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PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18293
...) holds great promise for patient-specific cardiotoxicity drug testing, disease modeling, and cardiac regeneration... 
DEFINED MEDIUM | MESODERM | MULTIDISCIPLINARY SCIENCES | ENDODERM | CARDIOMYOCYTE DIFFERENTIATION | INDUCTION | LINES | FUNCTIONAL-PROPERTIES | Body Patterning - drug effects | Oxygen - pharmacology | Antigens, CD34 - metabolism | Polyvinyl Alcohol - pharmacology | Humans | Fibroblast Growth Factor 2 - pharmacology | Mesoderm - drug effects | Mesoderm - cytology | Cell Culture Techniques - methods | Embryoid Bodies - metabolism | Embryoid Bodies - cytology | Adult | Induced Pluripotent Stem Cells - cytology | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Cell Line | Insulin - pharmacology | Induced Pluripotent Stem Cells - drug effects | Myocytes, Cardiac - cytology | Transgenes - genetics | Fetal Blood - cytology | Cell Adhesion - drug effects | Bone Morphogenetic Protein 4 - pharmacology | Genetic Vectors - genetics | Embryoid Bodies - drug effects | Animals | Myocytes, Cardiac - drug effects | Cell Differentiation - drug effects | Fibroblasts - drug effects | Culture Media - pharmacology | Myocytes, Cardiac - metabolism | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Electrophysiological Phenomena - drug effects | Physiological aspects | Bone morphogenetic proteins | Fibroblast growth factors | Embryonic stem cells | Analysis | Drugs | Neonates | Neuropathology | Variability | Embryo cells | Drug development | Blood | Contraction | Regeneration (physiology) | Alcohols | Engineering | Cord blood | Efficiency | Calcium-binding protein | Fibroblasts | Long QT syndrome | Modelling | Polyvinyl alcohol | Heart diseases | Biomedical engineering | Fibroblast growth factor 2 | CD34 antigen | Bone morphogenetic protein 4 | Oxygen | Anemia | Cardiomyocytes | Insulin | Embryos | Coronary artery disease | Medicine | Regeneration | Troponin | Troponin I | Plasmids | Stem cells | Epigenetics | Mutation | Differentiation | Pluripotency
Journal Article
Circulation Research, ISSN 0009-7330, 10/2012, Volume 111, Issue 9, pp. 1125 - 1136
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2010, Volume 56, Issue 25, pp. 2115 - 2125
Objectives In this study, we have investigated the effects of cannabidiol (CBD) on myocardial dysfunction, inflammation, oxidative/nitrative stress, cell death... 
Cardiovascular | Internal Medicine | diabetic complications | cannabinoids | inflammation | oxidative stress | CONSTITUENT | CARDIAC & CARDIOVASCULAR SYSTEMS | METALLOTHIONEIN | MITOCHONDRIAL | ENDOCANNABINOID SYSTEM | ANTAGONISM | PROTECTS | INHIBITION | DISEASE | NF-KAPPA-B | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Humans | Body Weight - drug effects | Male | NF-kappa B - metabolism | Glucose | Cannabidiol - therapeutic use | Drug Evaluation, Preclinical | Disease Models, Animal | Mice, Inbred C57BL | Cells, Cultured | Pancreas - drug effects | Cannabidiol - pharmacology | Myocardium - pathology | Pancreas - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Myocytes, Cardiac - drug effects | Fibrosis | Diabetic Cardiomyopathies - pathology | Hemodynamics - drug effects | Mice | Oxidative Stress - drug effects | Phosphates | Oxidative stress | Medical colleges | Niacinamide | Cardiomyopathy | Alcoholism | Radiation | Superoxide | Biochemistry | Dextrose | Purines | Cannabinoids | Cell death | Analysis | Nitric oxide | Protein kinases | Heart diseases | Adenosine triphosphatase | Jewish schools | Mortality | Cardiovascular disease | Cardiomyocytes | Kinases | Experiments | Variance analysis | Antioxidants | Proteins | Polymerase chain reaction | Rodents | Atherosclerosis | Diabetes | Pancreas | Apoptosis
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2010, Volume 48, Issue 2, pp. 367 - 378
...)-induced cardiac contractile and intracellular Ca2+ dysfunction, oxidative stress, endoplasmic reticulum (ER... 
