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Circulation research, ISSN 1524-4571, 2017, Volume 121, Issue 7, pp. 838 - 854
The normal function of the human myocardium requires the proper generation and utilization of energy and relies on a series of complex metabolic processes to... 
Noncompaction cardiomyopathy | Cardiac failure | Cardiomyopathy | Infiltrative cardiomyopathy | Metabolic cardiomyopathy | infiltrative cardiomyopathy | DISTINCT CARDIOMYOPATHY | CARDIAC & CARDIOVASCULAR SYSTEMS | DANON-DISEASE | NEUROMUSCULAR DISORDERS | BARTH-SYNDROME | DILATED CARDIOMYOPATHY | ACYLGLYCEROL KINASE | NON-COMPACTION CARDIOMYOPATHY | SKELETAL-MUSCLE | noncompaction cardiomyopathy | HYPERTROPHIC CARDIOMYOPATHY | metabolic cardiomyopathy | CONGENITAL HEART-DISEASE | PERIPHERAL VASCULAR DISEASE | cardiomyopathy | HEMATOLOGY | cardiac failure | Isolated Noncompaction of the Ventricular Myocardium - genetics | Prognosis | Humans | Mitochondrial Diseases - metabolism | Metabolism, Inborn Errors - therapy | Cardiomyopathies - genetics | Molecular Diagnostic Techniques | Myocardium - metabolism | Cardiomyopathies - diagnosis | Mitochondrial Diseases - therapy | Mitochondrial Diseases - genetics | Genetic Predisposition to Disease | Isolated Noncompaction of the Ventricular Myocardium - therapy | Risk Factors | Metabolism, Inborn Errors - metabolism | Myocardium - pathology | Genetic Markers | Metabolism, Inborn Errors - genetics | Cardiomyopathies - therapy | Metabolism, Inborn Errors - diagnosis | Phenotype | Animals | Biopsy | Cardiomyopathies - metabolism | Isolated Noncompaction of the Ventricular Myocardium - metabolism | Mitochondrial Diseases - diagnosis | Isolated Noncompaction of the Ventricular Myocardium - diagnosis | Inborn errors of metabolism | Cardiac muscle | Metabolism | Skeletal muscle | Storage diseases | Mitochondria | Myocardium | Cytoskeleton | Ventricle | Mutation | Heart diseases | Structure-function relationships
Journal Article
The Lancet (British edition), ISSN 0140-6736, 2015, Volume 386, Issue 9995, pp. 813 - 825
Summary Left ventricular non-compaction, the most recently classified form of cardiomyopathy, is characterised by abnormal trabeculations in the left... 
Internal Medicine | MEDICINE, GENERAL & INTERNAL | PRACTICE GUIDELINES | CONGENITAL HEART-DISEASE | ISOLATED NONCOMPACTION | AMERICAN-COLLEGE | ASSOCIATION TASK-FORCE | BARTH-SYNDROME | LONG QT SYNDROME | TERM CLINICAL-COURSE | TRANSCRIPTION FACTOR | SPONGY MYOCARDIUM | Cardiomyopathy, Dilated - classification | Echocardiography | Isolated Noncompaction of the Ventricular Myocardium - therapy | Humans | Isolated Noncompaction of the Ventricular Myocardium - classification | Magnetic Resonance Angiography | Anticoagulants - therapeutic use | Male | Treatment Outcome | Arrhythmias, Cardiac - etiology | Signal Transduction - genetics | Mutation - genetics | Animals | Defibrillators, Implantable | Electrocardiography | Cardiomyopathy, Dilated - diagnosis | Female | Mice | Cardiomyopathy, Dilated - therapy | Disease Models, Animal | Isolated Noncompaction of the Ventricular Myocardium - diagnosis | Heart failure | Heart | Genetic disorders | Cardiomyopathy | Heart diseases | Jaw | Regulators | Embryo cells | Elastic scattering | Infants | Biochemistry | Compaction | Incidence | Proteins | Signal transduction | Diagnosis | Cardiac muscle | Abnormalities | Fetuses | Benign | Recesses | Endothelium | Blood circulation | Morphology | Stem cells | Myocardium | Death | Adults | Notch protein | Cardiovascular diseases | Cardiac arrhythmia | Calcineurin | Risk | Valves | Chin | Transplantation | Defects | Morphogenesis | Children | Cardiology | Ultrasound | Age | Stroke | Health risks | Septum | Ribonucleic acid--RNA | Catheters | Solidification | Magnetic resonance imaging | Ventricle | Methylation | Differentiation
Journal Article
PLoS genetics, ISSN 1553-7404, 2018, Volume 14, Issue 7, p. e1007502
Left ventricular non-compaction (LVNC) is a rare cardiomyopathy associated with a hypertrabeculated phenotype and a large spectrum of symptoms. It is still... 
CONTRACTION DEFECTS | CONDUCTION SYSTEM | NEUROMUSCULAR DISORDERS | CONGENITAL HEART-DISEASE | MOUSE MODEL | MUTATION | GENETICS & HEREDITY | GENE-EXPRESSION | MICE | NONCOMPACTION CARDIOMYOPATHY | PURKINJE-FIBER NETWORK | Sequence Deletion | Up-Regulation | Isolated Noncompaction of the Ventricular Myocardium - genetics | Homeodomain Proteins - metabolism | Humans | Isolated Noncompaction of the Ventricular Myocardium - complications | Heart Ventricles - embryology | Homeobox Protein Nkx-2.5 - metabolism | Gene Expression Profiling | Isolated Noncompaction of the Ventricular Myocardium - pathology | Gene Expression Regulation, Developmental | Myocardium - metabolism | Female | Homeobox Protein Nkx-2.5 - genetics | Heart Ventricles - pathology | Disease Models, Animal | Severity of Illness Index | Morphogenesis - genetics | Heart Failure - genetics | Myocardium - pathology | Mice, Knockout | Purkinje Fibers - pathology | Animals | Fibrosis | Mice | Isolated Noncompaction of the Ventricular Myocardium - diagnosis | Development and progression | Genetic aspects | Cardiomyopathy | Heart diseases | Heart ventricle, Left | Heart | Heart failure | Hypoplasia | Conduction | Phenotypes | Transcription factors | Nuclear magnetic resonance--NMR | Funding | Fetuses | Cardiomyocytes | Conditional mutant | Compaction | Gene expression | Embryos | Pathogenicity | Morphogenesis | Etiology | Myocardium | Mutation | Ventricle | Supervision | Nkx2.5 protein | Life Sciences | Human health and pathology | Cardiology and cardiovascular system | Nuclear magnetic resonance | NMR
Journal Article
Journal of cell science, ISSN 1477-9137, 2008, Volume 121, Issue 22, pp. 3794 - 3802
Journal Article
Journal Article