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Diabetologia, ISSN 0012-186X, 5/2017, Volume 60, Issue 5, pp. 927 - 937
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0129743 - e0129743
Reactive oxygen species (ROS) produced by nicotinamide adenine dinucleotide phosphate oxidase (NOX) play a key role in liver injury and fibrosis. Previous... 
TRANSFORMATION | NADPH OXIDASE | HEPATOCYTE APOPTOSIS | OXIDATIVE STRESS | SIGNALING PATHWAYS | HEDGEHOG PATHWAY | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | NAD(P)H OXIDASES | PROLIFERATION | EXPRESSION | NADPH Oxidases - chemistry | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Platelet-Derived Growth Factor - genetics | Myofibroblasts - metabolism | Carbon Tetrachloride Poisoning - metabolism | Hedgehog Proteins - genetics | Hepatitis - genetics | Hepatitis - metabolism | Liver Cirrhosis - metabolism | NADPH Oxidases - genetics | NADH, NADPH Oxidoreductases - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | NADH, NADPH Oxidoreductases - genetics | Signal Transduction | NADPH Oxidases - antagonists & inhibitors | Hepatitis - pathology | NADPH Oxidase 4 | Pyrazoles - metabolism | Carbon Tetrachloride Poisoning - pathology | NADPH Oxidase 1 | Mice, Knockout | Platelet-Derived Growth Factor - metabolism | Animals | Pyridines - metabolism | Liver Cirrhosis - pathology | Carbon Tetrachloride Poisoning - genetics | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Niacinamide | Platelet-derived growth factor | Carbon tetrachloride | Fibrosis | Inflammation | Comparative analysis | Liver cirrhosis | Phosphates | Oxidative stress | Pathogenesis | Liver | Genes | Lipid peroxidation | Activation | NAD(P)H oxidase | Lipopolysaccharides | Veterinarians | Hepatitis | Cell activation | NOX4 protein | Rodents | Cell cycle | Peroxidation | Medical research | Stellate cells | Enzymes | NADPH-diaphorase | Adenine | CCL4 protein | Medicine | Cirrhosis | Signaling | Injury prevention | Inhibitors | Hedgehog protein | Nicotinamide adenine dinucleotide | Nicotinamide | Laboratory animals | Plastics | Apoptosis | Bile | Index Medicus
Journal Article
Clinical Science, ISSN 0143-5221, 02/2013, Volume 124, Issue 3, pp. 191 - 202
Nox (NADPH oxidase)-derived ROS (reactive oxygen species) have been implicated in the development of diabetic nephropathy. Of the Nox isoforms in the kidney,... 
Oxidative stress | Diabetes | NADPH oxidase | NOX inhibitor | Kidney | db/db mouse | KIDNEY-DISEASE | MEDICINE, RESEARCH & EXPERIMENTAL | DB/DB MICE | MECHANISMS | NEPHROPATHY | FAMILY | kidney | INDUCED OXIDATIVE STRESS | NOX4 | COMPLICATIONS | HYPERTENSION | diabetes | oxidative stress | NADPH OXIDASE INHIBITOR | Gene Expression Regulation, Enzymologic - drug effects | Superoxide Dismutase - genetics | Kidney - pathology | Diabetic Nephropathies - etiology | NADPH Oxidases - metabolism | Body Weight - drug effects | Male | Kidney - metabolism | Diabetic Nephropathies - physiopathology | NADPH Oxidases - genetics | Thiobarbituric Acid Reactive Substances - analysis | Blood Pressure - drug effects | Albuminuria - urine | NADH, NADPH Oxidoreductases - metabolism | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Superoxide Dismutase - metabolism | Disease Models, Animal | Pyrazoles - pharmacology | NADH, NADPH Oxidoreductases - genetics | Kidney - drug effects | Blood Glucose - analysis | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Diabetes Mellitus, Type 2 - urine | NADPH Oxidase 4 | Albuminuria - prevention & control | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | NADPH Oxidase 1 | Diabetes Mellitus, Type 2 - blood | Animals | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Superoxide Dismutase-1 | Vascular Cell Adhesion Molecule-1 - metabolism | Transforming Growth Factor beta - metabolism | Pyridones - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 06/2014, Volume 25, Issue 6, pp. 1237 - 1254
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 07/2012, Volume 53, Issue 2, pp. 289 - 296
Reactive oxygen species (ROS) play a key role in chronic liver injury and fibrosis. Homologs of NADPH oxidases (NOXs) are major sources of ROS, but the exact... 
