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Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 994 - 1003
The use of human pluripotent stem cells for in vitro disease modelling and clinical applications requires protocols that convert these cells into relevant... 
DIRECTED DIFFERENTIATION | IN-VIVO | MOUSE | DEFINITIVE ENDODERM | GROWTH-FACTOR | HUMAN BLASTOCYSTS | STROMAL CELLS | WNT | CULTURE | LINES | CELL BIOLOGY | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium | Index Medicus
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2015, Volume 464, Issue 4, pp. 1022 - 1027
The thrombospondins (TSPs) are matricellular proteins that exert multifunctional effects by binding cytokines, cell-surface receptors and other proteins. TSPs... 
Cellular proliferation | Vascular smooth muscles | Thrombospondins | Gene expression profiling | Cartilage oligomeric matrix protein | Cell migration | CHEMOTAXIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | OLIGOMERIC MATRIX PROTEIN | PATHOLOGY | BIOPHYSICS | GROWTH-FACTOR | EXPRESSION | Gene Expression - drug effects | Thrombospondins - genetics | Cartilage Oligomeric Matrix Protein - pharmacology | Neointima - etiology | Thrombospondin 1 - physiology | Humans | Cell Proliferation - physiology | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Chemotaxis - drug effects | Cell Movement - physiology | Cartilage Oligomeric Matrix Protein - genetics | Thrombospondins - pharmacology | Cell Movement - drug effects | Chemotaxis - physiology | Thrombospondins - physiology | Thrombospondin 1 - genetics | Thrombospondin 1 - pharmacology | Cell Proliferation - drug effects | Cartilage Oligomeric Matrix Protein - physiology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Proteins | Physiological aspects | Smooth muscle | Gene expression | Protein binding | Analysis | Genes | Index Medicus | HUMAN POPULATIONS | APOPTOSIS | PHYSIOLOGY | CELL PROLIFERATION | CARDIOVASCULAR SYSTEM | COMPARATIVE EVALUATIONS | 60 APPLIED LIFE SCIENCES | POLYMERASE CHAIN REACTION | LYMPHOKINES | DOSES | INFLAMMATION | GENES | RECEPTORS | MUSCLES | SURFACES | Cell Migration | Gene Expression Profiling | Cartilage Oligomeric Matrix Protein | Vascular Smooth Muscles | Cellular Proliferation
Journal Article
Circulation, ISSN 0009-7322, 10/2013, Volume 128, Issue 18, pp. 2047 - 2057
Journal Article
1997, ISBN 9780521482103, xv, 293
Smooth muscles line many internal organs and, in general, are involved in moving fluids and slurry around the body. They are controlled by the action of... 
Muscle, Smooth | physiology | Muscle cells | Smooth muscle | Muscle Contraction | Molecular aspects | Physiology
Book
Circulation, ISSN 0009-7322, 05/2010, Volume 121, Issue 20, pp. 2200 - 2210
Background-Hutchinson-Gilford progeria syndrome is a rare inherited disorder of premature aging caused by mutations in LMNA or Zmpste24 that disrupt nuclear... 
Nuclear lamina | Aging | Smooth | Muscle | Arteriosclerosis | Progeria | SYNDROME MUTATION | LOCALIZATION | DEFECTS | CARDIAC & CARDIOVASCULAR SYSTEMS | muscle, smooth | ATHEROSCLEROSIS | progeria | arteriosclerosis | LAMIN-A | HUTCHINSON-GILFORD-PROGERIA | MOUSE MODEL | DISEASE | nuclear lamina | CALCIFICATION | PERIPHERAL VASCULAR DISEASE | MICE | aging | Metalloendopeptidases - genetics | Up-Regulation | Mitosis | Humans | Oxidative Stress - physiology | Myocytes, Smooth Muscle - pathology | Metalloendopeptidases - metabolism | RNA, Messenger - metabolism | Time Factors | Membrane Proteins - metabolism | Muscle, Smooth, Vascular - physiology | Biomarkers - metabolism | Atherosclerosis - pathology | Membrane Proteins - genetics | Down-Regulation | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Cellular Senescence - physiology | Nuclear Proteins - metabolism | Lamin Type A - metabolism | Cell Nucleus - ultrastructure | Muscle, Smooth, Vascular - cytology | Atherosclerosis - metabolism | Protein Precursors - metabolism | Cell Division - physiology | Muscle, Smooth, Vascular - pathology | Aging - physiology | DNA Damage | Blood Vessels - physiology | Physiological aspects | Development and progression | Genetic aspects | Research | Vascular smooth muscle | Intermediate filament proteins | Index Medicus | Abridged Index Medicus
Journal Article