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Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 2014, Volume 63, Issue 6, pp. 1241 - 1250
Connective tissue growth factor (CTGF) is involved in the pathogenesis of various fibrotic disorders. However, its role in the heart is not clear. To... 
Hypertension | Inflammation | S100 proteins | Angiotensin II | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Immunohistochemistry | Calgranulin B - metabolism | Oligonucleotide Array Sequence Analysis | Myocytes, Cardiac - immunology | Calgranulin A - genetics | Inflammation - metabolism | Myocardium - metabolism | Antibodies - immunology | Antigens, Ly - metabolism | Calgranulin A - immunology | Hypertension - genetics | Cytokines - genetics | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Neutrophils - drug effects | Toll-Like Receptor 4 - genetics | Hypertension - immunology | Toll-Like Receptor 4 - immunology | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Fibroblasts - immunology | Mice | Receptors, Immunologic - genetics | Myocardium - immunology | Calgranulin A - metabolism | Transcriptome - immunology | Calgranulin B - immunology | Hypertension - prevention & control | Calgranulin B - genetics | Receptors, Immunologic - immunology | Cytokines - immunology | CD11b Antigen - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Myocardium - pathology | Inflammation - immunology | Transcriptome - drug effects | Angiotensin II - immunology | Antibodies - pharmacology | Animals | Antigens, Ly - immunology | Inflammation - genetics | CD11b Antigen - metabolism | Receptors, Immunologic - metabolism | Index Medicus
Journal Article
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 07/2015, Volume 195, Issue 2, pp. 672 - 682
Cardiac dysfunction is a major consequence of sepsis/septic shock and contributes to the high mortality of sepsis. Innate and inflammatory responses mediated... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Interleukin-1 Receptor-Associated Kinases - genetics | Myocardium - immunology | Carotid Arteries | Genetic Vectors - administration & dosage | Myocytes, Cardiac - immunology | NF-kappa B - immunology | Male | Sepsis - complications | TNF Receptor-Associated Factor 6 - antagonists & inhibitors | TNF Receptor-Associated Factor 6 - immunology | Lipopolysaccharides - antagonists & inhibitors | Heart Failure - therapy | Interleukin-1 Receptor-Associated Kinases - immunology | Heart Failure - immunology | Lentivirus - genetics | TNF Receptor-Associated Factor 6 - genetics | Heart Failure - etiology | Macrophages - immunology | Cytokines - immunology | Disease Models, Animal | NF-kappa B - antagonists & inhibitors | Sepsis - immunology | Macrophages - pathology | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Heart Failure - genetics | Myocardium - pathology | Immunity, Innate | MicroRNAs - immunology | Sepsis - genetics | Myocytes, Cardiac - pathology | Administration, Intravenous | Animals | Myocytes, Cardiac - drug effects | NF-kappa B - genetics | Interleukin-1 Receptor-Associated Kinases - antagonists & inhibitors | Sepsis - therapy | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | MicroRNAs - genetics | Cytokines - antagonists & inhibitors | Primary Cell Culture | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus | microRNA-146a | NF-κB | cardiac function | TRAF6 | sepsis
Journal Article
Cardiovascular research, ISSN 1755-3245, 06/2019, Volume 115, Issue 7, pp. 1117 - 1130
New therapies are required to reduce myocardial infarct ( MI) size and prevent the onset of heart failure in patients presenting with acute myocardial... 
Monocytes | Dendritic cells | Lymphocytes | Myocardial ischaemia/reperfusion injury | Fibroblasts | Inflammation | Acute myocardial infarction | Macrophages | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Myocardium - immunology | Dendritic Cells - immunology | Humans | Monocytes - metabolism | Monocytes - immunology | Myocardial Infarction - immunology | Heart Failure - prevention & control | Myocardial Infarction - therapy | Myocardial Reperfusion Injury - pathology | Anti-Inflammatory Agents - adverse effects | Lymphocytes - immunology | Myocardial Infarction - pathology | Myocardium - metabolism | Heart Failure - immunology | Inflammation Mediators - metabolism | Mast Cells - metabolism | Anti-Inflammatory Agents - therapeutic use | Dendritic Cells - drug effects | Neutrophils - metabolism | Dendritic Cells - metabolism | Fibroblasts - metabolism | Myocardial Reperfusion Injury - immunology | Inflammation Mediators - immunology | Lymphocytes - metabolism | Mast Cells - immunology | Signal Transduction | Neutrophils - drug effects | Ventricular Function, Left - drug effects | Neutrophils - immunology | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Myocardial Infarction - metabolism | Mast Cells - drug effects | Monocytes - drug effects | Cardiovascular Agents - therapeutic use | Myocardial Reperfusion Injury - metabolism | Animals | Fibroblasts - drug effects | Lymphocytes - drug effects | Fibroblasts - immunology | Cardiovascular Agents - adverse effects | Ventricular Remodeling - drug effects | Myocardial Reperfusion Injury - prevention & control | Index Medicus | Invited Spotlight Reviews | Myocardial ischaemia | reperfusion injury
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 8/2016, Volume 113, Issue 35, pp. 9816 - 9821
Clathrin, a cytosolic protein composed of heavy and light chain subunits, assembles into a vesicle coat, controlling receptor-mediated endocytosis. To... 
TGFβ | Clathrin light chain | Endocytosis | G protein-coupled receptors | Antibody isotype switch | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptors, Transforming Growth Factor beta - agonists | Receptors, Transforming Growth Factor beta - genetics | Myocardium - immunology | Receptors, Transforming Growth Factor beta - immunology | Spleen - immunology | Immunoglobulin A - biosynthesis | Humans | Immunoglobulin A - genetics | Receptors, Adrenergic, beta-2 - immunology | Receptors, Opioid, delta - immunology | Cerebral Cortex - cytology | Liver - immunology | Receptors, Opioid, delta - genetics | Gene Deletion | Receptors, CXCR4 - genetics | B-Lymphocytes - pathology | Clathrin Light Chains - genetics | Immunoglobulin Class Switching | Endocytosis - immunology | Receptors, Adrenergic, beta-2 - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Spleen - cytology | Organ Specificity | Mice, Knockout | Myocardium - cytology | Animals | B-Lymphocytes - immunology | Receptors, CXCR4 - immunology | T-Lymphocytes - cytology | Cerebral Cortex - immunology | Protein-Serine-Threonine Kinases - immunology | T-Lymphocytes - immunology | Liver - cytology | Mice | Clathrin Light Chains - immunology | Viral antibodies | Clathrin | Cell research | Antibodies | Physiological aspects | Research | Proteins | Signal transduction | Molecules | Cellular biology | Lymphocytes | Rodents | Index Medicus | antibody isotype switch | Biological Sciences | endocytosis | clathrin light chain
Journal Article