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PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50644
Defects in mitochondrial electron transport chain (ETC) function have been implicated in a number of neurodegenerative disorders, cancer, and aging.... 
INACTIVATION | LIFE-SPAN | GENE | MULTIDISCIPLINARY SCIENCES | DISEASE | MITOCHONDRIA | ROTENONE-INSENSITIVE NADH | MUTATIONS | SACCHAROMYCES-CEREVISIAE | GENOME | NADH-UBIQUINONE OXIDOREDUCTASE | Mitochondria - enzymology | Electron Transport Complex I - deficiency | Humans | Holoenzymes - chemistry | NADH Dehydrogenase - genetics | Drosophila melanogaster - genetics | Mitochondria - ultrastructure | Drosophila melanogaster - metabolism | Holoenzymes - metabolism | RNA Interference | Electron Transport Complex I - genetics | Holoenzymes - deficiency | NADH Dehydrogenase - deficiency | Mitochondrial Diseases - therapy | Mitochondrial Diseases - genetics | Gene Expression | Drosophila melanogaster - cytology | Mitochondria - metabolism | Saccharomyces cerevisiae Proteins - genetics | NADH Dehydrogenase - chemistry | NADH Dehydrogenase - metabolism | Sequence Homology, Amino Acid | Phenotype | Animals | Drosophila melanogaster - growth & development | Holoenzymes - genetics | Enzymes | Nervous system diseases | Analysis | Drosophila | Genomics | Personal narratives | Genomes | Genetic aspects | Genetic engineering | Electron transport | Baking yeast | Dehydrogenases | Disease | Oxidoreductase | Disorders | Transgenic | NADH dehydrogenase | Homology | Biosynthesis | Biology | Dehydrogenase | Electron transport chain | Mitochondria | Ubiquinone | NADH | Rodents | Aging | Evolution | Physiology | Ubiquinone oxidoreductase | Assembly | NADH-ubiquinone oxidoreductase | Neurodegenerative diseases | Gel electrophoresis | RNA-mediated interference | Gene expression | Insects | Nicotinamide adenine dinucleotide | Subassemblies | Mutation | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, p. e0144441
Despite advances in basic and clinical research, metastasis remains the leading cause of death in breast cancer patients. Genetic abnormalities in... 
EPITHELIAL-MESENCHYMAL TRANSITION | TARGET | ACTIVATION | COMPLEX-I | PATHWAY | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | GENES | AUTOPHAGY | TUMORIGENESIS | MOTILITY | NADH Dehydrogenase - antagonists & inhibitors | RNA, Small Interfering - genetics | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | NADH Dehydrogenase - genetics | Immunoenzyme Techniques | Breast Neoplasms - metabolism | Tandem Mass Spectrometry | DNA, Mitochondrial - genetics | Epithelial-Mesenchymal Transition | Female | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Signal Transduction | Down-Regulation | RNA, Messenger - genetics | Oxidative Phosphorylation | Mitochondria - metabolism | Mitochondria - pathology | NADH Dehydrogenase - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Breast Neoplasms - genetics | Cell Cycle | Breast Neoplasms - pathology | Proteomics | Cell Movement | Breast cancer | Genetic aspects | Mitochondrial DNA | Metastasis | TOR protein | Cell proliferation | Phosphorylation | Motility | Dehydrogenases | Laboratories | NADH dehydrogenase | AKT protein | Oncology | Dehydrogenase | Autophagy | Cell adhesion & migration | Metastases | Electron transport chain | Genetic abnormalities | Signal transduction | Mitochondria | Cell growth | NADH | Surgery | Deoxyribonucleic acid--DNA | NADH-ubiquinone oxidoreductase | Immunoglobulins | Abnormalities | Data processing | Gene expression | Metabolism | Chromatography | NAD | Oxidative phosphorylation | Nicotinamide adenine dinucleotide | Biomarkers | Mutation | Cell migration | Cancer | Deoxyribonucleic acid | DNA
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 4970 - 12
The viability of Mycobacterium tuberculosis (Mtb) depends on energy generated by its respiratory chain. Cytochrome bc1-aa3 oxidase and type-2 NADH... 
