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Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 210 - 218
Oxidative modification of low-density lipoprotein (LDL) plays a causative role in the development of atherosclerosis. In this study, we demonstrate that... 
Reactive oxygen species | Minimally oxidized LDL | RANTES | Atherosclerosis | NADPH oxidase 2 | atherosclerosis | AORTIC ENDOTHELIAL-CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | SYK | SUPEROXIDE-PRODUCTION | minimally oxidized LDL | CUTTING EDGE | NOX FAMILY | reactive oxygen species | LIPOPOLYSACCHARIDE | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | NF-KAPPA-B | Phosphorylation | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | Protein-Tyrosine Kinases - metabolism | NADPH Oxidases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Gene Knockdown Techniques | NADPH Oxidases - deficiency | Atherosclerosis - enzymology | Time Factors | Toll-Like Receptor 4 - deficiency | Interleukin-1beta - metabolism | Protein Kinase C - metabolism | Inflammation Mediators - metabolism | NADPH Oxidases - genetics | Chemokine CCL5 - metabolism | Lipoproteins, LDL - metabolism | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Syk Kinase | Cell Line | Phospholipase C gamma - metabolism | Atherosclerosis - immunology | Signal Transduction | Macrophages, Peritoneal - enzymology | Myeloid Differentiation Factor 88 - genetics | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | NADPH Oxidase 2 | Toll-Like Receptor 4 - metabolism | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Mice | Enzyme Activation | Myeloid Differentiation Factor 88 - metabolism | Membrane Glycoproteins - deficiency | Cell Movement | Index Medicus
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2018, Volume 123, pp. 62 - 71
The NADPH oxidase (NOX) enzyme family is a major source of reactive oxygen species (ROS) and contributor to the secondary pathology underlying traumatic brain... 
Traumatic brain injury | Neurogenesis | NADPH oxidase | NOX inhibitor | NOX2 | MOUSE-BRAIN | MIGRATION | ADULT NEUROGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | MECHANISMS | SUBVENTRICULAR ZONE | HIPPOCAMPUS | NEURAL STEM-CELLS | ENDOCRINOLOGY & METABOLISM | DIFFERENTIATION | EXPRESSION | Oxidases | Brain | Neurosciences | Brain research | Neurons | Stem cells | Physiological aspects | Injuries
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2016, Volume 291, Issue 13, pp. 7029 - 7044
Using high throughput screening-compatible assays for superoxide and hydrogen peroxide, we identified potential inhibitors of the NADPH oxidase (Nox2) isoform... 
high throughput screening (HTS) | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NADPH oxidase inhibitors | reactive oxygen species (ROS) | CANCER | NOX INHIBITORS | reactive nitrogen species (RNS) | REACTIVE OXYGEN | EXTRACELLULAR-SUPEROXIDE | NADPH oxidase | IN-VIVO | DISEASE | BIOLOGY | superoxide ion | NITRIC-OXIDE | fluorescence probes | THERAPEUTIC TARGETS | Small Molecule Libraries - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Membrane Glycoproteins - metabolism | Phenanthridines - chemistry | Humans | NADPH Oxidases - metabolism | Coumarins - chemistry | Chromatography, High Pressure Liquid | Fluorometry | Quaternary Ammonium Compounds - chemistry | Hydrogen Peroxide - chemistry | Membrane Glycoproteins - antagonists & inhibitors | Enzyme Inhibitors - chemistry | Superoxides - metabolism | Peroxynitrous Acid - antagonists & inhibitors | Superoxides - antagonists & inhibitors | Gene Expression | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Superoxides - chemistry | NADPH Oxidase 2 | Hydrogen Peroxide - metabolism | Peroxynitrous Acid - biosynthesis | Small Molecule Libraries - chemistry | Molecular Probes - chemistry | High-Throughput Screening Assays | HL-60 Cells | Oxazines - chemistry | Peroxynitrous Acid - chemistry | Macrophage Activation - drug effects | Hydrogen Peroxide - antagonists & inhibitors | Index Medicus | Medicinal Chemistry | Life Sciences | Biomolecules | Biochemistry, Molecular Biology | Chemical Sciences | Molecular Biophysics
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 05/2017, Volume 135, Issue 22, pp. 2163 - 2177
Journal Article
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2247 - 2247
Journal Article
Journal Article
Journal of Hypertension, ISSN 0263-6352, 02/2014, Volume 32, Issue 2, pp. 300 - 305
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 08/2015, Volume 23, Issue 5, pp. 358 - 374
Journal Article
Current opinion in hematology, ISSN 1065-6251, 9/2017, Volume 24, Issue 5, pp. 460 - 466
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2015, Volume 290, Issue 9, pp. 5449 - 5461
Sepsis-mediated endothelial Angiopoeitin-2 (Ang2) signaling may contribute to microvascular remodeling in the developing lung. The mechanisms by which... 
BRONCHOPULMONARY DYSPLASIA | ANGIOPOIETINS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH-FACTORS | NETWORK FORMATION | ACUTE LUNG INJURY | SMOOTH-MUSCLE-CELLS | SEPSIS | EXPRESSION | PATHOPHYSIOLOGY | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Membrane Glycoproteins - metabolism | Angiopoietin-2 - genetics | Humans | NADPH Oxidases - metabolism | Immunoblotting | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Angiopoietin-2 - metabolism | Transcription Factor AP-1 - metabolism | I-kappa B Kinase - metabolism | RNA Interference | NADPH Oxidases - genetics | Receptor, TIE-2 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Endothelial Cells - physiology | Microvessels - cytology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Receptor, TIE-2 - genetics | Animals | Signal Transduction - drug effects | Active Transport, Cell Nucleus - drug effects | Lipopolysaccharides - pharmacology | Lung - blood supply | Microscopy, Fluorescence | Endothelial Cells - drug effects | Index Medicus | Angiogenesis | Lung Injury | Endothelial Cell | LPS | Angiopoeitin-2 | Cell Biology | NADPH Oxidase
Journal Article