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Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7527, pp. 431 - 435
Journal Article
FASEB Journal, ISSN 0892-6638, 2005, Volume 19, Issue 13, pp. 1890 - 1892
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2016, Volume 311, Issue 2, pp. H347 - H363
Dramatic maturational changes in cardiac energy metabolism occur in the newborn period, with a shift from glycolysis to fatty acid oxidation. Acetylation and... 
Lysine acetylation | Lysine succinylation | Myocardial fatty acid oxidation | Newborn heart | MITOCHONDRIAL-PROTEIN-ACETYLATION | BIVENTRICULAR WORKING HEARTS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GLUCOSE-OXIDATION | lysine acetylation | NEONATAL RABBIT HEARTS | lysine succinylation | FATTY-ACID OXIDATION | INSULIN-RESISTANCE | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | OVERLOAD HYPERTROPHY | myocardial fatty acid oxidation | CONTRACTILE FUNCTION | newborn heart | Phosphoglycerate Mutase - metabolism | Immunoprecipitation | Mitochondria, Heart - metabolism | 3-Hydroxyacyl CoA Dehydrogenases - metabolism | Immunoblotting | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Lysine - metabolism | Acetylation | Fatty Acids - metabolism | Fetal Heart - metabolism | Animals, Newborn | Cell Line | Rabbits | Oxidation-Reduction | Acyl-CoA Dehydrogenase, Long-Chain - metabolism | Rats | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Energy Metabolism | Succinic Acid - metabolism | Glycolysis | Protein Processing, Post-Translational | Hexokinase - metabolism | In Vitro Techniques | Heart | Infants (Newborn) | Physiological aspects | Energy metabolism | Bioenergetics | Phosphorylation | RNA-protein interactions | Metabolism | Kinases | Fatty acids | Adenosine triphosphatase | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. e9 - e18
Persistent inflammatory response has adverse effects on left ventricular (LV) function and remodeling following acute myocardial infarction. We hypothesized... 
Myocardial infarction | Inflammation | Cardiac remodeling | Cytokines | IL-10 | CARDIAC-FUNCTION | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | myocardial infarction | INTERLEUKIN-10 | FACTOR-ALPHA | MATRIX METALLOPROTEINASES | TUMOR-NECROSIS-FACTOR | inflammation | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | cytokines | DYSFUNCTION | HEMATOLOGY | EXPRESSION | ELAV-Like Protein 1 | NIH 3T3 Cells | RNA-Binding Proteins - genetics | Phosphorylation | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Anti-Inflammatory Agents - metabolism | ELAV Proteins | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | RNA Interference | Time Factors | Matrix Metalloproteinase 9 - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Inflammation Mediators - metabolism | Interleukin-10 - administration & dosage | Antigens, Surface - metabolism | Interleukin-10 - metabolism | Anti-Inflammatory Agents - administration & dosage | Myocardial Infarction - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Capillaries - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Recombinant Proteins - metabolism | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Antigens, Surface - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Animals | Arterioles - metabolism | Fibrosis | Inflammation - prevention & control | Mice | Ventricular Remodeling - drug effects | Neovascularization, Physiologic | RNA-Binding Proteins - metabolism | Apoptosis | Inflammation - physiopathology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 79, pp. 275 - 283
Abstract Background Obesity leads to metabolic heart disease (MHD) that is associated with a pathologic increase in myocardial fatty acid (FA) uptake and... 
Cardiovascular | Obesity | Metabolic heart disease | Mitochondria | Lipid excess | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PROLIFERATOR-ACTIVATED RECEPTOR | KINASE-C | FATTY-ACIDS | CELL BIOLOGY | REDUCES DIABETIC CARDIOMYOPATHY | SKELETAL-MUSCLE | LIPOTOXIC CARDIOMYOPATHY | FAILING HEART | COACTIVATOR PGC-1 | TRANSCRIPTIONAL CONTROL | Mitochondria, Heart - ultrastructure | Phosphorylation | Mitochondria, Heart - metabolism | Carnitine - metabolism | RNA, Messenger - metabolism | Fatty Acid Transport Proteins - metabolism | Diglycerides - metabolism | Protein Kinase C - metabolism | Adenosine Triphosphate - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Carnitine - analogs & derivatives | Proto-Oncogene Proteins c-akt - metabolism | Myocardium - ultrastructure | Ceramides - metabolism | Sphingomyelins - metabolism | RNA, Messenger - genetics | Gene Expression Regulation | Oxygen Consumption | Lipids - adverse effects | Myocardium - pathology | Organ Specificity | Hydrogen Peroxide - metabolism | Catalase - metabolism | Transcription Factors - metabolism | Animals | Electron Transport Complex II - metabolism | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Myocytes, Cardiac - metabolism | Mice | PPAR alpha - metabolism | Fatty acids | Analysis | Mitochondrial DNA | Index Medicus | Metabolic Heart Disease | Lipid Excess
Journal Article
1983, ISBN 0471102490, x, 552
Book
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, pp. e35905 - e35905
Hematopoietic progenitor CD133(+)/c-kit(+) cells have been shown to be involved in myocardial healing following myocardial infarction (MI). Previously we... 
