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Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 2014, Volume 63, Issue 6, pp. 1241 - 1250
.... However, its role in the heart is not clear. To investigate the role of CTGF in regulating the development of cardiac fibrosis and heart failure, we subjected mice to thoracic aortic constriction (TAC... 
Hypertension | Inflammation | S100 proteins | Angiotensin II | MECHANISMS | MYELOID-RELATED PROTEIN-14 | RAGE | MAPK | MYELOID-RELATED-PROTEIN-8/14 | INHIBITION | inflammation | angiotensin II | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | DYSFUNCTION | hypertension | NF-KAPPA-B | EXPRESSION | Immunohistochemistry | Calgranulin B - metabolism | Oligonucleotide Array Sequence Analysis | Myocytes, Cardiac - immunology | Calgranulin A - genetics | Inflammation - metabolism | Myocardium - metabolism | Antibodies - immunology | Antigens, Ly - metabolism | Calgranulin A - immunology | Hypertension - genetics | Cytokines - genetics | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Neutrophils - drug effects | Toll-Like Receptor 4 - genetics | Hypertension - immunology | Toll-Like Receptor 4 - immunology | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Fibroblasts - immunology | Mice | Receptors, Immunologic - genetics | Myocardium - immunology | Calgranulin A - metabolism | Transcriptome - immunology | Calgranulin B - immunology | Hypertension - prevention & control | Calgranulin B - genetics | Receptors, Immunologic - immunology | Cytokines - immunology | CD11b Antigen - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Myocardium - pathology | Inflammation - immunology | Transcriptome - drug effects | Angiotensin II - immunology | Antibodies - pharmacology | Animals | Antigens, Ly - immunology | Inflammation - genetics | CD11b Antigen - metabolism | Receptors, Immunologic - metabolism
Journal Article
Circulation research, ISSN 1524-4571, 2016, Volume 118, Issue 2, pp. 241 - 253
...–activated receptor (PPAR)α and under normal conditions accounts for 70% of cardiac ATP content... 
heart failure | PPAR alpha | fatty acids | cardiac myocyte | heart | CARDIAC & CARDIOVASCULAR SYSTEMS | LIPID-METABOLISM | CONTRACTILE DYSFUNCTION | FATTY-ACID-METABOLISM | SUBSTRATE METABOLISM | MOUSE MODELS | ACTIVATED-RECEPTOR-ALPHA | FAILING HEART | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | ENERGY-METABOLISM | HEMATOLOGY | TRANSCRIPTIONAL REGULATION | Up-Regulation | Transcriptional Activation | Diabetes Mellitus, Experimental - genetics | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Sepsis - metabolism | Transcription, Genetic | Diabetes Mellitus, Experimental - metabolism | Kruppel-Like Transcription Factors - deficiency | Binding Sites | Fatty Acids - metabolism | Diabetes Mellitus, Experimental - physiopathology | Sodium-Glucose Transporter 2 - genetics | Binding, Competitive | Cardiomyopathy, Dilated - genetics | Cell Line | Promoter Regions, Genetic | Oxidation-Reduction | Signal Transduction | Sodium-Glucose Transporter 2 - metabolism | Mice, Inbred C57BL | Genotype | PPAR alpha - genetics | Sepsis - physiopathology | Cardiomyopathy, Dilated - metabolism | Sepsis - genetics | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Mice, Knockout | Triglycerides - metabolism | Phenotype | Animals | Cardiomyopathy, Dilated - physiopathology | Energy Metabolism | Proto-Oncogene Proteins c-jun - metabolism | Myocytes, Cardiac - metabolism | Protein Binding | PPAR alpha - metabolism | Kruppel-Like Transcription Factors - genetics | diabetic cardiacmyopathy | KLF5 | PPARα
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 10, pp. 3770 - 3783
.... However, the importance of TGF-beta-Smad2/3 signaling in fibroblast-mediated cardiac fibrosis has not been directly evaluated in vivo... 
