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Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 08/2012, Volume 90, Issue 8, pp. 1135 - 1144
Peroxisome proliferator-activated receptors (PPAR) regulate the expression of genes involved in lipid metabolism, energy production, and inflammation. Their... 
WY-14643 | activation du PPAR-α | ischémie–reperfusion du myocarde | myocardial ischaemia–reperfusion | PPAR-α activation | cardioprotection | PI3K–Akt | metabolic genes | gènes du métabolisme | GAMMA | INDUCED ARRHYTHMIAS | PHYSIOLOGY | KINASE | PI3K-Akt | INFARCT SIZE | HEART | PROLIFERATOR | PATHWAY | PPAR-alpha activation | RECEPTORS ALPHA | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | myocardial ischaemia-reperfusion | Phosphorylation | Pyrimidines - antagonists & inhibitors | Phosphatidylinositol 3-Kinase - physiology | Peroxisome Proliferators - therapeutic use | Rats, Wistar | Myocardial Reperfusion Injury - complications | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Male | Arrhythmias, Cardiac - physiopathology | Peroxisome Proliferators - antagonists & inhibitors | Up-Regulation - physiology | Myocardial Infarction - physiopathology | Protein Kinases - biosynthesis | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Arrhythmias, Cardiac - prevention & control | PPAR alpha - biosynthesis | Rats | Peroxisome Proliferators - pharmacology | Chymases - biosynthesis | Proto-Oncogene Proteins c-akt - physiology | Pyrimidines - pharmacology | Myocardial Reperfusion Injury - physiopathology | Up-Regulation - drug effects | Myocardial Infarction - complications | Myocardial Reperfusion Injury - metabolism | Animals | Androstadienes - pharmacology | Signal Transduction - drug effects | Pyrimidines - therapeutic use | PPAR alpha - physiology | Signal Transduction - physiology | Arrhythmias, Cardiac - complications | Myocardial Infarction - prevention & control | Protein Kinase Inhibitors - pharmacology | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Cardiovascular system | Prevention | Transcription factors | Ischemia | Cardiotonic agents | Analysis | Research | Cardiac glycosides
Journal Article
Journal of Heart and Lung Transplantation, ISSN 1053-2498, 2014, Volume 33, Issue 4, pp. 327 - 340
Although primary graft dysfunction (PGD) is fairly common early after cardiac transplant, standardized schemes for diagnosis and treatment remain contentious.... 
Surgery | consensus | outcomes | primary graft dysfunction | primary graft failure | cardiac transplantation | SURGERY | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | INTERNATIONAL-SOCIETY | REPERFUSION INJURY | LUNG-TRANSPLANTATION | NATRIURETIC PEPTIDE | TRANSPLANTATION | HEART-TRANSPLANTATION | RESPIRATORY SYSTEM | SUBARACHNOID HEMORRHAGE | TROPONIN-I LEVELS | BRAIN-DEATH | EXTRACORPOREAL MEMBRANE-OXYGENATION | Reoperation | Postoperative Complications - physiopathology | Myocardial Reperfusion Injury - diagnosis | Myocardial Reperfusion Injury - mortality | Primary Graft Dysfunction - physiopathology | Graft Rejection - physiopathology | Graft Rejection - therapy | Humans | Risk Factors | Cooperative Behavior | Postoperative Complications - therapy | Graft Rejection - diagnosis | Postoperative Complications - mortality | Myocardial Reperfusion Injury - physiopathology | Postoperative Complications - diagnosis | Interdisciplinary Communication | Myocardial Reperfusion Injury - therapy | Primary Graft Dysfunction - mortality | Ischemic Preconditioning, Myocardial | Graft Rejection - mortality | Primary Graft Dysfunction - diagnosis | Survival Analysis | Primary Graft Dysfunction - therapy | Heart Transplantation - mortality | Heart | Medical colleges | Conferences, meetings and seminars | Therapeutics | Transplantation | Conferences and conventions | Pulmonary hypertension | Homeopathy | Materia medica and therapeutics
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 04/2019, Volume 17, Issue 1, pp. 127 - 14
Background: The sodium-glucose cotransporter-2 (SGLT2) inhibitor canagliflozin has been shown to reduce major cardiovascular events in type 2 diabetic... 
Sodium-glucose cotransporter-2 inhibitor | Cardioprotection | Canagliflozin | Myocardial ischemia-reperfusion injury | MEDICINE, RESEARCH & EXPERIMENTAL | SGLT2 INHIBITORS | PHOSPHORYLATION | KINASE | CARDIOMYOCYTES | HEART | PRESSURE | AMPK | ARTERY | Apoptosis - drug effects | Canagliflozin - pharmacology | Myocardial Reperfusion Injury - complications | Systole - drug effects | Male | Aldehydes - metabolism | Liver - physiopathology | Cardiotonic Agents - therapeutic use | Liver - drug effects | Canagliflozin - therapeutic use | Diastole - drug effects | Aorta - physiopathology | Myocardial Reperfusion Injury - drug therapy | Phosphorylation - drug effects | Kidney - physiopathology | Biomarkers - metabolism | Endothelium - pathology | Glycosuria - physiopathology | Kidney - drug effects | Aorta - drug effects | Ventricular Function, Left - drug effects | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glycosuria - complications | Myocardial Reperfusion Injury - physiopathology | Endothelium - physiopathology | Aorta - pathology | Endothelium - drug effects | Animals | Signal Transduction - drug effects | Oxidative Stress - drug effects | Vasodilation - drug effects | Blood Glucose - metabolism | Myocardial Reperfusion Injury - prevention & control | Nitrosative Stress - drug effects | Heart failure | Diabetics | Rats as laboratory animals | Analysis | Clinical trials | Research | Drug therapy | Health aspects | Occlusion | Myocardial infarction | Heart | Oxidative stress | Phosphorylation | Drug delivery systems | Intravenous administration | Heart attacks | Bax protein | 4-Hydroxynonenal | Bcl-2 protein | AKT protein | Myocardial ischemia | mRNA | Size determination | Kinases | Vasodilation | Proteins | Reperfusion | Ischemia | Rodents | Calcium-binding protein | Aorta | Heart diseases | Injury analysis | Medical research | Adenosine monophosphate | AMP | Diabetes mellitus | Coronary artery | Gene expression | Nitric-oxide synthase | Endothelium | Sodium | Protein kinase | Adenosine kinase | Nitric oxide | Ventricle | Diabetes | Laboratory animals | Apoptosis | Myocardial ischemia–reperfusion injury | Sodium–glucose cotransporter-2 inhibitor
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Journal Article
Circulation, ISSN 0009-7322, 08/2019, Volume 140, Issue 9, pp. 751 - 764
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