CD31(+) CELLS | ENDOTHELIAL PROGENITOR CELLS | ISCHEMIA/REPERFUSION INJURY | ACTIVATION | ISCHEMIC VASCULAR-DISEASE | ANGIOGENIC ACTIVITY | BONE-MARROW | BIOLOGY | RECEPTOR | TOPCARE-AMI | EXPRESSION | Up-Regulation | Capillaries - pathology | Receptors, Notch - metabolism | Cardiomegaly - pathology | Angiopoietin-1 - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Serrate-Jagged Proteins | Bone Marrow - metabolism | Myocardium - metabolism | Endomyocardial Fibrosis - complications | Myocardial Infarction - physiopathology | Capillaries - metabolism | Apelin | Jagged-1 Protein | Capillaries - physiopathology | Signal Transduction | Cardiomegaly - physiopathology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Chemokine CXCL12 - metabolism | Hematopoietic Stem Cells - cytology | Endomyocardial Fibrosis - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Cardiomegaly - metabolism | Cell Movement | Actins - metabolism | Glycoproteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Myocardial Infarction - pathology | Endomyocardial Fibrosis - pathology | Membrane Proteins - metabolism | Heart Function Tests | Diabetes Mellitus, Type 2 - complications | Calcium-Binding Proteins - metabolism | Myocardium - pathology | Adipokines | AC133 Antigen | Cardiomegaly - complications | Myocardial Infarction - complications | Animals | Diabetes Mellitus, Type 2 - physiopathology | Bone Marrow - pathology | Receptor, Notch3 | Endomyocardial Fibrosis - physiopathology | Apoptosis | Advertising executives | Vascular endothelial growth factor | Adenoviruses | Heart attack | Myocardial infarction | Heart | Diabetic retinopathy | Heart attacks | Angiopoietin | Recovery of function | Smooth muscle | Cardiovascular disease | Recruitment | Angiogenesis | Toxicology | Cell growth | Ischemia | Data recovery | Rodents | Bone marrow | Repair | Heart diseases | Immunoglobulins | Diabetes mellitus | Coronary artery | Muscles | Pharmacology | c-Kit protein | Hemopoiesis | Studies | Ostomy | Coronary vessels | Fibrosis | Stem cells | Myocardium | Infarction | Diabetes | Laboratory animals | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2009, Volume 106, Issue 38, pp. 16381 - 16386
We used the muscle creatine kinase (MCK) conditional frataxin knockout mouse to elucidate how frataxin deficiency alters iron metabolism. This is of... 
Up regulation | Molecules | Mitochondria | Energy metabolism | Messenger RNA | Reverse transcriptase polymerase chain reaction | Lead | Iron | Friedreich ataxia | Down regulation | Frataxin | Iron transferrin | Heme synthesis | CELLS | heme synthesis | FRATAXIN HOMOLOG | HEME-SYNTHESIS | MULTIDISCIPLINARY SCIENCES | transferrin | INTERLEUKIN-6 | DEFICIENCY | frataxin | CLUSTER SYNTHESIS | METABOLISM | iron | MICE | PROTEINS | EXPRESSION | Heme - metabolism | Oligonucleotide Array Sequence Analysis | Friedreich Ataxia - pathology | Humans | Gene Expression Profiling | Antimicrobial Cationic Peptides - metabolism | Iron-Binding Proteins - metabolism | Friedreich Ataxia - genetics | Kidney - metabolism | Carbon-Sulfur Lyases - genetics | Hepcidins | Myocardium - metabolism | Antimicrobial Cationic Peptides - genetics | Disease Models, Animal | Friedreich Ataxia - metabolism | Liver - metabolism | Uroporphyrinogen III Synthetase - genetics | Uroporphyrinogen III Synthetase - metabolism | Mitochondria - metabolism | Coproporphyrinogen Oxidase - genetics | Reverse Transcriptase Polymerase Chain Reaction | Carbon-Sulfur Lyases - metabolism | Iron - metabolism | Blotting, Western | Mice, Knockout | Myocardium - cytology | Animals | Spleen - metabolism | Ferrochelatase - metabolism | Ferrochelatase - genetics | Iron-Binding Proteins - genetics | Mice | Coproporphyrinogen Oxidase - metabolism | Tissue | Rodents | Vitamin deficiency | Mutation | Kinases | Metabolism | Index Medicus | Biological Sciences
Journal Article
Endocrinology, ISSN 0013-7227, 02/2016, Volume 157, Issue 2, pp. 737 - 751
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2007, Volume 293, Issue 2, pp. 909 - 918
Recent studies have uncovered important cross talk between inflammation, generation of reactive oxygen and nitrogen species, and lipid metabolism in the... 
Pressure-volume relationship | Cardiac function | Endocannabinoids | Anandamide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANANDAMIDE HYDROLYSIS | HEART-FAILURE | ENDOGENOUS CANNABINOID SYSTEM | ENDOCANNABINOID SYSTEM | RECEPTOR | KNOCKOUT MICE | CORONARY-ARTERIES | HEMODYNAMIC PROFILE | pressure-volume relationship | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | cardiac function | endocannabinoids | VASCULAR ENDOTHELIAL-CELLS | anandamide | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Humans | Monocytes - metabolism | NF-kappa B - metabolism | Coronary Vessels - metabolism | Receptors, Cannabinoid - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Aging - genetics | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Amidohydrolases - deficiency | Amidohydrolases - metabolism | Endothelial Cells - metabolism | Reactive Nitrogen Species - metabolism | Amidohydrolases - genetics | Cells, Cultured | Gene Expression Regulation | Polyunsaturated Alkamides - metabolism | Cell Adhesion | Aging - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Myocardium - enzymology | Animals | Coronary Vessels - cytology | Inflammation - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Aging - metabolism | Apoptosis | Studies | Cardiovascular system | Rodents | Aging | Gene expression | Fatty acids | Index Medicus
Journal Article