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Circulation, ISSN 0009-7322, 08/2013, Volume 128, Issue 9, pp. 970 - 981
BACKGROUND—Sarcoplasmic reticulum (SR) Ca leak through ryanodine receptor type 2 (RyR2) dysfunction is of major pathophysiological relevance in human heart... 
Heart failure | Calcium calmodulin-dependent protein kinase type 2 | Sarcoplasmic reticulum | Protein kinases | Hypertrophy | heart failure | RYANODINE RECEPTOR PHOSPHORYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | CONTRACTILITY | hypertrophy | protein kinases | VENTRICULAR MYOCYTES | RELEASE | FAILING HEARTS | calcium-calmodulin-dependent protein kinase type 2 | sarcoplasmic reticulum | ATRIAL-FIBRILLATION | CAMKII | CALCIUM SPARKS | PKA PHOSPHORYLATION | PERIPHERAL VASCULAR DISEASE | HEART-FAILURE PROGRESSION | Phosphorylation | Calcium - metabolism | Humans | Middle Aged | Homeostasis | Cardiomegaly - pathology | Male | Ryanodine Receptor Calcium Release Channel - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - drug effects | Case-Control Studies | Myocardium - metabolism | Female | Sarcoplasmic Reticulum - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP-Dependent Protein Kinases - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Enzyme Inhibitors - pharmacology | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Disease Progression | Aged | Cardiomegaly - metabolism | Physiological aspects | Calcium channels | Research | Index Medicus | Abridged Index Medicus
Journal Article
Physiological genomics, ISSN 1094-8341, 10/2018, Volume 50, Issue 10, pp. 846 - 861
Numerous studies have demonstrated that Na+/H+ exchanger isoform 1 (NHE1) is elevated in myocardial diseases and its effect is detrimental. To better... 
miRNA-mRNA interaction | NHE1 | cardiovascular diseases | cardiac hypertrophy | high-throughput sequencing | PHYSIOLOGY | MYOCARDIAL-INFARCTION | DIASTOLIC CALCIUM | HEART-FAILURE | VENTRICULAR MYOCYTES | CELL BIOLOGY | HYPERTROPHY | NHE-1 INHIBITION | SODIUM-HYDROGEN EXCHANGER | H+ EXCHANGE | RAT MYOCARDIUM | GENETICS & HEREDITY | GENE-EXPRESSION
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 495, Issue 7439, pp. 107 - 110
Journal Article
Circulation, ISSN 0009-7322, 05/2009, Volume 119, Issue 19, pp. 2561 - 2567
Background-In classic Fabry patients, accelerated coronary atherosclerosis and left ventricular hypertrophy manifest in the fourth decade; however, signs of... 
Microcirculation | Pathology | Hypercholesterolemia | Ischemia | Hypertrophy | CARDIAC & CARDIOVASCULAR SYSTEMS | pathology | CHOLESTEROL | hypertrophy | CARDIOMYOPATHY | INVOLVEMENT | ATHEROSCLEROSIS | LOW-DENSITY LIPOPROTEIN | microcirculation | hypercholesterolemia | ALPHA-GALACTOSIDASE-A | ischemia | GLYCOSPHINGOLIPIDS | SERUM-LIPOPROTEINS | PERIPHERAL VASCULAR DISEASE | AGALSIDASE-ALPHA | ACCUMULATION | HEMATOLOGY | Recombinant Proteins - therapeutic use | alpha-Galactosidase - therapeutic use | Capillaries - pathology | Trihexosylceramides - analysis | Inclusion Bodies - chemistry | Humans | Lysosomes - enzymology | Male | Endocardium - pathology | Endothelium, Vascular - chemistry | Young Adult | Clinical Trials, Phase III as Topic | Adult | Female | Trihexosylceramides - metabolism | Inclusion Bodies - ultrastructure | Double-Blind Method | Capillaries - chemistry | Coronary Artery Disease - prevention & control | Fabry Disease - complications | Coronary Disease - etiology | Randomized Controlled Trials as Topic | Myocytes, Cardiac - pathology | Coronary Disease - pathology | Coronary Disease - prevention & control | Endocardium - ultrastructure | Biopsy | Adolescent | Coronary Disease - drug therapy | Myocytes, Cardiac - metabolism | Endothelium, Vascular - pathology | Prevention | Care and treatment | Heart enlargement | Atherosclerosis | Physiological aspects | Development and progression | Dosage and administration | Research | Recombinant molecules | Fabry's disease | Alpha galactosidases | Index Medicus | Abridged Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 08/2017, Volume 121, Issue 5, pp. 575 - 583
Journal Article
Circulation, ISSN 0009-7322, 04/2018, Volume 137, Issue 16, pp. 1712 - 1730
BACKGROUND:Here, we generated human cardiac muscle patches (hCMPs) of clinically relevant dimensions (4 cm × 2 cm × 1.25 mm) by suspending cardiomyocytes,... 
