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Circulation, ISSN 0009-7322, 06/2012, Volume 125, Issue 25, pp. 3170 - 3181
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 1, pp. 164 - 174
Nucleotide-binding oligomerization domain-Like Receptor with a Pyrin domain 3 (NLRP3) is considered necessary for initiating a profound sterile inflammatory... 
Heart | Myocardial infarction | Inflammasome | Ischaemia-reperfusion | IL-1β | Potassium - metabolism | Up-Regulation | Inflammasomes - metabolism | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Myocardial Infarction - physiopathology | Toll-Like Receptors - metabolism | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Fibroblasts - metabolism | Myocardial Reperfusion Injury - immunology | Rats | Receptors, Cytoplasmic and Nuclear - genetics | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Fibroblasts - drug effects | Lipopolysaccharides - pharmacology | Fibroblasts - immunology | Heart Ventricles - metabolism | Mice | Interleukin-18 - genetics | Interleukin-18 - metabolism | Myocardial Reperfusion Injury - prevention & control | Heart Ventricles - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Myocardial Infarction - genetics | Rats, Wistar | Ventricular Function, Left | Heart Ventricles - immunology | Myocardial Infarction - immunology | Cytoskeletal Proteins - deficiency | Myocardial Infarction - pathology | Heart Ventricles - pathology | Myocardial Contraction | Mice, Inbred C57BL | Cells, Cultured | Inflammasomes - genetics | Carrier Proteins - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Heart Ventricles - physiopathology | CARD Signaling Adaptor Proteins | Apoptosis | Index Medicus
Journal Article
Journal of Pineal Research, ISSN 0742-3098, 10/2018, Volume 65, Issue 3, pp. e12503 - n/a
Journal Article
Circulation Research, ISSN 0009-7330, 07/2015, Volume 117, Issue 4, pp. 352 - 363
RATIONALE:Necrosis is one of the main forms of cardiomyocyte death in heart disease. Recent studies have demonstrated that certain types of necrosis are... 
H19 long noncoding RNA | Myocardial infarction | Oxidative stress | Ischemia/reperfusion injury | MiRNA | Fas-associated death domain protein | Necrosis | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | NECROPTOSIS | ischemia/reperfusion injury | myocardial infarction | ISCHEMIA | CELL-DEATH | PROTECTS | HEART-DISEASE | necrosis | miRNA | PERIPHERAL VASCULAR DISEASE | RIP3 | HEMATOLOGY | EXPRESSION | oxidative stress | CYCLE | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Myocardial Infarction - genetics | Humans | Male | MicroRNAs - metabolism | Dose-Response Relationship, Drug | Myocardial Reperfusion Injury - pathology | Transfection | RNA Interference | Myocardial Infarction - pathology | HEK293 Cells | Myocardial Reperfusion Injury - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Fas-Associated Death Domain Protein - genetics | Hydrogen Peroxide - toxicity | Signal Transduction | Mice, Inbred C57BL | Fas-Associated Death Domain Protein - metabolism | Rats | RNA, Long Noncoding - genetics | Myocardial Infarction - metabolism | Tumor Necrosis Factor-alpha - toxicity | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | MicroRNAs - genetics | Myocardial Infarction - prevention & control | Oligonucleotides - administration & dosage | RNA, Long Noncoding - metabolism | Myocardial Reperfusion Injury - prevention & control | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Index Medicus | Original
Journal Article
Circulation, ISSN 0009-7322, 11/2010, Volume 122, Issue 20, pp. 2039 - 2047
Journal Article
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 03/2017, Volume 6, Issue 3, p. n/a
Background-The cardiac microvascular system ischemia/reperfusion injury following percutaneous coronary intervention is a clinical thorny problem. This study... 
apoptosis | ischemia/reperfusion injury | endothelial cell | mitochondria | Endothelial cell | Mitochondria | Ischemia/reperfusion injury | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PERMEABILITY | HEXOKINASE | NO-REFLOW PHENOMENON | REPERFUSION | ISCHEMIA | ENDOTHELIAL-CELLS | ACUTE MYOCARDIAL-INFARCTION | CYTOCHROME-C RELEASE | CARDIOPROTECTION | Immunohistochemistry | Mitochondria, Heart - ultrastructure | Microvessels - physiopathology | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Mitochondrial Dynamics - genetics | Apoptosis - drug effects | Male | Mitochondrial Proteins - genetics | Chromatography, High Pressure Liquid | Microcirculation - drug effects | Voltage-Dependent Anion Channel 1 - biosynthesis | Mitochondrial Membrane Transport Proteins - biosynthesis | Mitochondrial Membrane Transport Proteins - genetics | DNA, Mitochondrial - genetics | Hexokinase - biosynthesis | Hexokinase - genetics | Mitochondria, Heart - genetics | Myocardial Reperfusion Injury - genetics | Cardiolipins - genetics | Disease Models, Animal | Oxidation-Reduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Cells, Cultured | Gene Expression Regulation | Mitochondrial Proteins - biosynthesis | Blotting, Western | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Membrane Proteins - biosynthesis | Animals | Mitochondrial Dynamics - drug effects | Myocytes, Cardiac - metabolism | Cardiolipins - biosynthesis | Mice | Myocytes, Cardiac - ultrastructure | Voltage-Dependent Anion Channel 1 - genetics | Endothelial Cells - drug effects | Index Medicus
Journal Article
Apoptosis, ISSN 1360-8185, 7/2016, Volume 21, Issue 7, pp. 809 - 824
Recently, we demonstrated that melatonin reduced protein kinase RNA (PKR)-like ER kinase (PERK)-eukaryotic initiation factor 2 alpha (eIF2α)-activating... 
Biochemistry, general | Melatonin | Biomedicine | Myocardial ischemia–reperfusion injury | Survivor activating factor enhancement pathway | Cancer Research | Oncology | Reperfusion injury salvage kinase pathway | Cell Biology | Virology | Endoplasmic reticulum stress | H9C2 CARDIOMYOBLAST CELLS | ISCHEMIA/REPERFUSION INJURY | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | Myocardial ischemia-reperfusion injury | CARDIAC DYSFUNCTION | DIABETIC-RATS | CELLULAR STRESS | CELL BIOLOGY | IN-VITRO | SIGNALING PATHWAY | CARDIOVASCULAR-DISEASES | ACTIVATES STAT3 | Myocardial Ischemia - genetics | Myocardial Ischemia - metabolism | Phosphorylation | Oxidative Stress | eIF-2 Kinase - metabolism | Humans | Male | Phosphatidylinositol 3-Kinases - metabolism | Melatonin - administration & dosage | Myocardial Reperfusion Injury - enzymology | Myocardium - metabolism | Eukaryotic Initiation Factor-2 - metabolism | Myocardial Reperfusion Injury - drug therapy | Myocardial Reperfusion Injury - genetics | eIF-2 Kinase - genetics | Signal Transduction | Mice, Inbred C57BL | Activating Transcription Factor 4 - genetics | Glycogen Synthase Kinase 3 - metabolism | Phosphatidylinositol 3-Kinases - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Activating Transcription Factor 4 - metabolism | Glycogen Synthase Kinase 3 - genetics | Endoplasmic Reticulum Stress | Eukaryotic Initiation Factor-2 - genetics | Myocardial Ischemia - surgery | Index Medicus
Journal Article