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Journal of the American Society of Nephrology, ISSN 1046-6673, 06/2014, Volume 25, Issue 6, pp. 1237 - 1254
Journal Article
Diabetologia, ISSN 0012-186X, 5/2017, Volume 60, Issue 5, pp. 927 - 937
Journal Article
Circulation Research, ISSN 0009-7330, 04/2012, Volume 110, Issue 9, pp. 1217 - 1225
RATIONALE:The function of Nox4, a source of vascular H2O2, is unknown. Other Nox proteins were identified as mediators of endothelial dysfunction. OBJECTIVE:We... 
Hypertension | Angiogenesis | Carbon monoxide | Nitric oxide | Redox regulation | INDUCED ACTIVATION | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | redox regulation | angiogenesis | carbon monoxide | ANGIOTENSIN-II | BLOOD-PRESSURE | NAD(P)H OXIDASE | ENDOTHELIAL-CELLS | IN-VIVO | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HYDROGEN-PEROXIDE | HEMATOLOGY | hypertension | UP-REGULATION | Catalase - pharmacology | Heme Oxygenase-1 - metabolism | Humans | NADPH Oxidases - metabolism | Male | Ischemia - genetics | Carbonates - pharmacology | RNA, Messenger - metabolism | NADPH Oxidases - deficiency | Time Factors | NADPH Oxidases - genetics | Carbonates - metabolism | Hemin - pharmacology | Hypertension - enzymology | Hypertension - genetics | Ischemia - pathology | Cytoprotection | Disease Models, Animal | Muscle, Skeletal - blood supply | NADH, NADPH Oxidoreductases - genetics | Hypertension - pathology | Hydrogen Peroxide - metabolism | Mice, Knockout | Human Umbilical Vein Endothelial Cells - enzymology | Organometallic Compounds - metabolism | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Endothelial Cells - enzymology | Hypertrophy | Ischemia - enzymology | Boranes - metabolism | Transfection | NADH, NADPH Oxidoreductases - deficiency | Hypertension - chemically induced | NF-E2-Related Factor 2 - genetics | Membrane Proteins - metabolism | Nitric Oxide Synthase Type III - metabolism | Angiotensin II | Polyethylene Glycols - pharmacology | Carbon Dioxide - metabolism | Mice, Inbred C57BL | Cells, Cultured | Boranes - pharmacology | Antioxidants - pharmacology | Ischemia - physiopathology | NADPH Oxidase 4 | Hypertension - physiopathology | NADPH Oxidase 1 | Animals | Lung - blood supply | Neovascularization, Physiologic | Organometallic Compounds - pharmacology | Apoptosis | Endothelial Cells - drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0129743
Reactive oxygen species (ROS) produced by nicotinamide adenine dinucleotide phosphate oxidase (NOX) play a key role in liver injury and fibrosis. Previous... 
TRANSFORMATION | NADPH OXIDASE | HEPATOCYTE APOPTOSIS | OXIDATIVE STRESS | SIGNALING PATHWAYS | HEDGEHOG PATHWAY | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | NAD(P)H OXIDASES | PROLIFERATION | EXPRESSION | NADPH Oxidases - chemistry | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Platelet-Derived Growth Factor - genetics | Myofibroblasts - metabolism | Carbon Tetrachloride Poisoning - metabolism | Hedgehog Proteins - genetics | Hepatitis - genetics | Hepatitis - metabolism | Liver Cirrhosis - metabolism | NADPH Oxidases - genetics | NADH, NADPH Oxidoreductases - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | NADH, NADPH Oxidoreductases - genetics | Signal Transduction | NADPH Oxidases - antagonists & inhibitors | Hepatitis - pathology | NADPH Oxidase 4 | Pyrazoles - metabolism | Carbon Tetrachloride Poisoning - pathology | NADPH Oxidase 1 | Mice, Knockout | Platelet-Derived Growth Factor - metabolism | Animals | Pyridines - metabolism | Liver Cirrhosis - pathology | Carbon Tetrachloride Poisoning - genetics | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Niacinamide | Platelet-derived growth factor | Carbon tetrachloride | Fibrosis | Inflammation | Comparative analysis | Liver cirrhosis | Phosphates | Oxidative stress | Pathogenesis | Liver | Genes | Lipid peroxidation | Activation | NAD(P)H oxidase | Lipopolysaccharides | Veterinarians | Hepatitis | Cell activation | NOX4 protein | Rodents | Cell cycle | Peroxidation | Medical research | Stellate cells | Enzymes | NADPH-diaphorase | Adenine | CCL4 protein | Medicine | Cirrhosis | Signaling | Injury prevention | Inhibitors | Hedgehog protein | Nicotinamide adenine dinucleotide | Nicotinamide | Laboratory animals | Plastics | Apoptosis | Bile
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 12039 - 12051
Reactive oxygen species (ROS) modulate intracellular signaling but are also responsible for neuronal damage in pathological states. Microglia, the resident CNS... 
