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Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 167 - 178
Excessive levels of reactive oxygen species (ROS) and increased expression of NADPH oxidases (Nox) have been proposed to contribute to pulmonary artery... 
Epigenetics | Histone deacetylases | NADPH oxidase | Pulmonary artery hypertension | OXYGEN SPECIES PRODUCTION | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HDAC INHIBITION | CELL-PROLIFERATION | 5 NOX5 | NAD(P)H OXIDASE | THERAPEUTIC TARGET | DISEASE | ENDOCRINOLOGY & METABOLISM | EXPRESSION | RIGHT-VENTRICULAR HYPERTROPHY | Transcription, Genetic - drug effects | Antihypertensive Agents - pharmacology | Endothelial Cells | Luciferases - metabolism | Reactive Oxygen Species - metabolism | Epigenesis, Genetic | Humans | Monocrotaline | Male | RNA Polymerase II - metabolism | Hypertension, Pulmonary - chemically induced | Luciferases - genetics | p300-CBP Transcription Factors - genetics | Isoenzymes - metabolism | HEK293 Cells | Fibroblasts | Hypertension, Pulmonary - drug therapy | Genes, Reporter | Cell Line | Promoter Regions, Genetic | Histone Deacetylases - genetics | Signal Transduction | Isoenzymes - genetics | Rats | Histone Deacetylases - metabolism | Hypertension, Pulmonary - genetics | Rats, Sprague-Dawley | p300-CBP Transcription Factors - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | NADPH Oxidase 2 - metabolism | Histone Deacetylase Inhibitors - pharmacology | RNA Polymerase II - genetics | Mice | NADPH Oxidase 2 - antagonists & inhibitors | NADPH Oxidase 2 - genetics | Hypertension, Pulmonary - pathology | Isoenzymes - antagonists & inhibitors | Oxidases | Epigenetic inheritance | Chromatin | RNA | Analysis | Genes | Histones | Superoxide | DNA binding proteins | Genetic transcription | Pulmonary hypertension
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2247 - 15
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 01/2017, Volume 102, pp. 57 - 66
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2016, Volume 97, pp. 95 - 108
Amyotrophic lateral sclerosis (ALS) is an incurable neurodegenerative disease characterized by progressive loss of motor neurons, gliosis, neuroinflammation... 
SOD1G93A mice | Thioridazine | NADPH oxidase | NOX | Amyotrophic lateral sclerosis | Perphenazine | Microglia | Phenothiazine | DUOX dual oxidase | List of abbreviations ALS amyotrophic lateral sclerosis | WST-1 2-(4-Iodophenyl)-3-(4-nitrophenyl)-5-(2,4-disulfophenyl)-2H-tetrazolium, monosodium salt | PBS phosphate buffered saline | HOCl hypochlorous acid | BBB blood brain barrier | WT wild typ | 2-chloroethidium | 2-Cl-E | NOXO1 NADPH oxidase organiser type 1 | CNS central nervous system | DPI diphenyleneiodonium | ethidium | NOX NADPH oxidase | HE hydroethidine | qPCR quantitative polymerase chain reaction | MPO myeloperoxidase | LC/MS/MS Liquid chromatography-tandem mass spectrometry | GM-CSF granulocyte-macrophage colony-stimulating factor | PMA phorbol myristate acetate | TDP 43 TAR DNA binding protein 43 | DMEM Dulbecco's modified Eagle Medium | hydrogen peroxide | DNA deoxyribonucleic acid | ROS reactive oxygen species | superoxide radical anion | SOD superoxide dismutase | HBSS Hank's buffered salt solution | half maximal efficient concentration | 2-hydroxyethidium | NADPH nicotinamide adenine dinucleotide phosphate | NC non-carrier | FBS fetal bovine serum | 2-OH-E | FUS/TLS Fused in Sarcoma/Translocated in Sarcoma | DMSO Dimethylsulfoxide | SURVIVAL | SUPEROXIDE | HYDROETHIDINE | SOD1(G93A) mice | BIOCHEMISTRY & MOLECULAR BIOLOGY | SPINAL-CORD | REACTIVE OXYGEN | MOTOR-NEURON DEGENERATION | NOX FAMILY | ENDOCRINOLOGY & METABOLISM | ALS MICE | INHIBITOR | Spinal Cord - drug effects | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Male | NADPH Oxidase 1 - genetics | Thioridazine - administration & dosage | Amyotrophic Lateral Sclerosis - drug therapy | Motor Neurons - pathology | Gene Expression Regulation - drug effects | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | NADPH Oxidase 2 - metabolism | Spinal Cord - pathology | Superoxide Dismutase-1 - genetics | Adult | Female | Aged | Mice | Perphenazine - administration & dosage | NADPH Oxidase 2 - genetics | Superoxide Dismutase-1 - antagonists & inhibitors | Disease Models, Animal | Oxidases | Nervous system diseases | Analysis | Oncology, Experimental | Superoxide | Research | Cancer
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 234 - 243
Revascularization of ischemic tissue is a highly desirable outcome in multiple diseases, including cardiovascular diseases and ischemic retinopathies.... 
Angiogenesis | Oxidative stress | Inflammation | Ischemia | Nrf2 | NADPH oxidase | DIABETIC-RETINOPATHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANGIOTENSIN-II | RETINAL ISCHEMIA-REPERFUSION | NAD(P)H OXIDASE | PROTECTIVE ROLE | ENDOCRINOLOGY & METABOLISM | ANIMAL-MODEL | VESSEL LOSS | NEOVASCULARIZATION | PREMATURITY | Retina - drug effects | Retinitis - chemically induced | Retina - metabolism | Oxidative Stress | Receptors, Notch - metabolism | Receptors, Notch - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Ischemia - genetics | Oxygen - adverse effects | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Neovascularization, Pathologic - pathology | Retinitis - genetics | NF-E2-Related Factor 2 - genetics | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Ischemia - pathology | Animals, Newborn | Retinitis - pathology | Signal Transduction | Membrane Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ischemia - metabolism | Neovascularization, Pathologic - chemically induced | NF-E2-Related Factor 2 - deficiency | Mice, Knockout | Animals | NADPH Oxidase 2 - metabolism | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | NADPH Oxidase 2 - genetics | Retina - pathology | Retinitis - metabolism | Oxidases | Medical colleges | Ophthalmology | Vascular endothelial growth factor | Analysis | Endothelial growth factors
Journal Article
Journal Article