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1. Full Text Therapeutic potential of NADPH oxidase 1/4 inhibitors
by Teixeira, G and Szyndralewiez, C and Molango, S and Carnesecchi, S and Heitz, F and Wiesel, P and Wood, J M
British journal of pharmacology, ISSN 0007-1188, 06/2017, Volume 174, Issue 12, pp. 1647 - 1669
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Fibrosis - drug therapy | NADPH Oxidase 1 - antagonists & inhibitors | Anti-Inflammatory Agents - pharmacology | Pyridines - chemistry | Humans | Enzyme Inhibitors - pharmacology | Fibrosis - metabolism | Inflammation - metabolism | Pyrazoles - chemistry | Animals | Anti-Inflammatory Agents - chemistry | Inflammation - drug therapy | NADPH Oxidase 2 - metabolism | Enzyme Inhibitors - chemistry | NADPH Oxidase 1 - metabolism | Pyridines - pharmacology | Pyridones - chemistry | NADPH Oxidase 2 - antagonists & inhibitors | Pyridones - pharmacology | Pyrazoles - pharmacology | Oxidases | Physiological aspects | Cellular signal transduction | Health aspects | Analysis | Medical research | Oxidative stress | Reactive oxygen species | Disease | Oxidase | Organs | Clinical trials | Gene deletion | Modulators | NAD(P)H oxidase | Diseases | Signal transduction | Inhibitors | Pathways | CYBB protein | Isoforms | Fibrosis | Transduction | In vivo methods and tests | Inhibition | Localization | Index Medicus | Themed Section | Review
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2. Full Text Endothelial NADPH oxidase 4 protects ApoE-/- mice from atherosclerotic lesions
by Craige, Siobhan M and Kant, Shashi and Reif, Michaella and Chen, Kai and Pei, Yongmei and Angoff, Rebecca and Sugamura, Koichi and Fitzgibbons, Timothy and Keaney, John F
Free radical biology & medicine, ISSN 0891-5849, 12/2015, Volume 89, pp. 1 - 7
Reactive oxygen species | CXCL9 | NADPH Oxidase 4 | T regulatory cells | Atherosclerosis | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Endothelium, Vascular - cytology | T-Lymphocytes, Regulatory - metabolism | Cell Proliferation | Reactive Oxygen Species - metabolism | Oxidative Stress | Atherosclerosis - genetics | Humans | NADPH Oxidases - metabolism | Male | Aorta - metabolism | Interferon-gamma - metabolism | Immunoenzyme Techniques | NADPH Oxidases - genetics | Female | Real-Time Polymerase Chain Reaction | Cytokines - blood | Atherosclerosis - pathology | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Atherosclerosis - metabolism | Blotting, Western | Mice, Knockout | Animals | Endothelium, Vascular - metabolism | Apolipoproteins E - physiology | Mice | Aorta - cytology | Atherosclerosis - prevention & control | Oxidases | Genetic engineering | Biological response modifiers | T cells | Apolipoproteins | Cells | Cardiovascular agents | Interferon gamma | Endothelium | Proteins | Index Medicus | atherosclerosis | reactive oxygen species
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3. Full Text Wild-type and mutant p53 differentially regulate NADPH oxidase 4 in TGF-β-mediated migration of human lung and breast epithelial cells
by Boudreau, H.E and Casterline, B.W and Burke, D.J and Leto, T.L
British journal of cancer, ISSN 0007-0920, 05/2014, Volume 110, Issue 10, pp. 2569 - 2582
Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Medical sciences | Tumors | Reactive Oxygen Species - metabolism | Humans | Lung Neoplasms - metabolism | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Mutation, Missense | Lung - cytology | Breast Neoplasms - metabolism | Focal Adhesion Protein-Tyrosine Kinases - physiology | Tumor Suppressor Protein p53 - physiology | Epithelial Cells - physiology | RNA, Messenger - biosynthesis | Genes, p53 | Neoplasm Metastasis | Transfection | RNA Interference | Epithelial-Mesenchymal Transition | NADPH Oxidases - genetics | Female | NADPH Oxidases - biosynthesis | Neoplasm Proteins - genetics | Breast - cytology | Neoplasm Proteins - biosynthesis | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Transforming Growth Factor beta - physiology | Enzyme Induction | NADPH Oxidase 4 | RNA, Neoplasm - biosynthesis | Breast Neoplasms - pathology | Cell Line, Tumor | RNA, Neoplasm - genetics | Cell Line, Transformed | Cell Movement | Index Medicus | mutant p53 | Molecular Diagnostics | epithelial-to-mesenchymal transition (EMT) | NADPH oxidase | transforming growth factor-beta (TGF-β) | cell migration | reactive oxygen species (ROS)
