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Trends in endocrinology and metabolism, ISSN 1043-2760, 2014, Volume 25, Issue 9, pp. 452 - 463
Highlights • Endothelial cells express four NADPH oxidase isoforms: NOX1, 2, 4, and 5. • NOX1, 2, and 5 make superoxide and cause endothelial dysfunction and vascular disease... 
Endocrinology & Metabolism | hyperlipidemia | NADPH oxidases | vascular disease | selective NOX1/2 inhibitors | inflammation | reactive oxygen species | hypertension | diabetes | endothelial dysfunction | Hypertension | Vascular disease | Selective NOX1/2 inhibitors | Reactive oxygen species | Hyperlipidemia | Endothelial dysfunction | Inflammation | Diabetes | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | SPONTANEOUSLY HYPERTENSIVE-RATS | NITRIC-OXIDE SYNTHASE | LOW-DENSITY-LIPOPROTEIN | ANGIOTENSIN-II | REDOX SIGNALING PLATFORMS | INDUCED UP-REGULATION | ENDOCRINOLOGY & METABOLISM | HYDROGEN-PEROXIDE PRODUCTION | CORONARY-ARTERY-DISEASE | NADPH Oxidases - chemistry | Membrane Glycoproteins - metabolism | Humans | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | Endothelium, Vascular - enzymology | Molecular Targeted Therapy | Membrane Glycoproteins - antagonists & inhibitors | NADPH Oxidases - genetics | Membrane Proteins - metabolism | Vascular Diseases - drug therapy | Membrane Proteins - genetics | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Mice, Transgenic | NADPH Oxidase 5 | NADPH Oxidase 4 | Vascular Diseases - enzymology | Enzyme Inhibitors - therapeutic use | NADPH Oxidase 2 | NADPH Oxidase 1 | Cardiovascular Agents - pharmacology | Membrane Glycoproteins - genetics | Mice, Knockout | Cardiovascular Agents - therapeutic use | Animals | Membrane Proteins - antagonists & inhibitors | Models, Biological | Oxidases | Blood circulation disorders | Superoxide | Peroxides | Endothelium
Journal Article
Nature communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2247 - 15
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 08/2016, Volume 97, pp. 95 - 108
.... The aim of this study was to evaluate the involvement of NADPH oxidases (NOX) in the oxidative damage and progression of ALS neuropathology... 
SOD1G93A mice | Thioridazine | NADPH oxidase | NOX | Amyotrophic lateral sclerosis | Perphenazine | Microglia | Phenothiazine | DUOX dual oxidase | List of abbreviations ALS amyotrophic lateral sclerosis | WST-1 2-(4-Iodophenyl)-3-(4-nitrophenyl)-5-(2,4-disulfophenyl)-2H-tetrazolium, monosodium salt | PBS phosphate buffered saline | HOCl hypochlorous acid | BBB blood brain barrier | WT wild typ | 2-chloroethidium | 2-Cl-E | NOXO1 NADPH oxidase organiser type 1 | CNS central nervous system | DPI diphenyleneiodonium | ethidium | NOX NADPH oxidase | HE hydroethidine | qPCR quantitative polymerase chain reaction | MPO myeloperoxidase | LC/MS/MS Liquid chromatography-tandem mass spectrometry | GM-CSF granulocyte-macrophage colony-stimulating factor | PMA phorbol myristate acetate | TDP 43 TAR DNA binding protein 43 | DMEM Dulbecco's modified Eagle Medium | hydrogen peroxide | DNA deoxyribonucleic acid | ROS reactive oxygen species | superoxide radical anion | SOD superoxide dismutase | HBSS Hank's buffered salt solution | half maximal efficient concentration | 2-hydroxyethidium | NADPH nicotinamide adenine dinucleotide phosphate | NC non-carrier | FBS fetal bovine serum | 2-OH-E | FUS/TLS Fused in Sarcoma/Translocated in Sarcoma | DMSO Dimethylsulfoxide | SURVIVAL | SUPEROXIDE | HYDROETHIDINE | SOD1(G93A) mice | BIOCHEMISTRY & MOLECULAR BIOLOGY | SPINAL-CORD | REACTIVE OXYGEN | MOTOR-NEURON DEGENERATION | NOX FAMILY | ENDOCRINOLOGY & METABOLISM | ALS MICE | INHIBITOR | Spinal Cord - drug effects | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Male | NADPH Oxidase 1 - genetics | Thioridazine - administration & dosage | Amyotrophic Lateral Sclerosis - drug therapy | Motor Neurons - pathology | Gene Expression Regulation - drug effects | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | NADPH Oxidase 2 - metabolism | Spinal Cord - pathology | Superoxide Dismutase-1 - genetics | Adult | Female | Aged | Mice | Perphenazine - administration & dosage | NADPH Oxidase 2 - genetics | Superoxide Dismutase-1 - antagonists & inhibitors | Disease Models, Animal | Oxidases | Nervous system diseases | Analysis | Oncology, Experimental | Superoxide | Research | Cancer
Journal Article
American journal of physiology. Lung cellular and molecular physiology, ISSN 1040-0605, 02/2012, Volume 302, Issue 4, pp. L410 - L419
Goodson P, Kumar A, Jain L, Kundu K, Murthy N, Koval M, Helms MN. Nadph oxidase regulates alveolar epithelial sodium channel activity and lung fluid balance in vivo via O-2(-) signaling... 
