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Biochemical journal, ISSN 1470-8728, 2019, Volume 476, Issue 20, pp. 3033 - 3052
6-NADH and 6-NADPH are strong inhibitors of several dehydrogenases that may form spontaneously from NAD(P)H... 
HUMAN RENALASE | SYSTEM | REPAIR | ENZYME | NADH | 1,6-DIHYDRO-NAD | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | METABOLITE DAMAGE | IDENTIFICATION | 5-PHOSPHATE OXIDASE
Journal Article
Circulation research, ISSN 0009-7330, 08/2001, Volume 89, Issue 5, pp. 408 - 414
...(P)H oxidase in angiotensin (Ang) II–induced hypertension, suggesting de novo assembly in response to Ang II... 
gp91 | p47 | Superoxide | Angiotensin II | NAD(P)H oxidase
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 6, pp. 1117 - 1128
...)H oxidase, oxidative stress, and endothelial function. METHODS AND RESULTS:The markers of oxidative stress, NAD(P... 
NF-κ | AMPK | NAD(P)H oxidase | Proteasome | NF-κB | NAD(P)H Oxidase
Journal Article
Journal Article
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 09/2010, Volume 17, Issue 9, pp. 1474 - 1485
...). Hypoxia stimulated the expression of the endoplasmic-resident gp96, which co-immunoprecipitated TLR4, whereas silencing gp96 mRNA expression impaired hypoxia-induced apoptosis in TLR4-expressing RTECs. NAD(P) H oxidase 4 (NOX4... 
kidney | apoptosis | NAD(P)H oxidase 4 | gp96 | Toll-like receptor 4 | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEATH | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | ISCHEMIA-REPERFUSION INJURY | REACTIVE OXYGEN | EPITHELIAL-CELLS | NF-KAPPA-B | NADPH OXIDASES | INNATE IMMUNITY | RNA, Small Interfering - genetics | Cell Hypoxia - physiology | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | TNF Receptor-Associated Factor 2 - genetics | NADPH Oxidases - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Kidney - metabolism | Isoenzymes - metabolism | NADPH Oxidases - genetics | Female | p38 Mitogen-Activated Protein Kinases - metabolism | MAP Kinase Kinase Kinase 5 - metabolism | Kidney Tubules - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | bcl-2-Associated X Protein - metabolism | TNF Receptor-Associated Factor 2 - metabolism | Toll-Like Receptor 4 - genetics | Kidney - cytology | NADPH Oxidase 4 | Toll-Like Receptor 4 - metabolism | Membrane Glycoproteins - genetics | Mice, Knockout | Kidney Tubules - cytology | Animals | Signal Transduction - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - physiology | Mice | Protein Kinase Inhibitors - pharmacology | Apoptosis - physiology | Myeloid Differentiation Factor 88 - metabolism | Protein Binding - physiology
Journal Article
Circulation research, ISSN 0009-7330, 12/2002, Volume 91, Issue 12, pp. 1160 - 1167
...). Reactive oxygen species (ROS) derived from NAD(P)H oxidase are critically important in many aspects of vascular cell regulation, and both the small GTPase Rac1 and gp91... 
Angiogenesis | Reactive oxygen species | Vascular endothelial growth factor | Endothelial cells | NAD(P)H oxidase
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2013, Volume 127, Issue 18, pp. 1888 - 1902
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2016, Volume 36, Issue 12, pp. 2412 - 2420
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2007, Volume 27, Issue 12, pp. 2627 - 2633
...) is able to quench oxidative stress initiated by high glucose through prevention of NAD(P)H oxidase activation... 
Rosiglitazone | Oxidative stress | AMPK | Diabetes | NAD(P)H oxidase | RESPIRATORY BURST | ACTIVATED PROTEIN-KINASE | RECEPTOR-GAMMA | VEIN ENDOTHELIAL-CELLS | DIABETIC-PATIENTS | SKELETAL-MUSCLE | SIGNALING PATHWAY | INSULIN-RESISTANCE | NITRIC-OXIDE | rosiglitazone | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | diabetes | oxidative stress | CORONARY-ARTERY-DISEASE | Enzyme Activators - pharmacology | Phosphorylation | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | PPAR gamma - drug effects | Humans | NADPH Oxidases - metabolism | Multienzyme Complexes - metabolism | Male | Muscle, Skeletal - metabolism | PPAR gamma - metabolism | Phosphoproteins - metabolism | AMP-Activated Protein Kinases | Transfection | RNA Interference | Protein Kinase C - metabolism | Muscle, Skeletal - drug effects | Thiazolidinediones - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Muscle, Skeletal - enzymology | Endothelial Cells - metabolism | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Protein-Serine-Threonine Kinases - genetics | Rats | Multienzyme Complexes - genetics | Protein Kinase C - antagonists & inhibitors | Antioxidants - pharmacology | NADPH Oxidase 2 | Rats, Sprague-Dawley | Protein Transport | Animals | Signal Transduction - drug effects | Glucose - metabolism | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Oxidative Stress - drug effects | Endothelial Cells - enzymology | rac1 GTP-Binding Protein - metabolism | Endothelial Cells - drug effects | RNA, Small Interfering - metabolism
Journal Article