NADPH oxidase 4 | Liver fibrosis | Stellate cell activation | Hepatocyte apoptosis | NADPH OXIDASE | ACTIVATION | TGF-BETA | ISOFORMS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | FIBROGENESIS | MECHANISMS | HEPATIC STELLATE CELLS | ENDOCRINOLOGY & METABOLISM | NAD(P)H OXIDASES | STRESS | Liver - pathology | Pyrazoles - therapeutic use | Liver - enzymology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Bile Ducts - metabolism | Humans | NADPH Oxidases - metabolism | Hepatitis, Autoimmune - drug therapy | Hepatocytes - pathology | Smad3 Protein - metabolism | Liver Cirrhosis - enzymology | Hepatic Stellate Cells - cytology | Hepatitis, Autoimmune - pathology | Fas Ligand Protein - pharmacology | Ligation | Liver - drug effects | Gene Deletion | NADPH Oxidases - genetics | NADH, NADPH Oxidoreductases - metabolism | Hepatocytes - drug effects | Hepatic Stellate Cells - drug effects | Pyrazoles - pharmacology | Pyridines - therapeutic use | Liver Cirrhosis - drug therapy | NADH, NADPH Oxidoreductases - genetics | Bile Ducts - drug effects | NADPH Oxidases - antagonists & inhibitors | Rats | Bile Ducts - surgery | NADPH Oxidase 4 | NADPH Oxidase 1 | Reactive Oxygen Species - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Liver Cirrhosis - pathology | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Pyridines - pharmacology | Dactinomycin - pharmacology | Transforming Growth Factor beta - metabolism | Hepatitis, Autoimmune - enzymology | Hepatocytes - enzymology | Oxidases | Phosphates | Niacinamide | Purines | Liver diseases | Analysis | Liver | Fibrosis | Bone morphogenetic proteins | Transforming growth factors | Muscle proteins | Apoptosis | Index Medicus | stellate cell activation | liver fibrosis | hepatocyte apoptosis | nicotinamide adenine dinucleotide phosphate reduced oxidase 4
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 12039 - 12051
Reactive oxygen species (ROS) modulate intracellular signaling but are also responsible for neuronal damage in pathological states. Microglia, the resident CNS... 
Interleukin-1 | NADPH oxidase | Nitric oxide | Nox | Neuroinflammation | LPS | Microglia | nitric oxide | OXIDATIVE STRESS | interleukin-1 | CHRONIC GRANULOMATOUS-DISEASE | ALZHEIMERS-DISEASE | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | NEURONAL DAMAGE | neuroinflammation | SUPEROXIDE GENERATION | MOUSE MODEL | LIPOPOLYSACCHARIDE | HYDROGEN-PEROXIDE | microglia | PARKINSONS-DISEASE | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nitrites - metabolism | rac GTP-Binding Proteins - metabolism | Male | Gliosis - enzymology | Gliosis - physiopathology | Cytochrome b Group - metabolism | Zymosan - metabolism | Encephalitis - physiopathology | Inflammation Mediators - pharmacology | rac GTP-Binding Proteins - genetics | NADPH Oxidases - genetics | Female | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | Corpus Striatum - enzymology | NADH, NADPH Oxidoreductases - genetics | Encephalitis - enzymology | Corpus Striatum - physiopathology | Microglia - drug effects | Mice, Inbred C57BL | Microglia - enzymology | Neuropeptides - metabolism | Cytochrome b Group - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Neurotoxins - pharmacology | Proteins - genetics | Animals | Proteins - metabolism | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Oxidative Stress - drug effects | rac1 GTP-Binding Protein | Index Medicus | Reactive Oxygen Species | Oxidative Stress | Nitrites | Neuropeptides | Lipopolysaccharides | Proteins | Life Sciences | Immunology | NADH, NADPH Oxidoreductases | Membrane Glycoproteins | Zymosan | rac GTP-Binding Proteins | Encephalitis | Neurotoxins | Inflammation Mediators | Cytochrome b Group | Gliosis | Corpus Striatum | NADPH Oxidase
Journal Article
Arteriosclerosis Thrombosis and Vascular Biology, ISSN 1079-5642, 02/2016, Volume 36, Issue 2, pp. 295 - 307
OBJECTIVE—Oxidative stress is considered a hallmark of atherosclerosis. In particular, the superoxide-generating type 1 NADPH oxidase (NOX1) has been shown to... 
