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Gastroenterology, ISSN 0016-5085, 04/2018, Volume 154, Issue 5, pp. 1465 - 1479.e13
Cirrhosis results from accumulation of myofibroblasts derived from quiescent hepatic stellate cells (Q-HSCs); it regresses when myofibroblastic HSCs are... 
Metabolic Reprogramming | Liver Diseases | Hippo Pathway | Fibrogenesis | FIBROSIS | TRANSCRIPTIONAL ACTIVITY | PULMONARY-HYPERTENSION | FATTY LIVER-DISEASE | MECHANISMS | CANCER | METABOLISM | GLUCOSE | GROWTH | Fibro-genesis | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Liver - pathology | Cell Proliferation | Mitochondria, Liver - metabolism | Humans | Glutamine - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Smoothened Receptor - metabolism | Phosphoproteins - metabolism | Smoothened Receptor - genetics | Case-Control Studies | Cellular Reprogramming | Myofibroblasts - metabolism | Transfection | Hedgehog Proteins - genetics | RNA Interference | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis, Experimental - pathology | Cell Transdifferentiation | Ketoglutaric Acids - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | Mitochondria, Liver - pathology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Rats | Glutaminase - metabolism | Liver Cirrhosis, Experimental - genetics | Phosphoproteins - genetics | Mice, Knockout | Phenotype | Animals | Energy Metabolism | Adaptor Proteins, Signal Transducing - genetics | Liver Cirrhosis - pathology | Adaptor Proteins, Signal Transducing - metabolism | Liver Cirrhosis, Experimental - metabolism | Glucose metabolism | Metabolites | RNA | Analysis | Liver | Physiological aspects | Genetic research | Verteporfin | Development and progression | Mice | Liver cirrhosis | Glutamine | Index Medicus | Abridged Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2013, Volume 19, Issue 8, pp. 1047 - 1053
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 23, pp. 9621 - 9630
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 06/2012, Volume 11, Issue 1, pp. 73 - 73
Background: Alpha-lipoic acid (ALA), a naturally occurring compound, exerts powerful protective effects in various cardiovascular disease models. However, its... 
Alpha-Lipoic acid | Mitochondrial oxidative stress | Cardiac fibrosis | Extracellular matrix remodeling | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | METALLOTHIONEIN | KINASE | RATS | FIBROBLASTS | HEART | PATHOGENESIS | OVEREXPRESSION | ATTENUATION | ENDOCRINOLOGY & METABOLISM | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Diabetic Cardiomyopathies - metabolism | Mitochondria, Heart - metabolism | Diabetic Cardiomyopathies - etiology | Rats, Wistar | Actins - metabolism | Extracellular Matrix - metabolism | Male | Mitochondria, Heart - drug effects | Diabetic Cardiomyopathies - physiopathology | Collagen Type II - metabolism | Myofibroblasts - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Cardiac Catheterization | Myocardium - metabolism | Gelatinases - metabolism | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Myofibroblasts - pathology | Collagen Type I - metabolism | Matrix Metalloproteinase 2 - metabolism | Extracellular Matrix - drug effects | Ventricular Function, Left - drug effects | Rats | Myocardium - pathology | Thioctic Acid - pharmacology | Cardiotonic Agents - pharmacology | Myofibroblasts - drug effects | Blotting, Western | Enzyme Precursors - metabolism | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Fibrosis | Diabetes Mellitus, Experimental - pathology | Diabetic Cardiomyopathies - pathology | Enzyme Activation | Oxidative Stress - drug effects | Spectrophotometry | Transforming Growth Factor beta - metabolism | Ventricular Remodeling - drug effects | Mitogen-Activated Protein Kinases - metabolism | Proteins | Medical research | Cardiovascular disease | Diabetes | Rodents | Metabolic disorders | Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 03/2014, Volume 306, Issue 6, pp. L534 - L542
MicroRNAs are small noncoding RNAs that inhibit protein expression. We have previously shown that the inhibition of the microRNA let-7d in epithelial cells... 
