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Neuropsychopharmacology, ISSN 0893-133X, 04/2013, Volume 38, Issue 5, pp. 743 - 752
The NMDA-receptor antagonist ketamine has proven efficient in reducing symptoms of suicidality, although the mechanisms explaining this effect have not been... 
cytokine | quinolinic acid | depression | glutamate | suicide | PSYCHIATRY | RESISTANT DEPRESSION | ADD-ON TRIAL | D-ASPARTATE ANTAGONIST | NEUROSCIENCES | HIPPOCAMPAL VOLUME | EMERGENCY-DEPARTMENT | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | KYNURENIC ACID | MAJOR DEPRESSIVE DISORDER | Encephalitis - psychology | Excitatory Amino Acid Agonists - cerebrospinal fluid | Kynurenine - cerebrospinal fluid | Quinolinic Acid - cerebrospinal fluid | Excitatory Amino Acid Agonists - therapeutic use | Follow-Up Studies | Somatosensory Disorders - complications | Spinal Puncture | Humans | Middle Aged | Male | Mental Disorders - complications | Kynurenic Acid - cerebrospinal fluid | Mental Disorders - psychology | Young Adult | Psychiatric Status Rating Scales | Tritium - cerebrospinal fluid | Encephalitis - cerebrospinal fluid | Suicide - psychology | Adult | Female | Aged | Retrospective Studies | Interleukin-6 - metabolism | Index Medicus | Brain | Glutamic acid | Glutamic acid receptors (ionotropic) | Depression | Mass spectroscopy | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Inflammation | Cerebrospinal fluid | High-performance liquid chromatography | Suicide | Interleukin 6 | Gas chromatography | Ketamine | Glutamatergic transmission | Kynurenic acid | Quinolinic acid | Neurotransmission | Original | Medical and Health Sciences | Medicin och hälsovetenskap | MEDICIN | MEDICINE
Journal Article
Glia, ISSN 0894-1491, 11/2011, Volume 59, Issue 11, pp. 1600 - 1611
We have previously demonstrated that human astrocytes are GABAergic cells. Throughout the adult human brain, they express the GABA synthesizing enzyme GAD 67,... 
serine | calcium chelation | glycine | GABA transporters | GABA receptors | glutamate | glutamate receptors | Glutamate receptors | D-serine | Glutamate | Glycine | Calcium chelation | CA2+ INFLUX | CELLS | TRANSPORTER | RAT | SYNAPTIC-TRANSMISSION | RECEPTOR | IDENTIFICATION | NEUROSCIENCES | INDICATORS | BRAIN | Receptors, Glutamate - metabolism | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Humans | Calcium - analysis | RNA, Messenger - biosynthesis | Vigabatrin - pharmacology | Culture Media | Egtazic Acid - analogs & derivatives | Astrocytes - chemistry | Real-Time Polymerase Chain Reaction | Cell Survival - drug effects | Glutamic Acid - pharmacology | RNA, Messenger - genetics | Cells, Cultured | Excitatory Amino Acid Agonists - pharmacology | Chelating Agents - pharmacology | DNA Primers | Vesicular Glutamate Transport Proteins - metabolism | Vesicular Glutamate Transport Proteins - genetics | Blotting, Western | Vesicular Glutamate Transport Proteins - antagonists & inhibitors | Egtazic Acid - pharmacology | Cyclohexanecarboxylic Acids - pharmacology | Astrocytes - metabolism | Cell culture | Enzymes | Glutamic acid | Calcium (intracellular) | 4-Aminobutyrate transaminase | Calcium | Astrocytes | Glutamic acid receptors (ionotropic) | Data processing | N-Methyl-D-aspartic acid receptors | Glutamate decarboxylase | Glutamic acid receptors | mRNA | gamma -Aminobutyric acid transporter | gamma -Aminobutyric acid B receptors | Neurotransmitters | gamma -Aminobutyric acid A receptors | Chelating agents | Index Medicus
Journal Article
Brain Research, ISSN 0006-8993, 1993, Volume 613, Issue 1, pp. 88 - 95
Journal Article
Neurochemical Research, ISSN 0364-3190, 10/2018, Volume 43, Issue 10, pp. 1938 - 1946
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, pp. e95318 - e95318
Background: Dietary long-chain n-3 polyunsaturated fatty acid (PUFA) supplementation may be beneficial for chronic brain illnesses, but the issue is not agreed... 