atherosclerosis | endothelial cells | diabetes mellitus | NADPH oxidase | reactive oxygen species | OXIDATIVE STRESS | ACCELERATED ATHEROSCLEROSIS | CARDIOVASCULAR-SYSTEM | NECROSIS-FACTOR-ALPHA | COLLAGEN-SYNTHESIS | TRANSFORMING GROWTH-FACTOR-BETA-1 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIABETES-ASSOCIATED ATHEROSCLEROSIS | PHARMACOLOGICAL INHIBITION | HEMATOLOGY | Inflammation - pathology | Vascular Remodeling | Apolipoproteins E - deficiency | Membrane Glycoproteins - metabolism | Oxidative Stress | Atherosclerosis - genetics | Humans | NADPH Oxidases - metabolism | Male | Case-Control Studies | NADPH Oxidases - deficiency | Atherosclerosis - enzymology | Transfection | RNA Interference | Time Factors | Inflammation Mediators - metabolism | Superoxides - metabolism | Aortic Diseases - prevention & control | NADPH Oxidases - genetics | Aortic Diseases - enzymology | NADH, NADPH Oxidoreductases - metabolism | Aorta - enzymology | Aortic Diseases - pathology | Disease Models, Animal | Fibrillar Collagens - metabolism | Atherosclerosis - pathology | Cytokines - metabolism | Signal Transduction | Cells, Cultured | NADPH Oxidase 4 | Plaque, Atherosclerotic | NADPH Oxidase 2 | NADPH Oxidase 1 | Hydrogen Peroxide - metabolism | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Inflammation - prevention & control | Endothelial Cells - pathology | Endothelial Cells - enzymology | Inflammation - enzymology | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 38, pp. 20030 - 20041
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2016, Volume 97, pp. 95 - 108
Amyotrophic lateral sclerosis (ALS) is an incurable neurodegenerative disease characterized by progressive loss of motor neurons, gliosis, neuroinflammation... 
SOD1G93A mice | Thioridazine | NADPH oxidase | NOX | Amyotrophic lateral sclerosis | Perphenazine | Microglia | Phenothiazine | DUOX dual oxidase | List of abbreviations ALS amyotrophic lateral sclerosis | WST-1 2-(4-Iodophenyl)-3-(4-nitrophenyl)-5-(2,4-disulfophenyl)-2H-tetrazolium, monosodium salt | PBS phosphate buffered saline | HOCl hypochlorous acid | BBB blood brain barrier | WT wild typ | 2-chloroethidium | 2-Cl-E | NOXO1 NADPH oxidase organiser type 1 | CNS central nervous system | DPI diphenyleneiodonium | ethidium | NOX NADPH oxidase | HE hydroethidine | qPCR quantitative polymerase chain reaction | MPO myeloperoxidase | LC/MS/MS Liquid chromatography-tandem mass spectrometry | GM-CSF granulocyte-macrophage colony-stimulating factor | PMA phorbol myristate acetate | TDP 43 TAR DNA binding protein 43 | DMEM Dulbecco's modified Eagle Medium | hydrogen peroxide | DNA deoxyribonucleic acid | ROS reactive oxygen species | superoxide radical anion | SOD superoxide dismutase | HBSS Hank's buffered salt solution | half maximal efficient concentration | 2-hydroxyethidium | NADPH nicotinamide adenine dinucleotide phosphate | NC non-carrier | FBS fetal bovine serum | 2-OH-E | FUS/TLS Fused in Sarcoma/Translocated in Sarcoma | DMSO Dimethylsulfoxide | SURVIVAL | SUPEROXIDE | HYDROETHIDINE | SOD1(G93A) mice | BIOCHEMISTRY & MOLECULAR BIOLOGY | SPINAL-CORD | REACTIVE OXYGEN | MOTOR-NEURON DEGENERATION | NOX FAMILY | ENDOCRINOLOGY & METABOLISM | ALS MICE | INHIBITOR | Spinal Cord - drug effects | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Male | NADPH Oxidase 1 - genetics | Thioridazine - administration & dosage | Amyotrophic Lateral Sclerosis - drug therapy | Motor Neurons - pathology | Gene Expression Regulation - drug effects | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | NADPH Oxidase 2 - metabolism | Spinal Cord - pathology | Superoxide Dismutase-1 - genetics | Adult | Female | Aged | Mice | Perphenazine - administration & dosage | NADPH Oxidase 2 - genetics | Superoxide Dismutase-1 - antagonists & inhibitors | Disease Models, Animal | Oxidases | Nervous system diseases | Analysis | Oncology, Experimental | Superoxide | Research | Cancer | Index Medicus
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 03/2002, Volume 90, Issue 4, pp. e58 - e65
Angiotensin II infusion causes endothelial dysfunction by increasing NAD(P)H oxidase-mediated vascular superoxide production. However, it remains to be... 
cGMP-dependent protein kinase | NADPH OXIDASE | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MESSENGER-RNA EXPRESSION | DEPENDENT PROTEIN-KINASES | vasodilator-stimulated phosphoprotein | SYNTHASE | nox expression | PHOSPHOPROTEIN SERINE-239 PHOSPHORYLATION | angiotensin II | SUPEROXIDE GENERATION | HYPERTENSIVE RATS | PERIPHERAL VASCULAR DISEASE | nitric oxide synthase uncoupling | VASCULAR SUPEROXIDE | HEMATOLOGY | SMOOTH-MUSCLE CELLS | Angiotensin II - administration & dosage | Membrane Glycoproteins - metabolism | NADPH Dehydrogenase - metabolism | Rats, Wistar | NADPH Oxidases - metabolism | NADPH Dehydrogenase - genetics | Aorta - metabolism | Nitric Oxide Synthase - genetics | Phosphoproteins - metabolism | Cyclic GMP-Dependent Protein Kinases - metabolism | NADP - pharmacology | Protein Kinase C - metabolism | Cyclic GMP-Dependent Protein Kinase Type I | Superoxides - metabolism | NADPH Oxidases - genetics | Blood Pressure - drug effects | Vasodilation - physiology | NADH, NADPH Oxidoreductases - metabolism | Disease Models, Animal | NADH, NADPH Oxidoreductases - genetics | Vasodilator Agents - pharmacology | NAD - pharmacology | Nitric Oxide Synthase Type III | Infusions, Parenteral | Rats | Soluble Guanylyl Cyclase | NADPH Oxidase 4 | Phosphoproteins - genetics | Enzyme Activation - drug effects | Cell Adhesion Molecules - metabolism | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Animals | Cyclic GMP - metabolism | Guanylate Cyclase | Signal Transduction - drug effects | Microfilament Proteins | Membrane Transport Proteins | Signal Transduction - physiology | Nitric Oxide Synthase - metabolism | Vasodilation - drug effects | In Vitro Techniques | Nitric Oxide - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2016, Volume 36, Issue 5, pp. 846 - 854
Journal Article
Circulation Research, ISSN 0009-7330, 04/2012, Volume 110, Issue 9, pp. 1217 - 1225
RATIONALE:The function of Nox4, a source of vascular H2O2, is unknown. Other Nox proteins were identified as mediators of endothelial dysfunction. OBJECTIVE:We... 
Hypertension | Angiogenesis | Carbon monoxide | Nitric oxide | Redox regulation | INDUCED ACTIVATION | nitric oxide |