High-mobility group-A2 protein | Epithelial-to-mesenchymal transition | Idiopathic pulmonary fibrosis | Transforming growth factor-β | Fibrosis | microRNA | Slug | PHYSIOLOGY | METASTASIS | idiopathic pulmonary fibrosis | INDUCTION | EMT | high-mobility group-A2 protein | epithelial-to-mesenchymal transition | transforming growth factor-beta | TRANSITION | LOOP-HELIX PROTEINS | REGULATOR | RESPIRATORY SYSTEM | DISEASE | fibrosis | GROWTH-FACTOR | DIFFERENTIATION | IDIOPATHIC PULMONARY-FIBROSIS | Cell Proliferation | HMGB2 Protein - metabolism | Cadherins - metabolism | Humans | Actins - metabolism | Pulmonary Fibrosis - genetics | Lung - cytology | Cell Movement - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Myofibroblasts - metabolism | Transfection | HMGA2 Protein - metabolism | Pulmonary Alveoli - metabolism | Epithelial-Mesenchymal Transition | Lung - metabolism | Pulmonary Fibrosis - metabolism | Wound Healing - genetics | Zonula Occludens-1 Protein - metabolism | Snail Family Transcription Factors | Fibroblasts - metabolism | Calcium-Binding Proteins - metabolism | Cells, Cultured | Transcription Factors - genetics | Fibronectins - metabolism | Transcription Factors - metabolism | Idiopathic Pulmonary Fibrosis - pathology | Fibroblasts - cytology | MicroRNAs - genetics | Keratin-19 - metabolism | Transforming Growth Factor beta - metabolism | MicroRNA | Physiological aspects | Fibroblasts | Phenotypic plasticity | Genetic research | Research | Transforming growth factors | Genotype & phenotype | Protein expression | Wound healing | Ribonucleic acid--RNA | Cells | Index Medicus | transforming growth factor-β
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, pp. e0186615 - e0186615
Background Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal disease. Histone deacetylase 6 (HDAC6) alters function and fate of various... 
EPITHELIAL-MESENCHYMAL TRANSITION | LUNG FIBROSIS | IN-VITRO | TGF-BETA | HISTONE DEACETYLASE INHIBITION | MULTIDISCIPLINARY SCIENCES | TUBULIN ACETYLATION | DISEASE | AUTOPHAGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | Idiopathic Pulmonary Fibrosis - genetics | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Phosphoprotein Phosphatases - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Mechanistic Target of Rapamycin Complex 1 | Autophagosomes - drug effects | Multiprotein Complexes - metabolism | Tubulin - metabolism | Bleomycin | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Aged, 80 and over | Female | Indoles - pharmacology | Lung - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Hydroxamic Acids - pharmacology | Fibroblasts - metabolism | Lung - pathology | Collagen Type I - metabolism | Histone Deacetylases - genetics | Histone Deacetylase 6 | RNA, Messenger - genetics | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | Autophagosomes - metabolism | Mice, Knockout | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Hydroxamic Acids - therapeutic use | Indoles - therapeutic use | Aged | Transforming Growth Factor beta - metabolism | Idiopathic Pulmonary Fibrosis - drug therapy | Histone deacetylase | Deregulation | Collagen (type I) | Phosphorylation | Disease | Mesenchyme | Pathogenesis | Lung | Critical care | AKT protein | Leucine | Kinases | Autophagy | Proteins | Fibroblasts | Vascular endothelial growth factor | Internal medicine | Lung diseases | Environmental health | 1-Phosphatidylinositol 3-kinase | Medicine | Pulmonary fibrosis | Inhibitors | Lungs | Lysine | Deacetylation | Collagen | Fibrosis | Mice | Protein phosphatase | Phagocytosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2013, Volume 110, Issue 6, pp. 2324 - 2329
In many organs, myofibroblasts play a major role in the scarring process in response to injury. In liver fibrogenesis, hepatic stellate cells (HSCs) are... 