Cardiovascular | ER stress | Oxidative stress | Sepsis | Cardiomyocytes | Autophagy | Metallothionein | CARDIAC & CARDIOVASCULAR SYSTEMS | MITOCHONDRIAL BIOGENESIS | PROTEIN-KINASE | CELL-DEATH | CELL BIOLOGY | SEPTIC SHOCK | NITRIC-OXIDE | LIPOPOLYSACCHARIDE | ORGAN DYSFUNCTION | MYOCARDIAL DYSFUNCTION | Metallothionein - metabolism | Reactive Oxygen Species - metabolism | Calcium - metabolism | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Sepsis - complications | Autophagy - drug effects | Myocytes, Cardiac - enzymology | Endoplasmic Reticulum - pathology | Inflammation - complications | Lysosomes - metabolism | Sepsis - pathology | Adenylate Kinase - metabolism | Endoplasmic Reticulum - drug effects | Biomechanical Phenomena - drug effects | Lysosomes - drug effects | Intracellular Space - drug effects | Myocardium - pathology | Lethal Dose 50 | Enzyme Activation - drug effects | Sepsis - physiopathology | Feeding Behavior - drug effects | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Intracellular Space - metabolism | Lipopolysaccharides - pharmacology | Intracellular Membranes - drug effects | Mice | Oxidative Stress - drug effects | Intracellular Membranes - metabolism | Antioxidants | Analysis | Depression, Mental | Alternative medicine | Acetylcysteine | Mitogens | Glutathione | cardiomyocytes | autophagy | metallothionein | sepsis | oxidative stress
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2011, Volume 60, Issue 2, pp. 625 - 633
Journal Article
Biomacromolecules, ISSN 1525-7797, 09/2017, Volume 18, Issue 9, pp. 2820 - 2829
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 6656
.... Here we show that HKL blocks agonist-induced and pressure overload-mediated, cardiac hypertrophic responses, and ameliorates pre-existing cardiac hypertrophy, in mice... 
METABOLISM | GENE | SIRT3-MEDIATED DEACETYLATION | PATHWAY | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | HOMOLOG | AKT | SIRTUINS | CALORIE RESTRICTION | CELL-DEATH | Mitochondria - enzymology | Fibroblasts - enzymology | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | Sirtuin 3 - metabolism | Myofibroblasts - enzymology | Superoxide Dismutase - antagonists & inhibitors | Cardiomegaly - pathology | Isoproterenol | Lignans - pharmacology | Myocytes, Cardiac - enzymology | Phenylephrine - pharmacology | Biphenyl Compounds - pharmacology | Membrane Proteins - metabolism | Superoxide Dismutase - metabolism | Myofibroblasts - pathology | Sirtuin 3 - genetics | Signal Transduction | Membrane Proteins - genetics | Carrier Proteins - antagonists & inhibitors | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Myocardium - pathology | Cardiotonic Agents - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Fibroblasts - pathology | Myofibroblasts - drug effects | Adenosine Triphosphatases - antagonists & inhibitors | Carrier Proteins - genetics | Myocytes, Cardiac - pathology | Acetylation - drug effects | Myocardium - enzymology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Cardiomegaly - prevention & control | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | Membrane Proteins - antagonists & inhibitors | Cell Differentiation - drug effects | Fibroblasts - drug effects | Cardiomegaly - chemically induced | Adenosine Triphosphatases - genetics | Cell Proliferation - drug effects | Mice | Enzyme Activation | Primary Cell Culture | Cardiomegaly - genetics
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 03/2008, Volume 102, Issue 5, pp. 597 - 606
Heart failure is the leading cause of death in the elderly, but whether this is the result of a primary aging myopathy dictated by depletion of the cardiac progenitor cell (CPC) pool is unknown... 
