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Journal of allergy and clinical immunology, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 769 - 780
... on mitochondrial function in the pathophysiology of airway inflammation in ozone-exposed mice and human airway smooth muscle (ASM) cells... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | ASTHMA FEATURES | MECHANISMS | IMMUNOLOGY | FRAGMENTATION | HYPERRESPONSIVENESS | BETA | RESPONSES | ALLERGY | ENERGY-METABOLISM | EXPRESSION | COPD | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
Nature cell biology, ISSN 1476-4679, 2015, Volume 17, Issue 8, pp. 994 - 1003
.... Here, we report the rapid and efficient differentiation of human pluripotent stem cells into vascular endothelial and smooth muscle cells... 
DIRECTED DIFFERENTIATION | IN-VIVO | MOUSE | DEFINITIVE ENDODERM | GROWTH-FACTOR | HUMAN BLASTOCYSTS | STROMAL CELLS | WNT | CULTURE | LINES | CELL BIOLOGY | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium
Journal Article
Circulation research, ISSN 1524-4571, 2017, Volume 120, Issue 12, pp. 1903 - 1915
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2013, Volume 33, Issue 1, pp. 67 - 75
.... We investigated here whether Gal-3 is involved in Aldo-induced vascular fibrosis. Methods and Results-In rat vascular smooth muscle cells Gal-3 overexpression enhanced specifically collagen type I synthesis... 
collagen type I | aldosterone | HEART-FAILURE | LEFT-VENTRICULAR DYSFUNCTION | INFLAMMATION | ENDOTHELIAL-CELLS | fibrosis | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | galectin-3 | vascular smooth muscle cells | EXPRESSION | INSIGHTS | CARDIOTROPHIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Blood Pressure | Inflammation - chemically induced | Inflammation - pathology | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Humans | Galectin 3 - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aldosterone | Muscle, Smooth, Vascular - physiopathology | Galectin 3 - deficiency | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Hypertension - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Hypertension - prevention & control | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Vascular Stiffness | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Mice, Inbred C57BL | Cells, Cultured | Rats | Galectin 3 - genetics | Hypertension - pathology | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Collagen Type I - biosynthesis | Muscle, Smooth, Vascular - pathology | Animals | Galectin 3 - antagonists & inhibitors | Fibrosis | Inflammation - genetics | Inflammation - prevention & control | Mice | Inflammation - physiopathology | Cellular Biology | Life Sciences | Muscle, Smooth, Vascular | Mineralocorticoid Receptor Antagonists | Hypertension | Hematology | Biochemistry, Molecular Biology | Collagen Type I | Inflammation | Human health and pathology | Myocytes, Smooth Muscle | Galectin 3
Journal Article
Circulation research, ISSN 1524-4571, 2012, Volume 110, Issue 11, pp. 1484 - 1497
RATIONALE:Pulmonary arterial hypertension (PAH) is a lethal syndrome characterized by pulmonary vascular obstruction caused, in part, by pulmonary artery smooth muscle cell (PASMC) hyperproliferation... 
mitotic checkpoint | cyclin B1/cyclin-dependent kinase 1 | mitochondrial fission | hypoxia-inducible factor-1 | mitochondrial division inhibitor-1 | CARDIAC & CARDIOVASCULAR SYSTEMS | ENTRY | INDUCIBLE FACTOR 1-ALPHA | HYPOXIA | DRP1 | PATHWAY | ARTERIAL-HYPERTENSION | ENDOTHELIAL-CELLS | GENE-EXPRESSION | GERMLINE MUTATIONS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | K+ CHANNELS | Antihypertensive Agents - pharmacology | Humans | Monocrotaline | Male | Cyclin B1 - metabolism | Cobalt | Hypertension, Pulmonary - therapy | CDC2 Protein Kinase - metabolism | RNA Interference | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Myocytes, Smooth Muscle - drug effects | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Myocytes, Smooth Muscle - enzymology | Dynamins - genetics | Rats | Familial Primary Pulmonary Hypertension | Hypoxia - complications | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Cell Cycle Checkpoints | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Mitochondria, Muscle - drug effects | Enzyme Activation | Hypertension, Pulmonary - etiology | Muscle, Smooth, Vascular - enzymology | Dynamins - metabolism | Phosphorylation | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Serine | Myocytes, Smooth Muscle - pathology | Mitochondrial Proteins - genetics | Case-Control Studies | Transfection | Quinazolinones - pharmacology | Mitochondria, Muscle - enzymology | Mitochondria, Muscle - pathology | Cells, Cultured | Muscle, Smooth, Vascular - pathology | Animals | Mitosis - drug effects | Glycolysis | Cell Proliferation - drug effects | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Genetic Therapy - methods | Hypoxia-inducible factor 1 | CDK1-cyclin B1 | Mitochondrial division inhibitor-1 (Mdivi-1) | Mitochondrial fission | Mitotic check point
Journal Article
Acta biomaterialia, ISSN 1742-7061, 04/2012, Volume 8, Issue 4, pp. 1440 - 1449
.... However, few in vitro studies have been performed to identify the effects of environmental elasticity on the differentiation of MSC into vascular cell types... 
