperfusion Injury - prevention & control | Life Sciences | Current Opinion | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2017, Volume 7, Issue 1, p. 41337
Enhancing mitochondrial biogenesis and reducing mitochondrial oxidative stress have emerged as crucial therapeutic strategies to ameliorate diabetic myocardial... 
ISCHEMIA-REPERFUSION INJURY | ACTIVATED PROTEIN-KINASE | BIOGENESIS | METABOLISM | PROTECTION | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | CADMIUM-INDUCED HEPATOTOXICITY | DYSFUNCTION | STRESS | SIRT3 | AMP-Activated Protein Kinases - metabolism | Diabetes Mellitus, Experimental - drug therapy | Sirtuin 3 - metabolism | Apoptosis - drug effects | Myocardial Reperfusion Injury - complications | Streptozocin | Male | Diabetes Mellitus, Type 1 - complications | Cardiotonic Agents - therapeutic use | Myocardial Reperfusion Injury - pathology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Glucose - toxicity | Diabetes Mellitus, Experimental - complications | Myocardial Reperfusion Injury - drug therapy | Superoxide Dismutase - metabolism | Glucose Tolerance Test | Signal Transduction | Cytochromes c - metabolism | Diabetes Mellitus, Type 1 - pathology | Myocardium - pathology | Mitochondria - metabolism | Cardiotonic Agents - pharmacology | Mitochondria - drug effects | Rats, Sprague-Dawley | Diabetes Mellitus, Type 1 - drug therapy | Transcription Factors - metabolism | Animals | Melatonin - therapeutic use | Diabetes Mellitus, Experimental - pathology | Cytosol - metabolism | Mice | Oxidative Stress - drug effects | Melatonin - pharmacology | RNA, Small Interfering - metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50515
Recent studies have revealed the critical role of microRNAs (miRNAs) in regulating cardiac injury. Among them, the cardiac enriched microRNA-1(miR-1) has been... 
APOPTOSIS | HSP60 | MYOCARDIAL-INFARCTION | PROTECTION | MULTIDISCIPLINARY SCIENCES | MUSCLE-SPECIFIC MICRORNA | IN-VIVO | HEAT-SHOCK | CARDIOMYOCYTES | EXPRESSION | EPSILON | Oligonucleotides - genetics | L-Lactate Dehydrogenase - secretion | Humans | Caspase 3 - metabolism | Myocytes, Cardiac - secretion | Male | MicroRNAs - metabolism | Gene Knockdown Techniques | Creatine Kinase - secretion | Base Sequence | Chaperonin 60 - metabolism | Reperfusion Injury - complications | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Chaperonin 60 - genetics | Reperfusion Injury - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Protein Kinase C-epsilon - genetics | Mice, Transgenic | Myocardial Infarction - complications | Myocytes, Cardiac - pathology | Animals | Myocardial Ischemia - complications | Protein Kinase C-epsilon - metabolism | Myocytes, Cardiac - metabolism | Mice | MicroRNAs - genetics | Apoptosis | MicroRNA | Ischemia | Luciferase | Heat shock proteins | Genetic engineering | Health aspects | Protein kinases | Heart | Hsp60 protein | Animal models | Protein kinase C | Laboratories | Cyclin-dependent kinases | Transgenic mice | Caspase | Cardiomyocytes | Myocytes | Kinases | Ribonucleic acid--RNA | Caspase-3 | Reperfusion | Rodents | miRNA | Heart diseases | Injuries | Heat shock | RNA | Ribonucleic acid
Journal Article