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4. Full Text Regulation of ROS Signal Transduction by NADPH Oxidase 4 Localization
by Kai Chen and Michael T. Kirber and Hui Xiao and Yu Yang and John F. Keaney
The Journal of cell biology, ISSN 0021-9525, 6/2008, Volume 181, Issue 7, pp. 1129 - 1139
COS cells | Oxidases | Reactive oxygen species | Phosphorylation | Receptors | Smooth muscle myocytes | Antibodies | Oxidation | Physiological regulation | Endothelial cells | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Endoplasmic Reticulum - enzymology | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Cercopithecus aethiops | Protein Transport - drug effects | Antioxidants - pharmacology | NADPH Oxidase 4 | Mutation - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - metabolism | Receptor, Epidermal Growth Factor - metabolism | Animals | Substrate Specificity - drug effects | Signal Transduction - drug effects | Endothelial Cells - cytology | Endoplasmic Reticulum - drug effects | Oxidation-Reduction - drug effects | Cell Proliferation - drug effects | Mice | Epidermal Growth Factor - pharmacology | Phosphorylation - drug effects | COS Cells | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Evaluation | Vascular endothelium | Cell research | Cellular signal transduction | Properties | Endoplasmic reticulum | Antioxidants | Signal transduction | Enzymes | Oxygen | Biochemistry | Cells | Index Medicus | s
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5. Full Text Macrophages Generate Reactive Oxygen Species in Response to Minimally Oxidized Low-Density Lipoprotein: Toll-Like Receptor 4– and Spleen Tyrosine Kinase–Dependent Activation of NADPH Oxidase 2
by Bae, Yun Soo and Lee, Jee Hyun and Choi, Soo Ho and Kim, Sunah and Almazan, Felicidad and Witztum, Joseph L and Miller, Yury I
Circulation research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 210 - 218
Reactive oxygen species | Minimally oxidized LDL | RANTES | Atherosclerosis | NADPH oxidase 2 | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Cardiology. Vascular system | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Medical sciences | Vertebrates: cardiovascular system | Blood and lymphatic vessels | Phosphorylation | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | Protein-Tyrosine Kinases - metabolism | NADPH Oxidases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Gene Knockdown Techniques | NADPH Oxidases - deficiency | Atherosclerosis - enzymology | Time Factors | Toll-Like Receptor 4 - deficiency | Interleukin-1beta - metabolism | Protein Kinase C - metabolism | Inflammation Mediators - metabolism | NADPH Oxidases - genetics | Chemokine CCL5 - metabolism | Lipoproteins, LDL - metabolism | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Syk Kinase | Cell Line | Phospholipase C gamma - metabolism | Atherosclerosis - immunology | Signal Transduction | Macrophages, Peritoneal - enzymology | Myeloid Differentiation Factor 88 - genetics | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | NADPH Oxidase 2 | Toll-Like Receptor 4 - metabolism | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Mice | Enzyme Activation | Myeloid Differentiation Factor 88 - metabolism | Membrane Glycoproteins - deficiency | Cell Movement | Index Medicus
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6. Full Text NADPH oxidase 4 limits bone mass by promoting osteoclastogenesis
by Goettsch, Claudia and Babelova, Andrea and Trummer, Olivia and Erben, Reinhold G and Rauner, Martina and Rammelt, Stefan and Weissmann, Norbert and Weinberger, Valeska and Benkhoff, Sebastian and Kampschulte, Marian and Obermayer-Pietsch, Barbara and Hofbauer, Lorenz C and Brandes, Ralf P and Schröder, Katrin