Intratracheal instillation | Small G protein | Amiloride | Rac1 | Chest X-ray | LPS | amiloride | CELLS | PHYSIOLOGY | intratracheal instillation | small G protein | TRANSCRIPTION | ENAC | OXYGEN | INHIBITION | RESPIRATORY SYSTEM | MOUSE MODEL | SENSITIVE NA+ TRANSPORT | LIQUID CLEARANCE | LIPOPOLYSACCHARIDE | chest X-ray | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Lipopolysaccharides - administration & dosage | NADPH Oxidases - metabolism | rac GTP-Binding Proteins - metabolism | Lung - diagnostic imaging | Epithelial Sodium Channels - metabolism | Pulmonary Alveoli - enzymology | Lung - enzymology | Extracellular Fluid - diagnostic imaging | rac GTP-Binding Proteins - antagonists & inhibitors | Membrane Glycoproteins - physiology | Pulmonary Alveoli - metabolism | Sodium Channels - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Lung - metabolism | Aminoquinolines - pharmacology | Lipopolysaccharides - pharmacokinetics | Signal Transduction | Mice, Inbred C57BL | Neuropeptides - metabolism | Sodium Chloride - administration & dosage | Pyrimidines - pharmacology | Gene Knockout Techniques | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Mice, Knockout | Radiography | Extracellular Fluid - metabolism | Patch-Clamp Techniques | Animals | Sodium Chloride - pharmacokinetics | Mice | rac1 GTP-Binding Protein | NADPH Oxidases - physiology | Neuropeptides - antagonists & inhibitors
Journal Article
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2008, Volume 45, Issue 9, pp. 1223 - 1231
The importance of NADPH oxidase (Nox) in hypoxic responses in hypoxia-sensing cells, including pulmonary artery smooth muscle cells (PASMCs), remains uncertain... 
Reactive oxygen species | Mitochondria | Protein kinase C | Free radicals | Intracellular calcium | NADPH oxidase | Hypoxia | Pulmonary artery
Journal Article
PLoS pathogens, ISSN 1553-7374, 2011, Volume 7, Issue 2, p. e1001271
...), Nox2-containing NADPH oxidase, is a novel pharmacological target against the lung inflammation caused by influenza A viruses. Male WT (C57BL/6) and Nox2(-/y... 
IMMUNE-RESPONSE | CELLS | OXYGEN RADICALS | MICROBIOLOGY | PNEUMONIA | PATHOGENESIS | VIROLOGY | NITRIC-OXIDE | INFECTION | MICE | EXPRESSION | NADPH OXIDASES | PARASITOLOGY | Oxidoreductases - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Orthomyxoviridae Infections - complications | NADPH Oxidases - metabolism | Pneumonia - pathology | Pneumonia - prevention & control | Male | Oxidoreductases - physiology | Lung - enzymology | Orthomyxoviridae Infections - genetics | Membrane Glycoproteins - physiology | Membrane Glycoproteins - antagonists & inhibitors | Influenza A virus - pathogenicity | NADPH Oxidases - genetics | Bronchoalveolar Lavage Fluid - cytology | Lung - metabolism | Pneumonia - genetics | Influenza A virus - physiology | Lung - pathology | Pneumonia - etiology | Bronchoalveolar Lavage Fluid - virology | Macrophages - pathology | Oxidoreductases - metabolism | Orthomyxoviridae Infections - metabolism | Oxidoreductases - genetics | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Orthomyxoviridae Infections - pathology | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Enzyme Activation - drug effects | Enzyme Inhibitors - therapeutic use | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Macrophages - drug effects | Mice | Enzyme Activation - physiology | NADPH Oxidases - physiology | Lung - immunology | Oxidases | Influenza viruses | Lung diseases | Physiological aspects | Research | Health aspects | Risk factors | Proteins | Pandemics | Lungs | Cytokines | Rodents
Journal Article
The Plant cell, ISSN 1532-298X, 2008, Volume 20, Issue 5, pp. 1390 - 1406
...) and NADPH oxidase participate in the radical burst. Here, we show that mitogen-activated protein kinase (MAPK... 