Interleukin-1 | NADPH oxidase | Nitric oxide | Nox | Neuroinflammation | LPS | Microglia | nitric oxide | OXIDATIVE STRESS | interleukin-1 | CHRONIC GRANULOMATOUS-DISEASE | ALZHEIMERS-DISEASE | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | NEURONAL DAMAGE | neuroinflammation | SUPEROXIDE GENERATION | MOUSE MODEL | LIPOPOLYSACCHARIDE | HYDROGEN-PEROXIDE | microglia | PARKINSONS-DISEASE | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nitrites - metabolism | rac GTP-Binding Proteins - metabolism | Male | Gliosis - enzymology | Gliosis - physiopathology | Cytochrome b Group - metabolism | Zymosan - metabolism | Encephalitis - physiopathology | Inflammation Mediators - pharmacology | rac GTP-Binding Proteins - genetics | NADPH Oxidases - genetics | Female | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | Corpus Striatum - enzymology | NADH, NADPH Oxidoreductases - genetics | Encephalitis - enzymology | Corpus Striatum - physiopathology | Microglia - drug effects | Mice, Inbred C57BL | Microglia - enzymology | Neuropeptides - metabolism | Cytochrome b Group - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Neurotoxins - pharmacology | Proteins - genetics | Animals | Proteins - metabolism | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Oxidative Stress - drug effects | rac1 GTP-Binding Protein | Index Medicus | Reactive Oxygen Species | Oxidative Stress | Nitrites | Neuropeptides | Lipopolysaccharides | Proteins | Life Sciences | Immunology | NADH, NADPH Oxidoreductases | Membrane Glycoproteins | Zymosan | rac GTP-Binding Proteins | Encephalitis | Neurotoxins | Inflammation Mediators | Cytochrome b Group | Gliosis | Corpus Striatum | NADPH Oxidase
Journal Article
Hepatology, ISSN 0270-9139, 12/2012, Volume 56, Issue 6, pp. 2316 - 2327
Journal Article
Trends in Pharmacological Sciences, ISSN 0165-6147, 2016, Volume 37, Issue 4, pp. 318 - 327
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 443 - 454
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 38, pp. 20030 - 20041
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e14665
Reactive oxygen species, ROS, are regulators of endothelial cell migration, proliferation and survival, events critically involved in angiogenesis. Different... 
NAD(P)H OXIDASE | ROLES | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | CELL-PROLIFERATION | ANGIOTENSIN-II | NF-KAPPA-B | EXPRESSION | CANCER | NOX1 | ENDOTHELIAL GROWTH-FACTOR | Pyrazoles - therapeutic use | Humans | Molecular Targeted Therapy | Gene Knockdown Techniques | NADH, NADPH Oxidoreductases - physiology | Neoplasms - genetics | Angiogenesis Inhibitors - therapeutic use | Female | Pyrazoles - pharmacology | Gene Targeting | NADH, NADPH Oxidoreductases - genetics | Endothelial Cells - metabolism | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Neoplasms - blood supply | PPAR alpha - genetics | Signal Transduction - genetics | NADPH Oxidase 1 | Mice, Knockout | Neoplasms - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Neovascularization, Pathologic - drug therapy | PPAR alpha - physiology | Neovascularization, Pathologic - genetics | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Pyridones - therapeutic use | Pyridones - pharmacology | Endothelial Cells - drug effects | Regulators | Leukocyte migration | Metastasis | Kinases | NAD(P)H oxidase | Cell adhesion & migration | Proteins | Angiogenesis | Immunology | NOX4 protein | Rodents | CYBB protein | Tomography | Fibroblasts | Localization | Cues | NF-κB protein | Immunoglobulins | Medical imaging | Cell survival | Endothelial cells | Pathology | Signaling | Isoforms | Photographs | Cell migration | Cancer | Tumors
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2008, Volume 283, Issue 14, pp. 8885 - 8892
Journal Article