Phenotypes | Hepatocytes | Epithelial cells | Liver | Fibrosis | Cell lines | Antibodies | Hepatic stellate cells | Myofibroblasts | Liver cells | Epithelial-mesenchymal transition | Alpha-smooth muscle actin | Glisson's capsule | Podoplanin | alpha-smooth muscle actin | PATHWAYS | ACID | RAT | MULTIDISCIPLINARY SCIENCES | MOUSE | RECEPTOR | POPULATIONS | podoplanin | SMOOTH-MUSCLE | fibrosis | LINEAGE | Liver - pathology | WT1 Proteins - metabolism | Membrane Glycoproteins - metabolism | Hepatic Stellate Cells - metabolism | RNA, Messenger - metabolism | Liver - injuries | Myofibroblasts - metabolism | Biliary Tract - metabolism | Cell Transdifferentiation - drug effects | Liver Cirrhosis - metabolism | Chemical and Drug Induced Liver Injury - pathology | WT1 Proteins - genetics | Hepatic Stellate Cells - pathology | Liver Cirrhosis - etiology | Myofibroblasts - pathology | Epithelium - pathology | Gene Expression | Liver Regeneration | Epithelium - metabolism | Signal Transduction | Liver - metabolism | RNA, Messenger - genetics | Cells, Cultured | Mice, Transgenic | Chemical and Drug Induced Liver Injury - genetics | Carbon Tetrachloride - toxicity | Biliary Tract - pathology | Nerve Tissue Proteins - metabolism | Cell Lineage | Transforming Growth Factor beta - pharmacology | Animals | Chemical and Drug Induced Liver Injury - metabolism | Liver Cirrhosis - pathology | Mesoderm - metabolism | Mice | Mesoderm - pathology | Transforming Growth Factor beta - metabolism | Glycoproteins | Liver diseases | Rodents | Cells | Tumors | Index Medicus | Biological Sciences
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 88, pp. 120 - 123
Cardiovascular | Cardiac fibrosis | Marker | Proto-myofibroblast | Fibroblast activating protein | Myofibroblast | FIBROSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | MYOCARDIAL-INFARCTION | TISSUE | IDENTIFICATION | PERIOSTIN | CELL BIOLOGY | CARDIAC FIBROBLASTS | DISEASE | MYOFIBROBLASTS | EXPRESSION | Cell Adhesion Molecules - genetics | Vimentin - metabolism | Humans | Actins - metabolism | Ectodysplasins - metabolism | Actins - genetics | Procollagen-Proline Dioxygenase - metabolism | Myofibroblasts - metabolism | Discoidin Domain Receptors | Endomyocardial Fibrosis - diagnosis | Vimentin - genetics | Thy-1 Antigens - genetics | Gelatinases - metabolism | Serine Endopeptidases - genetics | Endomyocardial Fibrosis - genetics | Cell Differentiation | Endomyocardial Fibrosis - pathology | Membrane Proteins - metabolism | Fibroblasts - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Myofibroblasts - pathology | Ectodysplasins - genetics | Membrane Proteins - genetics | Gene Expression Regulation | Receptors, Mitogen - metabolism | Fibroblasts - pathology | Receptor Protein-Tyrosine Kinases - metabolism | Cell Adhesion Molecules - metabolism | Thy-1 Antigens - metabolism | Animals | Procollagen-Proline Dioxygenase - genetics | Receptor Protein-Tyrosine Kinases - genetics | Gelatinases - genetics | Endomyocardial Fibrosis - metabolism | Serine Endopeptidases - metabolism | Calcium-Binding Proteins - genetics | Receptors, Mitogen - genetics | Index Medicus
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 938 - 946
Background & Aims The pathogenesis of liver fibrosis involves activation of hepatic stellate cells, which is associated with depletion of intracellular lipid... 
Gastroenterology and Hepatology | Inflammation | Myofibroblasts | Energy Depletion | Mouse Model | RAT-LIVER | FIBROSIS | PATHWAY | DISEASE | MECHANISMS | RETINOL | GASTROENTEROLOGY & HEPATOLOGY | Liver - pathology | Kidney - pathology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Carbon Tetrachloride | Humans | Hepatic Stellate Cells - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Kidney - metabolism | Epoxy Compounds - pharmacology | Liver - drug effects | RNA Interference | Adenosine Triphosphate - metabolism | Liver Cirrhosis, Experimental - pathology | Lung - metabolism | Autophagy - genetics | Microtubule-Associated Proteins - deficiency | Hepatic Stellate Cells - pathology | Fibroblasts - metabolism | Hepatic Stellate Cells - drug effects | Cell Line | Lung - pathology | Oleic Acid - metabolism | Adenine - analogs & derivatives | Liver - metabolism | Mice, Inbred C57BL | Adenine - pharmacology | Liver Cirrhosis, Experimental - genetics | Fibroblasts - pathology | Mice, Knockout | Thioacetamide | Autophagy-Related Protein 7 | Animals | Autophagy-Related Protein 5 | Energy Metabolism | Fibroblasts - drug effects | Liver Cirrhosis, Experimental - chemically induced | Lipid Metabolism - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Mice | Liver Cirrhosis, Experimental - metabolism | Phosphates | Medical colleges | Neurosciences | Platelet-derived growth factor | Liver diseases | Albumin | Lipids | Muscle proteins | Fatty acids | Cells | Monosaccharides | Unsaturated fatty acids | Monounsaturated fatty acids | Actin | Analysis | Intermediate filament proteins | Sugars | Index Medicus | Abridged Index Medicus
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 2, pp. 189 - 198
Journal Article
Cell Stem Cell, ISSN 1934-5909, 01/2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | KIDNEY FIBROSIS | SONIC HEDGEHOG | BONE-MARROW NICHE | NG2 PROTEOGLYCAN | CARDIAC FIBROSIS | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects | Index Medicus
Journal Article