Mesenchymal stem cell | Elasticity | Vascular differentiation | Nanofiber | 3-D matrix | MATERIALS SCIENCE, BIOMATERIALS | STIFFNESS | ENGINEERING, BIOMEDICAL | EXTRACELLULAR-MATRIX | SCAFFOLD | Cross-Linking Reagents - pharmacology | Up-Regulation - radiation effects | Tensile Strength - drug effects | Elastic Modulus - drug effects | Polyethylene Glycols - chemistry | Methacrylates - chemistry | Spectroscopy, Fourier Transform Infrared | Tissue Scaffolds - chemistry | Polymerization - drug effects | Mesenchymal Stromal Cells - cytology | Ultraviolet Rays | Time Factors | Polymerase Chain Reaction | Compressive Strength - drug effects | Compressive Strength - radiation effects | Hydrogel, Polyethylene Glycol Dimethacrylate - pharmacology | Porosity - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Cell Differentiation - radiation effects | Nanofibers - chemistry | Mesenchymal Stromal Cells - drug effects | Nanofibers - ultrastructure | Polymerization - radiation effects | Endothelial Cells - metabolism | Mesenchymal Stromal Cells - metabolism | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Elasticity - drug effects | Endothelial Cells - radiation effects | Materials Testing | Elasticity - radiation effects | Muscle, Smooth, Vascular - cytology | Tensile Strength - radiation effects | Porosity - radiation effects | Elastic Modulus - radiation effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Cell Differentiation - drug effects | Endothelial Cells - cytology | Gene Expression Regulation - radiation effects | Endothelial Cells - drug effects | Actin | Polyethylene glycol | Stem cells | Smooth muscle | Muscle proteins | Polyols | Nanotechnology | Endothelium | mesenchymal stem cell | vascular differentiation | 3D matrix | nanofiber
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 10, p. e47807
.... We focused on the effect of Pi levels on human aortic vascular smooth muscle cells (VSMCs), with an emphasis on the role of microRNAs (miRNAs... 