Cardiac stem cells | Aging cardiomyopathy | Telomere-telomerase system | aging cardiomyopathy | CARDIAC & CARDIOVASCULAR SYSTEMS | LABEL-RETAINING CELLS | MULTIPOTENT | telomerase system | REGENERATION | telomere | IGF-1 | MECHANISMS | cardiac stem cells | FAILURE | DISEASE | STEM-CELL | PERIPHERAL VASCULAR DISEASE | STRESS | HEMATOLOGY | GROWTH-FACTOR-I | Hepatocyte Growth Factor - therapeutic use | Apoptosis - drug effects | Cell Count | Rats, Inbred F344 | Aging - drug effects | Heart Failure - physiopathology | Cellular Senescence - drug effects | Male | Heart Failure - therapy | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | Telomere - metabolism | Adult Stem Cells - pathology | Insulin-Like Growth Factor I - therapeutic use | Adult Stem Cells - drug effects | Disease Models, Animal | Drug Administration Routes | Heart - growth & development | Rats | Survival Rate | Heart Failure - pathology | Cell Division - drug effects | Aging - pathology | Cell Movement - drug effects | Myocytes, Cardiac - pathology | Adult Stem Cells - metabolism | Antigens, Differentiation - biosynthesis | Phenotype | Regeneration - drug effects | Animals | Myocytes, Cardiac - drug effects | Cell Differentiation - drug effects | Heart - drug effects | Myocytes, Cardiac - metabolism | Receptors, Growth Factor - metabolism
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 7, p. e67009
...), on cardiac dysfunction and cardiac cell apoptosis in streptozotocin (STZ)-induced diabetic rat... 
TRANSCRIPTION FACTORS | MONOAMINE-OXIDASE | NADPH OXIDASE | MYOCARDIAL-INFARCTION | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | LOW-DENSITY-LIPOPROTEIN | ANGIOTENSIN-II | NF-KAPPA-B | MEDIATED OXIDATIVE STRESS | GENE-TRANSCRIPTION | Gene Expression - drug effects | Rats, Wistar | Arachidonate 5-Lipoxygenase - genetics | Streptozocin | Humans | Heart Failure - physiopathology | Male | NF-kappa B - metabolism | Monoamine Oxidase - genetics | Diabetic Cardiomyopathies - physiopathology | Heart Failure - prevention & control | Heart Rate - drug effects | Diabetes Mellitus, Experimental - prevention & control | Lipid Peroxidation - drug effects | Diabetic Cardiomyopathies - prevention & control | Blood Pressure - drug effects | Diabetes Mellitus, Experimental - chemically induced | Heme Oxygenase (Decyclizing) - metabolism | Xanthine Oxidase - genetics | Heme Oxygenase (Decyclizing) - genetics | Diabetes Mellitus, Experimental - physiopathology | Heart - physiopathology | Xanthine Oxidase - metabolism | Arachidonate 5-Lipoxygenase - metabolism | Catalase - genetics | Rats | Antioxidants - pharmacology | Catalase - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | NF-kappa B - genetics | Heart - drug effects | Myocytes, Cardiac - metabolism | Cytokines - antagonists & inhibitors | Oxidative Stress - drug effects | Cytokines - biosynthesis | Monoamine Oxidase - metabolism | Antioxidants | Usage | Care and treatment | Cardiomyopathy | Heart cells | Health aspects | Heart diseases | Heart | Oxidative stress | Reactive oxygen species | Transcription factors | Diastolic pressure | Bax protein | Bcl-2 protein | Science | Prevention | Signal transduction | Cell activation | Developmental stages | Catalase | Rodents | Heme | Arachidonate 5-lipoxygenase | Blood pressure | Oxidation | Food | Enzymes | Cytokines | Cardiac muscle | Complications | Diabetes mellitus | Muscles | Cardiomyocytes | Inflammation | Gene expression | Amine oxidase (flavin-containing) | Heart rate | Studies | Cell death | Nitric oxide | γ-Interferon | Interleukin 10 | Xanthine oxidase | Interferon | Ventricle | Diabetes | Lipoxygenase | Binding sites | Apoptosis
Journal Article