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Circulation Research, ISSN 0009-7330, 04/2009, Volume 104, Issue 7, pp. 851 - 859
The characteristics of dilated cardiomyopathy (DCM) resulting from chronic viral myocarditis are remodeling processes of the extracellular matrix. Based on our... 
Infection | Myocarditis | Osteopontin | Inflammation | Remodeling | CELLS | infection | remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | VIRUS | myocarditis | osteopontin | AUTOIMMUNE MYOCARDITIS | DILATED CARDIOMYOPATHY | VIRAL MYOCARDITIS | PLASMINOGEN-ACTIVATOR | inflammation | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | HUMAN HEART | EXPRESSION | PULMONARY-FIBROSIS | Osteopontin - genetics | Cardiomyopathy, Dilated - pathology | Phosphorylation | Osteopontin - deficiency | Humans | Transforming Growth Factor beta1 - metabolism | Osteopontin - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Calcitriol - analogs & derivatives | Coxsackievirus Infections - virology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Macrophages - virology | Time Factors | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Biomarkers - metabolism | Acute Disease | Collagen Type I - metabolism | Coxsackievirus Infections - physiopathology | Coxsackievirus Infections - metabolism | Mice, Inbred C57BL | Rats | Matrix Metalloproteinase 3 - metabolism | Myocardium - pathology | Cardiomyopathy, Dilated - metabolism | Cardiomyopathy, Dilated - virology | Mice, Knockout | Coxsackievirus Infections - pathology | Myocarditis - virology | Macrophages - metabolism | Animals | Cardiomyopathy, Dilated - physiopathology | Urokinase-Type Plasminogen Activator - metabolism | Calcitriol - pharmacology | Fibrosis | Myocarditis - metabolism | Mice | Enterovirus B, Human - pathogenicity | Myocarditis - pathology | Chronic Disease | Myocarditis - physiopathology | Ventricular Remodeling - drug effects
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1310 - 1322
Coagulation is a host defense system that limits the spread of pathogens. Coagulation proteases, such as thrombin, also activate cells by cleaving PARs. In... 
TOLL-LIKE RECEPTOR-3 | MEDICINE, RESEARCH & EXPERIMENTAL | NATURAL-KILLER-CELLS | ISCHEMIA/REPERFUSION INJURY | PROTEASE-ACTIVATED RECEPTORS | VIRUS-INFECTION | IN-VIVO | ENDOTHELIAL-CELLS | FACTOR MESSENGER-RNA | INTERFERON-BETA | TISSUE FACTOR EXPRESSION | Receptor, PAR-1 - metabolism | Humans | Male | Troponin I - blood | STAT1 Transcription Factor - metabolism | Myocardium - metabolism | HEK293 Cells | Interferon-beta - genetics | Influenza A virus - immunology | Myocarditis - immunology | Influenza A virus - physiology | Orthomyxoviridae Infections - metabolism | Coxsackievirus Infections - metabolism | Liver - metabolism | Mice, Inbred C57BL | Enterovirus - physiology | Myocarditis - blood | Immunity, Innate | STAT1 Transcription Factor - genetics | Toll-Like Receptor 3 - metabolism | Receptor, PAR-1 - genetics | Mice, Knockout | Myocarditis - virology | Animals | Interferon-beta - metabolism | Thromboplastin - genetics | Enterovirus - immunology | Mice | HeLa Cells | Thrombin - metabolism | Thromboplastin - metabolism | Fibrin - metabolism | Chemokine CXCL10 - metabolism | Coxsackievirus Infections - immunology | Orthomyxoviridae Infections - immunology | Receptor, PAR-1 - physiology | Virus diseases | Cell receptors | Immune response | Physiological aspects | Development and progression | Genetic aspects | Research
Journal Article
Veterinary Immunology and Immunopathology, ISSN 0165-2427, 01/2013, Volume 151, Issue 1-2, pp. 49 - 62
The heart is considered the powerhouse of the cardiovascular system. Heart and skeletal muscle inflammation (HSMI), cardiomyopathy syndrome (CMS) and pancreas... 
Immunohistochemistry | CMS | Atlantic salmon | HSMI | RAINBOW-TROUT | CELL NUCLEAR ANTIGEN | NECROSIS-FACTOR-ALPHA | DNA-SYNTHESIS | IMMUNOLOGY | ONCORHYNCHUS-MYKISS WALBAUM | MHC CLASS-I | BETA-CHAIN | LYMPHOID-TISSUE | VETERINARY SCIENCES | TNF-ALPHA | AMEBIC-GILL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Myocardium - immunology | Caspase 3 - metabolism | Salmo salar - metabolism | Myositis - immunology | Histocompatibility Antigens Class I - metabolism | Pancreatic Diseases - pathology | Pancreatic Diseases - immunology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myocardium - metabolism | Histocompatibility Antigens Class II - metabolism | Myositis - pathology | Myocarditis - immunology | Cardiomyopathies - immunology | Biomarkers - metabolism | Salmo salar - anatomy & histology | Pancreatic Diseases - metabolism | Pancreatic Diseases - veterinary | Myositis - metabolism | Myositis - veterinary | Fish Proteins - metabolism | Cardiomyopathies - pathology | Myocardium - pathology | CD3 Complex - metabolism | Fish Diseases - pathology | Salmo salar - immunology | CD8 Antigens - metabolism | Animals | Cardiomyopathies - metabolism | Cardiomyopathies - veterinary | Myocarditis - metabolism | Myocarditis - veterinary | Myocarditis - pathology | Proliferating Cell Nuclear Antigen - metabolism | Fish Diseases - metabolism
Journal Article
Journal Article
International Journal of Cardiology, ISSN 0167-5273, 2010, Volume 151, Issue 1, pp. 18 - 33
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 2, pp. E155 - E164
Autoimmune response to cardiac troponin I (TnI) induces inflammation and fibrosis in the myocardium. High-mobility group box 1 (HMGB1) is a multifunctional... 
