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Journal of allergy and clinical immunology, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 769 - 780
... on mitochondrial function in the pathophysiology of airway inflammation in ozone-exposed mice and human airway smooth muscle (ASM) cells... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | ASTHMA FEATURES | MECHANISMS | IMMUNOLOGY | FRAGMENTATION | HYPERRESPONSIVENESS | BETA | RESPONSES | ALLERGY | ENERGY-METABOLISM | EXPRESSION | COPD | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2013, Volume 33, Issue 1, pp. 67 - 75
.... We investigated here whether Gal-3 is involved in Aldo-induced vascular fibrosis. Methods and Results-In rat vascular smooth muscle cells Gal-3 overexpression enhanced specifically collagen type I synthesis... 
collagen type I | aldosterone | HEART-FAILURE | LEFT-VENTRICULAR DYSFUNCTION | INFLAMMATION | ENDOTHELIAL-CELLS | fibrosis | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | galectin-3 | vascular smooth muscle cells | EXPRESSION | INSIGHTS | CARDIOTROPHIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Blood Pressure | Inflammation - chemically induced | Inflammation - pathology | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Humans | Galectin 3 - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aldosterone | Muscle, Smooth, Vascular - physiopathology | Galectin 3 - deficiency | Inflammation - metabolism | Transfection | RNA Interference | Time Factors | Hypertension - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Hypertension - prevention & control | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Vascular Stiffness | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Mice, Inbred C57BL | Cells, Cultured | Rats | Galectin 3 - genetics | Hypertension - pathology | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Collagen Type I - biosynthesis | Muscle, Smooth, Vascular - pathology | Animals | Galectin 3 - antagonists & inhibitors | Fibrosis | Inflammation - genetics | Inflammation - prevention & control | Mice | Inflammation - physiopathology | Cellular Biology | Life Sciences | Muscle, Smooth, Vascular | Mineralocorticoid Receptor Antagonists | Hypertension | Hematology | Biochemistry, Molecular Biology | Collagen Type I | Inflammation | Human health and pathology | Myocytes, Smooth Muscle | Galectin 3
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2015, Volume 64, Issue 5, pp. 1444 - 1449
Journal Article
Circulation research, ISSN 1524-4571, 2017, Volume 120, Issue 12, pp. 1903 - 1915
Journal Article
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2014, Volume 129, Issue 15, pp. 1551 - 1559
Journal Article
Circulation, ISSN 0009-7322, 04/2006, Volume 113, Issue 15, pp. 1857 - 1864
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2009, Volume 119, Issue 10, pp. 1424 - 1432
Background - Atherosclerosis is an inflammatory disease in which interferon (IFN)-gamma, the signature cytokine of Th1 cells, plays a central role. We... 
Smooth | Inflammation | Muscle | Interleukins | Lymphocytes | Coronary disease | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | lymphocytes | muscle, smooth | AUTOIMMUNE INFLAMMATION | HELPER-CELLS | ATHEROSCLEROSIS | IL-23 | interleukins | coronary disease | GROWTH-FACTOR-BETA | inflammation | DISEASE | IL-17 | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Vasculitis - physiopathology | Humans | Middle Aged | Receptors, Interleukin-17 - immunology | Vasculitis - etiology | Interleukin-17 - physiology | Myocytes, Smooth Muscle - pathology | Male | Interferon-gamma - metabolism | Chemokine CXCL10 - biosynthesis | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Interferon-gamma - antagonists & inhibitors | Adult | Female | Interleukin-8 - metabolism | Interleukins - pharmacology | Receptors, Interferon - antagonists & inhibitors | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Transforming Growth Factor beta1 - pharmacology | Cells, Cultured - drug effects | Coronary Vessels - drug effects | Interleukin-8 - biosynthesis | CD4-Positive T-Lymphocytes - metabolism | Interferon-gamma - physiology | Receptors, Interleukin-17 - antagonists & inhibitors | Interleukin-17 - biosynthesis | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Muscle, Smooth, Vascular - pathology | Receptors, Interferon - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Coronary Artery Disease - complications | Interleukin-6 - biosynthesis | Aged | Cells, Cultured - metabolism | Coronary Artery Disease - immunology | Chemokine CXCL10 - metabolism | Interferon-gamma - biosynthesis | muscle | smooth
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2015, Volume 35, Issue 3, pp. 535 - 546
OBJECTIVE—We previously showed that cholesterol loading in vitro converts mouse aortic vascular smooth muscle cells (VSMC... 
Macrophages | Atherosclerotic | Cholesterol | Atherosclerosis | Plaque | atherosclerosis | MYOCARDIN | FOAM CELL | ACTIVATION | FATTY STREAK | plaque, atherosclerotic | HIGH-DENSITY-LIPOPROTEIN | macrophages | METABOLISM | GENE-EXPRESSION | cholesterol | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | MASTER REGULATOR | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Oligonucleotide Array Sequence Analysis | Atherosclerosis - genetics | Coculture Techniques | Humans | Myocytes, Smooth Muscle - pathology | MicroRNAs - metabolism | Necrosis | Transfection | Time Factors | Trans-Activators - genetics | Cell Transdifferentiation | Sterol Regulatory Element Binding Protein 2 - metabolism | Nuclear Proteins - genetics | Binding Sites | Foam Cells - pathology | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Foam Cells - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Signal Transduction | Cholesterol, HDL - metabolism | Jurkat Cells | Mice, Inbred C57BL | Gene Expression Regulation | Aorta, Thoracic - metabolism | Gene Expression Profiling - methods | Nuclear Proteins - metabolism | Cholesterol - metabolism | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Cell Lineage | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Trans-Activators - metabolism | MicroRNAs - genetics | Phagocytosis
Journal Article
Circulation Research, ISSN 0009-7330, 02/2010, Volume 106, Issue 2, pp. 363 - 372
Journal Article