GENETIC MANIPULATION | MEDICINE, RESEARCH & EXPERIMENTAL | HYPERTROPHY | GROWTH-FACTOR-BETA | HEART-DISEASE | TGF-BETA | SMAD3 | MYOCARDIAL-INFARCTION | IN-VIVO | MYOFIBROBLAST DIFFERENTIATION | PROTECTS | Receptors, Transforming Growth Factor beta - genetics | Heart Diseases - metabolism | Male | Smad3 Protein - metabolism | Myofibroblasts - metabolism | Smad3 Protein - genetics | Gene Deletion | Myocardium - metabolism | Smad2 Protein - genetics | Protein-Serine-Threonine Kinases - metabolism | Myofibroblasts - pathology | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Mice, Transgenic | Myocardium - pathology | Organ Specificity | Myocytes, Cardiac - pathology | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Fibrosis | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Heart Diseases - genetics | Heart Diseases - pathology | Transcription factors | Phosphorylation | Heart attacks | Disease | Homeostasis | Smad3 protein | Gene deletion | Kinases | Proteins | Clonal deletion | Smad2 protein | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Growth factors | Heart failure | Cytokines | Cardiomyocytes | Gene expression | Pressure | Latency | Adenoviruses | Genetic engineering | Apoptosis
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2012, Volume 125, Issue 23, pp. 2844 - 2853
Journal Article
PloS one, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e30329
Journal Article
Journal Article
Circulation research, ISSN 1524-4571, 2013, Volume 113, Issue 5, pp. 603 - 616
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2011, Volume 50, Issue 7, pp. 777 - 793
.... Here, we review the main redox-regulated pathways that are involved in cardiac myocyte excitation... 
Heart failure | Reactive oxygen species | Mitochondria | Free radicals | NADPH oxidase | Cardiac myocyte | Redox signaling | Hypertrophy | HYPOXIA-INDUCIBLE FACTOR | NOX2-CONTAINING NADPH OXIDASE | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-KAPPA-B | MITOCHONDRIAL PERMEABILITY TRANSITION | EMBRYONIC STEM-CELLS | RECEPTOR-STIMULATED HYPERTROPHY | ENDOCRINOLOGY & METABOLISM | INDUCED CARDIOMYOCYTE HYPERTROPHY | XANTHINE OXIDOREDUCTASE INHIBITION | RAT VENTRICULAR MYOCYTES | Excitation Contraction Coupling - physiology | Myocardial Ischemia - metabolism | Rabbits | Cell Proliferation | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Oxidation-Reduction | Oxidative Stress | Heart Diseases - physiopathology | Humans | NADPH Oxidases - metabolism | Stress, Physiological | Rats | Mitochondria - metabolism | Hypertrophy - metabolism | Oxygen - metabolism | Hypertrophy - physiopathology | Animals | Myocytes, Cardiac - physiology | Myocardial Ischemia - physiopathology | Thioredoxins - metabolism | Mice | Cell Differentiation - physiology | ARC, apoptosis repressor with caspase recruitment domain | PKG, protein kinase G | AIF, apoptosis-inducing factor | Review | MAO-A, monoamine oxidase-A | MI, myocardial infarction | TNFα, tumor necrosis factor-α | ES, embryonic stem | GPCR, G-protein-coupled receptor | RyR, ryanodine receptor | PHD, prolyl hydroxylase dioxygenase | CTGF, connective tissue growth factor | mtDNA, mitochondrial DNA | ROS, reactive oxygen species | ETC, electron transport chain | HDAC, histone deacetylase | EB, embryoid body | Ca exchanger | G6PDH, glucose-6-phosphate dehydrogenase | PKC, protein kinase C | SR, sarcoplasmic reticulum | SERCA, sarcoplasmic reticulum calcium ATPase | VEGF, vascular endothelial growth factor | Hif, hypoxia-inducible factor | NOS, nitric oxide synthase | NCX, Na | MPTP, mitochondrial permeability transition pore | ECC, excitation–contraction coupling | CamKII, calmodulin kinase II | ER, endoplasmic reticulum | MMP, matrix metalloproteinase | PKA, protein kinase A | Trx1, thioredoxin1
Journal Article