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Journal of Hypertension, ISSN 0263-6352, 02/2014, Volume 32, Issue 2, pp. 306 - 317
AIMS:Guanosine 5′-triphosphate cyclohydrolase-1 (GTPCH-1) is a rate-limiting enzyme in de-novo synthesis of tetrahydrobiopterin (BH4), an essential cofactor... 
guanosine 5′-triphosphate cyclohydrolase-1 | endothelial nitric oxide synthase | tumor suppressor kinase liver kinase B1/adenosine 5′-monophosphateactivated protein kinase | hypertension | NAD(P)H:quinone oxidoreductase 1 | CELLS | SIGNALING PATHWAYS | guanosine 5 '-triphosphate cyclohydrolase-1 | tumor suppressor kinase liver kinase B1/adenosine 5 '-monophosphate-activated protein kinase | COMBINATION THERAPY | EXERCISE | SKELETAL-MUSCLE | SIRT1 ACTIVITY | TETRAHYDROBIOPTERIN | AMPK | PERIPHERAL VASCULAR DISEASE | HIGH BLOOD-PRESSURE | CALORIE RESTRICTION | Sirtuin 1 - metabolism | AMP-Activated Protein Kinases - metabolism | Antihypertensive Agents - pharmacology | Humans | Hypertension - drug therapy | Male | Aorta - metabolism | Blood Pressure - drug effects | Blood Pressure - physiology | Hypertension - enzymology | Nitric Oxide Synthase Type III - metabolism | Vasodilation - physiology | NAD - metabolism | Protein-Serine-Threonine Kinases - metabolism | Rats, Inbred SHR | Cell Line | Aorta - drug effects | Cells, Cultured | Rats | Naphthoquinones - pharmacology | Enzyme Activation - drug effects | GTP Cyclohydrolase - metabolism | Sirtuin 1 - antagonists & inhibitors | Hypertension - physiopathology | NAD(P)H Dehydrogenase (Quinone) - antagonists & inhibitors | Animals | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Vasodilation - drug effects | Endothelial Cells - enzymology | Endothelial Cells - drug effects
Journal Article
Hypertension, ISSN 0194-911X, 02/2011, Volume 57, Issue 2, pp. 245 - 254
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 02/2015, Volume 42, Issue 2, pp. 179 - 185
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, p. e0148097
High reactive oxygen species (ROS) levels and enhanced vascular smooth muscle cells (VSMC) proliferation are observed in numerous cardiovascular diseases. The... 
MIGRATION | PATHWAYS | COMPLEX | PROTEIN | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | GROWTH | CARDIOVASCULAR-DISEASE | RECEPTOR | RAC1 | DOWNSTREAM ACTIVATION | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Receptor, Angiotensin, Type 1 - genetics | Receptor, Epidermal Growth Factor - metabolism | MAP Kinase Kinase 4 - metabolism | ADP-Ribosylation Factors - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | ADP-Ribosylation Factors - metabolism | Myocytes, Smooth Muscle - drug effects | NADH, NADPH Oxidoreductases - metabolism | ADP-Ribosylation Factors - genetics | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Muscle, Smooth, Vascular - drug effects | Vasoconstrictor Agents - pharmacology | Cell Line | NADH, NADPH Oxidoreductases - genetics | Angiotensin II - pharmacology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Gene Expression Regulation | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Muscle, Smooth, Vascular - cytology | NADPH Oxidase 1 | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | MAP Kinase Kinase 4 - genetics | Cell Proliferation - drug effects | rac1 GTP-Binding Protein - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | rac1 GTP-Binding Protein - genetics | Cell proliferation | Angiotensin | Physiological aspects | Smooth muscle | Genetic aspects | Research | Guanosine triphosphatase | ADP-ribosylation factor | Molecular machines | GTP-binding protein | Oxidase | Intracellular signalling | Activation | Hormones | Kinases | ADP | ADP-ribosylation factor 6 | NAD(P)H oxidase | Cell adhesion & migration | Proteins | Signal transduction | Cell growth | Epidermal growth factor | Rodents | Aorta | Ribosylation | Angiotensin II | Heart diseases | ADP-ribosylation | Enzymes | Immunoglobulins | Epidermal growth factor receptors | RNA-mediated interference | Muscles | Extracellular signal-regulated kinase | MAP kinase | Rac1 protein | JNK protein | Superoxide | Pharmacology | Gene expression | Ribonucleic acid--RNA | Medicine | Cardiovascular diseases | Guanosinetriphosphatase | RNA | Ribonucleic acid
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 49, pp. 18933 - 18943
Mitochondrial dysfunction is a key pathological feature of many different types of neurodegenerative disease. Sterile alpha and Toll/interleukin receptor... 
