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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2007, Volume 293, Issue 2, pp. 909 - 918
Recent studies have uncovered important cross talk between inflammation, generation of reactive oxygen and nitrogen species, and lipid metabolism in the pathogenesis of cardiovascular aging... 
Pressure-volume relationship | Cardiac function | Endocannabinoids | Anandamide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANANDAMIDE HYDROLYSIS | HEART-FAILURE | ENDOGENOUS CANNABINOID SYSTEM | ENDOCANNABINOID SYSTEM | RECEPTOR | KNOCKOUT MICE | CORONARY-ARTERIES | HEMODYNAMIC PROFILE | pressure-volume relationship | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | cardiac function | endocannabinoids | VASCULAR ENDOTHELIAL-CELLS | anandamide | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Humans | Monocytes - metabolism | NF-kappa B - metabolism | Coronary Vessels - metabolism | Receptors, Cannabinoid - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Aging - genetics | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Amidohydrolases - deficiency | Amidohydrolases - metabolism | Endothelial Cells - metabolism | Reactive Nitrogen Species - metabolism | Amidohydrolases - genetics | Cells, Cultured | Gene Expression Regulation | Polyunsaturated Alkamides - metabolism | Cell Adhesion | Aging - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Myocardium - enzymology | Animals | Coronary Vessels - cytology | Inflammation - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Aging - metabolism | Apoptosis
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 515, Issue 7527, pp. 431 - 435
Journal Article
Journal Article
Journal of internal medicine, ISSN 0954-6820, 2013, Volume 274, Issue 1, pp. 25 - 40
Obesity increases the risk of metabolic diseases, including insulin resistance and type 2 diabetes, as well as cardiovascular disease. In addition to lipid... 
lipids | NAFLD | storage | lipid droplets | insulin resistance | lipotoxicity | Insulin resistance | Lipids | Storage | Lipotoxicity | Lipid droplets | TRIGLYCERIDE TRANSFER PROTEIN | DE-NOVO LIPOGENESIS | LIVER-SPECIFIC DELETION | LOW-DENSITY LIPOPROTEIN | SKELETAL-MUSCLE | MEDICINE, GENERAL & INTERNAL | TYROSINE-PHOSPHATASE 1B | FATTY-ACID OXIDATION | APOLIPOPROTEIN-B SECRETION | PLECKSTRIN HOMOLOGY DOMAIN | DIFFERENTIATION-RELATED PROTEIN | Humans | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Phosphoproteins - metabolism | Inflammation - metabolism | Myocardium - metabolism | Lipoproteins - metabolism | Diabetes Mellitus, Type 2 - etiology | Lipogenesis | Fatty Liver - metabolism | Signal Transduction | Obesity - complications | Liver - metabolism | Insulin Resistance | Lipid Metabolism | Heart Failure - metabolism | Immunity, Innate | Toll-Like Receptor 4 - metabolism | Obesity - metabolism | Triglycerides - metabolism | Animals | Carrier Proteins - metabolism | Metabolic Diseases - metabolism | Lipoproteins, VLDL - metabolism | alpha-2-HS-Glycoprotein - metabolism | Perilipin-1 | Type 2 diabetes | Muscles | Development and progression | pleckstrin homology domain | Klinisk medicin | fatty-acid oxidation | low-density lipoprotein | apolipoprotein-b secretion | triglyceride transfer protein | Clinical Medicine | deletion | tyrosine-phosphatase 1b | endoplasmic-reticulum stress | droplet-associated protein | differentiation-related protein | liver-specific
Journal Article
Circulation research, ISSN 1524-4571, 2009, Volume 104, Issue 2, pp. e9 - e18
Persistent inflammatory response has adverse effects on left ventricular (LV) function and remodeling following acute myocardial infarction. We hypothesized... 
Myocardial infarction | Inflammation | Cardiac remodeling | Cytokines | IL-10 | CARDIAC-FUNCTION | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | myocardial infarction | INTERLEUKIN-10 | FACTOR-ALPHA | MATRIX METALLOPROTEINASES | TUMOR-NECROSIS-FACTOR | inflammation | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | cytokines | DYSFUNCTION | HEMATOLOGY | EXPRESSION | ELAV-Like Protein 1 | NIH 3T3 Cells | RNA-Binding Proteins - genetics | Phosphorylation | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Anti-Inflammatory Agents - metabolism | ELAV Proteins | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | RNA Interference | Time Factors | Matrix Metalloproteinase 9 - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Inflammation Mediators - metabolism | Interleukin-10 - administration & dosage | Antigens, Surface - metabolism | Interleukin-10 - metabolism | Anti-Inflammatory Agents - administration & dosage | Myocardial Infarction - physiopathology | p38 Mitogen-Activated Protein Kinases - metabolism | Capillaries - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Recombinant Proteins - metabolism | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Antigens, Surface - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Animals | Arterioles - metabolism | Fibrosis | Inflammation - prevention & control | Mice | Ventricular Remodeling - drug effects | Neovascularization, Physiologic | RNA-Binding Proteins - metabolism | Apoptosis | Inflammation - physiopathology | RNA, Small Interfering - metabolism
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 05/2007, Volume 34, Issue 5, pp. 425 - 431
SUMMARY • It is known that stress causes disruption of homeostasis and an imbalanced anti-oxidant status in several organs. The aim of the present study was to... 
