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Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2017, Volume 37, Issue 1, pp. 26 - 34
The importance of maintaining contractile function in aortic smooth muscle cells (SMCs) is evident by the fact that heterozygous mutations in the major... 
mutation | thoracic aorta | aortic aneurysm | aortic dissection | vascular smooth muscle cells | Marfan syndrome | LIGHT-CHAIN KINASE | ACTIN POLYMERIZATION | DOMAIN-STRUCTURE | TGFBR2 MUTATIONS | MYOSIN HEAVY-CHAIN | BETA RECEPTOR | MARFAN-SYNDROME | MOUSE MODEL | DISEASE | PERIPHERAL VASCULAR DISEASE | PATENT DUCTUS-ARTERIOSUS | HEMATOLOGY | Myosin-Light-Chain Kinase - genetics | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Genetic Testing | Muscle, Smooth, Vascular - metabolism | Pulsatile Flow | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Myosin Heavy Chains - genetics | Cyclic GMP-Dependent Protein Kinase Type I - genetics | Muscle, Smooth, Vascular - physiopathology | Myosin Heavy Chains - metabolism | Actins - genetics | Cyclic GMP-Dependent Protein Kinase Type I - metabolism | Aneurysm, Dissecting - physiopathology | Aortic Aneurysm, Thoracic - pathology | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Calcium-Binding Proteins - metabolism | Vasoconstriction - genetics | Elastin - metabolism | Aneurysm, Dissecting - metabolism | Myosin-Light-Chain Kinase - metabolism | Heredity | Aneurysm, Dissecting - pathology | Genetic Markers | Aortic Aneurysm, Thoracic - physiopathology | Mechanotransduction, Cellular | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Aortic Aneurysm, Thoracic - metabolism | Mutation | Calcium-Binding Proteins - genetics | elastin-contractile unit | Contractile Function | Vascular Biology | Aneurysm | Genetics | vascular smooth muscle cell | Aortic Dissection | thoracic
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2016, Volume 36, Issue 2, pp. 266 - 273
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1330 - 1340.e1
Background & Aims Progression of liver fibrosis in experimental models depends on gut-derived bacterial products, but little is known about mechanisms of... 
Gastroenterology and Hepatology | Intestinal Inflammation | Endotoxin | Microbiota Composition | Microbiome | CELLS | BARRIER FUNCTION | RATS | BACTERIAL TRANSLOCATION | FACTOR-ALPHA | INFLAMMATION | CIRRHOSIS | DISEASE | CARBON-TETRACHLORIDE | GASTROENTEROLOGY & HEPATOLOGY | EPITHELIAL PERMEABILITY | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Toll-Like Receptor 2 - genetics | Cholestasis - complications | Cell Count | Male | Monocytes - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Ligation | Liver Cirrhosis - metabolism | Statistics, Nonparametric | Cardiac Myosins - metabolism | Disease Models, Animal | Tight Junctions - metabolism | Signal Transduction | Mice, Inbred C57BL | Intestinal Mucosa - microbiology | Toll-Like Receptor 2 - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Bacterial Translocation | Mice, Knockout | Animals | Bile Ducts | rho GTP-Binding Proteins - metabolism | Liver Cirrhosis - pathology | Liver Cirrhosis - microbiology | Mice | Myosin Light Chains - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Liver diseases | Tumor necrosis factor | Analysis | Liver | Fibrosis | Myosin | Fluorescence | Muscle proteins | Transforming growth factors | Tumors | Necrosis | Medical research | Microbiota (Symbiotic organisms) | Medicine, Experimental | microbiota composition | intestinal inflammation | microbiome | endotoxin
Journal Article
Genes and Development, ISSN 0890-9369, 05/2008, Volume 22, Issue 9, pp. 1231 - 1243
During vasculogenesis and angiogenesis, endothelial cell responses to growth factors are modulated by the compositional and mechanical properties of a... 
Angiogenesis | Extracellular matrix | Actomyosin | Endothelial cells | Fibronectin | VASCULAR DEVELOPMENT | endothelial cells | GROWTH-CONTROL | ASSEMBLY SITES | angiogenesis | MOUSE EMBRYOS | DEVELOPMENTAL BIOLOGY | CELL-CYCLE PROGRESSION | CELL BIOLOGY | IN-VITRO | fibronectin | actomyosin | BALLISTIC INTRACELLULAR NANORHEOLOGY | ENDOTHELIAL-CELLS | GENETICS & HEREDITY | EXTRACELLULAR MATRIX-CYTOSKELETON | extracellular matrix | Neovascularization, Physiologic - drug effects | Phosphorylation | Humans | Vinculin - metabolism | Extracellular Matrix - metabolism | Immunoblotting | Green Fluorescent Proteins - genetics | Hepatocyte Growth Factor - pharmacology | Recombinant Fusion Proteins - metabolism | Extracellular Matrix - chemistry | Transfection | Myosins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Culture Techniques | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Green Fluorescent Proteins - metabolism | Collagen Type I - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Cell Adhesion | Fibronectins - metabolism | Protein Folding | Microscopy, Confocal | Neovascularization, Physiologic - physiology | Vinculin - genetics | Fibronectins - chemistry | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Cytoskeleton - metabolism | Recombinant Fusion Proteins - genetics | Cytoskeleton - physiology | Fibrin - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Fibronectins | Research | Neovascularization | Research Paper
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2007, Volume 292, Issue 5, pp. 1660 - 1671
Although p38 MAPK activation is essential for myogenesis, the upstream signaling mechanism that activates p38 during myogenesis remains undefined. We recently... 
Myosin heavy chain | Myogenin | Myocyte enhancer factor-2C | Mitogen-activated protein kinase | Akt | Tumor necrosis factor-α | p21 | myocyte enhancer factor-2C | myosin heavy chain | PROTEIN-KINASE PATHWAY | PHYSIOLOGY | myogenin | UBIQUITIN LIGASE | INFLAMMATORY MYOPATHIES | AUTOCRINE SECRETION | CELL BIOLOGY | tumor necrosis factor-alpha | mitogen-activated protein kinase | SKELETAL-MUSCLE | TUMOR-NECROSIS-FACTOR | SIGNALING PATHWAY | GENE-EXPRESSION | TERMINAL DIFFERENTIATION | GROWTH-FACTOR-I | Tumor Necrosis Factor-alpha - metabolism | Cobra Cardiotoxin Proteins | Muscle, Skeletal - metabolism | Muscle Fibers, Skeletal - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Dose-Response Relationship, Drug | MAP Kinase Kinase 6 - metabolism | Receptors, Tumor Necrosis Factor, Type I - deficiency | Myoblasts - metabolism | Muscular Diseases - physiopathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Muscle, Skeletal - drug effects | Cell Differentiation | p38 Mitogen-Activated Protein Kinases - metabolism | Muscular Diseases - chemically induced | Disease Models, Animal | Autocrine Communication | Cell Line | Muscular Diseases - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Tumor Necrosis Factor-alpha - pharmacology | Regeneration - drug effects | Animals | Muscle Development - drug effects | Muscle, Skeletal - physiopathology | Mice | Enzyme Activation | Muscle, Skeletal - pathology | tumor necrosis factor-α
Journal Article
Journal Article