PARKIN | AXONAL DEGENERATION | ACTIVATION | PINK1 | UBIQUITIN | TRIGGERS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | LINKS | CELL-DEATH | DELETION | NAD | cell death | nicotinamide adenine dinucleotide (NAD) | mitochondria | c-Jun N-terminal kinase (JNK) | SARM1 | ATP | Cell Biology
Journal Article
Journal Article
Infection and Immunity, ISSN 0019-9567, 06/2012, Volume 80, Issue 6, pp. 2121 - 2132
Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
INFECTIOUS DISEASES | OXIDATIVE STRESS | TH1 CYTOKINES | INTERFERON-GAMMA | IFN-GAMMA | ER STRESS | GLYCOSYLPHOSPHATIDYLINOSITOLS | LIPOPOLYSACCHARIDE | MEDIATED APOPTOSIS | IMMUNOLOGY | NF-KAPPA-B | CELL-DEATH | Toxoplasmosis, Animal - metabolism | Transcription Factor CHOP - genetics | MAP Kinase Signaling System - physiology | Reactive Oxygen Species - metabolism | Oxidative Stress | Apoptosis - drug effects | Glutathione - metabolism | Endoplasmic Reticulum - metabolism | RNA, Messenger - metabolism | Trophoblasts - drug effects | MAP Kinase Kinase 4 - metabolism | Female | Trophoblasts - cytology | MAP Kinase Kinase Kinase 5 - metabolism | NADH, NADPH Oxidoreductases - metabolism | Stress, Physiological - physiology | NADH, NADPH Oxidoreductases - genetics | MAP Kinase Kinase Kinase 5 - genetics | Caspase 12 - genetics | Gene Expression Regulation - physiology | Caspase 12 - metabolism | DNA - metabolism | Mice, Inbred ICR | NADPH Oxidase 1 | Placenta - metabolism | Pregnancy | Animals | MAP Kinase Kinase 4 - genetics | Mice | Placenta - parasitology | Lipid Peroxidation | Transcription Factor CHOP - metabolism | Toxoplasmosis, Animal - pathology | Index Medicus | Glutathione peroxidase | Oxidative stress | tachyzoites | Animal models | Reactive oxygen species | Caspase-12 | Fetuses | Acetylcysteine | MAP kinase | Abortion | Malondialdehyde | NAD(P)H oxidase | Infection | Polymerase chain reaction | Teratogenesis | peroxide | Trophoblasts | Placenta | c-Jun amino-terminal kinase | 8-Hydroxydeoxyguanosine | Endoplasmic reticulum | Apoptosis | Host Response and Inflammation
Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 11/2013, Volume 15, Issue 11, pp. 993 - 999
Aim To examine whether protein kinase C (PKC) and associated downstream mechanisms are involved in hyperglycaemia (HG)‐evoked blood–brain barrier (BBB) damage.... 
blood‐brain barrier | hyperglycaemia | occludin | oxidative stress | protein kinase C | Oxidative stress | Protein kinase C | Hyperglycaemia | Blood-brain barrier | Occludin | blood-brain barrier | PKC-BETA | DIABETIC-RATS | BLOOD-BRAIN-BARRIER | STROKE | NAD(P)H OXIDASE | INHIBITION | ENDOCRINOLOGY & METABOLISM | MEDIATED DISRUPTION | NITRIC-OXIDE | EXPRESSION | Endothelium, Vascular - cytology | NADPH Oxidases - chemistry | Blood-Brain Barrier - cytology | Pericytes - drug effects | Coculture Techniques | Humans | Pericytes - cytology | Blood-Brain Barrier - physiopathology | Microvessels - metabolism | NADPH Oxidases - metabolism | Capillary Permeability - drug effects | Endothelium, Vascular - drug effects | Occludin - metabolism | Brain - metabolism | Brain - blood supply | Matrix Metalloproteinase 2 - chemistry | Hyperglycemia - physiopathology | Tight Junctions - drug effects | Astrocytes - cytology | Microvessels - cytology | Tight Junctions - metabolism | Astrocytes - drug effects | Brain - cytology | Matrix Metalloproteinase 2 - metabolism | Pericytes - metabolism | Cells, Cultured | Microvessels - drug effects | Electric Impedance | Down-Regulation - drug effects | Enzyme Activation - drug effects | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Brain - drug effects | Hyperglycemia - metabolism | Endothelium, Vascular - metabolism | Hyperglycemia - enzymology | Protein Kinase C beta - chemistry | Protein Kinase C beta - metabolism | Protein Kinase Inhibitors - pharmacology | Protein Kinase C beta - antagonists & inhibitors | Astrocytes - metabolism
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2006, Volume 281, Issue 43, pp. 32207 - 32216
AMP-activated protein kinase (AMPK) is a heterotrimeric protein kinase that is crucial for cellular energy homeostasis of eukaryotic cells and organisms. Here... 
CELLS | ENERGY HOMEOSTASIS | PHOSPHORYLATION | SUBSTRATE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | UPSTREAM KINASE | CELLULAR-ENERGY | CREATINE-KINASE | SUBUNIT | BETA | Protein Kinases - metabolism | Protein-Serine-Threonine Kinases - analysis | Phosphorylation | Multienzyme Complexes - metabolism | AMP-Activated Protein Kinases | Aminoimidazole Carboxamide - pharmacology | Recombinant Proteins - isolation & purification | Ribonucleotides - pharmacology | Protein Isoforms - metabolism | Allosteric Regulation - physiology | Adenosine Triphosphate - metabolism | Protein-Serine-Threonine Kinases - metabolism | Recombinant Proteins - metabolism | Adenosine Monophosphate - metabolism | NAD - pharmacology | Protein-Serine-Threonine Kinases - genetics | Glutathione Transferase - metabolism | Multienzyme Complexes - genetics | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Mice | Enzyme Activation | Multienzyme Complexes - analysis | Mutation | In Vitro Techniques | Protein Isoforms - genetics | Multienzyme Complexes | Allosteric Regulation | Protein-Serine-Threonine Kinases | Biochemistry, Molecular Biology | Adenosine Triphosphate | Recombinant Proteins | Adenosine Monophosphate | NAD | Life Sciences | Aminoimidazole Carboxamide | Ribonucleotides | Protein Kinases | Protein Isoforms | Glutathione Transferase
Journal Article