UNANESTHETIZED RATS | METABOLISM | DOCOSAHEXAENOIC ACID | MULTIDISCIPLINARY SCIENCES | FRONTAL-CORTEX | LIVER | DOUBLE-BLIND | POLYUNSATURATED FATTY-ACIDS | ARACHIDONIC-ACID | ALPHA-LINOLENIC ACID | BRAIN | Brain Chemistry - drug effects | Brain Diseases - metabolism | Dietary Fats - adverse effects | Nerve Growth Factor - metabolism | Rats | Male | Excitatory Amino Acid Agonists - pharmacology | Brain Diseases - chemically induced | N-Methylaspartate - pharmacology | N-Methylaspartate - adverse effects | Animals | Brain Diseases - pathology | Group IV Phospholipases A2 - metabolism | Dietary Fats - pharmacology | Fatty Acids, Omega-3 - pharmacology | Interleukin-1beta - metabolism | Lipid Metabolism - drug effects | Excitatory Amino Acid Agonists - adverse effects | Chronic Disease | Fatty Acids, Omega-3 - adverse effects | Physiological aspects | Methyl aspartate | Unsaturated fatty acids | Nerve growth factor | Essential fatty acids | Dietary supplements | Brain | Neurosciences | Polyunsaturated fatty acids | Weaning | Bipolar disorder | Arachidonic acid | Docosahexaenoic acid | Illnesses | Rodents | Fish | Lipid metabolism | Supplementation | Deprivation | Phospholipase A2 | Fish oils | Glial fibrillary acidic protein | Markers | IL-1β | Metabolism | Fatty acids | Brain-derived neurotrophic factor | Linolenic acid | Diet | Excitotoxicity | Fats and oils | N-Methyl-D-aspartic acid | Index Medicus
Journal Article
Neurochemical Research, ISSN 0364-3190, 11/2017, Volume 42, Issue 11, pp. 3279 - 3288
Irregular N-methyl-d-aspartate receptor (NMDAR) function is one of the main hypotheses employed to facilitate understanding of the underlying disease state of... 
Neurochemistry | Biochemistry, general | Enzyme occupancy | Neurology | d -Serine | Neurosciences | Biomedicine | Sodium benzoate | d -Amino acid oxidase (DAO) | N -methyl- d -aspartate receptor (NMDAR) | Cell Biology | d-Amino acid oxidase (DAO) | N-methyl-d-aspartate receptor (NMDAR) | d-Serine | D-Serine | BIOCHEMISTRY & MOLECULAR BIOLOGY | CINNAMON | N-methyl-D-aspartate receptor (NMDAR) | SCHIZOPHRENIA | ASTROCYTES | NEUROSCIENCES | IN-VITRO | DOUBLE-BLIND | D-Amino acid oxidase (DAO) | PLACEBO-CONTROLLED TRIAL | NMDA RECEPTOR FUNCTION | METABOLITE | T-CELLS | BRAIN | Biomarkers - metabolism | D-Amino-Acid Oxidase - antagonists & inhibitors | Humans | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Sodium Benzoate - chemistry | Crystallography, X-Ray | Male | Serine - metabolism | Animals | Enzyme Inhibitors - chemistry | Mice | Sodium Benzoate - pharmacology | D-Amino-Acid Oxidase - metabolism | Oxidases | Medical research | Therapeutics | Medicine, Experimental | Schizophrenia | Amino acids | Alzheimer's disease | Homeopathy | Materia medica and therapeutics | Benzoic acid | Cerebellum | Enzymes | Mental disorders | Oxidase | Clinical trials | Pyruvic acid | D-Amino-acid oxidase | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Glycine | D-Amino acids | Inhibitors | Sodium | Biomarkers | Bioindicators | Amino acid oxidase | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2018, Volume 144, Issue 4, pp. 408 - 420
We propose that ascorbate or dehydroascorbate promotes the internalization of excitatory aminoacid transporter type 3 (EAAT3) and thereby decreases glutamate... 
