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PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e30329 - e30329
Background: Endogenous cardiac progenitor cells are a promising option for cell-therapy for myocardial infarction (MI). However, obtaining adequate numbers of... 
PROGENITOR CELLS | HEART | STEM-CELLS | INFARCTION | THERAPY | MULTIPOTENT | REGENERATION | BIOLOGY | DIFFERENTIATION | CARDIOMYOCYTES | EXPRESSION | Immunohistochemistry | Myocardial Ischemia - metabolism | Leukocyte Common Antigens - metabolism | LIM-Homeodomain Proteins - metabolism | Stem Cells - metabolism | Stem Cell Transplantation | Flow Cytometry | Time Factors | Myocardium - metabolism | Myocardial Ischemia - physiopathology | Antigens, Ly - metabolism | Myocardial Infarction - physiopathology | Cell Differentiation | Membrane Proteins - metabolism | Myocytes, Smooth Muscle - metabolism | Heart - physiopathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Myocardium - pathology | Myocardial Infarction - metabolism | Transcription Factors - metabolism | Animals | Myocytes, Cardiac - metabolism | Mice | Neovascularization, Physiologic | Apoptosis | Myocardial infarction | Heart | Therapy | Heart attacks | Smooth muscle | CD45 antigen | Angiogenesis | Cell growth | Ischemia | Precursors | Rodents | Animal tissues | Bone marrow | Cardiology | Heart diseases | Medical research | Cardiac muscle | Muscles | Cardiomyocytes | Gene expression | Vectors (Biology) | Endothelial cells | Medicine | Islet-1 protein | Stem cells | Cells (biology) | Myocardium | Hypoxia | Infarction | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 05/2012, Volume 110, Issue 11, pp. 1465 - 1473
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 502, Issue 7471, pp. 372 - 376
Ca2+/calmodulin-dependent protein kinase II (CaMKII) is an enzyme with important regulatory functions in the heart and brain, and its chronic activation can be... 
N-ACETYLGLUCOSAMINE | MYOCYTES | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | CARDIAC-HYPERTROPHY | RECEPTOR | CYTOSOLIC PROTEINS | CARDIOMYOCYTES | MELLITUS | CA2+/CALMODULIN-DEPENDENT PROTEIN-KINASE | INSIGHT | Calcium - metabolism | Humans | Brain - enzymology | Hyperglycemia - complications | Glycosylation - drug effects | Acetylglucosamine - metabolism | Myocytes, Cardiac - enzymology | Diazooxonorleucine - pharmacology | Sarcoplasmic Reticulum - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Arrhythmias, Cardiac - metabolism | Diabetes Complications - metabolism | Rats | Glucose - pharmacology | Enzyme Activation - drug effects | Sulfonamides - pharmacology | Myocardium - cytology | Brain - drug effects | Hyperglycemia - metabolism | Myocardium - enzymology | Animals | Arrhythmias, Cardiac - enzymology | Glucose - metabolism | Hyperglycemia - enzymology | Myocytes, Cardiac - metabolism | Arrhythmias, Cardiac - complications | Diabetes Complications - enzymology | Mice | Benzylamines - pharmacology | Enzymes | Medical research | Arrhythmia | Protein biosynthesis | Glycosylation | Research | Risk factors | Complications and side effects | Hyperglycemia | Physiological aspects | Medicine, Experimental | Regulation | Protein kinases | Proteins | Brain | Phosphorylation | Rodents | Cardiomyocytes | Diabetes | Kinases | Index Medicus
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2017, Volume 474, Issue 16, pp. 2785 - 2801
Journal Article
Cell, ISSN 0092-8674, 03/2018, Volume 173, Issue 1, pp. 104 - 116.e12
Human diseases are often caused by loss of somatic cells that are incapable of re-entering the cell cycle for regenerative repair. Here, we report a... 
heart failure | cyclin | proliferation | cell cycle | regeneration | cytokinesis | cardiomyocyte | cell division | CDK | heart | HYPERTROPHY | INFARCTION | EXPRESSION ANALYSIS | AMPLIFICATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICE | ZEBRAFISH HEART REGENERATION | FAILURE | CANCER | FIBROBLASTS | PROGRAM | CELL BIOLOGY | Cyclin D1 - metabolism | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Cyclin-Dependent Kinase 4 - genetics | Heart - physiology | Humans | Myosin Heavy Chains - genetics | Cyclin B1 - metabolism | CDC2 Protein Kinase - metabolism | Cell Cycle Proteins - antagonists & inhibitors | Myocardial Infarction - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Myocardial Infarction - veterinary | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | CDC2 Protein Kinase - genetics | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Cyclin B1 - genetics | Rats | Mice, Transgenic | Nuclear Proteins - metabolism | Myocardial Infarction - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Cytokinesis | Regeneration | Animals | Cyclin D1 - genetics | Nuclear Proteins - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Heart | Bone morphogenetic proteins | Transforming growth factors | Cell cycle | Stem cells | Index Medicus | Cell Cycle | Cardiomyocyte | Cell Division | Cyclin
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 06/2009, Volume 296, Issue 6, pp. 1363 - 1373
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 06/2015, Volume 26, Issue 6, pp. 1290 - 1302
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