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Heart, ISSN 1355-6037, 10/2015, Volume 101, Issue 20, pp. 1639 - 1645
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 02/2017, Volume 16, Issue 1, p. 19
Background: Many cardioprotective pharmacological agents failed to exert their protective effects in diabetic hearts subjected to myocardial... 
Ischemia-reperfusion | Diabetes | Mitochondrial fission | Drp1 | OXIDATIVE STRESS | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | FUSION | ISCHAEMIA/REPERFUSION INJURY | MITOCHONDRIAL BIOGENESIS | CARDIAC DYSFUNCTION | RESTRICTION | HEART | INDUCED CARDIOPROTECTION | ENDOCRINOLOGY & METABOLISM | FISSION | Myocardial Reperfusion Injury - etiology | Diabetes Mellitus, Experimental - drug therapy | Dynamins - metabolism | Mitochondria, Heart - metabolism | Apoptosis - drug effects | Mitochondria, Heart - pathology | Streptozocin | Male | Mitochondria, Heart - drug effects | Troponin I - blood | Myocardial Reperfusion Injury - pathology | Diet, High-Fat | Myocardial Infarction - pathology | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Myocardial Infarction - etiology | Quinazolinones - pharmacology | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Dynamins - antagonists & inhibitors | Mice, Inbred C57BL | Myocardial Infarction - metabolism | Cardiovascular Agents - pharmacology | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Mitochondrial Dynamics - drug effects | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | L-Lactate Dehydrogenase - blood | Myocytes, Cardiac - metabolism | Myocardial Infarction - prevention & control | Oxidative Stress - drug effects | Myocardial Reperfusion Injury - prevention & control
Journal Article
STEM CELLS, ISSN 1066-5099, 08/2014, Volume 32, Issue 8, pp. 2123 - 2134
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 12/2014, Volume 745, pp. 1 - 9
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 01/2013, Volume 17, Issue 1, pp. 181 - 191
Hyperglycaemia during acute myocardial infarction is common and associated with increased mortality. Thioredoxin‐interacting protein (Txnip) is a modulator of... 
Hyperglycaemia | Akt | p38 mitogen‐activated protein kinase | Txnip | oxidative stress | myocardial ischaemia/reperfusion | P38 mitogen-activated protein kinase | Oxidative stress | Myocardial ischaemia/reperfusion | MEDICINE, RESEARCH & EXPERIMENTAL | myocardial ischaemia | MITOCHONDRIAL-FUNCTION | ISCHEMIA | ISCHEMIA/REPERFUSION-INJURY | SUPPRESSION | DISULFIDE | CELL BIOLOGY | INFARCT SIZE | INSULIN | p38 mitogen-activated protein kinase | reperfusion | EXPRESSION | RNA, Small Interfering - genetics | Oxidative Stress | Myocardial Reperfusion Injury - complications | Hyperglycemia - complications | Male | Proto-Oncogene Proteins c-akt - genetics | Myocardial Reperfusion Injury - pathology | Myocardium - metabolism | Hyperglycemia - pathology | Superoxides - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Malondialdehyde - metabolism | Signal Transduction | Carrier Proteins - antagonists & inhibitors | Cells, Cultured | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Myocardium - pathology | Glucose - pharmacology | Rats, Sprague-Dawley | Hyperglycemia - metabolism | Carrier Proteins - genetics | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Apoptosis | Glucose metabolism | Hyperglycemia | Glucose | Thioredoxin | Mitogens | Dextrose | Myocardial infarction | Heart | Phosphorylation | Dehydrogenases | Heart attacks | AKT protein | Activation | Kinases | Proteins | Infusion | Reperfusion | Ischemia | Cell cycle | Inhibition | Redox properties | Injuries | Cytokines | Incubation | RNA-mediated interference | Coronary artery | MAP kinase | Cardiomyocytes | Superoxide | siRNA | Gene expression | Studies | Nitric oxide | Coronary vessels | Insulin resistance | Diabetes | Laboratory animals | Veins & arteries | Original
Journal Article
Science Translational Medicine, ISSN 1946-6234, 08/2014, Volume 6, Issue 248, pp. 248ra105 - 248ra105
Journal Article