The Journal of clinical investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4731 - 4738
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Reactive Oxygen Species - metabolism | Bone Density - physiology | Humans | Middle Aged | Osteoporosis, Postmenopausal - pathology | Bone Density - genetics | Cell Differentiation - genetics | NADPH Oxidases - deficiency | Bone Resorption - genetics | RANK Ligand - physiology | Monocytes - pathology | NADPH Oxidases - genetics | Female | Osteoclasts - physiology | Monocytes - physiology | Cell Differentiation - physiology | Disease Models, Animal | Bone Resorption - physiopathology | Osteoclasts - pathology | Osteoporosis, Postmenopausal - genetics | NADPH Oxidase 4 | Mice, Knockout | Animals | Bone Resorption - pathology | Osteoporosis, Postmenopausal - physiopathology | Mice | Polymorphism, Single Nucleotide | Macrophage Colony-Stimulating Factor - physiology | NADPH Oxidases - physiology | Oxidases | NADP (Coenzyme) | Physiological aspects | Development and progression | Genetic aspects | Bone diseases | Research | Risk factors | Enzymes | Cell culture | Normal distribution | Colleges & universities | Bones | Bone density | Index Medicus | Abridged Index Medicus
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7. Full Text Activation of TLR4 is required for the synergistic induction of dual oxidase 2 and dual oxidase A2 by IFN-γ and lipopolysaccharide in human pancreatic cancer cell lines
by Wu, Yongzhong and Lu, Jiamo and Antony, Smitha and Juhasz, Agnes and Liu, Han and Jiang, Guojian and Meitzler, Jennifer L and Hollingshead, Melinda and Haines, Diana C and Butcher, Donna and Roy, Krishnendu and Doroshow, James H
The Journal of immunology (1950), ISSN 0022-1767, 02/2013, Volume 190, Issue 4, pp. 1859 - 1872
Life Sciences & Biomedicine | Immunology | Science & Technology | Neoplasm Transplantation | Pancreatic Neoplasms - metabolism | Reactive Oxygen Species - metabolism | Humans | Signal Transduction - immunology | Pancreatitis - immunology | Female | NADPH Oxidases - biosynthesis | Tumor Cells, Cultured | Lipopolysaccharides - physiology | Pancreatic Neoplasms - enzymology | Interferon-gamma - physiology | Random Allocation | Toll-Like Receptor 4 - metabolism | Pancreatitis - enzymology | Toll-Like Receptor 4 - physiology | Membrane Proteins - biosynthesis | Animals | Mice, Nude | Pancreatic Neoplasms - immunology | Cell Line, Tumor | Mice | Pancreatitis - metabolism | Dual Oxidases | Chronic Disease | Index Medicus | Abridged Index Medicus
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8. Full Text Activation of NADPH Oxidase 4 in the Endoplasmic Reticulum Promotes Cardiomyocyte Autophagy and Survival During Energy Stress Through the Protein Kinase RNA-Activated-Like Endoplasmic Reticulum Kinase/Eukaryotic Initiation Factor 2α/Activating Transcription Factor 4 Pathway
by Sciarretta, Sebastiano and Zhai, Peiyong and Shao, Dan and Zablocki, Daniela and Nagarajan, Narayani and Terada, Lance S and Volpe, Massimo and Sadoshima, Junichi
Circulation research, ISSN 0009-7330, 11/2013, Volume 113, Issue 11, pp. 1253 - 1264
fasting | myocardial ischemia | autophagy | NOX4 protein | endoplasmic reticulum | reactive oxygen species | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Stress, Physiological - physiology | Reactive Oxygen Species - metabolism | Myocytes, Cardiac - cytology | Oxidative Stress - physiology | Endoplasmic Reticulum - physiology | Autophagy - physiology | NADPH Oxidase 4 | Eukaryotic Initiation Factor-2 - physiology | Mice, Knockout | NADPH Oxidases - deficiency | Animals | eIF-2 Kinase - physiology | Myocytes, Cardiac - physiology | Up-Regulation - physiology | NADPH Oxidases - genetics | Models, Animal | Signal Transduction - physiology | Mice | Energy Metabolism - physiology | In Vitro Techniques | NADPH Oxidases - physiology | Activating Transcription Factor 4 - physiology | Cell Survival - physiology | Index Medicus
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