MIR-145 | METABOLISM | MULTIDISCIPLINARY SCIENCES | GROWTH | GENE-EXPRESSION | PHENOTYPE | CALCIFICATION | MICE | MICRORNA-223 | Cell Proliferation | Apolipoproteins E - deficiency | Gene Expression - drug effects | Muscle, Smooth, Vascular - metabolism | Humans | Vascular Calcification - genetics | MicroRNAs - metabolism | Aorta - metabolism | rhoB GTP-Binding Protein - metabolism | Vascular Calcification - metabolism | Versicans - genetics | Kruppel-Like Transcription Factors - metabolism | rhoB GTP-Binding Protein - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Phosphates - pharmacology | Actin Cytoskeleton - genetics | MEF2 Transcription Factors | Muscle, Smooth, Vascular - drug effects | Actin Cytoskeleton - metabolism | Myogenic Regulatory Factors - genetics | Aorta - drug effects | MADS Domain Proteins - genetics | Myogenic Regulatory Factors - metabolism | Down-Regulation - drug effects | Muscle, Smooth, Vascular - cytology | Mice, Knockout | MADS Domain Proteins - metabolism | Up-Regulation - drug effects | Cell Movement - drug effects | Phosphates - metabolism | Animals | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Versicans - metabolism | Aorta - cytology | Primary Cell Culture | Kruppel-Like Transcription Factors - genetics | Phosphates | Vascular smooth muscle | Research | Biological markers | Cell proliferation | Calcification (ectopic) | Leukocyte migration | Syngeneic grafts | Downstream effects | Smooth muscle | Risk factors | Biomedical materials | Cell growth | Apolipoprotein E | Actin | Rodents | Aorta | Biocompatibility | miRNA | Physiology | Versican | Complications | Health risks | Muscles | Metabolism | Gene expression | Medicine | Osteoclastogenesis | MicroRNAs | Pharmacy | Coronary vessels | Calcification | Biomarkers | Cytoskeleton | Cell migration | Apoptosis | Kidney transplantation
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2014, Volume 34, Issue 2, pp. 355 - 364
OBJECTIVE—Vascular remodeling occurs after endothelial injury, resulting in smooth muscle cell (SMC... 
placental growth factor | receptors mineralocorticoid | vascular endothelial growth factor receptor-1 | aldosterone | myocytes smooth muscle | MORTALITY | OXIDATIVE STRESS | EVENTS | receptors | smooth muscle | myocytes | mineralocorticoid | BLOOD-PRESSURE | SPIRONOLACTONE | PERIPHERAL VASCULAR DISEASE | EPLERENONE | HYPERTENSION | HEMATOLOGY | EXPRESSION | BLOCKER | GENE-TRANSCRIPTION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Receptors, Mineralocorticoid - agonists | Receptors, Mineralocorticoid - genetics | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Muscle, Smooth, Vascular - metabolism | Pregnancy Proteins - genetics | Myocytes, Smooth Muscle - pathology | Male | Pregnancy Proteins - metabolism | RNA, Messenger - metabolism | Receptors, Mineralocorticoid - deficiency | Time Factors | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aldosterone - pharmacology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Animals | Fibrosis | Placenta Growth Factor | Carotid Arteries - pathology | Cell Proliferation - drug effects | Mice | Endothelial Cells - pathology | Endothelial Cells - drug effects | smooth muscle cells | mineralocorticoid receptor | vascular remodeling | VEGF
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2009, Volume 119, Issue 10, pp. 1424 - 1432
Background - Atherosclerosis is an inflammatory disease in which interferon (IFN)-gamma, the signature cytokine of Th1 cells, plays a central role. We... 
Smooth | Inflammation | Muscle | Interleukins | Lymphocytes | Coronary disease | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | lymphocytes | muscle, smooth | AUTOIMMUNE INFLAMMATION | HELPER-CELLS | ATHEROSCLEROSIS | IL-23 | interleukins | coronary disease | GROWTH-FACTOR-BETA | inflammation | DISEASE | IL-17 | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Vasculitis - physiopathology | Humans | Middle Aged | Receptors, Interleukin-17 - immunology | Vasculitis - etiology | Interleukin-17 - physiology | Myocytes, Smooth Muscle - pathology | Male | Interferon-gamma - metabolism | Chemokine CXCL10 - biosynthesis | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Interferon-gamma - antagonists & inhibitors | Adult | Female | Interleukin-8 - metabolism | Interleukins - pharmacology | Receptors, Interferon - antagonists & inhibitors | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Transforming Growth Factor beta1 - pharmacology | Cells, Cultured - drug effects | Coronary Vessels - drug effects | Interleukin-8 - biosynthesis | CD4-Positive T-Lymphocytes - metabolism | Interferon-gamma - physiology | Receptors, Interleukin-17 - antagonists & inhibitors | Interleukin-17 - biosynthesis | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Muscle, Smooth, Vascular - pathology | Receptors, Interferon - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Coronary Artery Disease - complications | Interleukin-6 - biosynthesis | Aged | Cells, Cultured - metabolism | Coronary Artery Disease - immunology | Chemokine CXCL10 - metabolism | Interferon-gamma - biosynthesis | muscle | smooth
Journal Article