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Molecular Psychiatry, ISSN 1359-4184, 03/2018, Volume 23, Issue 3, pp. 597 - 608
Several preclinical studies have reported the rapid antidepressant effects of N-methyl-D-aspartate receptor (NMDAR) antagonists, although the underlying... 
PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | D-ASPARTATE-RECEPTOR | OPPOSING ROLES | GLUTAMATE UPTAKE | SYNAPTIC PLASTICITY | NEUROSCIENCES | KETAMINE | IN-VITRO | MOOD DISORDERS | MEDIAL PREFRONTAL CORTEX | MAJOR DEPRESSIVE DISORDER | DIFFERENTIAL ROLES | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Depression - physiopathology | Excitatory Amino Acid Antagonists - therapeutic use | Phosphorylation | Receptors, N-Methyl-D-Aspartate - metabolism | Death-Associated Protein Kinases - metabolism | Rats | Male | Rats, Sprague-Dawley | Animals | Signal Transduction - drug effects | Depressive Disorder - physiopathology | Stress, Psychological - metabolism | Prefrontal Cortex - metabolism | Antidepressive Agents - pharmacology | Glutamic Acid - metabolism | Chronic Disease | Disease Models, Animal | Care and treatment | Depression, Mental | Physiological aspects | Development and progression | Glutamate | Protein kinases | Health aspects | Neurotransmitter receptors | RNA viruses | Cortex (prefrontal) | Death-associated protein kinase | Glutamic acid receptors (ionotropic) | Astrocytes | N-Methyl-D-aspartic acid receptors | Viruses | Pharmacology | Glutamic acid receptors | Mental depression | Kinases | Ribonucleic acid--RNA | Critical components | Resistance | Antidepressants | Rodents | Conductance | Glutamic acid transporter | Conditioning | Transporter | Original
Journal Article
Journal Article
Neurochemical Research, ISSN 0364-3190, 10/2017, Volume 42, Issue 10, pp. 2712 - 2729
Previously we have demonstrated that brain-derived neurotrophic factor (BDNF) contributes to spinal long-term potentiation (LTP) and pain hypersensitivity... 
Neurochemistry | Biochemistry, general | Neurology | Spinal dorsal horn | Neurosciences | Biomedicine | Brain-derived neurotrophic factor | Long-term potentiation | Fyn | GluN2B-containing NMDA receptor | Cell Biology | Neuropathic pain | CENTRAL SENSITIZATION | NEUROTROPHIC FACTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | SYNAPTIC-TRANSMISSION | NR2B PHOSPHORYLATION | PATHOLOGICAL PAIN | D-ASPARTATE RECEPTOR | NEUROSCIENCES | DORSAL-HORN | SRC FAMILY KINASES | NEONATAL HYPOXIA-ISCHEMIA | Phosphorylation | Hyperalgesia - metabolism | Neuralgia - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Spinal Nerves - metabolism | Rats, Sprague-Dawley | Tyrosine - metabolism | Animals | Proto-Oncogene Proteins c-fyn - metabolism | Long-Term Potentiation - drug effects | Peripheral Nerve Injuries - metabolism | Long-Term Potentiation - physiology | Brain-Derived Neurotrophic Factor - metabolism | Tyrosine | Methyl aspartate | Allergy | Neurons | Phenols | Allergic reaction | Neurophysiology | Potentiation | Brain | Glutamic acid receptors (ionotropic) | Glutamic acid receptors | Neuropathy | Receptors | Pain | Filaments | Surgery | Rodents | Pretreatment | Localization | Dorsal horn | Fyn protein | Pain perception | Hypersensitivity | Rats | N-Methyl-D-aspartic acid receptors | Injury prevention | Molecular modelling | TrkB receptors
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 48, pp. 20504 - 20509
Journal Article