Oxidases | Leaves | Pathogens | Inoculation | Reactive oxygen species | Cell death | Oxides | Gene expression regulation | Plants | Plant cells | TOBACCO-MOSAIC-VIRUS | ARABIDOPSIS-THALIANA | ACTIVATED PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | POTATO-TUBER TISSUES | PLANT SCIENCES | CELL BIOLOGY | HYPERSENSITIVE CELL-DEATH | REACTIVE OXYGEN | SALICYLIC-ACID | PLANT DEFENSE RESPONSE | PHYTOPHTHORA-INFESTANS | DISEASE RESISTANCE | MAP Kinase Signaling System - physiology | Reactive Oxygen Species - metabolism | NADPH Oxidases - metabolism | Molecular Sequence Data | Immunoblotting | Nitric Oxide Synthase - genetics | Proteins | MAP Kinase Signaling System - genetics | NADPH Oxidases - genetics | Plant Proteins - metabolism | MAP Kinase Kinase 2 - genetics | Mutagenesis, Site-Directed | Plant Proteins - physiology | Tobacco - metabolism | Phytophthora - physiology | MAP Kinase Kinase 2 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Colletotrichum - physiology | Host-Pathogen Interactions | Plant Proteins - genetics | Algal Proteins - pharmacology | MAP Kinase Signaling System - drug effects | Tobacco - genetics | Mitogen-Activated Protein Kinases - genetics | Nitric Oxide Synthase - metabolism | Tobacco - microbiology | Nitric Oxide - metabolism | Mitogen-Activated Protein Kinases - metabolism | Protein kinases | Nitric oxide | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, p. e0145750
...Author(s): Leroy C. Joseph [sup.1], Emanuele Barca [sup.2], Prakash Subramanyam [sup.3], Michael Komrowski [sup.1], Utpal Pajvani [sup.1], Henry M. Colecraft... 
NADPH OXIDASE | MYOCARDIAL-INFARCTION | (ROS)-INDUCED ROS RELEASE | ACIDS | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | SUDDEN CARDIAC DEATH | RISK | ELECTRON-TRANSPORT | ATRIAL-FIBRILLATION | Electron Transport | Antioxidants - chemistry | Calcium - metabolism | Homeostasis | Male | Membrane Glycoproteins - antagonists & inhibitors | Reactive Oxygen Species - chemistry | Gene Deletion | NADPH Oxidases - genetics | Palmitates - adverse effects | Sarcoplasmic Reticulum - metabolism | Heart Ventricles - pathology | Cell Line | Myocytes, Cardiac - cytology | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Oxygen Consumption | Palmitates - chemistry | Mitochondria - metabolism | Mitochondria - pathology | NADPH Oxidase 2 | Antimycin A - chemistry | Membrane Glycoproteins - genetics | Mice, Knockout | Muscle Cells - cytology | Animals | Protein Kinase C - chemistry | Mice | Oxidative Stress - drug effects | Apoptosis | Prevention | Oxidases | Oxidative stress | Mitochondria | Abnormalities | Heart cells | Physiological aspects | Saturated fatty acids | Health aspects | Heart | Cardiac arrhythmia | Protein kinase C | Calcium | Arrhythmia | Physicians | Leak channels | Clinical trials | Myocytes | Palmitic acid | Epidemiology | NAD(P)H oxidase | Depolarization | Antioxidants | Clonal deletion | Sarcoplasmic reticulum | Rodents | CYBB protein | Physiology | Inhibition | Heart diseases | Calcium homeostasis | Heart failure | Obesity | Oxygen | Cardiac muscle | Diabetes mellitus | Cardiomyocytes | Biophysics | Metabolism | Fatty acids | Calcium (mitochondrial) | Coronary artery disease | Medicine | Studies | Molecular modelling | Surgeons | Ventricle | Diabetes | Electron transport | Laboratory animals | Calcium (reticular)
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 10/2010, Volume 285, Issue 43, pp. 33197 - 33208
The X+-linked chronic granulomatous disease (X+-CGD) variants are natural mutants characterized by defective NADPH oxidase activity but with normal Nox2 expression... 
FERREDOXIN-NADP(+) REDUCTASE | NOX-FAMILY | CHRONIC GRANULOMATOUS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | FLAVOCYTOCHROME B | FUNCTIONAL-ANALYSIS | GP91(PHOX) SUBUNIT |