CLEARANCE | READ ALIGNMENT | INHIBITION | METABOLISM | II NADH DEHYDROGENASE | MULTIDISCIPLINARY SCIENCES | ADAPTATION | REGIMEN | INFECTION | 1-AMINOBENZOTRIAZOLE | DISCOVERY | NADH Dehydrogenase - antagonists & inhibitors | Electron Transport | Electron Transport Complex III - genetics | Electron Transport Complex IV - antagonists & inhibitors | NADH Dehydrogenase - genetics | Tuberculosis, Pulmonary - drug therapy | Mycobacterium tuberculosis - drug effects | Gene Knockdown Techniques | Piperidines - pharmacology | Adaptation, Physiological - genetics | Tuberculosis - drug therapy | Mycobacterium tuberculosis - metabolism | Antitubercular Agents - therapeutic use | Lung - pathology | Callithrix | Imidazoles - pharmacology | Electron Transport Complex IV - genetics | Tuberculosis, Pulmonary - pathology | Animals | Electron Transport Complex III - antagonists & inhibitors | Lung - drug effects | Mice | Pyridines - pharmacology | Drug Development | In Vitro Techniques | Mycobacterium tuberculosis - genetics | Cytochrome | Clofazimine | Respiratory enzymes | Oxidase | NADH dehydrogenase | Drug development | Fatty acids | Disease control | Deletion mutant | Tuberculosis | NADH | Cytochrome bc1 | Growth media | Plasticity | Nicotinamide adenine dinucleotide | Cytochromes | Plastic properties | Lesions | Electron transport | Viability
Journal Article
Applied microbiology and biotechnology, ISSN 1432-0614, 2015, Volume 99, Issue 13, pp. 5573 - 5582
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 4, p. e61677
Despite the fact that mitochondrial dysfunction has an important role in tumorigenesis and metastasis, the underlying mechanism remains to be elucidated.... 
BREAST-CANCER | TUMOR PROGRESSION | GRIM-19 | MULTIDISCIPLINARY SCIENCES | ROS | FIBRONECTIN | EXTRACELLULAR-MATRIX | DEATH | INTERFERON-BETA | IDENTIFICATION | DEFICIENCY | Mitochondria - enzymology | RNA, Small Interfering - genetics | Cell Proliferation | Reactive Oxygen Species - metabolism | Cadherins - metabolism | Humans | Extracellular Matrix - metabolism | NADH Dehydrogenase - genetics | Electron Transport Complex I - metabolism | Integrins - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | Neoplasm Metastasis | Electron Transport Complex I - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Shape | Epithelial-Mesenchymal Transition | Apoptosis Regulatory Proteins - genetics | NADH, NADPH Oxidoreductases - metabolism | Spheroids, Cellular | NADH, NADPH Oxidoreductases - genetics | Gene Expression | NADH Dehydrogenase - metabolism | Cell Adhesion Molecules - metabolism | Fibronectins - metabolism | Apoptosis Regulatory Proteins - metabolism | Phenotype | Cell Line, Tumor | Fibronectins - genetics | HeLa Cells | Cell Movement | Proteins | Development and progression | Metastasis | Electron transport | Phosphorylation | Laboratories | Oxidoreductase | Science | Biology | Mitochondrial DNA | Metastases | Cell adhesion & migration | Electron transport chain | Mitochondria | Ubiquinone | NADH | Cell adhesion | Extracellular matrix | Tumorigenesis | Oxidation | Ubiquinone oxidoreductase | NADH-ubiquinone oxidoreductase | Medical research | Oxygen | Invasiveness | Breast cancer | Tumor cell lines | Gene expression | Morphology | Nicotinamide adenine dinucleotide | Cell migration | Cancer | Apoptosis
Journal Article
Applied microbiology and biotechnology, ISSN 1432-0614, 2019, Volume 103, Issue 10, pp. 3965 - 3978
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2008, Volume 28, Issue 46, pp. 12039 - 12051
Reactive oxygen species (ROS) modulate intracellular signaling but are also responsible for neuronal damage in pathological states. Microglia, the resident CNS... 
Interleukin-1 | NADPH oxidase | Nitric oxide | Nox | Neuroinflammation | LPS | Microglia | nitric oxide | OXIDATIVE STRESS | interleukin-1 | CHRONIC GRANULOMATOUS-DISEASE | ALZHEIMERS-DISEASE | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | NEURONAL DAMAGE | neuroinflammation | SUPEROXIDE GENERATION | MOUSE MODEL | LIPOPOLYSACCHARIDE | HYDROGEN-PEROXIDE | microglia | PARKINSONS-DISEASE | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nitrites - metabolism | rac GTP-Binding Proteins - metabolism | Male | Gliosis - enzymology | Gliosis - physiopathology | Cytochrome b Group - metabolism | Zymosan - metabolism | Encephalitis - physiopathology | Inflammation Mediators - pharmacology | rac GTP-Binding Proteins - genetics | NADPH Oxidases - genetics | Female | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | Corpus Striatum - enzymology | NADH, NADPH Oxidoreductases - genetics | Encephalitis - enzymology | Corpus Striatum - physiopathology | Microglia - drug effects | Mice, Inbred C57BL | Microglia - enzymology | Neuropeptides - metabolism | Cytochrome b Group - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Neurotoxins - pharmacology | Proteins - genetics | Animals | Proteins - metabolism | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Oxidative Stress - drug effects | rac1 GTP-Binding Protein | Index Medicus | Reactive Oxygen Species | Oxidative Stress | Nitrites | Neuropeptides | Lipopolysaccharides | Proteins | Life Sciences | Immunology | NADH, NADPH Oxidoreductases | Membrane Glycoproteins | Zymosan | rac GTP-Binding Proteins | Encephalitis | Neurotoxins | Inflammation Mediators | Cytochrome b Group | Gliosis | Corpus Striatum | NADPH Oxidase
Journal Article