immobilization-cold stress | rat | glutathione | protein oxidation | cold stress | anti-oxidant enzymes | lipid peroxidation | immobilization stress | anti‐oxidant enzymes | immobilization–cold stress | Cold stress | Protein oxidation | Rat | Anti-oxidant enzymes | Immobilization-cold stress | Lipid peroxidation | Immobilization stress | Glutathione | CATALASE | ENZYME-ACTIVITIES | DEFENSE | PHYSIOLOGY | GLUCOCORTICOIDS | GLUTATHIONE-PEROXIDASE | BODY-WEIGHT | METABOLISM | AGE-RELATED-CHANGES | PHARMACOLOGY & PHARMACY | BRAIN | EXPOSURE | Protein Carbonylation | Rats, Wistar | Glutathione - metabolism | Zinc - metabolism | Antioxidants - metabolism | Male | Stress, Psychological - psychology | Immobilization - psychology | Kidney - metabolism | Myocardium - metabolism | Copper - metabolism | Corticosterone - blood | Weight Gain | Superoxide Dismutase - metabolism | Glutathione Peroxidase - metabolism | Cold Temperature | Liver - metabolism | Rats | Thiobarbituric Acid Reactive Substances - metabolism | Kidney - cytology | Liver - chemistry | Lipid Peroxidation - physiology | Myocardium - cytology | Catalase - metabolism | Animals | Selenium - metabolism | Models, Biological | Weight Loss | Kidney - chemistry | Liver - cytology | Liver, cytology | Thiobarbituric Acid Reactive Substances, metabolism | Superoxide Dismutase, metabolism | Zinc, metabolism | Antioxidants, metabolism | Kidney, metabolism | Glutathione, metabolism | Myocardium, metabolism | Immobilization, psychology | Corticosterone, blood | Catalase, metabolism | Selenium, metabolism | Liver, chemistry | Cold | Copper, metabolism | Lipid Peroxidation, physiology | Myocardium, cytology | Kidney, chemistry | Liver, metabolism | Glutathione Peroxidase, metabolism | Stress, Psychological, psychology | Kidney, cytology | Antioxidants | Comparative analysis | Oxidation-reduction reaction
Journal Article
The FASEB Journal, ISSN 0892-6638, 11/2005, Volume 19, Issue 13, pp. 1890 - 1892
ABSTRACTWe investigated a possible beneficial role for bilirubin, one of the products of heme degradation by the cytoprotective enzyme heme oxygenase‐1 in... 
nitric oxide | oxygen free radicals | OXIDATIVE STRESS | SUPEROXIDE | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | BILIVERDIN | CELL BIOLOGY | SYNTHASE EXPRESSION | ANTIOXIDANT | IN-VIVO | BIOLOGY | HEME OXYGENASE-1 | SERUM BILIRUBIN | Nitric Oxide Synthase Type II - biosynthesis | Bilirubin - metabolism | Antioxidants - metabolism | Kidney - enzymology | Malondialdehyde - chemistry | NADPH Oxidases - metabolism | Male | Aorta - metabolism | Kidney - metabolism | Time Factors | Myocardium - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | Glutathione Peroxidase - metabolism | Free Radicals | Down-Regulation | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Rats, Sprague-Dawley | Blotting, Western | Nitrates - metabolism | Macrophages - metabolism | Rats, Gunn | Models, Biological | Lipopolysaccharides - chemistry | Mice | Nitric Oxide Synthase - metabolism | Blood Pressure | Jaundice - pathology | Tumor Necrosis Factor-alpha - metabolism | Nitric Oxide Synthase Type II - chemistry | Nitrites - metabolism | Shock, Septic - prevention & control | Oxygen - metabolism | Shock, Septic - metabolism | Heme - chemistry | Escherichia coli - metabolism | Shock, Septic - drug therapy | Interleukin-10 - metabolism | Aorta - enzymology | Superoxide Dismutase - metabolism | Cell Line | Bilirubin - chemistry | Models, Statistical | Homozygote | Myocardium - enzymology | Animals | Nitric Oxide - metabolism
Journal Article
Journal Article