Heart | Myocardial infarction | Models | Pluripotent stem cells | Tissue engineering | Animal | models, animal | tissue engineering | CARDIAC & CARDIOVASCULAR SYSTEMS | pluripotent stem cells | myocardial infarction | TISSUE | HEART-DISEASES | MATURATION | heart | FUNCTIONAL CONSEQUENCES | ENDOTHELIAL PROGENITOR CELLS | WALL STRESS | REPAIR | CARDIOMYOCYTE APOPTOSIS | PERIPHERAL VASCULAR DISEASE | EXOSOMES MEDIATE | OPTICAL MEASUREMENTS | Myocardial Infarction - genetics | Ventricular Function, Left | Humans | Endothelial Cells - transplantation | Myocytes, Smooth Muscle - pathology | Transplantation, Heterologous | Recovery of Function | Tissue Scaffolds | Ventricular Remodeling | Time Factors | Regeneration - genetics | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Cell Differentiation | Myocytes, Smooth Muscle - transplantation | Disease Models, Animal | Myocardial Infarction - surgery | Tissue Engineering - methods | Induced Pluripotent Stem Cells - physiology | Endothelial Cells - metabolism | Induced Pluripotent Stem Cells - transplantation | Cells, Cultured | Gene Expression Regulation | Myocardium - pathology | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Stem Cell Transplantation - methods | Animals | Endothelial Cells - pathology | Sus scrofa | Care and treatment | Swine | Heart cells | Stem cells | Research | Health aspects | Heart attack | Index Medicus | Abridged Index Medicus | Porcine | Myocardial Infarction | Tissue Engineering
Journal Article
Current molecular medicine, 2016, Volume 16, Issue 1, p. 12
Calreticulin is a Ca(2+)-binding chaperone in the endoplasmic reticulum (ER) which is crucial to the correct embryological development of the heart in mice.... 
Heart - physiopathology | Myocytes, Cardiac - pathology | Animals | Heart Diseases - metabolism | Calcium - metabolism | Humans | Calreticulin - metabolism | Embryonic Development - physiology | Endoplasmic Reticulum - metabolism | Myocytes, Cardiac - metabolism | Heart Diseases - pathology
Journal Article
Circulation, ISSN 0009-7322, 01/2013, Volume 127, Issue 1, pp. 74 - 85
Background-Cardiac consequences of obesity include inflammation, hypertrophy, and compromised energy metabolism. Glucagon-like peptide-1 is an incretin hormone... 
AMP-activated protein kinase | inflammation | heart | obesity | glucagon-like peptide-1 | connexin-43 | CARDIAC & CARDIOVASCULAR SYSTEMS | ACTIVATED PROTEIN-KINASE | MYOCARDIAL-INFARCTION | HEART-FAILURE | ENDOPLASMIC-RETICULUM STRESS | DIABETIC CARDIOMYOPATHY | TUMOR-NECROSIS-FACTOR | INSULIN-RESISTANCE | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | RAT-HEART | MICE | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Obesity - drug therapy | Monocytes - cytology | Humans | Heart Diseases - prevention & control | Hypercholesterolemia - drug therapy | Heart Diseases - epidemiology | Connexin 43 - genetics | Disease Models, Animal | Hypercholesterolemia - epidemiology | Muscle, Smooth, Vascular - drug effects | Cell Line | Endoplasmic Reticulum Stress - drug effects | Myocytes, Cardiac - cytology | Glucagon-Like Peptide 1 - analogs & derivatives | Mice, Inbred C57BL | Risk Factors | Glucagon-Like Peptide 1 - pharmacology | Insulin Resistance | Cardiotonic Agents - pharmacology | Muscle, Smooth, Vascular - cytology | Nitric Oxide Synthase Type III - genetics | Blood Glucose - drug effects | Monocytes - drug effects | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Coronary Vessels - cytology | Endothelial Cells - cytology | Obesity - epidemiology | Mice | Liraglutide | Endothelial Cells - drug effects | Prevention | Obesity | Care and treatment | Physiological aspects | Research | Gastrointestinal hormones | Cardiovascular diseases | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2013, Volume 123, Issue 12, pp. 5284 - 5297
Basal autophagy is a crucial mechanism in cellular homeostasis, underlying both normal cellular recycling and the clearance of damaged or misfolded proteins,... 
MEDICINE, RESEARCH & EXPERIMENTAL | HEMODYNAMIC STRESS | DESMIN-RELATED CARDIOMYOPATHY | MONITORING AUTOPHAGY | HEART-FAILURE | PREAMYLOID OLIGOMER | DISEASE | BASAL AUTOPHAGY | BODY FORMATION | EXERCISE | TRANSGENIC MICE | Proteostasis Deficiencies - metabolism | alpha-Crystallin B Chain - genetics | Microtubule-Associated Proteins - genetics | Autophagy - physiology | Ventricular Dysfunction - prevention & control | Muscular Dystrophies - genetics | Recombinant Fusion Proteins - metabolism | Cardiomyopathies - genetics | Heart Failure - therapy | Muscle Proteins - metabolism | Heart Failure - etiology | Physical Conditioning, Animal | Disease Models, Animal | Muscular Dystrophies - metabolism | Cardiomyopathies - pathology | Mice, Transgenic | Heart Failure - metabolism | Heart Failure - pathology | Microtubule-Associated Proteins - biosynthesis | Muscular Dystrophies - pathology | Microtubule-Associated Proteins - physiology | Gene Expression Regulation - drug effects | Myocytes, Cardiac - pathology | Autophagy-Related Protein 7 | Animals | Cardiomyopathies - metabolism | Myocytes, Cardiac - metabolism | Mice | Doxorubicin - pharmacology | Crosses, Genetic | Homeostasis | Autophagy (Cytology) | Care and treatment | Research | Heart diseases | Heart | Disease | Mortality | Genes | Storm damage | Cardiomyocytes | Autophagy | Proteins | Studies | Aggregates | Neurodegeneration | Rodents | Laboratory animals | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 09/2012, Volume 126, Issue 11, pp. S54 - S64
Journal Article
Circulation, ISSN 0009-7322, 2013, Volume 128, Issue 13, pp. 1420 - 1432
Journal Article