Myocarditis | Cytokines | AAV | STEM-CELLS | MYOCARDIAL-INFARCTION | myocarditis | MULTIDISCIPLINARY SCIENCES | RECEPTOR | ISCHEMIA-REPERFUSION INJURY | T-CELL-ACTIVATION | MOBILITY GROUP BOX-1 | cytokines | RAT-HEART | GENE-TRANSFER | CARDIAC TROPONIN-I | GLYCATION END-PRODUCTS | Inflammation - pathology | Transcription, Genetic - drug effects | NF-kappa B - metabolism | Inflammation - blood | Freund's Adjuvant - immunology | Inflammation - complications | HMGB1 Protein - metabolism | Myocardium - metabolism | Protein Binding - drug effects | Heart Diseases - complications | Inflammation Mediators - metabolism | Heart Diseases - blood | Toll-Like Receptors - metabolism | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Heart Function Tests | Myocarditis - genetics | Receptor for Advanced Glycation End Products - blood | Troponin - metabolism | Immunization | Dependovirus - metabolism | Myocarditis - complications | Receptor for Advanced Glycation End Products - metabolism | Myocardium - pathology | Down-Regulation - drug effects | Mice, Knockout | Up-Regulation - drug effects | Animals | Signal Transduction - drug effects | Biopsy | Fibrosis | Inflammation - genetics | Matrix Metalloproteinases - metabolism | Myocarditis - pathology | Myocarditis - physiopathology | HMGB1 Protein - blood | Heart Diseases - genetics | Heart Diseases - pathology | Immunologic Factors - pharmacology | Physiological aspects | Immune response | Observations | Heart diseases | Biological Sciences | PNAS Plus
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 484, Issue 7397, pp. 251 - 255
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 04/2017, Volume 113, Issue 5, pp. 475 - 487
Monocytes/macrophages response plays a key role in post-infarction inflammation that contributes greatly to post-infarction ventricular remodelling and cardiac... 
Myocardial infarction | Topiramate | Monocyte | GABAA receptor | Macrophage | Monocytes - metabolism | Time Factors | Fructose - pharmacology | Myocardial Infarction - pathology | Myocardium - metabolism | Receptors, GABA-A - drug effects | Myocarditis - prevention & control | Antigens, Ly - metabolism | Myocardial Infarction - physiopathology | Heart Rupture, Post-Infarction - prevention & control | Heart Ventricles - pathology | Disease Models, Animal | Receptors, GABA - drug effects | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Myocardium - pathology | Myocardial Infarction - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Monocytes - drug effects | Ventricular Dysfunction, Left - metabolism | Collagen - metabolism | Fructose - analogs & derivatives | Macrophages - metabolism | Phenotype | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Fibrosis | GABA Agonists - pharmacology | Myocarditis - metabolism | Receptors, GABA - metabolism | Macrophages - drug effects | Heart Rupture, Post-Infarction - metabolism | Heart Ventricles - metabolism | Receptors, GABA-A - metabolism | Myocarditis - pathology | Heart Rupture, Post-Infarction - physiopathology | Myocarditis - physiopathology | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects
Journal Article
Journals of Gerontology - Series A Biological Sciences and Medical Sciences, ISSN 1079-5006, 08/2011, Volume 66, Issue 8, pp. 823 - 834
The effect of a chronic combined treatment with growth hormone (GH) plus melatonin (Mel) on different age-related processes in cytosolic and nuclear fractions... 
Heart | Aging | Oxidative stress | Inflammation | Apoptosis | OXIDATIVE STRESS PARAMETERS | BODY-COMPOSITION | OLD FEMALE RATS | GERIATRICS & GERONTOLOGY | INFLAMMATORY GENE-EXPRESSION | NITRIC-OXIDE | GERONTOLOGY | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | BCL-2 FAMILY-MEMBERS | SENESCENCE-ACCELERATED MICE | OXIDE-INDUCED APOPTOSIS | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - genetics | Oxidative Stress | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | Nitric Oxide Synthase - genetics | RNA, Messenger - metabolism | Melatonin - administration & dosage | Cell Nucleus - metabolism | Time Factors | Isoenzymes - metabolism | Injections, Subcutaneous | Myocardium - metabolism | Muscle Proteins - metabolism | Interleukin-10 - metabolism | Human Growth Hormone - administration & dosage | Heme Oxygenase (Decyclizing) - metabolism | Heme Oxygenase (Decyclizing) - genetics | Biomarkers - metabolism | Interleukin-1 - metabolism | Drug Administration Schedule | Administration, Oral | Isoenzymes - genetics | Drug Synergism | Animals | NF-kappa B - genetics | Interleukin-10 - genetics | Cytosol - metabolism | Mice | Nitric Oxide Synthase - metabolism | Myocarditis - physiopathology | Aging - metabolism | Animal experimentation | Somatotropin | Usage | Care and treatment | Melatonin | Messenger RNA | Physiological aspects | Research | Gene expression | Health aspects
Journal Article