ascorbate | dehydroascorbate | CREB | aspartate uptake | purified and mixed retinal cultures | d-aspartate uptake | CELLS | CREB PHOSPHORYLATION | ACTIVATION | REDOX SITE | BIOCHEMISTRY & MOLECULAR BIOLOGY | D-aspartate uptake | DEHYDROASCORBIC ACID | RELEASE | ASCORBIC-ACID | NEUROSCIENCES | IN-VITRO | XENOPUS OOCYTES | PROTEIN-KINASE-II | Retina - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | Retina - metabolism | Biotinylation | Cells, Cultured | Extracellular Space - drug effects | Glutamates - metabolism | Chick Embryo | Vitamins - pharmacology | Extracellular Space - metabolism | Animals | Excitatory Amino Acid Transporter 3 - metabolism | Signal Transduction - drug effects | Chickens | Ascorbic Acid - pharmacology | Methyl aspartate | Aspartate | Government securities | Glutamate | Vitamin C | Protein binding | Phosphorylation | Ascorbic acid | Calcium | Glutamic acid receptors (ionotropic) | Central nervous system | Amino acids | Retina | Glutamic acid receptors | Kinases | Cell surface | Receptors | Calcium-binding protein | Ca2+/calmodulin-dependent protein kinase | Inhibition | Calmodulin | Retinal cells | Imaging techniques | N-Methyl-D-aspartic acid receptors | Excitatory amino acid transporter | Signaling | Receptor mechanisms | Glutamic acid transporter | Glutamatergic transmission | Transport | Transporter | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2012, Volume 109, Issue 2, pp. 627 - 632
Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disorder involving an extensive loss of motoneurons. Aberrant excitability of motoneurons has... 
Oxidases | Enzymes | Pathology | Brain | Spinal cord | Astrocytes | Neurons | Neuroglia | Amyotrophic lateral sclerosis | Mice | Neurodegeneration | Enzyme histochemistry | Excitotoxicity | 2D-HPLC | Motor neuron disease | GLYCINE | motor neuron disease | MULTIDISCIPLINARY SCIENCES | MOUSE | neurodegeneration | AMYOTROPHIC-LATERAL-SCLEROSIS | RACEMASE | enzyme histochemistry | PERFORMANCE LIQUID-CHROMATOGRAPHY | MOTOR-NEURON DEGENERATION | NMDA | XENOPUS-OOCYTES | RAT-BRAIN | excitotoxicity | D-ALANINE | Superoxide Dismutase - genetics | Mice, Inbred C57BL | D-Amino-Acid Oxidase - genetics | Gene Expression Regulation - physiology | DNA Primers - genetics | Chromatography, High Pressure Liquid | Serine - metabolism | Blotting, Western | Mutation, Missense - genetics | Mice, Knockout | Animals | Cell Death - physiology | Histological Techniques | Mutagenesis | Cloning, Molecular | Amyotrophic Lateral Sclerosis - enzymology | Amyotrophic Lateral Sclerosis - etiology | Superoxide Dismutase-1 | D-Amino-Acid Oxidase - metabolism | Real-Time Polymerase Chain Reaction | Astrocytes - metabolism | Physiological aspects | Motor neurons | Amino acids | Health aspects | Serine | Mutation | Rodents | Animal models | Neurodegenerative diseases | D-Serine | Excitability | D-Amino-acid oxidase | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Inflammation | reticulospinal tract | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, pp. e113954 - e113954
The etiology of multiple sclerosis (MS) is currently unknown. However, one potential mechanism involved in the disease may be excitotoxicity. The elevation of... 
EXCITOTOXICITY | ENCEPHALOMYELITIS | MK-801 | GROUP-I | MULTIPLE-SCLEROSIS | MULTIDISCIPLINARY SCIENCES | CALCIUM | NMDA | MODEL | ALTERED EXPRESSION | GLT-1 | Receptors, Glutamate - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Rats, Inbred Lew | Receptors, N-Methyl-D-Aspartate - metabolism | Rats | Excitatory Amino Acid Antagonists - pharmacology | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - metabolism | Brain - drug effects | Brain - metabolism | Animals | Female | Glutamic Acid - metabolism | Glutamic Acid - analysis | Methyl aspartate | Brain | Aspartate | RNA | Glutamate | Multiple sclerosis | Traumatic brain injury | Glutamic acid receptors | Cerebrospinal fluid | Sclerosis | Proteins | Receptors | Ischemia | Neurodegeneration | Etiology | Binding | Neurochemistry | Medical research | Amantadine | Cytokines | Astrocytes | Gene expression | Studies | Memantine | Ligands | Brain damage | Glutamatergic transmission | Transport | Glutamic acid receptors (metabotropic) | Transporter | Encephalomyelitis | Laboratories | Glutamic acid receptors (ionotropic) | Amino acids | mRNA | Experiments | RNA transport | Rodents | Excitation | Protein transport | Nerve endings | Neurons | Amyotrophic lateral sclerosis | N-Methyl-D-aspartic acid receptors | Excitatory amino acid transporter | Experimental allergic encephalomyelitis | Brain research | Excitotoxicity | Phenols | Regulation | Glutamic acid transporter | Brain injury | Apoptosis | Index Medicus
Journal Article